93
Anterior Cruciate Ligament Injury Ed Mulligan, PT, DPT, OCS, SCS, ATC Assistant Professor D t t f Ph i l Th Department of Physical Therapy Dallas, TX

Anterior Cruciate Ligament Injury - Continuing ED Knee ACL Lecture handout... · Step Down Test 6 point rating scale 1. ... Adequate balance (arm strategy – subject keeps ... •

Embed Size (px)

Citation preview

Anterior Cruciate Ligament Injury

Ed Mulligan, PT, DPT, OCS, SCS, ATC

Assistant ProfessorD t t f Ph i l ThDepartment of Physical TherapyDallas, TX

Lets start with the …

ACL Epidemiology

Incidence Gender Preference MOI MOI

ACL Injury Incidence

250K injuries/year in US ‐ 100K surgeries/year in US

70% of injuries are non‐contact in nature

Incidence in athletic population over 10 years = 20%

Incidence at West Point Study over 4 years– Female = 3.5%; Males = 3.2%– When comparing common sportsp g p

Incidence ratio for females = 1.5

Multiple other studies suggest females                                        2 6 x higher incidence2‐6 x higher incidence

ACL Mechanism of Injury

Non‐contact (70%) Approaching 90% in females Approaching 90% in females

rotational, deceleration injury poor landing mechanics

Contact (30%)• Lateral contact  valgus force

• ACL, MCL, meniscus• Terrible Triad (usually lateral meniscus 

acutely and medial meniscus chronically)• Anterior Contact  hyperextension 

• ACL, PCL

High Risk Sports

Football Skiing Soccer Basketball VolleyballThe incidence of non‐contact ACL injuries is greater in sports that require multidirectional activities, such as rapid deceleration, pivoting, cutting, and landing from jumps

Non-Contact MOI

Pivot‐Pop‐Puffy

Non-Contact MOI

VAROOM– VAlgus– ROtationOut of Control– Out of Control

– Muscle Dysynchrony

P O P P.O.P– Planted– Out of ControlOut of Control– “Pop”

Position of No Return

Forward body flexion Hip adduction/IR 20‐30° of knee flexion and valgus

Tibi l ER Tibial ER Forefoot pronation Landing: Landing:

– On balls of feet with the body weight forward and out of control

ModifiableModifiableNEUROMUSCULARNEUROMUSCULAR

NonNon--ModifiableModifiableSTRUCTURAL

NEUROMUSCULARNEUROMUSCULARTiming and Control Adaptations TechniqueTiming and Control Adaptations Technique

Risk InfluenceRisk Influence

STRUCTURALNarrow NotchSmall ACL

↑ External Tibial Torsion

NonNon--ModifiableModifiableHORMONALcollagen strength ↑ External Tibial Torsion

Genu ValgumGenu Recurvatum

collagen strengthjoint laxity

ModifiableModifiableBIOMECHANICALBIOMECHANICAL

Extrinsic FactorsExtrinsic Factors –– Shoes/Playing SurfaceShoes/Playing SurfaceExtrinsic Factors Extrinsic Factors  Shoes/Playing SurfaceShoes/Playing Surface↑ genu valgus and hip flexion angles; ↓ knee flexion s ffness during athle c maneuvers↑ genu valgus and hip flexion angles; ↓ knee flexion s ffness during athle c maneuvers

↑ STJ prona on/navicular drop↑ STJ prona on/navicular dropLigament Dominance Ligament Dominance –– Quadricep Dominance Quadricep Dominance –– Dominant Extremity Imbalance Dominant Extremity Imbalance –– CORE CORE 

weaknessweaknessweaknessweakness

Modifiable Neuromuscular Differences

Diminished Muscular Forces (Ligament Dominance)– altered Q/H ratios– poor core stability and control– Poor control of femoral IR/Add– Absence of muscle control in the frontal plane or 

with high ground reaction forces Dependence on Quads for Stability (Quadricep 

Dominant)– Activate quads in position of risk

• Prefer knee extensors for stabilization over kneePrefer knee extensors for stabilization over knee flexors/hip extensors)

– More upright posture Lower Extremity Ambidexterity

– Unilateral weakness, uncoordination

Modifiable Neuromuscular Differences

Diminished proximal neuromuscular control  (Trunk Dominance)– Momentum of trunk is not controlled sufficiently leading to– Momentum of trunk is not controlled sufficiently leading to 

uncontrolled motion of the COG during deceleration and movement of the GRF vectors to the lateral side of the joint

