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Causal Attribution 1 A Connectionist Approach to Causal Attribution Frank Van Overwalle and Dirk Van Rooy Vrije Universiteit Brussel, Belgium === FINAL VERSION === Chapter prepared for : S. J. Read & L. C Miller (Eds.) Connectionist and PDP models of Social Reasoning and Social Behavior . Lawrence Erlbaum. We are very grateful to the editors for their helpful comments on earlier versions of the manuscript. The research reported in this chapter was in part supported by the Belgian National Foundation of Scientific Research (N.F.W.O.) under grant 8.0192.95. Address for correspondence: Frank Van Overwalle, Department of Psychology, Vrije Universiteit Brussel, Pleinlaan 2, B - 1050 Brussel, Belgium; or by e-mail: [email protected]. No license: PDF produced by PStill (c) F. Siegert - http://www.this.net/~frank/pstill.html

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Causal Attribution 1

A Connectionist Approach to Causal Attribution

Frank Van Overwalle and Dirk Van Rooy

Vrije Universiteit Brussel, Belgium

=== FINAL VERSION ===

Chapt er pr epar ed f or : S. J. Read & L. C Mi l l er ( Eds. )

Connect i oni st and PDP model s of Soci al Reasoni ng and Soci al

Behavior . Lawrence Erlbaum.

We ar e ver y gr at ef ul t o t he edi t or s f or t hei r hel pf ul

comment s on ear l i er ver si ons of t he manuscr i pt . The r esear ch

r epor t ed i n t hi s chapt er was i n par t suppor t ed by t he Bel gi an

Nat i onal Foundat i on of Sci ent i f i c Resear ch ( N. F. W. O. ) under gr ant

8. 0192. 95. Addr ess f or cor r espondence: Fr ank Van Over wal l e,

Depar t ment of Psychol ogy, Vr i j e Uni ver si t ei t Br ussel , Pl ei nl aan 2,

B - 1050 Brussel, Belgium; or by e - mail: [email protected].

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Causal Attribution 2

Introduction

At t r i but i ng a cause t o an event i s an i ndi spensabl e ment al

capaci t y t hat enabl es humans t o i dent i f y t he f act or s i n t hei r

envi r onment r esponsi bl e f or t hei r har dshi p or wel l - bei ng, t o

pr edi ct s i mi l ar event s i n t he f ut ur e and t o i ncr ease t hei r cont r ol

over t hei r occur r ences. I n f act , not onl y humans but al so ani mal s

use t he capaci t y t o det ect cause- ef f ect r el at i onshi ps i n or der t o

pr osper and saf eguar d t hei r ever yday adapt at i on and l ong- term

sur vi val . Gi ven t hi s l ong evol ut i onar y hi st or y f r om si mpl e

i nver t ebr at es l i ke t he mol l usk ( Hawki ns, 1989) on, i t seems

r easonabl e t o assume t hat much, i f not al l causal l ear ni ng i s

gover ned by ver y el ement ar y and si mpl e cogni t i ve pr ocesses,

oper at i ng i n ani mal s as wel l as humans. I n t hi s paper , we wi l l

ar gue t hat t he causal l ear ni ng pr ocess i nvol ves t he devel opment of

ment al associations or connections bet ween pot ent i al causes and

t he ef f ect , and t hat t hi s l ear ni ng pr ocess can be pr of i t abl y

analyzed from a connectionist perspective.

The associ at i ve appr oach t o causal l ear ni ng gr ew f r om

resea r ch on ani mal condi t i oni ng ( e. g. , Rescor l a & Wagner , 1972) ,

and has gai ned i ncr easi ng suppor t i n cur r ent cogni t i ve r esear ch on

human causal i t y and cat egor i zat i on ( f or r evi ews see Al l en, 1993;

Shanks, 1993, 1995) and i n connect i oni st or adapt i ve net wor k

model s of human memor y and t hi nki ng ( e. g. , Gl uck & Bower , 1988a;

McCl el l and & Rumel har t , 1988) . The var i ous t heor et i cal pr oposal s

put f or war d i n t hese di ver se ar eas of r esear ch seem t o conver ge t o

a f ew f undament al pr i nci pl es. For i nst ance, t he popul ar

asso ci at i ve model of ani mal condi t i oni ng pr oposed by Rescor l a and

Wagner i n 1972 i s, i n f act , f or mal l y equi val ent t o a speci f i c

c l ass of t wo- l ayer connect i oni st model s based on t he delta

l ear ni ng al gor i t hm ( i . e. , pat t er n associ at or s; McCl el l and &

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Causal Attribution 3

Rumelhart, 1988).

I n cont r ast t o cogni t i ve psychol ogi st s ' r i s i ng i nt er est i n

associ at i ve or connect i oni st l ear ni ng pr i nci pl es, soci al

psychol ogi st s have been sl ow i n i ncor por at i ng t hese i deas i n t hei r

t heor i es of human causal i t y j udgment s. Rat her t han t he

descendant s of a l ong evol ut i onar y past , t hey v i ew humans as nai ve

sci ent i st s ( Kel l ey, 1967) , l ogi c i ans ( Hewst one & Jaspar s, 1987) or

st at i st i c i ans ( Cheng & Novi ck, 1990) , who comput e causal i t y i n

anal ogy t o l ogi cal and st at i st i cal pr ocedur es and nor ms. Al t hough

r esear ch has r epeat edl y demonst r at ed t hat humans somet i mes make

bi ased i nf er ences t hat devi at e f r om nor mat i ve pr obabi l i t i es ( cf . ,

Kahneman, Sl ovi c & Tver sky, 1982) , such f i ndi ngs have been

r out i nel y i nt er pr et ed i n t er ms of unf avor abl e condi t i ons or l ack

of cogni t i ve r esour ces t o cal cul at e t he cor r ect i nf er ences r at her

t han as evi dence of an al t er nat i ve l ear ni ng pr ocess. Even

r esear ch i n soci al cogni t i on whi ch adopt ed not i ons f r om ear l i er

net wor k st r uct ur es ( e. g. , i n per son i mpr essi on, Hami l t on, Dr i scol l

& Wor t h, 1989) or f r om r ecent connect i oni st net wor k model s ( e. g. ,

i n causal knowl edge st r uct ur es, Read & Mar cus- Newhal l , 1993) has

been mai nl y concer ned wi t h t he r et r i eval of exi st i ng memor i es,

r at her t han wi t h t he l ear ni ng i t sel f of new concept s and causal

rel ationships.

How mi ght we under st and t he causal l ear ni ng pr ocess ? What

i nsi ght s do connect i oni st appr oaches of f er t hat go beyond t hose

of f er ed by al t er nat i ve r ul e- based ( e. g. , st at i st i cal ) model s ? To

addr ess t hese f undament al quest i ons, we begi n t hi s chapt er wi t h a

r evi ew of t he nor mat i ve pr obabi l i s t i c t heor y of causal at t r i but i on

as exempl i f i ed by t he cont r ast model devel oped by Cheng and Novi ck

( 1990) and ext ended by Van Over wal l e ( 1996a; Van Over wal l e &

Heyl i ghen, 1995) ; and compar e i t wi t h a connectionist

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Causal Attribution 4

i mpl ement at i on of Rescor l a and Wagner ' s ( 1972) model of l ear ni ng.

Next , we wi l l pr esent evi dence i ndi cat i ng t hat t he connect i oni st

appr oach pr ovi des a bet t er expl anat i on f or some phenomena of

causal compet i t i on l i ke di scount i ng and augment at i on ( cf . Kel l ey,

1971) . Fi nal l y, we wi l l pr esent t he configural model by Pear ce

( 1994) whi ch r epr esent s a maj or advance i n connect i oni st model i ng

t hat over comes sever al l i mi t at i ons of ear l i er connect i oni st model s

( Rescor l a & Wagner , 1972; McCl el l and & Rumel har t , 1988) , and we

wi l l r evi ew some dat a suggest i ng t hat t hi s model i s super i or i n

deal i ng wi t h gener al i zat i on of causal i t y t o f act or s t hat ar e

similar to the true cause.

Probabilistic Approach

Kel l ey ( 1967) , one of t he f ounder s of at t r i but i on t heor y i n

soci al psychol ogy, pr oposed t hat per cei ver s i dent i f y t he causes of

an ef f ect by usi ng a pr i nci pl e of covariation whi ch speci f i es t hat

an " ef f ect i s at t r i but ed t o t hat condi t i on whi ch i s pr esent when

t he ef f ect i s pr esent and whi ch i s absent when t he ef f ect i s

absent " ( p. 194) . He speci f i ed t hr ee maj or compar i sons t hat ar e

i mpor t ant i n det ect i ng covar i at i on and causal i t y i n t he soci al

domai n, and l at er r esear ch ( e. g. , Hewst one & Jaspar s, 1987; Hi l t on

& Sl ugoski , 1986; Cheng & Novi ck, 1990) demonst r at ed t hat each of

t hese compar i sons gener at es an at t r i but i on : Low consensus ( t he

ef f ect occur s when t hi s per son i s pr esent but not when ot her s ar e)

pr oduces at t r i but i ons t o t he person ; hi gh distinctiveness ( t he

ef f ect occur s when t hi s st i mul us i s pr esent but not when other

st i mul i ar e) gener at es at t r i but i ons t o t he stimulus ; and l ow

consistency ( t he ef f ect i s pr esent at t hi s occasi on but not at

ot her occasi ons) pr oduces at t r i but i ons t o t he occasion . Al t hough

Kel l ey ' s ( 1967) covar i at i on i dea i s now wi del y accept ed i n t he

at t r i but i on l i t er at ur e, t her e i s di sagr eement about t he under l y i ng

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Causal Attribution 5

pr ocesses by whi ch covar i at i on and causal i t y i s det ect ed. Some

at t r i but i on r esear cher s have t aken a pr obabi l i s t i c appr oach t o

describe this process in more detail.

Probabilistic Contras ts

Accor di ng t o r esear cher s t aki ng a pr obabi l i s t i c per spect i ve,

per cei ver s st or e i n memor y t he f r equenci es about cause- effect

occur r ences, and t hen per f or m some quasi - st at i st i cal comput at i on

on t hese f r equenci es i n or der t o pr oduce a causal j udgment ( f or an

over vi ew see Al l en, 1993; Cheng & Novi ck, 1990; Shanks, 1993) . I n

Fi gur e 1, t he r el evant f r equenci es ( denot ed by a - d) ar e

r epr esent ed i n a 2 x 2 cont i ngency t abl e, wher e one axi s r ef l ect s

t he pr esence or absence of t he pot ent i al cause C, and t he second

axis the presence or absence of the effect.

