Burn lecture.ppt

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    BURNINJURIES& ITSMANAGEMENT

    Dr I braheem Bashayreh, RN, PhD

    4/1/2011

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    BURNS

    Wounds caused by exposure to:

    1. excessive heat

    2. Chemicals

    3. fire/steam4. radiation

    5. electricity

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    BURNS

    Results in 10-20 thousand deaths annually

    Survival best at ages 15-45

    Children, elderly, and diabetics

    Survival best burns cover less than 20% of TBA

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    TYPES OF BURNS

    Thermal

    exposure to flame or a hot object

    Chemical

    exposure to acid, alkali or organic substancesElectrical

    result from the conversion of electrical energy into heat.Extent of injury depends on the type of current, the pathway offlow, local tissue resistance, and duration of contact

    Radiation

    result from radiant energy being transferred to the bodyresulting in production of cellular toxins

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    CHEMICALBURN

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    ELECTRICAL BURN

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    BURN WOUND ASSESSMENT

    Classified according to depth of injury and

    extent of body surface area involved

    Burn wounds differentiated depending on

    the level of dermis and subcutaneous tissueinvolved

    1. superficial (first-degree)

    2. deep (second-degree)3. full thickness (third and fourth degree)

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    SUPERFICIAL BURNS

    (FIRST DEGREE)

    Epidermal tissue only affected

    Erythema, blanching on pressure, mild swelling

    no vesicles or blister initially

    Not serious unless large areas involved i.e. sunburn

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    DEEP (SECOND DEGREE)

    *Involves the epidermis and deep layer of the dermis

    Fluid-filled vesiclesred, shiny, wet, severe pain

    Hospitalization required if over 25% of body surface

    involvedi.e. tar burn, flame

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    FULL THICKNESS (THIRD/FOURTH

    DEGREE)

    Destruction of all skin layers

    Requires immediate hospitalization

    Dry, waxy white, leathery, or hard skin, no pain

    Exposure to flames, electricity or chemicals cancause 3rddegree burns

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    TOTAL BODY SURFACE AREA (TBSA)

    Superficial burns are not involved in the calculation

    Lund and Browder Chart is the most accurate

    because it adjusts for age

    Rule of nines divides the bodyadequate for initialassessment for adult burns

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    LUND BROWDER CHART USED FOR

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    LUNDBROWDERCHARTUSEDFOR

    DETERMININGBSA

    4/1/2011 22Evans, 18.1, 2007)

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    RULES OF NINES

    Head & Neck = 9%

    Each upper extremity (Arms) = 9%

    Each lower extremity (Legs) = 18%

    Anterior trunk= 18% Posterior trunk = 18%

    Genitalia (perineum) = 1%

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    VASCULAR CHANGES RESULTING

    FROM BURN INJURIES

    Circulatory disruption occurs at the burnsite immediately after a burn injury

    Blood flow decreases or cease due tooccluded blood vessels

    Damaged macrophages within the tissuesrelease chemicals that cause constriction ofvessel

    Blood vessel thrombosis may occurcausing necrosis Macrophage:A type of white blood that ingests (takes in) foreign material.

    Macrophages are key players in the immune responseto foreign invaderssuch as infectious microorganisms.

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    http://www.medterms.com/script/main/art.asp?articlekey=3906http://www.medterms.com/script/main/art.asp?articlekey=3906
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    FLUID SHIFT

    Occurs after initial vasoconstriction, then

    dilation

    Blood vessels dilate and leak fluid into the

    interstitial spaceKnown as third spacing or capillary leak

    syndrome

    Causes decreased blood volume and blood

    pressure

    Occurs within the first 12 hours after the

    burn and can continue to up to 36 hours

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    FLUID IMBALANCES

    Occur as a result of fluid shift and cell

    damage

    Hypovolemia

    Metabolic acidosisHyperkalemia

    Hyponatremia

    Hemoconcentration (elevated bloodosmolarity, hematocrit/hemoglobin) due to

    dehydration

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    CURLINGS ULCER

    Acute ulcerative gastro duodenal disease

    Occur within 24 hours after burn

    Due to reduced GI blood flow and mucosal damage

    Treat clients with H2 blockers, mucoprotectants,

    and early enteral nutrition

    Watch for sudden drop in hemoglobin

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    EMERGENT PHASE

    *Immediate problem is fluid loss, edema,reduced blood flow (fluid and electrolyteshifts)