Poor trunk neuromuscular control is predictive of knee                     injuryZazulak B, et al, Am J Sports Med, 2007

Core proprioceptive neuromuscular training improved                      body sway controlPaterno M, et al, J Orthop Sports Phys Ther, 2004 

Evaluating for N l D fi it Neuromuscular Deficits

Single Leg Squat or Step Down

Frontal Plane Pelvic Control– Trendelenburg or trunk lateral flexion

Uncontrolled LE internal rotation Increased genu valgus Hip over patella over 2nd metatarsal Poor control of descent Subtalar compensatory pronation

Step Down Test6 point rating scale

1. Adequate trunk control (lumbar flexion or side bendingbending

2. Adequate hip control (pelvic frontal or sagittal plane change)

3. Adequate knee control (tibial tuberosity medial to 2nd metatarsal (+1); medial to medial border of foot (+2)

4. Adequate balance (arm strategy – subject keeps hands on waist and does not use arms to balance

5 Adequate STJ control (no “wobble” during5. Adequate STJ control (no wobble during descent)

Good – Fair – Poor Experiment

If at higher level of function can evaluate the jump landing position at:

Toe touch Deepest position of landing Early concentric “take off”

Contrast of Landing Strategies

Stiff Landing Soft Landing• ed Knee Ext Moments• ed Quad Activation• ed Hip Ext Moments d Gl M EMG

• ed Knee Ext Moments• ed Quad Activation• ed Hip Ext Moments d Gl M EMG• ed Glut Max EMG

Increased valgus loading

• ed Glut Max EMG

Decreased valgus loading

KNEE STRATEGY“female oriented”

HIP STRATEGY“male oriented”

Consequence of Decreased Hip Strength

Hamstringi

Adductor Magnusi

TFL DominanceDominance

Weakness of G. Max

Dominance

Weakness of G. Max

Dominance

Weakness of G. Medweakness

Overuse hamstrings as hip extensors

Overuse Add. Magnus as hip extensors

Overuse TFL as hip abductor

compensation

Increased hip adduction

Increased hip adduction

Increased hipinternal rotation

result

MoralMoral

Cover the “butt” in rehab

Valgus Position - Varus Position

Operative vs. Non-Op Considerations

• Activity level and demands on knee “ dif k t lif t l• “modify knee to lifestyle or modify lifestyle to knee”

• Degree of instability and associated injuries• Degree of instability and associated injuries• 1% increased risk for meniscal tear for each 

month post‐injury up to 2 years

• Age, status of growth plates

• Patient motivations/compliance

Non-Op or Prehab Initial Points of Emphasis

Restore Joint Mobility PASSIVE t h i t t t i PASSIVE techniques to restore extension 

– ottoman stretch– prone hangs

h i / lf h– hamstring/calf stretches Heel Slides; Wall Slides, and Stationary Cycling to restore flexion ROM

Restore Muscle PerformanceRestore Muscle Performance Quad and Ham Isometrics (augmented with E. Stim prn) 4 plane Straight Leg Raises

P t t t d i t i t l t d Progress to protected arc isotonics as tolerated

General Rehab Progression

Initiate Weight BearingPWB progressing to FWB as patient demonstrates a• PWB progressing to FWB as patient demonstrates a normal gait pattern

Mimic Function• Mini‐Squats• Heel Raises• Lateral Step UpsLateral Step Ups

Build muscular and cardiovascular endurance                                      with ACL safe weight‐bearing activities

Initial Goals

Full ROM Controllable or absent effusion Good quad control 

– 60‐80% of uninvolved No extensor lag; normal gait

“quiet joint”

Decision Making

Traditionally, we know that 20‐40% of appropriate patients40% of appropriate patients are successful with a non‐

operative approach

With the use of specific selection criteria, this success may , yincrease to as high as 80%

Non-Operative Candidate Evaluation

4 criteria that have been shown to correlate with a successful non operative (potential coper) approach are:successful non‐operative (potential coper) approach are:

1. > 80% on 20’ unilateral timed hop test2 > 80% score on KOS ADL Scale2. > 80% score on KOS ADL Scale3. > 60% on Global Rating Scale 4. No more than one “giving way” episode                                   g g y p

since the original traumatic event

Fitzgerald GK, Axe MJ, Snyder‐Mackler L.JOSPT 30(4):194 203 2000

JOSPT 30(4):194‐203, 2000

ACL deficient copers, adapters, and non-copers have distinct differences in their pattern of gait recovery by 40 days post injury