--------------------------

Insert Figure 1 about here

--------------------------

For r easons t hat wi l l become cl ear l at er , t he f i r st axi s al so

di spl ays a second f act or X, r epr esent i ng t he backgr ound or cont ext

agai nst whi ch t he cause C occur s. Thi s cont ext i s i nvar i ant l y

pr esent , i r r espect i ve of t he pr esence or absence of t he t ar get

f act or C. To di st i ngui sh bet ween t he t wo t ypes of f act or s, t he

f act or C i s t er med a contrast f act or ( because i t i s pr esent i n one

case but not t he ot her ) , and t he f act or X i s t er med a context

f act or ( because i t r ef l ect s t he cont ext pr esent i n al l cases under

obser vat i on) . I t shoul d be not ed t hat we def i ne cont r ast or

cont ext f act or s wi t h r espect t o a f ocal set of obser vat i ons

sel ect ed by t he per cei ver or pr ovi ded by t he exper i ment er ; t hey

ar e not necessar i l y cont r ast i ve or cont ext ual under al l possi bl e

observations of interest.

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Causal Attribution 6

Accor di ng t o pr obabi l i s t i c t heor y, causal j udgment s i nvol ve

t he cal cul at i on of t he cont r ast bet ween t he pr obabi l i t y of t he

ef f ect gi ven t he pr esence of t he cause mi nus t he same pr obabi l i t y

gi ven t he absence of t he cause ( see Al l en, 1993; Cheng & Novi ck,

1990; Shanks, 1993) . St at ed mor e si mpl y, causal i t y i s at t r i but ed

t o a cause t hat di f f er s f r om an i nvar i ant backgr ound or cont ext

wher e t hat cause i s absent . Thi s i s mat hemat i cal l y expr essed by a

probabilistic contrast 1 :

∆PC = P[Effect|C] - P[Effect|~C] [1]

= a - c a + b c + d

i n whi ch P r epr esent s t he pr opor t i on i n whi ch t he ef f ect

occur s when t he cause i s pr esent ( C) or absent ( ~C; a t i l de

denot es t he absence of a f act or ) ; and t he smal l l et t er s a- d denot e

t he f r equenci es i n Fi gur e 1. Thi s pr obabi l i s t i c cont r ast i s

c l osel y r el at ed t o common st at i st i cal measur es of cor r el at i on

bet ween t wo st i mul i such as t he χ2 st at i st i c, and has t her ef or e

received a normative status.

How can t he pr obabi l i s t i c cont r ast equat i on be appl i ed t o

Kel l ey ' s ( 1967) covar i at i on pr i nci pl e ? An i nnovat i ve at t empt t o

t ackl e t hi s quest i on was devel oped by Cheng and Novi ck ( 1990) i n

t hei r pr obabi l i s t i c cont r ast model . As t hei r appr oach i s al so

i mpor t ant i n under st andi ng connect i oni st appl i cat i ons, we wi l l

di scuss one exampl e i n some mor e det ai l . Let us t ake t he t ar get

event " Sar ah l aughed" , and l et us f ocus on Sarah as t he pot ent i al

cause of t he ef f ect laughed . Because t he ef f ect ( l aughed) i s

al ways pr esent when t he t ar get per son ( Sar ah) i s pr esent , t hi s can

be expr essed as P[ Laughed| Sar ah] = 1. Now, t o est i mat e Sar ah' s

causal r ol e, we need t o cont r ast t hi s pr obabi l i t y wi t h a r el evant

causal backgr ound wher e t he t ar get per son i s absent , f or exampl e,

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Causal Attribution 7

wi t h ot her per sons such as Sar ah' s f r i ends. The l ow consensus

i nf or mat i on t hat her f r i ends di d not l augh at t he same event can

be expr essed as P[ Laughed| ~Sar ah] = 0. Then, accor di ng t o

Equat i on 1, t he cont r ast bet ween t ar get and compar i son per sons

wi l l be hi gh, or ∆PSarah = 1, and Sar ah' s l aught er wi l l

consequent l y be at t r i but ed t o her sel f . I f , on t he ot her hand, t he

hi gh consensus i nf or mat i on i s gi ven t hat Sar ah' s f r i ends al so

l aughed dur i ng t he same event or P[ Laughed| ~Sar ah] = 1, t hen

∆PSarah = 0 and causal i t y wi l l not be at t r i but ed t o her . The same

cont r ast l ogi c appl i es f or hi gh di st i nct i veness and l ow

consi st ency i nf or mat i on. Gener al l y, a f act or wi l l be desi gnat ed

t he cause when i t s out come i s di f f er ent f r om t hat of t he

compar i son cases. Empi r i cal r esear ch has conf i r med t hat peopl e

make at t r i but i ons t o t he per son, st i mul us or occasi on i n l i ne wi t h

the probabilistic contrast model (Cheng & Novick, 1990) .

However , an i mpor t ant shor t comi ng of pr obabi l i s t i c t heor y i s

t hat i t does not speci f y how at t r i but i ons ar e made about t he

causal backgr ound gi ven a f ocal set of obser vat i ons. For exampl e,

when j udgi ng t he causal r ol e of Sar ah, her behavi or was cont r ast ed

wi t h t hat of ot her per sons who const i t ut ed a r el evant causal

backgr ound or cont ext ; but t hi s causal backgr ound i t sel f coul d not

be est i mat ed. Cheng and Novi ck gave t he causal cont ext onl y a

r at her shal l ow i nt er pr et at i on as enabl i ng condi t i on or i r r el evant

f act or , and speci f i ed t hat compar i sons wi t h an al t er nat i ve set of

obser vat i ons wer e necessar y t o j udge t hei r st r engt h. However , as

we wi l l see, i n connect i oni st appr oaches, causal cont ext s can be

est i mat ed and do pl ay a cr uci al r ol e i n det er mi ni ng t he causal

st r engt h of a cont r ast f act or , even wi t hi n a gi ven set of

obser vat i ons. Gi ven t hat t hi s i s t he case, we mi ght s i mpl y st op

her e and not e t hat t he pr obabi l i s t i c model i s ser i ousl y def i c i ent

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Causal Attribution 8

and much mor e l i mi t ed t han connect i oni st appr oaches. However , an

al t er nat i ve appr oach whi ch seems much mor e i nf or mat i ve, i s t o

ext ent pr obabi l i s t i c t heor y wi t h addi t i onal pr edi ct i ons f or causal

cont ext s, so t hat we st i l l can make sensi bl e compar i sons bet ween

the two approaches.

Probabilistic Contexts

What i s a cont ext f act or pr eci sel y, and how can i t s causal

st r engt h be i nf er r ed f r om a f ocal set of obser vat i ons ?

For t unat el y, t hi s i ssue was al r eady addr essed i n our ear l i er wor k

on causal at t r i but i on ( Van Over wal l e, 1996a; Van Over wal l e &

Heyl i ghen, 1995) . A cont ext i s def i ned as a r el at i vel y constant

background condi t i on consi st i ng, f or i nst ance, of " s i t uat i onal

st i mul i ar i s i ng f r om t he . . . envi r onment " ( Rescor l a & Wagner ,

1972, p. 88) . Because r esear cher s on t he f undament al di mensi ons

of human causal i t y ( e. g. , Abr amson, Sel i gman & Teasdal e, 1978;

Wei ner , 1986) al r eady i nt r oduced a t er mi nol ogy f or f act or s t hat

cor r espond ver y much t o our not i on of const ant backgr ound

condi t i ons, we bor r owed t hei r t er ms. Hence, t he cont ext of a

per son i s denot ed as external c i r cumst ances ( whi ch r emai n const ant

acr oss di f f er ent per sons) ; t he cont ext of a st i mul us as a global

cause ( whi ch r emai ns const ant acr oss di f f er ent st i mul i ) ; and t he

cont ext of an occasi on as a stable cause ( whi ch r emai ns per manent

over t i me) . As can be seen, each cont ext r ef l ect s a const ant

condi t i on agai nst whi ch a cont r ast cause may be di st i ngui shed.

Each pai r r ef l ect s t he t wo ext r emes of st andar d di mensi ons of

causal i t y, i ncl udi ng locus ( per sonal vs. ext er nal ) , globality

(stimulus - speci f i c vs. gener al ) and stability ( occasi onal vs.

st abl e) . Al t hough t hi s t er mi nol ogy st i l l l eaves open t he quest i on

whi ch speci f i c el ement s i n t he cont ext ar e r esponsi bl e f or t he

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Causal Attribution 9

ef f ect , t hi s same pr obl em i s i n f act al so t r ue f or cont r ast

causes. For i nst ance, an at t r i but i on t o t he per son does not

i ndi cat e whi ch el ement i nsi de t he per son i s causal i t y r el evant --

hi s or her abi l i t y , mot i vat i on, and so on ( see f or exampl es of

contrast and context causes, Weiner, 1986).

To comput e t he causal st r engt h of a cont ext f act or , Van

Over wal l e ( 1996a) devel oped a anal ogous pr obabi l i s t i c equat i on

which is mathematically expressed by a probabilistic context :

∆PX = P[Effect|~C] [2]

= c c + d

The l ogi c behi nd t hi s equat i on i s st r ai ght f or war d, because i t

has t he same gener al f or mat as t he cont r ast equat i on ( see Equat i on

1) , but r et ai ns onl y t he second t er m whi ch r epr esent s t he causal

cont ext X. I t speci f i es t hat t he st r engt h of a causal cont ext can

be est i mat ed f r om t he pr obabi l i t y of t he ef f ect gi ven t he r el evant

comparison cases where the target factor C is absent.

The pr obabi l i s t i c cont ext equat i on can al so easi l y be appl i ed

t o Kel l ey ' s ( 1967) covar i at i on var i abl es. I n our pr evi ous

exampl e, t he l ow consensus i nf or mat i on t hat most of Sar ah' s

f r i ends di d not l augh can be expr essed as P[ Laughed| ~Sar ah] = 0,

and t hi s l ow pr obabi l i t y i ndi cat es t hat t he causal cont ext ( e. g. ,

ext er nal c i r cumst ances) had l i t t l e causal ef f ect on Sar ah' s l augh.

I n cont r ast , t he hi gh consensus i nf or mat i on t hat most of her

f r i ends l aughed can be expr essed as P[ Laughed| ~Sar ah] = 1 so t hat

i n t hi s case causal i t y wi l l be st r ongl y at t r i but ed t o t he ext er nal

cont ext . The same l ogi c appl i es t o l ow di st i nct i veness and t o

hi gh consi st ency. Thus, i n gener al , a causal cont ext wi l l acqui r e

a subst ant i al amount of causal wei ght when bot h t he t ar get and

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Causal Attribution 10

comparison cases obtain the same effect.

