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14–17 September 2014, Barcelona, Spain Electronic poster abstracts
P02.14: Table 1.
CasesWeeks atdiagnosis Ultrasound findings
Cyst(number) Karyotype
Fetalsex Type of delivery
Weight atbirth
(grams)Apgar 1/5(minutes) pH
1 12 + 4 None 2 46XY Male Normal(Suctionpad)
2830 9/10
2 12 None 1 46XY Male Pending delivery3 12 None 1 Female Normal 2640 9/10 7,334 16 4 None None
2 Male Normal3440 9/10 7,35
2 Male Normal 3440 9/10 7,35
5 33 + 6 None 1 46XY Female Normal 3760 9/10 7,456 16 None 1 46XY 46XY
MalePendingdelivery
Male PendingDelivery 37609/10 7,45 6Pendingdelivery
7 32 None 1 Female Normal 3760 9/108 32 Triplet 1 3 Males Cesarean section 1571,
1400,1100
9/10; 9/10;6/9
9 12 Omphalocele Multiple Pending delivery10 20 Choroid plexus cyst 1 Pending delivery11 12 ISUA + bilateral
pyelic ectasia +oligohydramnios
1 1 46XY TOP 46XY TOP
12 12 Hidropsfetalis + partialmolar pregnancy
Multiple TOP
13 11 + 6 Omphalocele + clawhands + choroidplexus cyst +EIUGR
1 18 TRISOMY Stillbirth
14 12 Mega-cysticbladder +holoprosencephaly+ extremitymalposition
Multiple 18 TRISOMY TOP
15 12 Body wall complex/body stalk anomaly
1 45X0 TOP
16 14 Oligohydramnios +partial molarpregnancy
1 TRIPLOIDY TOP
TOP: termination of pregnancy.ISUA: Isolated single umbilical artery.EIUGR: Early intrauterine growth restriction.
P02.16Cause of fetal demise in first trimester parvovirus fetalinfection: anemia or myocarditis?
G.E. Chalouhi1, S. Benedetti1, C. Alby1,2, N. Benzina1,Y. Ville1
1Department of Obstetrics and Fetal Medicine,Necker-Enfants-Malades Hospital, APHP, Paris V University,Paris, France; 2Department ofGenetics-Pathology-Embryology and Cytogenetics,Necker-Enfants-Malades Hospital, APHP, Paris V University,Paris, France
Increased nuchal translucency and/or fetal hydrops during firsttrimester ultrasound examination have been reported as signs ofcongenital infection with parvovirus infection.
We report the case of a 35-year-old woman, gravida 3 para2 with no prior relevant history who underwent routine US
examination at 13 weeks’ by both dates and CRL (69 mm) anda nuchal translucency (NT) of 3.6 mm (>99th centile). The fetuswas hydropic with generalized subcutaneous edema, pleural, andpericardial effusion as well as ascites. Hemodynamics assessmentshowed tricuspid regurgitation, and reverse flow in the umbilicalartery. The MCA-PSV was 24.06 cm/s.
Although the reference table of MCA-PSV doesn’t cover thisearly gestational age, fetal anemia was suspected. An intra-uterinespontaneous fetal demise was diagnosed 2 days later. Complemen-tary investigations were performed showing a normal karyotype,positive maternal IgG and IgM for Parvovirus B19, the virologyanalysis on amniocytes culture showed positive Parvovirus B19DNA.
Maternal Parvoviral seroconversion rate varies between 3-34%and the risk of vertical transmission is approximately 30%. Fetalhydrops develops in 0 to 12.5% of infected fetus with a peak atbetween between 17 and 24 weeks, while fetal demise is estimatedto occur in 5-10%, with or without the diagnosis of fetal hydropsfollowing an unclear physiopathology.
Ultrasound in Obstetrics & Gynecology 2014; 44 (Suppl. 1): 181–369. 193