Button K, et al, Brit J Sports Med, 2006

Critical Element of Rehabilitation

Perturbation Training

• Stork Standd• BAPS Board

• Bi/Multiaxial Boards• Roller Board Translations• Roller Board Translations• Tilt Board• Roller Board Stabilization

Critical Element of Rehabilitation

Perturbation Training

Re‐establishes neuromuscular control and d i t bilit th h d li f fdynamic stability through delivery of forces to the lower extremity in various directions when the patient is on an unstable or punpredictable surface

R i i iReaction training to                                          unexpected  forces

Delaware Perturbation Training Protocol

Roller Board Rocker Board Roller Board-Platform

detailed handout in your Article Resources folder

Perturbation Variables

• Double to Single LimbWeight Shifts and Transitional Movements• Weight Shifts and Transitional Movements

• Head Position• Vision – Eyes Open Closedy p• Vestibular – Cervical Motion• Surface Stability

E i t Ch i

Shuttle Balance

• Equipment Choices• Extremity Motion

• UE/LE• Motion Direction, Speed, and Resistance Teton Tremor

D t b ti t i i g Does perturbation training really make a difference?

Inclusion of perturbation training  resulted in fivefold increase in a successful non‐operative outcome – Fitzgerald, Phys Ther, 2000successful non operative outcome Fitzgerald, Phys Ther, 2000

Reduces inappropriate co‐contraction compensations and normalized knee kinematics – Chmielewski, Phys Ther, 2005

When comparing neuromuscular retraining program to a strength training program  – Improved self‐report and functional outcome scores with similar strength 

and functional testing results – Risberg, Phys Ther, 2007, Am J Sports Med, 2010I l i f t b ti t i i i t h b lt d i Inclusion of perturbation training in post‐op rehab resulted in a more symmetrical gait pattern and higher levels of strength – Hartigan, J Orthop Res, 2009

CONCLUSION A bl h i l f h bili iCONCLUSION: Arguably – the most important element of rehabilitation

Extensive, Activity-Specific Agility/Running Program

– Bounds/Hops/Jumps– Walk – Jog – Run Progression– Figure 8s– Backpedaling– Sidestepping– Cariocas– Open and Crossover Cutsp– Starts and Stops

C it i B d P g i f Criteria-Based Progression for Return to Sport Phase Example Myer GD, J Orthop Sports Phys Ther, 2006

Structured, measurable objective criteria to guide terminal phasescriteria to guide terminal phases and return to athletic activities

Suggestions on Return to Sport JOSPT Theme Issue 42:4 – April 2012

Soccer – Skiing – Football – Basketball 

Non-Operative Prognosis

Systematic Review– Good short to mid‐term outcomes in terms of knee function (87/100 Lysholm) andfunction (87/100 Lysholm) and performance (symmetrical hop tests), however, activity levels decreased on average by 21%

Muaidi QI, et al, Sports Med, 2007

Rehab + Early Reconstruction vs. Rehab Alone

RCT of 121 young, active adults62 surgical subjects

• 39 i t i t KOOS

59 non‐operative subjects              (23 underwent delayed reconstruction)

39 i t i t KOOS

No significant between groups difference on SF‐36 or Tegner Activity Scale

• 39 point improvement on KOOS • 39 point improvement on KOOS

Tegner Activity Scale

Frobell RB, et al, N Engl J Med, 2010

P t O ti M tPost‐Operative ManagementACL Reconstruction

Surgical Variables

Graft– Tensile Strength

Surgeon variables– Preferencesg

– Single/Double Bundle– Auto vs. Allograft– Donor Source

– Protocols– Timing of Surgery

Patient goals/expectations Surgical considerations

– Surgical Approach– Graft Placement/Isometricity

Co‐morbidities present– Other ligament involvement– Meniscal health

– Intra‐operative graft tension– Method of fixation– Ligament augmentation– PRP

– Osteoarthritis– Osteochondral bruising

Rare to have an “isolated” injuryPRP– Physeal Status

j y

ACL Injury Pathoanatomy

“Isolated ACL tear” is misnomer– ACL only < 10%– Meniscus injuries ‐ 60‐75%“B b i ” MRI 80%– “Bone bruises” on MRI ‐ 80%