The pr obabi l i s t i c pr edi ct i ons f or bot h cont r ast and cont ext

causes wer e combi ned i n what we have t er med t he j oi nt model ( Van

Over wal l e, 1996a; Van Over wal l e & Heyl i ghen, 1995) t o r ef l ect t he

j oi nt oper at i on of t he t wo pr obabi l i s t i c equat i ons. The j oi nt

model bor r ows Equat i on 1 f r om Cheng and Novi ck ' s ( 1990) cont r ast

model f or t he comput at i on of cont r ast f act or s, and adds Equat i on 2

f or t he comput at i on of cont ext f act or s. Hence, t he t wo equat i ons

ar e used separ at el y f or est i mat i ng cont r ast and cont ext f act or s.

Resear ch on t he j oi nt model has conf i r med t hat peopl e make

at t r i but i ons t o cont r ast and cont ext causes i n l i ne wi t h t he

pr edi ct i ons of t he j oi nt model ( Van Over wal l e & Heyl i ghen, 1995;

Van Overwalle, 1996a).

Connectionist Approach

Al t hough t he empi r i cal r esul t s ar e suppor t i ve of a

pr obabi l i s t i c anal ysi s of causal at t r i but i on, i t seems qui t e

i mpl ausi bl e t hat ani mal s and humans possess t he capaci t y t o t al l y

and memor i ze f r equenci es i n t he pr esence and absence of al l

pot ent i al causes and expl i c i t l y comput e t he r el evant cont r ast and

cont ext pr obabi l i t i es. The ear l y associ at i ve model s and t he mor e

r ecent connect i oni st appr oaches addr essed t hese cogni t i ve

l i mi t at i ons by assumi ng t hat t he per cei ved st r engt h of causes i s

di r ect l y st or ed i n memor y under t he f or m of ment al connect i ons

bet ween t he pot ent i al cause and t he ef f ect . These cause- effect

connect i ons ar e gr adual l y adj ust ed gi ven i nf or mat i on on t he co-

occur ence of t he cause and t he ef f ect . Var i ous l ear ni ng

mechani sms descr i bi ng t hese adj ust ment s have been pr oposed i n t he

ani mal and human l ear ni ng l i t er at ur e, but t he l ear ni ng al gor i t hm

devel oped by Rescor l a and Wagner i n 1972 gai ned most popul ar i t y.

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Causal Attribution 11

Because t hi s al gor i t hm pr eceded r ecent devel opment s i n

connect i oni st model i ng and st i l l i s a maj or sour ce of i nspi r at i on

i n t he f i el d of associ at i ve l ear ni ng ( cf . , Pear ce, 1994) , we wi l l

f i r st di scuss t he Rescor l a- Wagner model and t hen t ur n t o a

connectionist implementation of it.

Rescorla - Wagner Associative Model

A cent r al not i on of t he Rescor l a- Wagner l ear ni ng al gor i t hm i s

t hat " or gani sms onl y l ear n when event s v i ol at e t hei r expect at i ons"

( p. 75) . Thus, changes i n associ at i ve wei ght s of a causal f act or

are dr i ven by r educi ng t he di f f er ence bet ween t he act ual ef f ect

and t he ef f ect expect ed by t he or gani sm. The r educt i on of t hi s

di f f er ence or er r or t akes pl ace af t er each t r i al i n whi ch t he

f act or i s pr esent , and t he r esul t ant adj ust ment i n wei ght or ∆w of

the factor, is expressed in the following learning formula :

∆w = α βw ( λ - Σw). [3]

wher e α r epr esent s t he sal i ence or t he pr obabi l i t y of

at t endi ng t o t he f act or , and i s nor mal l y set t o 1 i f t he f act or i s

pr esent and t o 0 i f absent ; and βw i s a l ear ni ng r at e par amet er ,

r angi ng bet ween 0 and 1, whi ch r ef l ect s t he speed of l ear ni ng.

The λ var i abl e denot es t he magni t ude of t he ef f ect , and i s

t ypi cal l y set t o 1 when t he ef f ect i s pr esent and t o 0 when

absent ; and Σw r epr esent s t he expect ed ef f ect based on t he summed

association weights of all factors present on the trial.

The cour se of l ear ni ng i n t he s i mpl e case wi t h one causal

f act or ( Σw = wC) i s st r ai ght f or war d. The wei ght wC st ar t s at

zer o, and successi ve t r i al s cause an i ncr ease or decr ease until

t he poi nt i s r eached when t he er r or or ( λ - wC) i s zer o, i mpl y i ng

t hat t he ef f ect i s per f ect l y pr edi ct ed by t he cause C, and no

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Causal Attribution 12

f ur t her changes ar e necessar y. I n t hat case, we say t hat l ear ni ng

has r eached asymptote . The l ear ni ng r at e par amet er βw det er mi nes

t he pr opor t i on or speed by whi ch t he di scr epancy or er r or bet ween

expect ed and act ual ef f ect ar e t aken i n t o account f or adj ust i ng

associ at i ve wei ght s. Thus, causal l ear ni ng i s concei ved as a

gr adual pr ocess t hat i s cont i nuousl y updat ed r at her t han a f i nal

j udgment at t he end of a ser i es of obser vat i ons as assumed by t he

probabilistic approach.

Connectionist Implementation

An i mpor t ant f eat ur e of Rescor l a and Wagner ' s associ at i ve

model i s t hat t hei r l ear ni ng al gor i t hm ( Equat i on 3) i s i dent i cal

t o t he delta or Wi dr ow- Hof f updat i ng al gor i t hm whi ch has pl ayed a

maj or r ol e i n some f eedf or war d connect i oni st model s ( McCl el l and &

Rumel har t , 1988) . Consequent l y, t he Rescor l a- Wagner model can be

easi l y i mpl ement ed by a t wo- l ayer f eedf or war d ar chi t ect ur e, as

i l l ust r at ed i n Fi gur e 2 ( see al so Gl uck & Bower , 1988a; McCl el l and

& Rumel har t , 1988) . The f i r st l ayer compr i ses i nput nodes t hat

encode t he pr esence of causal f act or s, and t he second l ayer

compr i ses t he out put node r epr esent i ng t he pr edi ct ed ef f ect . The

i nput nodes ar e connect ed t o t he out put node vi a adj ust abl e

connections or weights, denoted by dashed lines in Figure 2.

--------------------------

Insert Figure 2 about here

--------------------------

When a f act or i s pr esent at a t r i al , t he act i vat i on of i t s

i nput node i s t ur ned on at val ue 1; and when a f act or i s absent ,

t he act i vat i on i s t ur ned of f t o 0. Ther e ar e t wo f eat ur es i n t hi s

i nput codi ng t hat di f f er somewhat f r om a t ypi cal f eedf or war d

net wor k. Fi r st , t he absence of a f act or i s coded as an act i vat i on

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Causal Attribution 13

of 0 r at her - 1 ( McCl el l and & Rumel har t , 1988) . Thi s codi ng scheme

r ef l ect s t he common obser vat i on i n associ at i ve r esear ch t hat

ani mal s and humans l ear n l i t t l e about obj ect s t hat ar e absent .

Second, t her e i s al ways a const ant cont ext f act or X pr esent on

ever y t r i al , i r r espect i ve of t he pr esence of t he t ar get cause C,

( see some i l l ust r at i ve codi ng i n Fi gur e 2) . As not ed bef or e, t hi s

cont ext assumpt i on was al so i nt r oduced i n t he pr obabi l i s t i c model

by Van Over wal l e and Heyl i ghen ( 1995) . Al t hough a ver y s i mi l ar

i dea can be i mpl ement ed i n connect i oni st model s by t he use of bias

t er ms ( see McCl el l and & Rumel har t , 1988, p. 121) , associ at i ve

appr oaches assume t hat t hese cont ext f act or s r ef l ect i mpor t ant and

meani ngf ul el ement s of t he envi r onment i n whi ch l ear ni ng t akes

place (e.g., the animal's cage).

Next , act i vat i on f r om t he i nput nodes spr eads aut omat i cal l y

t o t he out put node i n pr opor t i on t o t hei r connect i on wei ght s, and

ar e summed t o det er mi ne t he act i vat i on of t he out put node. Thi s

out put act i vat i on i s t hus a l i near f unct i on of t he i nput

act i vat i on, and r epr esent s t he st r engt h of t he ef f ect pr edi ct ed by

t he net wor k. The act ual l y obt ai ned ef f ect i s r epr esent ed by a

t eachi ng val ue ( not shown i n t he f i gur e) , usi ng t he same +1 and 0

codi ng scheme as t he i nput nodes. Thi s t eachi ng val ue ser ves as

i nput t o t he out put node; and t he er r or bet ween t he out put

( expect ed ef f ect ) and t eachi ng act i vat i ons ( act ual ef f ect )

det er mi nes t he adj ust ment s t o t he st r engt hs of t he connect i ons i n

t he net wor k, as i n t he Rescor l a- Wagner model . The change i n t he

connect i on wei ght of f act or j ( ∆wj) i s pr opor t i onal t o t hi s er r or ,

and t hi s i s f or mal l y expr essed by t he del t a l ear ni ng al gor i t hm

(McClelland & Rumelhart, 1988, p. 87) :

∆wj = ε (t - o) a j . [4]

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Causal Attribution 14

wher e ε i s t he l ear ni ng r at e, and wher e t he ot her symbol s t ,

o, and aj denot e, r espect i vel y, t he t eachi ng, out put and i nput

act i vat i ons. Compar i ng t hi s equat i on wi t h Equat i on 3 of Rescor l a-

Wagner , shows t hat t hey ar e compl et el y i dent i cal , except f or some

not at i onal var i at i ons. The l ear ni ng r at e βw i n t he Rescor l a-

Wagner equat i on i s r epr esent ed her e by ε, t he magni t ude of t he

act ual ef f ect λ i s r epl aced by t he t eachi ng act i vat i on t , the

expect ed ef f ect based on t he summed associ at i on wei ght s Σw i s

denot ed her e by t he out put act i vat i on o, and t he sal i ence α of a

f act or i s r ef l ect ed i n t he act i vat i on of t he cor r espondi ng i nput

node a j of all factors present on the trial

I n r esear ch on causal l ear ni ng, af t er t he subj ect s have gone

t hr ough a t r i al - by - t r i al acqui s i t i on phase, t hey ar e pr esent ed

wi t h quest i ons concer ni ng t he causal i nf l uence of some f act or s.

