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1 Tasers and In- custody Deaths: The EMS Perspective Michael D. Curtis, MD EMS Medical Director Saint Michael’s Hospital – Stevens Point Saint Clare’s Hospital – Weston Ministry Health Care

Tasers and In-custody Deaths: The EMS Perspective

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Tasers and In-custody Deaths: The EMS Perspective. Michael D. Curtis, MD EMS Medical Director Saint Michael’s Hospital – Stevens Point Saint Clare’s Hospital – Weston Ministry Health Care. Objectives. Tasers Excited Delirium Physical Restraints Medical Management. - PowerPoint PPT Presentation

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Page 1: Tasers and In-custody Deaths: The EMS Perspective

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Tasers and In-custody Deaths: The EMS Perspective

Michael D. Curtis, MDEMS Medical DirectorSaint Michael’s Hospital – Stevens PointSaint Clare’s Hospital – WestonMinistry Health Care

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Objectives

Tasers Excited Delirium Physical Restraints Medical Management

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Approximately half of the 620 law

enforcement agencies in Wisconsin use

Tasers.

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Advantages of the Taser

Less risk of injury to law enforcement officers when subjects actively resist

Less risk of injury or death to subjects from law enforcement use of force

Photo Source: Taser International Instructor Certification Course V12, November 2004

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Thomas A. Swift’s Electric Rifle (TASER)

M26 Taser. Manufactured by Taser International

Source: http://www.keme.co.uk/~mack/M26.jpgSource: http://www.pointshooting.com/m26black.jpg

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Probes

Blast Doors

AFIDs Laser Sight

LIL: Low Intensity Lights (LEDs)

Trigger

Enhanced GripZones

DPM: Digital Power Magazine

DPM ReleaseButton

Stainless SteelShock Plates

SafetyIlluminationSelector

Stainless SteelSerial No. Plate

High Visibility Sights

Air Cartridge

TASER Wire

X26 Taser

Source: Taser International Instructor Certification Course V12, November 2004

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M26 Taser

Source: Taser International Instructor Certification Course V12, November 2004

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Tasers, in and of themselves,

are not lethal weapons.

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Tasers Use Electricity

Taser: 50,000 Volts

Static Electricity door knob 35,000 – 100,000

Volts Van De Graaff

Generator: 1 – 20 Million Volts

Photo Source: Taser International Instructor Certification Course V12, November 2004

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Tasers Use Electricity

It’s not the voltage it’s the amperage that is dangerous

Tasers use high voltage, but very low amperage M26: 3.6 milliamps (average current) M26:1.76 joules per pulse X26: 2.1 milliamps (average current) X26: 0.36 joules per pulse

X26 Taser delivers 19 pulses per second

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Tasers Use Electricity

Cardiac Defibrillators use 150 – 400 joules per pulse

The safety index for the fibrillation threshold ranges from 15 – 42 depending on the weight of the subject Source: PACE 2005; 28:S284-S287. Pig study Variable current/constant pulse frquency

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Aim like a standard firearm at center of mass Use sights and/or laser Rule of Thumb: 1 foot (.3m) spread for every 7

feet (2.1m) of travel

(m) .6 1.5m 2.1m 3m 4.5m 6.4m 7.6m

Target Distance (ft) 2′ 5' 7' 10' 15′ 21′ 25′ Spread (in) 4″ 9" 13" 18" 26" 36″ 38″

(cm) 10cm 23cm 33cm 46cm 66cm 91cm 109cm

Probe Trajectory

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Taser Effects

High voltage affects nerves Leads to intense muscle contraction Does not affect muscles directly

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Tasers have caused injuries, but most

Taser-related injuries are minor.