– Collateral ligament tears  MCL & LCL: 5‐24%

– Articular cartilage injuries up to 46%

ACL R i ACL Reconstruction Time Frames for Healing

Process of ligamentization takes 12‐18 months

Between post‐op weeks 1‐6, avascular necrosis and revascularization occur simultaneously

l f l Complete graft revascularization                                                                 takes 8‐20 weeks

One year post op until the graft One year post‐op until the graft                                                                   resembles the vascular, histologic                                                               al, and structural properties of                                                                     the native ACLthe native ACL

ACL R i ACL Reconstruction Criteria Based Progression

individualized decisions based on the patient’s function symptoms stability strength and motionfunction, symptoms, stability, strength, and motion …

rather than solely on therather than solely on the                                                         passage of time

Rehabilitation Pearls

Perfect surgery can be nullified by inappropriate rehabilitation                        ‐ perfect rehabilitation can not overcome inadequate surgery

All patients need to be challenged athletically

No specific protocol other than to PREVENT COMPLICATIONS

Improve overall conditioning without placing                                            unnecessary stress on the healing graft

Slow down for allografts, hamstring grafts, concurrent                                g , g g ,meniscal repairs or articular cartilage procedures

Functional, activity specific rehab to retrain                                            neuromuscular systems for anticipated demandsy p

ACL Rehab Points of Emphasis

Avoid flexion contractures and/or                                   h fib iarthrofibrosis

the three most common complications of knee ligament surgery are:of knee ligament surgery are:

1. flexion contractures2. quadriceps weakness3. patellofemoral irritabilitythese complications are strongly interrelated

RA Sachs, DM Daniel, ML Stone, RF GarfeinAm. J. Sports Med., Nov 1989; 17: 760 - 765

How soon should you get full extension?

Early, aggressive passive extension training without any restriction does not affect post‐op knee laxity – Isberg, et al. Knee Surg Sports Traumatol Arthrosc. 2006

How much extension do you want to get?

symmetrical extension to                         i l d iduninvolved side up to                                             5‐

10° hyperextensioni d t i th• increased strain on the                                                  ACL in the hyperextended position

Markolf KL, et al. J Bone Joint Surg Am. 1996

• hamstrings are ineffective in this range in               providing posterior restraint of the tibia

78(11):1720-7

p g p

ACL Rehab Points of Emphasis

• Prevent Osteoarthritis • a successful outcome is not how fast you get back to activity but what your knee looks like in 5‐15 years

• small deficits in terminal extension• small deficits in terminal extension                                                 range increased presence of OA at                                                 10 years particularly if concurrent                                                   meniscal or articular cartilage damagemeniscal or articular cartilage damage

Shelbourne KD, et al, Am J Sports Med, 2008

ACL Rehab Points of Emphasis

Monitor for Graft Stretch Outh ll h f l k f• theoretically, the graft progressively weakens from its original strength at implantation to its weakest point between 6‐12 weeks after surgery, and then begins to strengthen again

• this process continues for 12‐18 months

Theoretical Graft Strength Curve of a Patellar Tendon Autograft JOSPT, June 1992

16014 mm graft

120140

100ngth

14 mm graft

100

6080

40% s

tren 10 mm graft

02040%

12 24 36 52Weeks

820

Impact of Osteochondral Bone Bruise

80% of traumatic ACL injuries sustain osteochondral lesionsosteochondral lesions– more common with non‐contact MOI ‐

Viskontis, et al, AJSM, 2008– most common on lateral tibial plateau and 

LFC (bruising on medial side associated with more severe injuries)Bisson, et al, AJSM, 2013

h k l These patients take a longer time to reach normal homeostasis (pain, swelling, ROM, NM control) and may have to delay reconstruction

MRI image of knee with a small geographic bone bruise in the weight-bearing lateral femoral condyle and an extensive bruise of the lateral tibial plateau in association with have to delay reconstruction

Johnson, et al. AJSM 28(2):152‐155, 2000

an ACL rupture

OOther Factors Associated with Poor Outcomes

In addition to severe osteochondrosis,

– Obesity (BMI > 30) had 0.35 times the odds of success

– Smoking had 0.36 times the odds of success

Kowalchuk DA, et al, Arthroscopy, 2009

ACL Rehab Points of Emphasis

restoration of function through training of  the neuromuscular recognitionneuromuscular recognition

of the “position of stability”

unilateral stance with kneeunilateral stance with knee flexed 20‐30°

t ti t l d i l idmust ensure patient loads surgical side as research indicates patients avoid symmetrical weight bearing for up to 6 months despite their lack of perception of the asymmetrytheir lack of perception of the asymmetry