An appr opr i at e measur e i n t he net wor k f or subj ect s ' causal

j udgment s i s s i mpl y t he act i vat i on of t he out put node, gi ven t hat

t he appr opr i at e i nput nodes ar e t ur ned on. Thus, t o t est t he

pr edi ct i ons of t he net wor k, t he f act or s t o- be- t est ed ar e t ur ned on

at t he i nput l ayer as bef or e, except t hat t he st r engt h of a

cont r ast f act or i s now t est ed separ at el y wi t hout i t s accompanyi ng

cont ext ( see some i l l ust r at i ve codi ng i n Fi gur e 2) . I n t he

pr esent t wo- l ayer net wor k, t he out put act i vat i on i s i dent i cal t o

t he connect i on wei ght of each f act or t est ed, because t hi s i s t he

sol e f act or wi t h i t s i nput act i vat i on t ur ned on. I n t he r emai nder

of t hi s chapt er , t hi s connect i oni st i mpl ement at i on of associ at i ve

learning will be referred to as the Rescorla - Wagner network.

Fi gur e 3 i l l ust r at es t wo l ear ni ng hi st or i es wi t h 6 t r i al s f or

f act or s C and X, s i mul at ed by a connect i oni st net wor k j ust

descr i bed. I n t he f i r st exampl e wher e onl y t he cont r ast f act or

and i t s cont ext ar e f ol l owed by t he ef f ect ( CX → Ef f ect ; X → No

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Causal Attribution 15

Ef f ect ; l ef t panel ) , t he wei ght s t end t o asympt ot i c val ues of wC

= 1. 00 and wX = 0. 00. The cont r ast f act or acqui r es much posi t i ve

st r engt h because i t i s al ways f ol l owed by t he ef f ect . Al t hough

t he cont ext acqui r es some posi t i ve wei ght when pr esent ed t oget her

wi t h t he cont r ast f act or ( when CX → Ef f ect ) , t hi s wei ght i s

neut r al i zed ever y t i me the context is presented alone (when X → No

ef f ect ) , so t hat t he net r esul t i s l i t t l e causal st r engt h.

Conver sel y, i n t he second exampl e wher e onl y t he cont ext i s

f ol l owed by t he ef f ect ( X → Ef f ect , CX → No Ef f ect ; r i ght panel ) ,

t he wei ght s t end t o asympt ot i c val ues of wX = 1. 00 and wC = - 1.00.

The cont ext acqui r es st r ong posi t i ve st r engt h as i t i s al ways

f ol l owed by t he ef f ect . However , t he cont r ast f act or at t ai ns

st r ong negat i ve causal st r engt h, because when i t i s pr esent ed

t oget her wi t h t he cont ext t he ef f ect i s not pr oduced, so t hat i t

must compensate for the positive weight of the context.

--------------------------

Insert Figure 3 about here

--------------------------

An i mpor t ant char act er i st i c of t he Rescor l a- Wagner net wor k i s

t hat , gi ven suf f i c i ent l ear ni ng t r i al s, i t wi l l ar r i ve at t he same

causal pr edi ct i ons as t he pr obabi l i s t i c cont r ast and j oi nt model s

wi t hout st or i ng f r equenci es. Thi s somewhat sur pr i s i ng r esul t was

demonst r at ed mat hemat i cal l y by sever al aut hor s ( Chapman & Robbi ns,

1990; Van Over wal l e, 1996b) , and suggest s t hat t he nor mat i ve

pr edi ct i ons f r om pr obabi l i s t i c t heor y ar e, i n f act , al so emer gent

pr oper t i es of t he Rescor l a- Wagner l ear ni ng pr ocess. Fr om an

evol ut i onar y per spect i ve, i t seems i mper at i ve t hat associ at i ve

t hi nki ng i n ani mal s and humans has evol ved i n such a way t hat

pr edi ct i ve f act or s ar e i dent i f i ed wi t h r easonabl e accur acy

(Shanks, 1995).

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Causal Attribution 16

I n sum, t he t wo most i mpor t ant char act er i st i cs of an

connect i oni st appr oach t o causal l ear ni ng t hat di f f er ent i at es i t

f r om a pr obabi l i s t i c appr oach ar e t hat ( a) t he connect i oni st

appr oach does not r equi r e t hat f r equency i nf or mat i on on al l

pr evi ous t r i al s must be t al l i ed and memor i zed dur i ng t he l ear ni ng

per i od, but onl y t he cause- ef f ect connect i ons; and ( b) t he

connect i oni st appr oach does not r equi r e an expl i c i t and l abor i ous

comput at i onal pr ocess, but i mmedi at el y i nt egr at es i ncomi ng

i nf or mat i on of t he l ast t r i al by t he aut omat i c spr eadi ng of

act i vat i on f r om i nput t o out put and, i f i nconsi st enci es ar i se,

i mmedi at el y adj ust s t he connect i ons i n memor y so t hat causal

j udgment s ar e r eadi l y avai l abl e. I t i s evi dent t hat t he cogni t i ve

s i mpl i c i t y and evi dent gener al i t y of connect i oni st model s makes

t hem a ver y at t r act i ve al t er nat i ve t o pr obabi l i s t i c model s.

However , t he val i di t y of t he t wo appr oaches t o causal i t y depends

ul t i mat el y on t hei r empi r i cal conf i r mat i on. I t i s t o such

empirical findings that we turn now.

Discounting and Augmentation

To di st i ngui sh bet ween t he pr obabi l i s t i c and connect i oni st

account s, we expl or e t hei r pr edi ct i ons i n t wo si t uat i ons wher e a

per cei ver must l ear n whi ch one among mul t i pl e f act or s caused an

event . Despi t e t he cent r al pl ace accor ded i n at t r i but i on t heor y

t o t he pr i nci pl e of covar i at i on, Kel l ey ( 1971) r eal i zed t hat t hi s

pr i nci pl e al one was not suf f i c i ent t o expl ai n how compet i ng causal

expl anat i ons ar e sel ect ed, and he t her ef or e suggest ed t wo

auxiliary principles of discounting and augmentation .

The di scount i ng pr i nci pl e speci f i es t hat when t he i nf l uence

of one or mor e causes i s al r eady est abl i shed, per cei ver s wi l l

di sr egar d ot her possi bl e causes as l ess r el evant . A common

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Causal Attribution 17

exampl e i s when at t r i but i ons t o t he per son ar e di scount ed gi ven

evi dence on t he pot ent i nf l uence of ext er nal pr essur e. The

r ever se t endency i s descr i bed i n t he augment at i on pr i nci pl e, whi ch

suggest s t hat gi ven t wo opposi ng ( f aci l i t at or y vs. i nhi bi t or y)

causes, per cei ver s wi l l val ue t he st r engt h of t he cause t hat

pr oduces t he ef f ect hi gher t o compensat e f or t he i nhi bi t or y

i nf l uence of t he ot her cause. For i nst ance, success wi l l be mor e

st r ongl y at t r i but ed t o a per son' s capaci t i es when t he t ask was

har d r at her t han easy. Numer ous i nvest i gat i ons have shown t hat

t hese t wo compet i t i ve pr i nci pl es oper at e i n causal j udgment s of

adul t s ( e. g. , Hansen & Hal l , 1985; Kr ugl anski , Schwar t z, Mai des &

Hamel , 1978) and chi l dr en of 3- 4 year s of age ( e. g. , Kassi n &

Lowe, 1979; Kassin, L owe & Gibbons, 1980; Newman & Ruble, 1992).

Di scount i ng and augment at i on show a r emar kabl e s i mi l ar i t y

wi t h ef f ect s known i n t he associ at i ve l i t er at ur e as blocking and

condi t i oned i nhi bi t i on r espect i vel y ( cf . Val l ée- Tour angeau, Baker

& Mer ci er , 1994) . These t er ms r ef er t o speci f i c pr ocedur es by

whi ch compet i t i ve ef f ect s have been di scover ed, f i r st i n ani mal

condi t i oni ng ( e. g. , Kami n, 1968) , and subsequent l y i n causal

j udgment t asks wi t h humans ( e. g. , Baker , Mer ci er , Val ée-

Tour angeau, Fr ank & Pan, 1993; Chapman & Robbi ns, 1990; Chapman,

1991; Shanks, 1985, 1991) . As expl ai ned bef or e, a cent r al f eat ur e

of t he Rescor l a- Wagner and ot her connect i oni st model s i s t hat t he

adj ust ment s of cause- ef f ect connect i ons ar e det er mi ned by t he

di scr epancy bet ween t he act ual and expect ed ef f ect , gi ven not onl y

t he t ar get cause but all ot her s i mul t aneousl y pr esent ed causes, or

Σw. Hence, compet i t i on f or pr edi ct i ve st r engt h bet ween all causes

pr esent i s an i nher ent pr oper t y of associ at i ve or connect i oni st

learning.

We conduct ed an exper i ment ( Van Rooy & Van Over wal l e, 1996)

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Causal Attribution 18

i n whi ch, i n an i ni t i al phase, t he i nf l uence of t he cont ext was

st r engt hened i n or der t o expl or e whet her t hi s woul d l ead t o t he

di scount i ng or augment at i on of at t r i but i ons t o t he t ar get per son

or st i mul us l at er on. The causal st r engt h of t he cont ext was

enhanced by i ncr easi ng t he number of compar i son per sons or st i mul i

-- whi ch i mpl y t he cont ext -- f r om one ( smal l compar i son set ) t o

five ( l ar ge compar i son set ) . As di scussed bef or e, when sever al

compar i son per sons or st i mul i shar e t he same out come, st r ong

at t r i but i ons wi l l be made t o t he ext er nal or gl obal cont ext ,

r espect i vel y ( Van Over wal l e & Heyl i ghen, 1995; Van Over wal l e,

1996a) . For i nst ance, when many at hl et es r ecor d a ver y f ast t i me

i n a spr i nt r ace, i t i s mor e l i kel y t o at t r i but e t hi s out come t o

ext er nal c i r cumst ances such as a f avor abl e back wi nd r at her t han

t o per sonal t al ent . The cr uci al poi nt now i s t hat we expect

gr eat er di scount i ng or augment i ng ef f ect s when f i ve r at her t han

onl y one compar i son per son or st i mul us i s avai l abl e. For exampl e,

at t r i but i ons t o per sonal at hl et i c t al ent wi l l be mor e di scount ed

t he mor e ot her at hl et es al so r ecor ded f ast t i mes. Conver sel y,

per sonal t al ent wi l l be mor e augment ed t he mor e ot her at hl et es

r ecor ded sl ow r at her t han f ast r unni ng t i mes. To what ext ent can

probabilistic or connectionist models reproduce these predictions ?

