1) Basic ECG

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    THE

    ELECTROCARDIOGRAM(ECG / EKG)

    By: Dr Yasir Mansour i

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    How To Interpret ECG?

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    Highlights

    Always interpret the ECG in clinical

    context

    Always read the ECG systematically

    Put the data together & try to find pathology

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    Objectives

    To recognize the normal ECG of the heart

    To recognize the most common ECG

    Abnormalities

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    The QRS Complex

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    ECG ANALYSIS

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    Normal ECG with normal QRS transition from V1V6

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    ECG ANALYSIS

    RATE

    RHYTHM

    AXIS

    P WAVE

    P-R INTERVAL

    QRS COMPLEX

    ST SEGMENT

    T WAVE

    U WAVE

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    ECG Paper

    Speed Amplitude and

    Deflection

    Calibration

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    Standardisation

    10mv

    Paper Speed

    1 small square

    = 1mv (mm)

    1 big square

    = 5 small squares

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    ECG ANALYSIS

    RATE

    Dividing

    300 by the number of big squares,

    OR

    1500 by the number of small squares

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    THE RATE

    NORMAL (60100 beats / min)

    FAST (>100 beats / min) = TACHYCARDIA

    SLOW (< 60 beats / min) = BRADYCARDIA

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    Pacemakers of the Heart

    SA Node - Dominant pacemaker with an

    intrinsic rate of 60 - 100 beats/minute.

    AV Node - Back-up pacemaker with anintrinsic rate of 40 - 60 beats/minute.

    Ventricular cells - Back-up pacemaker with

    an intrinsic rate of 20 - 45 beats/minute.

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    HR > 150/minNote Paper Speed12.5mm/sec

    Same ECG: HR 75/minat Paper Speedof 25mm/sec

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    THE RHYTHM

    REGULAR or IRREGULAR

    There may be a combination of :

    Abnormal Rate + Abnormal Rhythm e.g.

    Atrial Fibrillation (fast and irregular)

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    REGULAR RHYTHM

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    IRREGULAR RHYTHM

    InspirationExpiration

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    NORMAL SINUS RHYTHM

    Regular P-P interval

    Regular R-R interval ( Rate between 60100/m )

    Each P wave is followed by a QRS complex

    P T

    R R R

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    ECG ANALYSIS

    RATE

    RHYTHM

    AXIS

    P WAVE

    P-R INTERVAL

    QRS COMPLEX

    ST SEGMENT

    T WAVE

    U WAVE

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    Determination of Electrical Axis of the Heart

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    NORMAL AXIS

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    LEFT AXIS DEVIATION

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    RIGHT AXIS DEVIATIONRIGHT

    AXIS DEVIATION

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    THE AXIS

    LADEXTREME

    RAD

    RADNORMAL

    I

    aVF

    I

    aVF

    I

    aVF

    I

    aVF

    0

    -90

    +90

    180

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    Normal AxisPositive R in I

    Positive R in II

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    Right Axis DeviationDeep S in I (Small R )

    Tall R in II

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    Left Axis DeviationTall R in I

    Deep S in AVF

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    ECG ANALYSIS

    RATE

    RHYTHM

    AXIS

    P WAVE

    P-R INTERVAL

    QRS COMPLEX

    ST SEGMENT

    T WAVE

    U WAVE

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    P Wave

    - Duration

    - Amplitude

    - Shape

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    P Wave

    Duration: 0.080.12sec(2-3 small squares)Amplitude: 2 - 2.5mm(22.5small squares)

    Shape:Roundedand upright, in Leads I, II, aVF, V4-V6.

    Inverted in aVR.

    Flat, inverted or biphasicin III, V1, V2

    P

    P

    P

    Normal

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    ECG ANALYSIS

    RATE

    RHYTHM

    AXIS

    P WAVE

    P-R INTERVAL

    QRS COMPLEX

    ST SEGMENT

    T WAVE

    U WAVE

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    PR Interval

    P

    R

    T

    PR Interval

    Normal - 0.120.20sec

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    PR Interval

    Normal Short Prolonged

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    ECG ANALYSIS

    RATE

    RHYTHM

    AXIS

    P WAVE

    P-R INTERVAL

    QRS COMPLEX

    ST SEGMENT

    T WAVE

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    QRS Complex

    1. Duration ( Normal 0.060.11sec)

    2. Components ( Q, R, S )

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    QRS Complex - Duration

    Narrow - Normal (0.060.11sec)