Athletic Ready Position

Co-activation Exercise Activities

Begalle RL, J Athl Train, 2012

Appreciation for Quad Function

Quad inhibition/avoidance is common following ACL injuryI d k i 9 h Inadequate knee extension moments present 9 months post‐op 

Ernst, et al Phys Ther 2000

Non‐copers adopt immature co‐contraction tendencies, altered hip/ankle movement strategies and function in more upright positionsmovement strategies and function in more upright positions

Chmielewski, et al Phys Ther 2005

Pre‐op quad strength is a significant predictor (better than demographic or other impairments) of self‐report outcomes at 6 monthsp ) p

Logerstedt D, Knee, 2013

Need to emphasize and progress quad function after patellar t d bidit ltendon morbidity resolves

Contrast of OKC and CKC Knee Extension JOSPT J 1992JOSPT, June 1992

20 Knee Extension with 25% BW

ant.tibial translation with leg extension at angles above 64 Yack, AJSM, 1993.

men

t (m

m) 16

12Squat with 25% BW

Knee Extension with 25% BW

Knee Extension

al Di

splac

e

8

4Squat

Tibi

0

4

Zone of SafetyZone of Safety

Knee Flexion Angle (°)

Anterior tibial displacement during active knee extension ( ith d ith t i t ) d t t ( ith

90° 60° 30° 0°

(with and without resistance) compared to a squat (with and without resistance) in chronic ACL deficient knees

Common Problems to Overcome:

1. Reflexive inhibition of the quadriceps

2 Tendency to hold the knee in2. Tendency to hold the knee in resting position 20‐30° flexion to accommodate swellingaccommodate swelling

3. Flexed knee gait

ACL Rehab Points of Emphasis

Used to prevent the onset of infrapatellar fat pad syndromeinfrapatellar fat pad syndrome(Paulos, 1987) that cause loss of sagittal plane motion and isg pcharacterized by exaggeratedpathological fibrous hyperplasiaf th t i ft tiof the anterior soft tissues

beyond that which is associatedwith normal healingwith normal healing

CKC SQUAT vs. LEG EXTENSION

Foot fixed  Foot mobile– proximal/distal motion

load application axial

– isolated knee motion

force perpendicular to facilitating co‐contraction

tibia that causes shear

Weight Bearing RehabilitationCKC Training Rationale

• stability derived from the loaded extremity and mid‐ROM activities where rotary motion is minimizedROM activities where rotary motion is minimi ed

• appropriate proprioceptive feedback

• replicates stabilizing, accelerating, and decelerating proximal and distal muscle function

• functional movement patterns and development of synergistic relationshipssynergistic relationships

• simulates the planting, absorbing, loading, and change of direction forces encountered in function

ACL Rehab Points of Emphasis

1. Dynamic control of valgus moment at the knee joint• Avoid medial collapseAvoid medial collapse

2. Train for fast speeds and reaction timing3. Train hip and trunk control ‐ “avoid the corkscrew”

• Hip collapses in stance or landing (hip IR/add)Hip collapses in stance or landing (hip IR/add)4. Train the hip muscles to assist in stabilization

• Hams/Quads generally control the sagittal plane; need hip muscles to control frontal/transverse planeto control frontal/transverse plane

5. Train to control knee extension (knee stability)• Land and function in greater knee flexion

6 Enhance NM control and protective pattern reflexes6. Enhance NM control and protective pattern reflexes• Keep foot under the center of mass

7. Train muscular endurance

ACL Exercise Considerations

• “0‐40° Range Safe” in early rehab Henning 1985 Voight 1991 Ohkoshi 1991 Lutz 1993 Escamilla 2012 Henning, 1985, Voight, 1991, Ohkoshi, 1991, Lutz, 1993, Escamilla, 2012

• Technique• Dynamic frontal plane control• Dynamic frontal plane control• Vertical Squat (feet under hips)• Slight forward trunk incline (increases gluteal                                           

and hamstring activity)and hamstring activity)• Push through heels (keep knees behind toes)• As resistance increases later in rehab may                                          

t t li it t i l t i b twant to limit terminal extension range but                                         monitor for patellofemoral irritation

Hamstring Dominant Exercise

Non Weight Bearing• Ham CurlsHam Curls• Reverse Leg Lifts• Roller Stool Forward

Weight BearingWeight Bearing• Front Lunge – long stride• Bridging 

uni/bilateral; stable‐unstable• Kneeling Weight Shift• Roman Chair Hip Extension• Good Morning Hip Extension (RDL)• Retrograde Stair Climbing

Why the emphasis on the Hamstrings?