Connectionist Predictions

Accor di ng t o t he Rescor l a- Wagner net wor k model , an i ncr easi ng

number of compar i son cases ( or t r i al s) wi t h a s i mi l ar out come wi l l

cause an i ncr ease i n t he per cei ved i nf l uence of t he cont ext . Thi s

i s i l l ust r at ed f or a per son f act or and an ext er nal cont ext i n

Fi gur e 4. As can be seen i n bot h t he t op and bot t om panel , t he

ext er nal cont ext acqui r es a much st r onger st r engt h af t er 5 t r i al s

i n t he l ar ge compar i son set ( wE = . 97) , t han af t er 1 t r i al i n t he

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Causal Attribution 19

smal l compar i son set ( wE = . 50) . Thi s cour se of causal l ear ni ng

was f ol l owed i n t he f i r st phase of our di scount i ng and

augmentation conditions.

--------------------------

Insert Figure 4 about here

--------------------------

The t wo condi t i ons di f f er ed i n t he second phase, af t er t he

causal st r engt h of t he cont ext had been est abl i shed. I n t he

di scount i ng condi t i on, a novel cont r ast per son or st i mul us was

i nt r oduced shar i ng t he same out come as t he pr evi ous compar i son

cases. Accor di ng t o t he Rescor l a- Wagner net wor k, i n t he l ar ge

compar i son set , t he r ol e of t hi s cont r ast f act or wi l l be st r ongl y

di scount ed or bl ocked because t he cont ext f act or al r eady f ul l y

pr edi ct s t hi s out come. Thi s can be seen i n t he t op panel of

Fi gur e 4, as mor e di scount i ng of t he cont r ast per son f act or i s

obser ved i n t he l ar ge compar i son set wher e t he cont ext has

pr evi ousl y acqui r ed st r ong causal st r engt h, t han i n t he smal l

comparison set where the context has acquired less strength.

On t he ot her hand, i n t he augment at i on pr ocedur e, a novel

per son or st i mul us cont r ast case i s i nt r oduced wi t h an out come

opposite t o t hat of t he pr ecedi ng compar i son cases. Accor di ng t o

t he Rescor l a- Wagner model , i n t he l ar ge compar i son set , t hi s

out come i s t ot al l y opposi t e t o t hat pr edi ct ed by t he cont ext

f act or so t hat t he cont r ast f act or wi l l acqui r e st r ong negat i ve

st r engt h t o compensat e f or t hi s di scr epancy. As can be seen f r om

t he bot t om panel i n Fi gur e 4, t he negat i ve st r engt h of t he

cont r ast f act or wi l l be mor e augment ed ( i . e. , mor e negat i ve) i n

t he l ar ge compar i son set wher e t he cont ext has acqui r ed a st r ong

causal r ol e, t han i n t he smal l set wher e t he cont ext has acqui r ed

less strength.

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Causal Attribution 20

Probabilistic Predictions

These pr edi ct i ons ar e, however , pr obl emat i c f or pr obabi l i s t i c

t heor y. The r eason i s t hat al l cur r ent pr obabi l i s t i c model s of

causal i t y ( Cheng & Novi ck, 1990; Cheng & Hol yoak, 1995; Van

Over wal l e, 1996a; Wal dmann & Hol yoak, 1992) base t hei r pr edi ct i ons

on a pr obabi l i s t i c cal cul at i on of t he ef f ect , t hat i s , t he

r el at i ve proportion of cause- ef f ect covar i at i on r at her t han i t s

raw f r equency. Consequent l y, al l model s pr edi ct t hat t he

pr obabi l i t y of t he ef f ect gi ven t he cont ext i s t he same whet her

t he number of compar i son cases i s one or f i ve ( i . e. , ∆PX = 1/ 1 and

5/ 5 r espect i vel y f or bot h di scount i ng and augment at i on) , so t hat

t he cont r ast f act or i s expect ed t o r ecei ve t he same causal

st r engt h i n each condi t i on ( see Equat i on 1) . Thus, i ncr easi ng t he

number of compar i son cases shoul d not make any di f f er ence t o

subjects' causal judgments.

Experiments and Results

Subj ect s wer e pr esent ed wi t h di f f er ent st or i es cont ai ni ng

i nf or mat i on about f i ve compar i son per sons or st i mul i i n t he l ar ge

compar i son set , or onl y one compar i son per son or st i mul us i n t he

smal l compar i son set . Thi s i nf or mat i on was f ol l owed by one t ar get

per son or st i mul us wi t h t he same out come ( di scount i ng) or a

opposi t e out come ( augment at i on) . The f ol l owi ng descr i pt i on

i l l ust r at es t he di scount i ng of a per son f act or i n t he l ar ge set

( wi t h t he smal l set bet ween br acket s) : " Fi ve ot her sal esl adi es

[ One ot her sal esl ady] and al so Anni e at t ai ned hi gh sal es f i gur es

f or per f umes. " The next descr i pt i on depi ct s t he augment at i on of a

per son f act or : " Fi ve ot her spor t swomen [ One ot her spor t swoman]

f el l dur i ng t he spr i nt r ace, but Sandr a di d NOT f al l dur i ng t he

spr i nt r ace. " Si mi l ar descr i pt i ons wer e gi ven t o mani pul at e t he

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Causal Attribution 21

discounting and augmentation of a stimulus factor.

I n one condi t i on, t he i nf or mat i on was pr esent ed i n a pr e-

packed summar y f or mat ( as i n t he exampl es above) whi ch capt ur es

some aspect s of peopl e' s ver bal i nt er act i ons wi t h one anot her , and

i n anot her condi t i on t he i nf or mat i on was pr esent ed i n a sequent i al

f or mat ( i . e. , case- after - case) whi ch r ef l ect s peopl e' s i nci dent al

l ear ni ng dur i ng ever yday l i f e. Af t er r eadi ng each st or y, subj ect s

r at ed t he causal i nf l uence of f our f act or s i ncl udi ng t he per son

( i . e. , somet hi ng about Anni e) , t he ext er nal cont ext ( i . e. ,

somet hi ng ext er nal out s i de Anni e) , t he st i mul us ( i . e. , something

about per f umes) and t he gl obal cont ext ( somet hi ng gener al about

toiletry ) , usi ng a 11- poi nt r at i ng scal e r angi ng f r om 0

( absolutely no influence ) to 10 ( very strong influence ).

The most r el evant at t r i but i on r at i ngs ar e l i s t ed i n Tabl e 1.

I n l i ne wi t h t he connect i oni st pr edi ct i ons, i n t he di scount i ng

condi t i on, t he mean at t r i but i ons t o t he per son and t he st i mul us

wer e mor e di scount ed i n t he l ar ge t han t he smal l compar i son set ,

and i n t he augment at i on condi t i on, t hey wer e mor e augment ed i n t he

l ar ge t han smal l compar i son set . Thus, i ncr easi ng t he number of

compar i son cases l ead t o mor e di scount i ng and mor e augment at i on.

Thi s ef f ect was si gni f i cant i n s i x out of ei ght compar i sons, and

most consi st ent i n t he summar y pr esent at i on condi t i on. Over al l ,

t hese r esul t s ar e consi st ent wi t h t he Rescor l a- Wagner net wor k

model, but clearly contradict probabilistic models.

-------------------------

Insert Table 1 about here

-------------------------

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Causal Attribution 22

Model Simulations

I n or der t o assess t he over al l per f or mance of t he t wo model s,

we comput ed si mul at i ons of Van Over wal l e' s ( 1996a) pr obabi l i s t i c

j oi nt model usi ng Equat i ons 1 and 2, and of t he Rescor l a- Wagner

net wor k usi ng Equat i on 3 ( or 4) wi t h wei ght updat es af t er each

t r i al . The i nf or mat i on f r om t he st or i es was encoded i n t he model s

i n exact l y t he same or der and number as pr ovi ded t o our subj ect s.

Gi ven t hat t he Rescor l a- Wagner net wor k has one f r ee par amet er , t he

l ear ni ng r at e βw, we sought t he best f i t f or t hi s model by r unni ng

si mul at i ons f or t he whol e r ange of admi ssi bl e par amet er val ues

( bet ween 0 and 1) , and t hen sel ect ed t he si mul at i on wi t h t he

hi ghest cor r el at i on bet ween si mul at ed and obser ved r esponses ( see

bel ow) . Al t hough t hi s pr ocedur e may r ef l ect some capi t al i zat i on

on chance, t he mer e exi st ence of a f r ee par amet er mi ght be

consi der ed as yet anot her way i n whi ch connect i oni st model s ar e

super i or . To our knowl edge, t her e i s no publ i shed r esear ch i n

whi ch t he Rescor l a- Wagner l ear ni ng r at e par amet er was est i mat ed on

soci al dat a, so t hat an appr opr i at e val ue can onl y be est abl i shed

post hoc. Not e, however , t hat t he r epor t ed best - f i t par amet er

val ues ar e gener al l y qui t e r obust , and t hat devi at i ons of . 10 i n

the parameter values decrease the fit only minimally.

The sequent i al and summar y f or mat s wer e s i mul at ed separ at el y

wi t h i ndependent l ear ni ng r at es βw, because we sur mi sed t hat t he

f or mat i n whi ch i nf or mat i on was pr esent ed dur i ng t he exper i ment

mi ght have af f ect ed t he speed of l ear ni ng of our subj ect s. Tabl e

2 di spl ays t he cor r el at i on R bet ween t he si mul at ed and obser ved

r esponses f or t he cont r ast and cont ext f act or s ( aver aged over al l

subj ect s) . Thi s measur e pr ovi des an i ndex of t he summar y f i t of

t he model s ( Gl uck & Bower , 1988a) . As can be seen, al t hough t he

pr obabi l i s t i c model capt ur ed some var i ance i n t he dat a ( mean R =

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Causal Attribution 23

. 314) , t he Rescor l a- Wagner net wor k model f i t t ed t he dat a much

bet t er ( mean R = . 802) . Cont r ar y t o our suspi c i on, t he l ear ni ng

r at e at t ai ned a hi gh val ue of . 80 i n bot h pr esent at i on f or mat s.

Thi s may suggest t hat t he det ect i on of covar i at i on among soci al

st i mul i occur r ed r el at i vel y f ast r egar dl ess of pr esent at i on mode,

per haps because t he soci al st or i es used i n our r esear ch wer e qui t e

simple and perhaps familiar to our subjects.