    Wide - Abnormal (> 0.11sec)

    Causes of Wide QRS Complex

    1. PVC

    2. Intraventricular Conduction Delay (IncompleteBBB)3. BBB

    4. Paced Beat

    5. W-P-W Syndrome

    6. Aberrant Conduction

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    QRS Complex - Duration

    Causes of Wide QRS Complex

    Premature Ventricular Contraction (PVC)

    PVC

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    Intraventricular Conduction Delay -

    (Incomplete) Bundle Branch BlockQRS = 0.11sec

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    Left Bundle Branch

    Block QRS > 0.12 QRS 4x0.04

    = 0.16

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    (Complete) Right Bundle

    Branch Block QRS > 0.12 QRS 4x0.04

    = 0.16

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    RBBB - Wide QRS complex Deep wide S in I, II, V5, V6

    Prominent R / RsR in V1, V2

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    QRS Complex - Duration

    Causes of Wide QRS Complex

    Paced Beat

    Pacemaker Impulse

    Paced Ventricular Beat

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    Type A W-P-W: Short PR Interval Wide QRS complex (Delta Wave)

    Prominent R in V1, V2

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    QRS Complex - Components

    Q Wave

    R Wave

    S Wave

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    Q WAVE

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    QRS Complex - Components

    Normal Q Wave

    Narrow (1mm duration)

    Amplitude less than of the accompanying R wave

    Q wave

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    QRS Complex - Components

    Abnormal Q Wave

    Wide (> 1mm duration) and / or

    Amplitude > than of the accompanying R wave

    Q

    R

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    R WAVE

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    R Wave

    Normal R Wave

    R wave

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    R Wave -Amplitude

    Abnormal R Wave:

    Low - Causes: i. Emphysemaii. Pleural Effusion

    iii. Pericardial Effusion

    iv. Dilated Cardiomyopathy

    Tall - LVH- RVH

    - Biventricular Hypertrophy

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    LVH

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    RVH - Prominent R in V1 and V2

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    Biventricular Hypertrophy ( LVH + RVH )

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    Differential Diagnosis of Prominent

    R or rsR in V1

    RVH

    RBBB

    True Posterior Myocardial Infarct

    Type A Wolffe Parkinson White (WPW)

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    RVH

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    RBBB - Wide QRS complex Deep wide S in I, II, V5, V6

    Prominent R / RsR in V1, V2

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    True Posterior MI - Prominent R / Rsr in V! and V2

    Rsr

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    Type A W-P-W Short PR Interval Wide QRS complex (Delta Wave)

    Prominent R in V1, V2

    ECG ANALYSIS

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    ECG ANALYSIS

    RATE RHYTHM

    AXIS

    P WAVE

    P-R INTERVAL

    QRS COMPLEX

    ST SEGMENT

    T WAVE

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    S-T Segment Changes

    ST segment Elevation

    ST Segment Depression

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    May be due to -

    i. Myocardial Infarction- Convexupwards

    - Related to the area of infarct

    - Abnormal Q wave may be present- Reciprocal ST depression may be present

    ii. Early Repolarisation

    (Elevated J Junction)

    iii. Acute Pericarditis- Concave upwards

    - Generalised ( in all the leads )

    - No abnormal Q wave

    Q

    R

    (i)

    (ii)

    J junction

    (iii)

    S-T Segment Elevation

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    S-T Segment Depression

    May be due to:

    1. Myocardial Ischaemia

    2. LVH with strain3. Unstable Angina

    4. Non-ST segment Elevation Myocardial Infarction

    5. Digoxin Effect / Toxicity6. Hypokalaemia

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    QT Interval

    Measured from the beginning of Q to the end of T

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    Measured from the beginning of Q to the end of T

    QT Interval

    QT Interval

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    QT Interval

    Measured from the beginning of Q to the end of T

    QTinterval < 50% preceding R-R interval

    QTc( corrected QT interval)

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    ECG ANALYSIS

    RATE RHYTHM

    AXIS

    P WAVE

    P-R INTERVAL

    QRS COMPLEX

    ST SEGMENT

    T WAVE

    T W

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    T Wave

    Normal T Wave

    Abnormal T Wave - Tall T wave

    - Inverted T wave

    Tall T waveInverted T wave

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    Normal ECG with normal QRS transition from V1V6

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