Synergist to the ACL– direct stress of ACL has moderate inhibitory effect on the quads and excites the hamstrings

– enhanced joint stabilizing role in ACL injured subject

Solomonow, AJSM 1987

H d H t i PTG How does Hamstring vs. PTG graft rehab differ?

Delayed hamstring strengthening– Submax isometrics begin at 4‐6 wks– Light isotonics at 6‐8 weeksStandard progression after two months– Standard progression after two months

Delayed functional progressionN i il l 3 h– No running until at least 3 months

– No jumping/plyometrics for at least 4 months– No unrestricted return to activity for at least 6 months

Quadriceps Dominant Exercise

NWB• Leg Extension (90‐45°)Leg Extension (90 45 )

− Early initiation is effective and safe – Fukada TY, Am J Sports Med, 2013• Roller Stool Backward

Weight Bearingg g• Leg Press 45‐100°• Wall Squat or Sit• Lateral Step Ups

PF Protection• weight back on heels• Move hands forward to maintain COG• 90‐45° in OKC and 45‐0° in CKC

Stationary Cycle

• cycling produced 7% elongation of an 80 lb. Lachman’s test and produced 1/5 of the ACLLachman s test and produced 1/5 of the ACL strain as walking on level ground

Henning CE, et al. .Am J Sports Med. 13:22–26,1985

• ACL stresses during cycling was 1/20 of unprotected isokinetic knee extension

Ericson MO Nisell R et al: Scand J Rehabil Med 18:165–172 1986Ericson MO, Nisell R, et al: Scand J Rehabil Med, 18:165 172,1986 

• Insignificant strain on the ACL during stationary cycling

Fleming BC, Beynnon BD, et al. Am J Sports Med. 26:109‐18, 1998

What is the need for regular, supervised physical therapy visits …

Can you get the same benefitwith a comprehensive HEP and periodic check ups?and periodic check ups?

7 visits (HEP based visits) vs. 20 (structured PT visits) had similar out‐comes in regards to ROM, isokinetic strength, and subjective ratingD C l M l J O h S Ph Th 1997DeCarlo M, et al, J Orthop Sports Phys Ther, 1997

Supervised exercise, in addition to a HEP, had minimal extra benefit for patients who had undergone ACL reconstruction in regards tofor patients who had undergone ACL reconstruction in regards to function, activity level, muscle strength, and anterior tibial translationBeard DJ, et al, J Orthop Sports Phys Ther, 1998Beard DJ, et al, J Orthop Sports Phys Ther, 1998

Recreational athletes undergoing ACL reconstruction had                           a higher % of subjects have a successful ROM outcome at                           

f3 months post‐op with a HEP consisting of 4 visits than a                            group who attended therapy twice/week (ave. 17 visits)Grant JA, et al,  Am J Sports Med, 2005

but, that Grant study just looked at short term impairment outcomes …

Follow‐up on 70% of those subjects at 2‐4 years showed2 4 years showed– ACL QoL score significantly higher 

for home exercise cohortNo significant difference in– No significant difference in Knee ROM Knee laxity Quad/Ham strength Quad/Ham strength IKDC score

So, do we need to change our paradigm?

Current Best Evidence of Selected Interventions based on Systematic Reviews

Intervention Value

CPM d t i li ht f tCPM no advantage in light of cost

Early weight‐bearingand ROM

no adverse effect(given good quad control and no concurrent surgery that necessitates limiting weight‐bearing for healing)necessitates limiting weight bearing for healing)

Post‐op/Functionalbracing

Neither necessary or beneficial(Additionally, Preponderance of the data indicates that the current brace designs cannot prevent injury)

Home‐based PT Effective in motivated patients

OKC vs. CKC exercise Safe to start OKC at 6 wks (or earlier)

“Accelerated” protocol Safe, but needs additional verificationAccelerated  protocol Safe, but needs additional verification

ACL Clinical GuidelinesKnee Stability and Movement Coordination Impairments - Knee Ligament SprainOrthopedic Section of the APTA