-------------------------

Insert Table 2 about here

-------------------------

Configural Attributions

So f ar , we have deal t wi t h s i ngl e cont r ast f act or s and t hei r

cont ext s whi ch make up si ngl e di mensi ons of causal i t y. However ,

i n r eal l i f e, humans ar e most of t en conf r ont ed wi t h a mor e compl ex

s i t uat i on wher e mul t i pl e di mensi ons ar e per cei ved si mul t aneousl y.

As Kel l ey ( 1967) not ed, we do not onl y obser ve and compar e

r egul ar l y wi t h ot her peopl e, but al so wi t h ot her s i t uat i ons or

st i mul i , and wi t h ot her t i me occasi ons. When anal yzi ng

covar i at i on wi t h mul t i pl e di mensi ons, causal i t y can not onl y be

at t r i but ed t o s i ngl e causes, but al so t o t hei r interactions or

configurations . For exampl e, a car acci dent i s of t en due t o a

concur r ence of c i r cumst ances wher e var i ous causal f act or s must be

pr esent ( e. g. , speedi ng, bad weat her , et c. ) t o pr oduce t he ef f ect .

Such i nt er act i ve causes ar e pr edi ct ed by t he pr obabi l i s t i c model

( Cheng & Novi ck, 1990; Van Over wal l e & Heyl i ghen, 1995; Van

Over wal l e, 1996a) , but ar e pr obl emat i c f or t he or i gi nal Rescor l a-

Wagner model . Sever al connect i oni st amendment s t o t he Rescor l a-

Wagner model have been pr oposed t o deal wi t h conf i gur at i ons, but

none wer e par t i cul ar l y el egant or pl ausi bl e ( e. g. , Gl uck & Bower ,

1988b, Van Over wal l e, 1996b) . Recent l y, however , Pear ce ( 1994)

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Causal Attribution 24

devel oped an connect i oni st ext ensi on t o Rescor l a and Wagner ' s

model whi ch deal s qui t e ni cel y wi t h conf i gur at i ons. We wi l l f i r st

di scuss Pear ce' s model and t hen expl or e some r el evant empi r i cal

data.

Pearce's Configural Network Model

Pear ce was i nspi r ed by t he common obser vat i on i n condi t i oni ng

r esear ch t hat ani mal s r espond not onl y t o t he t r ai ni ng cue but

al so t o ot her cues t hat ar e s i mi l ar t o i t , a phenomenon t hat i s

t er med generalization . The mor e t he ot her cues r esembl e t he

t r ai ni ng cue, t he mor e t he or gani sm expect s t he same ef f ect and

r esponds i n a s i mi l ar manner ( Pear ce, 1987, 1994) . Si mi l ar l y,

humans may not onl y at t r i but e causal i t y t o t he t r ue cause t hat

covar i es wi t h t he ef f ect , but al so t o ot her f act or s t hat shar e

s i mi l ar f eat ur es wi t h i t . However , such si mi l ar f act or s, i n

t hemsel ves, do not necessar i l y covar y wi t h t he ef f ect . Al t hough

gener al i zat i on may t hus be qui t e subopt i mal f r om a st at i st i cal

poi nt of v i ew, i t does have adapt i ve val ue i n r eal l i f e. Ther e

may al ways be some doubt about t he cr i t i cal f eat ur es i n t he t r ue

cause t hat pr oduced t he ef f ect . Because t hese cr i t i cal f eat ur es

may be pr esent i n ot her , s i mi l ar f act or s, i t may be advant ageous

t o gener al i ze causal i t y t o t hese f act or s. Hence, gener al i zat i on

pr ovi des t he basi s f or bui l di ng gener i c knowl edge t hat can be

appl i ed i n many mor e si t uat i ons t han t he or i gi nal l ear ni ng

situation.

To addr ess t he phenomenon of gener al i zat i on, Pear ce ( 1994)

pr oposed a connect i oni st net wor k whi ch assumes t hat per cei ver s

st or e i n memor y r epr esent at i ons of conf i gur al exempl ar s.

Exempl ar s r ef l ect t he whol e st i mul us conf i gur at i on as i t i s

encount er ed i n t he envi r onment ( e. g. , a per son i n a par t i cul ar

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Causal Attribution 25

s i t uat i on at a par t i cul ar t i me) r at her t han i sol at ed f act or s

( e. g. , a per son) . Pear ce' s ( 1994) connect i oni st net wor k consi sts

of t hr ee l ayer s, i n whi ch t he exempl ar s ar e r epr esent ed as

conf i gur al nodes si t uat ed at an i nt er medi at e l ayer i n bet ween t he

i nput and out put l ayer . These conf i gur al nodes di f f er f r om

st andar d hi dden nodes i n ot her wel l - known connect i oni st model s

( McCl el l and & Rumel har t , 1988) . An i l l ust r at i on of t he net wor k i s

gi ven i n Fi gur e 5 f or f our possi bl e combi nat i ons of consensus and

di st i nct i veness i nf or mat i on. Assumi ng t hat t he ext er nal ( E) and

gl obal ( G) cont ext f act or s ar e al ways pr esent , t he P* E* S* G

con f i gur al node r ef l ect s t he pr esence of bot h t he t ar get per son

( P) and st i mul us ( S) , P* E* G r ef l ect s t he pr esence of t he t ar get

per son, E* S* G i ndi cat es t he pr esence of t he t ar get st i mul us, and

E*G denotes that both the target person and stimulus are absent.

--------------------------

Insert Figure 5 about here

--------------------------

Pear ce' s ( 1994) net wor k c l osel y f ol l ows t he Rescor l a- Wagner

speci f i cat i ons wi t h r espect t o t he i nput and t ar get act i vat i ons,

t hat i s , act i vat i on i s set t o 1 f or a f act or or ef f ect t hat i s

pr esent , ot her wi se t he act i vat i on i s 0. The i nput act i vat i on t hen

spr eads t o t he conf i gur al nodes i n pr opor t i on t o t hei r s i mi l ar i t y

( see bel ow) . The mor e a conf i gur al node i s s i mi l ar t o t he pat t er n

of i nput nodes, t he mor e i t wi l l be act i vat ed. Hence, act i vat i on

f r om i nput can spr ead t o var i ous ot her conf i gur at i ons and so

i ndi r ect l y t o ot her f act or s due t o t hei r mut ual s i mi l ar i t y r at her

t han act ual covar i at i on wi t h t he ef f ect . Thi s pr oduces t he ef f ect

of generalization.

The si mi l ar i t y bet ween t he i nput and conf i gur al nodes i s

f i xed ( i ndi cat ed i n t he f i gur e by st r ai ght l i nes) and det er mi ned

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Causal Attribution 26

by the number of common factors, as formalized below :

i

Sh

= n

cn

i

nc

nh

∗ [5]

I n t hi s expr essi on, whi ch i s a s i mpl i f i ed ver si on of t he

or i gi nal f or mul a by Pear ce ( 1987, p. 65; 1994, p. 599) , nc i s t he

number of f act or s common t o t he i nput node and t he conf i gur al

node, ni i s t he number of f act or s pr esent i n t he i nput node, and

nh i s t he number of f act or s pr esent i n t he conf i gur al ( hi dden)

node.

Af t er t he conf i gur al nodes have been act i vat ed, t hei r

act i vat i on spr eads t hr ough adj ust abl e connect i ons ( i ndi cat ed by

br oken l i nes) t o t he out put node. As not ed bef or e, due t o

gener al i zat i on, al l conf i gur al nodes wi l l r ecei ve some degr ee of

act i vat i on, and al l of t hem cooper at e i n act i vat i ng t he out put

node. However , onl y t he conf i gur al node whi ch encodes exact l y t he

i nput i nf or mat i on wi l l r ecei ve maxi mum act i vat i on ( because i Sh =

1) . I t i s onl y t he connect i on bet ween t hi s maximally act i vat ed

conf i gur at i on and t he out put t hat i s adj ust ed af t er each t r i al ,

whi l e t he connect i ons bet ween t he ot her conf i gur at i ons r emai n

unchanged. The adj ust ment f ol l ows t he Rescor l a- Wagner l ear ni ng

al gor i t hm ( Equat i on 3) except , of cour se, t hat t he l i near

summat i on of t he i nput nodes ( Σw) i s now r epl aced by t he l i near

summat i on of t he act i vat i on r ecei ved f r om of t he conf i gur al nodes.

Pear ce' s ( 1994) assumpt i on t hat onl y t he connect i on of t he

maxi mal l y act i vat ed conf i gur at i on i s adj ust ed, st ems f r om t he

or i gi nal Rescor l a- Wagner model wher e onl y t he f act or s pr esent wer e

updated.

Li ke i n t he Rescor l a- Wagner model , an appr opr i at e measur e f or

subj ect s ' causal j udgment s i s s i mpl y t he act i vat i on of t he out put

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Causal Attribution 27

node gi ven t hat t he appr opr i at e i nput nodes ar e t ur ned on. Thi s

pr ocedur e wor ks f i ne when onl y one cont r ast - cont ext pai r ( or

causal di mensi on) i s i nvol ved. Usi ng a s i mi l ar l ogi c as Chapman

and Robbi ns ( 1990) , i t can be shown mat hemat i cal l y t hat Pear ce' s

model -- l i ke t he Rescor l a- Wagner model -- wi l l ar r i ve at

probabilistic norms given sufficient learning trials.

However , when mul t i pl e cont r ast - cont ext pai r s ar e i nvol ved,

t hi s pr ocedur e mi ght not be ent i r el y sat i sf act or y f or Pear ce' s

( 1994) net wor k, because t he act i vat i on of one f act or gener al i zes

t o ot her f act or s f r om ot her di mensi ons vi a t he conf i gur al nodes,

so t hat t he st r engt h of a f act or i s conf ounded and cannot be

t est ed i n i sol at i on. One possi bi l i t y t o r emedy t hi s shor t comi ng,

i s by assumi ng t hat when t he i nput nodes ar e t ur ned on f or one

contrast - cont ext di mensi on, t he at t ent i on or sal i ence f or t he

ot her di mensi ons i s dr ast i cal l y at t enuat ed. For exampl e, when t he

per cei ver t est s t he st r engt h of t he P or E f act or ( i . e. , l ocus

di mensi on) , t hen he or she may st r ongl y r educe t he at t ent i on t o

f act or s f r om ot her di mensi ons. Thi s assumpt i on i s consi st ent wi t h

t he common obser vat i on t hat many di mensi ons of causal i t y ar e

r el at i vel y i ndependent ( cf . Wei ner , 1986) , and t hus can be

at t ended t o separ at el y. We r ef er t o t hi s sel ect i ve at t ent i on

mechanism as the attention strength for other dimensions or αo.