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Taser Injuries

Muscle Contraction Injuries Stress fractures Muscle or tendon strain or tears Back injuries Joint injuries

Injuries from Falls May be serious depending on the height

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Taser Injuries

Minor Surface Burns Due to arcing

Tasers will ignite flammable liquids and gasses Potential for serious burns

Penetrating Eye Injuries

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Taser Darts

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Taser Dart Injuries

The skin at the puncture site is cauterized A swift tug will remove the barb easily

Taser users receive this training Wipe site with alcohol prep Consider a band-aid

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21 Source: Taser International X26 User Course V12, November 2004

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News media sources have implied a cause and effect relationship between Tasers and in-custody deaths…

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Concern About Tasers 147 in-custody taser-related deaths

since 1999 Source: Robert Anglen, Arizona Republic

August 8, 2005 The number is growing Draws significant negative media attention Outcry from human rights activists

Amnesty International http://web.amnesty.org/library/index/ENGAMR511392004

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24 Source: Seattle Post-Intelligencer

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There is no scientific evidence to date of a

cause and effect relationship between

Tasers and in-custody deaths.

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Taser Use in Police Training

Over 150,000 police volunteers No deaths

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In-Custody Deaths…

Why do some people die following a violent confrontation with police?

What role does the taser play, if any? What can police officers do to prevent in-

custody deaths?

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Typical Scenario

Male subject creating a disturbance Triggers 911 call Obvious to police that subject will resist Struggle ensues with multiple officers

May involve OC, Taser, choke holds, batons, etc.

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Typical Scenario

Physical restraints applied Subject subdued in a prone position Officers kneeling on subjects back Handcuffs, ankle cuffs Hogtying, hobble restraint or TARP

Prone vs. lateral positioning Transported in a squad car to jail

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Typical Scenario

Continued struggle against restraints Sometimes damages squad car

Apparent resolution period Subject becomes calm or slips into

unconsciousness Labored or shallow breathing Followed unexpectedly by…

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Typical Scenario

Death Resuscitation efforts are futile Los Angeles County EMS Study

18 ED deaths witnessed by paramedics (all were restrained)

In 13 – rhythm documentedVT and asystole were most common

No ventricular fibrillationAll failed resuscitation

Source: Am J Emerg Med; 2001:19(3), 187-191

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Typical Scenario

The press: Subject “died after being shocked with

taser” Implies cause and effect

The Fallacy: “Post hoc ergo proptor hoc”

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Typical Aftermath

Several weeks later – autopsy results… Cause of Death

Excited delirium Illicit stimulant drug abuse Concurrent medical problems Minimal injury from police confrontation

It wasn’t the taser after all Officers exonerated

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Typical Aftermath

Meanwhile the officers… Placed on administrative leave Subjected to investigation Face threat of potential criminal charges Face threat of potential civil litigation Subjected to public outcry Experience personal and family stress Contemplate a career change

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Several forensic pathology studies have cited excited

delirium, not Tasers, as the cause of death.

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What is Excited Delirium?

A controversial theory An imminently life threatening medical

emergency… Not a crime in progress!

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What is Excited Delirium?

Diagnostic criteria Characteristic behavioral components Metabolic Acidosis Hyperthermia Identifiable cause

Stimulant drugs Psychiatric disease

It does not explain all behavior that leads to confrontation with police

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Pathophysiology

Central nervous system effects: Changes in dopamine transporter and

receptors Accounts for behavioral changes Accounts for hyperthermia

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Behavioral Components: Delirium Delirium:

“Off the track” Confusion Clouding of consciousness Shifting attention Disorientation Hallucinations Onset rapid – acute Duration brief – transient

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Behavioral Components:Psychosis

Psychosis: Bizarre behavior and thoughts Hallucinations, paranoia

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Behavioral Components:Excited (Agitated) Extreme agitation, increased activity

Aggravated by efforts to subdue and restrain

Not likely to comply after one or two tasers

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Behavioral Components:Excited (Agitated)

Violent or aggressive behavior Towards inanimate objects, especially

smashing glass Towards self, others or police

Noncompliant with requests to desist Superhuman strength Insensitive to pain

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Excited Delirium

Hyperthermia High body temperature 105 – 113 oF Drug’s effect on temperature control center

in brain (hypothalamus) Tell-tale signs:

Profuse sweating Undressing – partial or complete

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Excited Delirium

Hyperthermia Aggravated by

increased activity the ensuing struggle warm humid weather (summertime) dehydration certain therapeutic medications