U /Intervention Implement

Unnecessary/ Not as 

Beneficial

Level of EvidenceSupporting 

Recommendation

Recommendation Grade

CPM for Post‐Op Pain II

CPM for Post‐Op ROM

Cryotherapy for Post‐Op Pain

Early Weight‐Bearing

II

II

IIEarly Weight‐Bearing

Mobilization Early Delayed

Post‐Op Bracing

II

I ‐ II

I ‐ III

Functional Bracing

ACL Deficient Bracing

Home Program Supplementation

I ‐ II

II ‐ III

I ‐ IIg pp

NMES (re‐education/strengthening)

I ‐ II

I ‐ II

I ‐ II

Therapeutic Exercise (OKC an CKC)

Eccentric Strengthening

“Accelerated” Rehab Safety

I ‐ II

II

Medial Knee Injuries

Grade Presentation

I T d i t bilitI Tenderness, no instability

II Broad tenderness, partial tear, laxity to stress

III End point absent with valgus stress in RPCC OO

Supported by posterior oblique ligament and superficial and deep tibial collateral ligaments

sMCLsMCL -- POLPOL

dMCLdMCL

Evaluated by valgus stress tests at 0/30° and anterior drawer test in external rotation

Medial Knee Rehabilitation

Hinged knee brace and protected arc of motion 20 90° for first 2 6 wks dependentmotion 20‐90 for first 2‐6 wks dependent upon grade severity

Emphasis on  frontal and transverse plane p p(internal rotators) dynamic stabilizers

Protocol provided in appendix of Clinical C t ti l i filCommentary article in your resource file

LaPrade RF, et al, J Orthop Sports Phys Ther, 2012 

Posterior Cruciate Ligament

Vertical orientationP 94% f Prevents 94% of posterior translation of tibia on femur

Prevents hyperextension

Posterior Cruciate Ligament Injury

Isolated PCLIsolated PCL Combine PCL InjuryCombine PCL Injury

Grade IGrade I Grade IIGrade II Grade IIIGrade IIIPCL/LCLPCL/LCL PCL/MCLPCL/MCL PCL/ACLPCL/ACL

NonNon‐‐op rehab op rehab with quadwith quad

NonNon‐‐op rehab op rehab unless pain &unless pain &

PCL/LCLPCL/LCL

Surgery within 2 weeks Surgery within 2 weeks –– PCL PCL reconstruction and acute repair ofreconstruction and acute repair of

PCL/MCLPCL/MCL PCL/ACLPCL/ACL

with quad with quad emphasis and emphasis and activity activity modsmods

unless pain & unless pain & instability instability continuescontinues

reconstruction and acute repair of reconstruction and acute repair of collateral ligaments and/or ACL collateral ligaments and/or ACL 

reconstructionreconstruction

I

Posterior InstabilityII

III

Grade III immobilized in full extension for 2‐4 weeks with FWB to toleranceFWB to tolerance

Quad emphasis for dynamic stability

A id f i l d h i i i Avoidance of isolated hamstring activity

“Mirror” perspective to ACL                                                         rehabilitation philosophyrehabilitation philosophy

Multi-Ligamentous Knee Injuries

• Pathoanatomy ‐ “dislocation” with significant soft tissue disruption with at leastsignificant soft tissue disruption with at least 3 of 4 ligaments involved

• 60% chance of tibial or femoral fracture • 50% chance of vascular injury with A/P• 50% chance of vascular injury with A/P dislocation 

• 25% chance for common peroneal nerve injuryinjury 

• Prognosis: poor− complications frequent and rarely does knee return to pre‐injury statereturn to pre‐injury state

MLKI Management

Initial Treatment– Immediate reduction and evaluation of vascular status confirmed by– Immediate reduction and evaluation of vascular status confirmed by 

arterial duplex ultrasound or CT angiography– Limited indication for non‐operative care

Operative Intervention Operative Intervention– emergent surgical intervention indicated if:

• vascular injury repair (takes precedence)f d di l ti• open fx and open dislocation

• irreducible dislocation• compartment syndrome

d l d li i / i– delayed ligamentous reconstruction/repair 

MLKI Management continued -

Delayed Surgical Reconstruction/Repair ‐– 1 or 2 staged intervention with PCL + medial and lateral structures repaired– 1 or 2 staged intervention with PCL + medial and lateral structures  repaired acutely and ACL reconstructed at a later time as needed

– improved outcomes with early treatment (within 3 weeks)

Post‐Operative TherapyPost Operative Therapy – early mobilization with functional bracing

ComplicationsStiffness (arthrofibrosis) is the most common complication (particularly with– Stiffness (arthrofibrosis) is the most common complication (particularly with delayed mobilization)

– Laxity and instability (37%)– Peroneal nerve injury (25%) (particularly with posterolateral dislocations)Peroneal nerve injury (25%) (particularly with posterolateral dislocations)

Impact of Pain and Fear

Pain is consistently associated with function across the time span of the rehabilitation processspan of the rehabilitation process

Fear of movement and re‐injury gradually decrease as function is restored and is a factor in the return to activityy

Do we need a FABQ modified for the knee?