The αo mechani sm was i mpl ement ed i n Pear ce' s s i mi l ar i t y

Equat i on 5 as f ol l ows : I f at l east one f act or of a di mensi on i s

pr esent at i nput , t hen t he def aul t at t ent i on ( = 1) i s used t o

est i mat e t he number of t hese f act or s i n Equat i on 5; conver sel y, i f

no f act or of a di mensi on i s pr esent at i nput , t hen t hei r number i s

est i mat ed by t he ( much r educed) αο at t ent i on val ue. A l ow αo

at t ent i on val ue i mpl i es t hat t he s i mi l ar i t y and st r engt h of a

f act or ( e. g. , P) i s i nf l uenced l ess by conf i gur al nodes cont ai ni ng

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Causal Attribution 28

many di f f er ent f act or s ( e. g. , P* E* S* G) and mor e by conf i gur al

nodes cont ai ni ng l i t t l e el se but t hi s f act or ( e. g. , P* E* G) . Thi s

i s t r ue f or bot h cont r ast and cont ext f act or s. I t i s i mpor t ant t o

not e t hat t hi s sel ect i ve at t ent i on mechani sm af f ect s onl y t he

t est i ng phase of t he model , because dur i ng l ear ni ng al l t he

cont ext f act or s ar e assumed t o be pr esent at i nput , so t hat al l

f act or s ar e equal l y act i ve and cont r i but e equal l y t o t he l ear ni ng

of conf i gur at i ons. Gi ven t hat Pear ce ( 1994) al l owed f or var yi ng

i nt ensi t y or at t ent i on i n t he t er ms of hi s s i mi l ar i t y equat i on, i n

t he net wor k s i mul at i ons descr i bed l at er , αo was f r eel y est i mat ed

bet ween 0 and 1. We expect t hat l ow val ues cl ose t o 0 wi l l r esul t

in a better fit of the model.

Other Networks with Hidden Nodes

Kr uschke ( 1992) r ecent l y pr oposed an exempl ar net wor k wi t h a

s i mi l ar i t y gr adi ent ver y s i mi l ar t o Pear ce' s conf i gur al net wor k,

but t hi s net wor k has f al l en out of f avor because i t s pr edi ct i ons

depend t oo much on l ear ni ng or der ( Lewandowsky, 1995) . Comput er

s i mul at i ons wi t h our dat a pr esent ed i n t he next sect i on, al so

showed t hat t hi s net wor k expl ai ned l ess t han hal f of t he var i ance.

Therefore, we will not further discuss this model.

Anot her c l ass of ver y popul ar connect i oni st net wor ks t hat

make use of an i nt er medi at e l ayer ar e back- pr opagat i on net wor ks

( McCl el l and & Rumel har t , 1988) . These net wor ks di f f er , however ,

f r om t he conf i gur al net wor k i n sever al r espect s. Fi r st , t he

i nt er medi at e nodes i n t he conf i gur al net wor k ar e t r anspar ent as

t hey exact l y copy t he i nput pat t er ns, wher eas back- propagation

net wor ks consi st s of nodes t hat ar e hi dden i n t he sense t hat t he

net wor k i t sel f sear ches f or an opt i mal r epr esent at i on wi t hout

di r ect i nt er vent i on f r om t he i nput . Second, t he l i nks bet ween

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Causal Attribution 29

i nput and conf i gur al nodes ar e f i xed and det er mi ned by t hei r

s i mi l ar i t y, wher eas t he connect i ons bet ween i nput and hi dden nodes

i n back- pr opagat i on net wor ks ar e adapt i ve and det er mi ned by t he

di scr epancy bet ween expect ed and act ual ef f ect t hat i s pr opagat ed

f r om out put l ayer t o t he hi dden l ayer , and t hen t o t he i nput

l ayer . Thi r d, and per haps mor e i mpor t ant l y, t he t wo net wor ks wer e

st i mul at ed by a di f f er ent t heor et i cal concer n. Wher eas t he

conf i gur al net wor k i s based on empi r i cal f i ndi ngs of

gener al i zat i on ef f ect s wi t h ani mal s ( Pear ce, 1987) , back-

pr opagat i on net wor ks have been devel oped mai nl y f r om mat hemat i cal

consi der at i ons on how t o pr opagat e t he er r or di scr epancy

ef f i c i ent l y t hr ough t he net wor k ( Rumel har t , Dur bi n, Gol den &

Chauvi n, 1995) . We sur mi se t hat t he st r onger empi r i cal r oot s of

t he conf i gur al net wor k make i t a mor e appr opr i at e candi dat e f or

model i ng causal l ear ni ng t han convent i onal back- propagation

net wor ks. Because t he conf i gur al net wor k has al r eady been t est ed

ext ensi vel y wi t h ani mal s ( see Pear ce, 1994) , we now t ur n t o some

empi r i cal evi dence wi t h human subj ect s t o compar e t he behavi or of

the configural network with the predictions of other networks.

Experiments and Results

We conduct ed t wo exper i ment s ( Van Over wal l e & Van Rooy, 1996)

i n whi ch subj ect s wer e pr esent ed di f f er ent scenar i os r ef l ect i ng

al l possi bl e t wo- by - t wo combi nat i ons of Kel l ey ' s t hr ee covar i at i on

var i abl es. Af t er r eadi ng each scenar i o, t he subj ect s r at ed t he

causal i nf l uence of s i ngl e cont r ast f act or s ( e. g. , somet hi ng about

t he per son, stimulus , or occasion ) , s i ngl e cont ext f act or s ( e. g. ,

somet hi ng ext er nal , global , or stable ) as wel l as combi nat i ons of

t hese f act or s, usi ng a scal e r angi ng f r om 0 ( no cause) t o 10 ( most

compl et e cause). Li ke i n t he pr evi ous exper i ment , t he i nf or mat i on

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Causal Attribution 30

was pr esent ed under a summar y f or mat or a sequent i al t r i al - by -

t r i al f or mat . The pr edi ct i ons of t he t heor et i cal model s under

consi der at i on ar e i l l ust r at ed i n Tabl e 3 f or consensus and

di st i nct i veness, and can be r eadi l y ext ended t o ot her combi nat i ons

of covar i at i on var i abl es. To f aci l i t at e t he di scussi on, under

each i nf or mat i on pat t er n we l i s t ed t he conf i gur al nodes i mpl i ed by

Pear ce' s ( 1994) model t hat wi l l acqui r e t he st r ongest connect i on

weights.

------ -------------------

Insert Table 3 about here

-------------------------

Si nce t he pr obabi l i s t i c model f ai l ed t o pr edi ct t he i mpor t ant

phenomenon of di scount i ng and augment at i on, we t ur n i mmedi at el y t o

t he pr edi ct i ons of Pear ce' s pr edecessor , t he Rescor l a- Wagner

net wor k. As suggest ed bef or e, gi ven suf f i c i ent l ear ni ng t r i al s,

t hi s net wor k wi l l conver ge t o pr obabi l i s t i c pr edi ct i ons, pr ovi ded

t hat t he i nput nodes ar e coded separ at el y f or each cont r ast -

cont ext di mensi on and t hei r i nt er act i ons ( Van Over wal l e, 1996b).

Hence, t he Rescor l a- Wagner net wor k pr edi ct s t hat when t her e i s a

cont r ast i n t he obser vat i ons, t hen at t r i but i ons ar e made t o

cont r ast causes; conver sel y when t her e i s no such cont r ast ,

at t r i but i ons ar e made t o t he cont ext . Appl i ed t o Tabl e 3, t hi s

i mpl i es t hat at t r i but i ons wi l l be made t o t he per son ( P) gi ven l ow

consensus, and t o ext er nal ( E) causes gi ven hi gh consensus; and

si mi l ar l y, t hat at t r i but i ons wi l l be made t o t he st i mul us ( S)

gi ven hi gh di st i nct i veness, and t o gener al ( G) causes gi ven l ow

di st i nct i veness. These pr edi ct i ons can be combi ned t o pr oduce

at t r i but i ons t o i nt er act i ons ( see t op panel ) . I n sum, t he

Rescorla - Wagner model assumes t hat t hese si ngl e f act or s and t hei r

i nt er act i ons wi l l r ecei ve s i gni f i cant positive causal st r engt h,

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Causal Attribution 31

wher eas al l ot her f act or s ( i . e. , not t abl ed i n t he t op panel ) ar e

assumed to have approximately null causal strength.

Pear ce' s conf i gur al net wor k model makes si mi l ar pr edi ct i ons

wi t h r espect t o t he causal f act or s depi ct ed i n t he t op panel of

Tabl e 3, but f or ent i r el y di f f er ent r easons. As expl ai ned bef or e,

t he conf i gur al net wor k pr edi ct s t hat most causal st r engt h wi l l be

acqui r ed by t he conf i gur al node t hat i s al ways f ol l owed by t he

ef f ect ( see t abl e header ) . For i nst ance, hi gh consensus and hi gh

di st i nct i veness i ndi cat es t hat t he cont r ast st i mul us ( S) , t oget her

wi t h t he ext er nal ( E) and gl obal ( G) cont ext s, ar e f ol l owed by t he

ef f ect . Thi s r ei nf or ces most st r ongl y t he connect i on wei ght of

t he E* S* G conf i gur al node. Thi s wei ght wi l l t hen gener al i ze t o

other causes t hat f or m a par t of t he conf i gur at i on ( e. g. , E* S* G

gener al i zes t o E, S & E* S) . Ther ef or e, t hese causes wi l l r ecei ve

subst ant i al positive causal st r engt h. At hi s poi nt , Pear ce' s

conf i gur al net wor k makes ver y s i mi l ar pr edi ct i ons as t he Rescor l a-

Wagner model . I n addi t i on, Pear ce' s net wor k makes t he uni que

pr edi ct i on t hat some amount of t he causal st r engt h of t he

maxi mal l y connect ed conf i gur al node wi l l al so gener al i ze t o ot her

i nt er act i ons t hat ar e not par t of i t , but shar e a f act or ( e. g. ,

E*S* G gener al i zes t o P* G & E* G) . These i nt er act i ons ar e denot ed

generalized causes because t hey do not covar y at al l wi t h t he

ef f ect , but wi l l r ecei ve some causal st r engt h ( see mi ddl e panel ) .

The st r engt h of t hese gener al i zed causes shoul d be st r onger t han

t hat of t he r emai ni ng causes whi ch ar e compl et el y di ssi mi l ar , and

therefore receive null causal strength (see bottom panel).