    Goals:

    1. secure airway2. support circulation by fluid replacement3. keep the client comfortable withanalgesics

    4. prevent infection through wound care5. maintain body temperature6. provide emotional support

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    EMERGENT PHASE

    Knowledge of circumstances surrounding the burninjury

    Obtain clients pre-burn weight (dry weight) tocalculate fluid rates

    Calculations based on weight obtained after fluidreplacement is started are not accurate because ofwater-induced weight gain

    Height is important in determining body surfacearea (BSA) which is used to calculate nutritionalneeds

    Know clients health history because thephysiologic stress seen with a burn can make alatent disease process develop symptoms

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    CLINICAL MANIFESTATIONS IN THE

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    CLINICAL MANIFESTATIONS IN THE

    EMERGENT PHASE Clients with major burn injuries and with inhalation injury are at

    risk for respiratory problems

    Inhalation injuries are present in 20% to 50% of the clientsadmitted to burn centers

    Assess the respiratory system by inspecting the mouth, nose, andpharynx

    Burns of the lips, face, ears, neck, eyelids, eyebrows, and

    eyelashes are strong indicators that an inhalation injury may bepresent

    Change in respiratory pattern may indicate a pulmonary injury.

    The client may: become progressively hoarse, develop a brassycough, drool or have difficulty swallowing, produce expiratorysounds that include audible wheezes, crowing, and stridor

    Upper airway edema and inhalation injury are most common inthe trachea and mainstem bronchi

    Auscultate these areas for wheezes

    If wheezes disappear, this indicates impending airway obstructionand demands immediate intubation

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    CLINICAL MANIFESTATIONS

    Cardiovascular will begin immediately

    which can include shock (Shock is a

    common cause of death in the emergent

    phase in clients with serious injuries)Obtain a baseline EKG

    Monitor for edema, measure central and

    peripheral pulses, blood pressure, capillary

    refill and pulse oximetry

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    CLINICAL MANIFESTATIONS

    Changes in renal function are related todecreased renal blood flow

    Urine is usually highly concentrated and hasa high specific gravity

    Urine output is decreased during the first 24hours of the emergent phase

    Fluid resuscitation is provided at the rate

    needed to maintain adult urine output at 30to 50- mL/hr.

    Measure BUN, creat and NA levels

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    CLINICAL MANIFESTATIONS

    Sympathetic stimulation during theemergent phase causes reduced GI motilityand paralytic ileus

    Auscultate the abdomen to assess bowel

    sounds which may be reducedMonitor for n/v and abdominal distentionClients with burns of 25% TBSA or who are

    intubated generally require a NG tube

    inserted to prevent aspiration and removalof gastric secretions

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    SKIN ASSESSMENT

    Assess the skin to determine the size and

    depth of burn injury

    The size of the injury is first estimated in

    comparison to the total body surface area(TBSA). For example, a burn that involves

    40% of the TBSA is a 40% burn

    Use the rule of nines for clients whose

    weights are in normal proportion to theirheights

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    IV FLUID THERAPY

    Infusion of IV fluids is needed to maintain sufficient bloodvolume for normal CO Clients with burns involving 15% to 20% of the TBSA

    require IV fluid Purpose is to prevent shock by maintaining adequate

    circulating blood fluid volume Severe burn requires large fluid loads in a short time to

    maintain blood flow to vital organs Fluid replacement formulas are calculated from the time

    of injury and not from the time of arrival at the hospital Diuretics should not be given to increase urine output.