Ch i l ki TL Ph Th 2008Chmielewski TL, Phys Ther, 2008

Functional Testing ResearchTesting Reliability has been Established

5-6”.3 sec

Test (Uninjured Patients) ICC SEMSingle Hop for Distance .96 4.56 cm

Reid A, et al. Phys Ther, 2007

6 m Hop for Time .66 .13 secondsTriple Hop for Distance .95 15.44 cmCrossover Hop for Distance .95 15.95 cmp .95 15.95 cmBolgla, et al. J Orthop Sports Phys Ther. 26:138‐143, 1997

Hop Test Examples

• Single Leg Hop for Distance

• Single Leg Triple Hop for Distance

• Single Leg 20’ (6m) Hop for Timeg g ( ) p

• Single Leg Crossover Hop for Time or Distance 

• Single Leg Vertical Jump

Hop Testing Research

• 50% have asymmetrical scores on distance hop 62% have asymmetry on distance or time hop• 62% have asymmetry on distance or time hop Noyes F, et al, Amer J Sports Med, 1991

Gustavsson A, et al. Knee Surg Sports Traumatol Arthrosc, 2006

Strength Test Examples

• PSLC Single Leg Squats

• Lateral or Front Step Downs

• Single Leg Wall SitSingle Leg Wall Sit

• Squat Depth Excursion

d l• Sport Cord Single Leg Squats

Dynamic Postural Control Tests

Balance Tests– Maintain COG over a base of support that is unpredictable 

M i t i COG fi d b f t– Maintain COG over a fixed base of support                                 while body segments move (reach tests)

Time to Stabilization Tests Time to Stabilization Tests– Vertical Hop or Dynamic Movement

functional tests

T run Box run Carioca test Carioca test Shuttle runS i t T t Sprint Test

Landing Stability

Resource Folder on your CD-ROM

Logerstedt DS, Snyder‐Mackler L, Ritter RC, Axe MJ. Knee Stability and Movement Coordination Impairments: Knee Ligament Sprain.  J Orthop S t Ph Th 2010 40(6) A1 A35Sports Phys Ther. 2010:40(6):A1‐A35

Wilk KE, Macrina LC, Cain EL, Dugas JR, Andrews JR. Recent advances in the rehabilitation of anterior cruciate ligament injuries. J Orthop Sports Phys Ther. 2012 Mar;42(3):153‐71.y ; ( )

van Grinsven S, van Cingel RE, Holla CJ, van Loon CJ. Evidence‐based rehabilitation following anterior cruciate ligament reconstruction. Knee Surg Sports Traumatol Arthrosc. 2010 Aug;18(8):1128‐44. 

Logerstedt DS, Snyder‐Mackler L, Ritter RC, Axe MJ; Orthopedic Section of the American Physical Therapy Association. Knee pain and mobility impairments: meniscal and articular cartilage lesions. J Orthop Sports Phys Ther. 2010 Jun;40(6):A1‐A35. 

Resource Folder on your CD-ROM

Laprade RF, Wijdicks CA. The management of injuries to the medial side of the knee. J Orthop Sports Phys Ther. 2012 Mar;42(3):221‐33.

Howells NR, Burnton LR, Robinson J, Porteus AJ, Eldridge JD, Murray JR. Acute knee dislocation: An evidence based approach to the management of the multiligament injured knee. Injury. 2011. 42(11):1198‐1204

Rosenthal MD, Rainey CE, Tognoni A, Worms R. Evaluation and management of posterior cruciate ligament injuries. Phys Ther Sport. Epub May 18, 2012

h ff bl d l ll ll Mithoeffer K, Hambly K, Logerstedet D, Ricci M, Silvers H, Della Villa S. Current concepts for rehabilitation and retun to sport after knee articular cartilage repair in the athlete.  J Orthop Sports Phys Ther 2012;42(3):254‐273

Questions - Discussion