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Excited Delirium

Metabolic Acidosis Potentially life threatening

Elevated blood potassium level Factors: dehydration, increased activity

Survivors: Kidney damage due to muscle breakdown May require dialysis

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Excited Delirium: The Usual Suspects

#1 Cause: Stimulant Drug Abuse Acute intoxication Superimposed on chronic abuse Acute intoxication triggers the event

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Excited Delirium: The Usual Suspects Underlying psychiatric disease

First described in 1849 before cocaine was first extracted from cocoa leaf

Mania (Bipolar Disorder) Psychosis (Schizophrenia)

Noncompliance with medications to control psychosis or bipolar disorder Unusual – #2 Cause

Rare: New onset schizophrenia

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Stimulant Drugs

Cocaine The major offender On the rise due to “crack epidemic”

Toxicology studies show… Low to moderate levels of cocaine High levels of benzoylecognine (the major

breakdown product of cocaine) Suggests recent use superimposed on

chronic abuse

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Stimulant Drugs

Other known culprits include: Methamphetamine Phencyclidine (PCP) LSD

Cocaethylene = Cocaine + Alcohol Toxic to the heart Unknown role in excited delirium deaths

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Concurrent Health Conditions

Obesity Heart Disease

Coronary artery disease Cardiomegaly Hypertrophic cardiomyopathy Myocarditis Fibrotic heart

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Autopsy Proof

Specialized laboratories can identify changes in brain chemistry that are characteristic of excited delirium

Blood and brain tissue levels of benzoylecognine and cocaine Typical ratio 5:1

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Tasers and Excited Delirium Deaths It’s not the Taser Many in-custody deaths long before

tasers were ever used Documented in 1980s medical literature

Deaths of persons not in custody Found naked in bathrooms Wet towels Empty ice cube trays scattered about A futile effort to cool themselves

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Tasers and Excited Delirium Deaths It is unknown whether tasers have

different adverse effects on people with excited delirium than on healthy volunteers

Tasers No proximate temporal relationship

between taser use and death Multiple or continuous taser shocks

Taser International’s recent warning against repeated shocks

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Whether repeated or continuous Taser

shocks is safe remains unknown.

They should probably be avoided, if

possible.

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Restraints and In-Custody Deaths

What roles do physical restraint, restraining technique and restraint position play in excited delirium deaths?

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Physical Restraints

Source: Prehosp Emerg Care, 2003:7(1); 48-55.

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Physical Restraint Issues

Positional Asphyxia Deaths have occurred with subjects

restrained in a prone position Theory: restricts breathing The role of the position is unclear Little data to support causality Other factors are the likely culprits

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Physical Restraint Issues

No clinically significant changes in pulmonary function tests in healthy volunteers Am J Forensic Med Pathol. 1998

Sep;19(3):201-5.

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Physical Restraint Issues

Restraint Asphyxia Increased deaths in restrained patients Rat Study

3 fold increase in cocaine-related deaths among “restrained” rats

Life Sci. 1994;55(19):PL379-82. Whether these may be contributory

remains controversial, but still possible Not considered causal

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Physical Restraint Issues

Compression asphyxia What are the adverse effects on breathing

and circulation when one or more officers kneel on the subjects back as they handcuff him?

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Excited delirium is an imminently life-

threatening medical emergency.

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The “Freight Train to Death”

How police restrain or position the subject will not stop “the freight train to death”

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The behavioral features of excited

delirium include criminal acts, but…

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Excited delirium is not a crime in progress,

and responders must recognize the

difference, before it’s too late.