    Change the amount and rate of fluid administration.Diuretics do not increase CO; they actually decreasecirculating volume and CO by pulling fluid from thecirculating blood volume to enhance diuresis

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    COMMON FLUIDS

    Protenate or 5% albumin in isotonic saline (1/2given in first 8 hr; given in next 16 hr)

    LR (Lactate Ringer) without dextrose (1/2 given infirst 8 hr; given in next 16 hr)

    Crystalloid (hypertonic saline) adjust to maintainurine output at 30 mL/hr

    Crystalloid only (lactated ringers)

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    ACUTE PHASE OF BURN INJURY

    Lasts until wound closure is complete

    Care is directed toward continued assessment andmaintenance of the cardiovascular and respiratory system

    Pneumonia is a concern which can result in respiratoryfailure requiring mechanical ventilation

    Infection (Topical antibioticsSilvadene) Tetanus toxoid

    Weight daily without dressings or splints and compare topre-burn weight

    A 2% loss of body weight indicates a mild deficit

    A 10% or greater weight loss requires modification ofcalorie intake

    Monitor for signs of infection

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    LOCAL AND SYSTEMIC SIGNS OF

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    LOCAL AND SYSTEMIC SIGNS OFINFECTION- GRAM NEGATIVE

    BACTERIA

    Pseudomonas, Proteus May led to septic shock

    Conversion of a partial-thickness injury to a full-thicknessinjury

    Ulceration of health skin at the burn site

    Erythematous, nodular lesions in uninvolved skin Excessive burn wound drainage

    Odor

    Sloughing of grafts

    Altered level of consciousness

    Changes in vital signs Oliguria

    GI dysfunction such as diarrhea, vomiting

    Metabolic acidosis

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    LAB VALUES

    Nahyponatremia or Hypernatremia

    KHyperkalemia or Hypokalemia

    WBC10,000-20,000

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    NURSING DIAGOSIS IN THE ACUTE

    PHASE

    Impaired skin integrity

    Risk for infection

    Imbalanced nutrition

    Impaired physical mobility Disturbed body image

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    PLANNING AND IMPLEMENTATION

    Nonsurgical management: removal of exudates and

    necrotic tissue, cleaning the area, stimulating

    granulation and revascularization and applying

    dressings. Debridement may be needed

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    DRESSING THE BURN WOUND

    After burn wounds are cleaned and debrided,

    topical antibiotics are reapplied to prevent infection

    Standard wound dressings are multiple layers of

    gauze applied over the topical agents on the burn

    wound

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    REHABILITATIVE PHASE OF BURN

    INJURY

    Started at the time of admissionTechnically begins with wound closure and

    ends when the client returns to the highestpossible level of functioning

    Provide psychosocial supportAssess home environment, financial

    resources, medical equipment, prostheticrehab

    Health teaching should include symptoms ofinfection, drugs regimens, f/u appointments,comfort measures to reduce pruritus

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    DIET

    Initially NPO

    Begin oral fluids after bowel sounds return

    Do not give ice chips or free water lead to

    electrolyte imbalance

    High protein, high calorie

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    GOALS

    Prevent complications (contractures)Vital signs hourly

    Assess respiratory functionTetanus boosterAnti-infective

    AnalgesicsNo aspirinStrict surgical asepsisTurn q2h to prevent contractures

    Emotional support

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    DEBRIDEMENT

    Done with forceps and curved scissor or through

    hydrotherapy (application of water for treatment)

    Only loose eschar removed

    Blisters are left alone to serve as a protector

    controversial

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    SKIN GRAFTS

    Done during the acute phase

    Used for full-thickness and deep partial-thickness

    wounds

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    POST CARE OF SKIN GRAFTS

    Maintain dressing

    Use aseptic technique

    Graft should look pink if it has taken after 5 days

    Skeletal traction may be used to prevent

    contractures Elastic bandages may be applied for 6 mo to 1 year

    to prevent hypertrophic scarring

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    THE END

    QUESTIONS

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