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Recognizing Excited Delirium

How they act How they look What they say and how they say it What they are doing How they make you feel How they respond to you How they respond to force How they respond to the taser

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Recognizing Excited Delirium

Agitation or Excitement = Increased activity and intensity Aggressive, threatening or combative –

gets worse when challenged or injured Amazing feats of strength Pressured loud incoherent speech Sweating (or loss of sweating late) Dilated pupils/less reactive to light Rapid breathing

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Recognizing Excited Delirium

Delirium = Confusion Disoriented

Person, place, time, purpose Rapid onset over a short period of recent time

“He just started acting strange” Easily distracted/lack of focus Decreased awareness and perception Rapid changes in emotions (laughter, anger,

sadness)

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Recognizing Excited Delirium

Psychotic = bizarre behavior Thought content inappropriate for

circumstances Hallucinations (visual or auditory) Delusions (grandeur, paranoia or

reference) Flight of ideas/tangential thinking Makes you feel uncomfortable

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Mnemonic: NOT A CRIME Naked – and sweating from hyperthermia Objects – violence against, especially glass Tough – unstoppable, insensitive to pain

Acute onset – “He just snapped!”

Confused – person, place, purpose, perception Resistant – will not follow commands to desist Incoherent speech – shouting, bizarre content Mental Health or Makes you uncomfortable Early EMS Back-up

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Bad Behavior: Other Reasons

Alcohol intoxication or withdrawal Other drug use problems

Example: Cocaine psychosis Pure psychiatric disease Head injury Dementia (Alzheimer’s Disease) Hypoglycemia Hyperthyroidism

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Patients with excited delirium need rapid aggressive medical

intervention.

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Alternative Strategy

Attempt verbal de-escalation Summon back-up quickly Summon EMS as early as possible Use taser before a struggle ensues Jump the subject and administer tranquillizer Back off and contain the subject without

restraint Once calm transport (no restraints?) Minimize struggle and restraints Unrealistically simplified?? – Maybe!

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The first goal of therapy is to gain

control of the violent behavior.

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The “Ideal” Drug

Rapid effective tranquilization No repeat dosing

No significant adverse effects respiratory depression cardiovascular depression neurological adverse effects

Easy to administer (IM) Allows easy assessment of neurological

status on ED arrival

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In Search of The “Ideal” Drug

Benzodiazepines Neuroleptics Atypical antipsychotics Ketamine

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Benzodiazepines

Effective But usually require repeat doses Adverse reactions:

Hypotension Respiratory Depression Over sedation

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Neuroleptics and Atypical Antipsychotics

Rapid onset (10 – 15 minutes or less) Can be very effective in a single dose Prolong the QT Interval (Droperidol) Target dopamine D2 receptors

May exacerbate hyperthermia

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Ketamine

Very rapid onset of action (<5 minutes) Highly effective in a single dose Favorable safety profile in healthy

patients Potential adverse effects:

Adrenergic over stimulation in excited delirium

“Emergence reactions” in adults

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The second goal of therapy is to stabilize

the underlying pathophysiologic

processes.

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Other ALS Interventions

Dehydration/Metabolic Acidosis: IV NS X 2 W/O

Hyperthermia: Cool environment, disrobe, tepid mist and

fanning, cooling blankets Hyperkalemia?:

Fluids, Calcium Chloride, Sodium Bicarbonate, Albuterol

Rapid transport

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Fall Back Position

Proceed to customary practices at any point when This strategy appears to fail Safety appears to be endangered It is necessary to escalate the level of force

based on the threat level Don’t transport in a squad car Use the least amount of force needed

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Caveats

Never place an agitated and combative patient in an ambulance without physical restraints

Never transport a restrained patient without an officer present who can unlock the restraints

Should the transporting officer disable his/her weapons?

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Potential Pitfalls

Can’t wait for back-up or EMS ALS not available Struggle and restraints cannot be

avoided

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Summary Excited Delirium is an imminently life

threatening medical emergency, not a crime in progress

In-custody deaths likely related to excited delirium

Tasers – if used early – may help (remains unproven)

ALS medics can give potent tranquilizers Rapid aggressive medical stabilization

needed

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Summary Beware of potential side effects of

therapeutic drugs Treat for hyperthermia, dehydration,

metabolic acidosis and potential hyperkalemia

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The End

Questions? Thank You!

Michael D. Curtis, MDEMS Medical DirectorSaint Michael’s HospitalSaint Clare’s [email protected]