A summar y of t he r esul t s i s gi ven i n Tabl e 4. As can be

seen, t he r at i ngs pr ovi ded by our subj ect s wer e most consi st ent

wi t h Pear ce' s ( 1994) conf i gur al net wor k. Ther e was st r ong suppor t

f or t he pr edi ct i on, shar ed wi t h t he Rescor l a- Wagner model , t hat

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Causal Attribution 32

t he at t r i but i on r at i ngs f or posi t i ve f act or s ar e hi gher t han f or

gener al i zed and nul l f act or s. Conf or m t o t he uni que pr edi ct i on of

t he conf i gur al net wor k, however , most gener al i zed causes r ecei ved

at t r i but i on r at i ngs t hat wer e subst ant i al l y hi gher t han nul l

causes.

-------------------------

Insert Table 4 about here

-------------------------

Model Simulations

To pr ovi de addi t i onal conf i r mat i on f or t hese r esul t s and t o

t est our pr oposed amendment wi t h r espect t o t he αo par amet er , we

comput ed si mul at i ons of Pear ce' s conf i gur al net wor k and compar ed

t hem wi t h t he pr edi ct i ons of t he Rescor l a- Wagner model . I n

addi t i on, we wi l l al so pr esent s i mul at i ons wi t h t he wi del y used

back - propagation network.

We used t he same pr ocedur e as i n t he ear l i er s i mul at i ons.

The i nf or mat i on pr ovi ded t o t he subj ect s was encoded i n each

net wor k, and connect i on wei ght s wer e updat ed af t er each t r i al .

For t he Rescor l a- Wagner net wor k, t her e wer e separ at e out put nodes

f or each cont r ast - cont ext di mensi on and f or t hei r i nt er act i ons,

wi t h a common t eachi ng val ue and a common βw l ear ni ng r at e

par amet er . Thi s ar chi t ect ur e guar ant ees t hat t he net wor k wi l l

conver ge t owar ds pr obabi l i s t i c nor ms ( Van Over wal l e, 1996b) . For

Pear ce' s conf i gur al net wor k, t he t r i al i nf or mat i on i mpl i ed f our

conf i gur al nodes ( see Tabl e 3) , wi t h a βw l ear ni ng r at e and a αo

sel ect i ve at t ent i on par amet er . For r easons of compar abi l i t y , we

t ook t he same number of hi dden nodes i n t he back- propagation

net wor k, and al so t he same number of par amet er s, i ncl udi ng a βw

l ear ni ng r at e and a αm moment um par amet er . The moment um par amet er

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Causal Attribution 33

r ef l ect s t he ef f ect of t he past wei ght updat e on t he cur r ent

updat e, and so ef f ect i vel y f i l t er s out st r ong osci l l at i ons i n t he

updat es ( McCl el l and & Rumel har t , 1988, p. 136) . Because, as not ed

ear l i er , t he cont ext f act or s act as a k i nd of bi as t er ms, we di d

not i ncl ude addi t i onal bi as wei ght s ( McCl el l and & Rumel har t ,

1988) . As bef or e, f or al l model s, we cal cul at ed al l admi ssi bl e

par amet er s val ues bet ween 0 and 1, and t hen sel ect ed t he val ues

whi ch at t ai ned t he hi ghest cor r el at i on bet ween si mul at ed and

observed data.

Because t he i nf or mat i on i n t he exper i ment s was ei t her

pr esent ed r andoml y ( i n t he sequent i al f or mat ) or wi t hout any

par t i cul ar or der ( i n t he summar y f or mat ) , f or each st or y, we r an

100 si mul at i ons wi t h di f f er ent r andom t r i al or der s2. Tabl e 5

pr esent s t he summar y f i t R f or each model . The r esul t s show t hat

al t hough t he Rescor l a- Wagner net wor k obt ai ned an adequat e f i t

( mean R = . 759) , i t was t he conf i gur al net wor k whi ch r eached a

s l i ght l y bet t er f i t over al l ( mean R = . 793) . The l ear ni ng r at e of

bot h model s was hi ghest f or t he sequent i al pr esent at i on f or mat ,

suggest i ng ( per haps cont r ar y t o i nt ui t i on) t hat t he somewhat mor e

compl ex st i mul us mat er i al i n t he pr esent exper i ment s was l ear ned

most qui ckl y when pr esent ed t r i al - by - t r i al . I n cont r ast , t he

back - pr opagat i on s i mul at i on f ai l ed t o r epr oduce our dat a t o any

reasonable degree ( mean R = .171).

Per haps, t he hi gh f i t of t he conf i gur al model was par t l y due

t o t he i nt r oduct i on of t he novel αo par amet er . As expect ed, t he

est i mat es of t hi s par amet er wer e ver y l ow, conf i r mi ng our i dea

t hat t o t est causal st r engt hs i ndependent l y, t he i nf l uence of

ot her di mensi ons needs t o be cancel ed out . Omi t t i ng t hi s

par amet er so t hat al l di mensi ons wer e equal l y act i vat ed dur i ng

t est i ng, r educed t he R f i t measur e f or t he conf i gur al net wor k by

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Causal Attribution 34

. 086 and . 146 i n t he sequent i al and summar y f or mat r espectively.

However , on t he basi s of t he pr esent r esul t s al one, i t i s

di f f i cul t t o assess t he gener al i t y of our αo sol ut i on f or ot her

material.

Why di d t he back- pr opagat i on model per f or m so poor l y ?

Addi t i onal s i mul at i ons may pr ovi de some hi nt s. When 20 i nst ead of

4 hi dden nodes wer e used, t he f i t di d not i mpr ove. However ,

r epeat i ng t he or i gi nal t r i al i nf or mat i on of each st or y i mpr oved

t he f i t subst ant i al l y , but not t o t he same degr ee as t he ot her

model s ( mean R = . 277 wi t h 10 r epet i t i ons and mean R = . 430 wi t h

100 r epet i t i ons, al l wi t h t he same par amet er val ues of Tabl e 5) .

Al t hough per haps mor e r epet i t i ons or s l i ght l y di f f er ent par amet er

val ues mi ght have accommodat ed t he dat a bet t er , t he model seems

unabl e t o l ear n t he conf i gur at i ons ( and t hei r hi dden

r epr esent at i on) at a r easonabl e speed. Thi s suggest s t hat

Pear ce' s ( 1994) exempl ar r epr esent at i on of hi dden conf i gur at i ons

is crucial in the superior and faster performance of his model.

-------------------------

Insert Table 5 about here

---------------- ---------

Conclusions

The dat a pr esent ed i n t hi s chapt er c l ear l y demonst r at e t hat a

connect i oni st appr oach has much pr omi se f or our under st andi ng of

t he pr ocesses under l y i ng causal r easoni ng. We have shown how t he

Rescorla - Wagner net wor k can easi l y deal wi t h di scount i ng and

augment at i on ef f ect s and how Pear ce' s conf i gur al net wor k pr edi ct s

gener al i zat i on of causal i t y, t wo f i ndi ngs whi ch ar e pr obl emat i c

f or t he or i gi nal covar i at i on pr i nci pl e of Kel l ey ( 1967, 1971) as

wel l as f or a pr obabi l i s t i c concept i on of i t ( Cheng & Novi ck,

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Causal Attribution 35

1990) . Ot her cogni t i ve r esear ch wi t h humans has document ed t hat

t he Rescor l a- Wagner model i s super i or t o t he pr obabi l i s t i c

appr oach i n expl ai ni ng compet i t i on ef f ect s l i ke di scount i ng and

augment at i on ( Baker et al . , 1993; Chapman & Robbi ns, 1990;

Chapman, 1991; Gl uck, & Bower , 1988a, 1988b; Shanks, 1985, 1993,

1995; Val l ée- Tour angeau, Baker & Mer ci er , 1994) , and t her e i s al so

i ncr easi ng evi dence t o suggest t hat ani mal s pr ocess covar i at i on

i nf or mat i on i n conf i gur al uni t s r at her t han el ement al f eat ur es

( f or a r evi ew see Pear ce, 1989, 1994) . On a t heor et i cal l evel ,

t he connect i oni st appr oach may expl ai n how humans ar e capabl e t o

det ect causal r el at i onshi ps whi l e usi ng l i t t l e cogni t i ve sour ces

and ef f or t , so t hat i t pr ovi des a mor e pl ausi bl e account of causal

reasoning during the hurry of everyday social life.

The pr esent connect i oni st appr oach t o at t r i but i on l eaves a

number of unr esol ved i ssues. The pi ct ur e of l ear ni ng t hat emer ges

f r om connect i oni st net wor ks i s of a r at her passi ve pr ocess, i n

whi ch act i vat i ons spr ead aut omat i cal l y and wei ght s ar e adj ust ed

i mmedi at el y. However , humans may al so t ake a mor e act i ve r ol e i n

whi ch t hey consi der var i ous causal hypot heses t hat may expl ai n an

out come. Recent l y, Read ( Read & Mar cus- Newhal l , 1993) pr oposed a

connect i oni st net wor k t o account f or t hi s pr ocess of hypot hesi s

sel ect i on. He suggest ed t hat humans' causal knowl edge i n a

par t i cul ar domai n can be r epr esent ed by a l ar ge net wor k st r uct ur e,

wi t h each node r epr esent i ng a domai n- r el evant causal f act or . Al l

t hese f act or s compet e t o acqui r e some degr ee of act i vat i on, but

onl y t he node wi t h t he hi ghest act i vat i on i s chosen as t he most

pl ausi bl e hypot hesi s. However , because t he connect i ons i n Read' s

net wor k ar e not adapt i ve, mor e wor k needs t o be done t o i nt egr at e

it with the present approach.

Anot her i nt r i gui ng quest i on i s how pr eci sel y connect i ons ar e

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Causal Attribution 36

adj ust ed gi ven summar i zed i nf or mat i on. The connect i oni st appr oach

has no obvi ous means of expl ai ni ng t hi s because t her e ar e no

separ at e t r i al s dur i ng whi ch i nput act i vat i on spr eads t hr ough t he

net wor k and wei ght s ar e adj ust ed. Our s i mul at i ons of t he summar y

dat a wer e, i n f act , car r i ed out by i mposi ng phant om " t r i al s" t o

t he net wor ks. I t i s possi bl e t hat t he pr i mi t i ve mechani sm of

associ at i ve l ear ni ng has been adapt ed dur i ng human evol ut i on f or

t he novel t ask of i nt er pr et i ng ver bal summar y st at ement s, because

nat ur e t ypi cal l y r e- uses subsyst ems t hat ar e al r eady capabl e of

f unct i oni ng on t hei r own ( Beecher , 1988) . One possi bi l i t y i s t hat

humans por t r ay t he summar y i nf or mat i on i n t he f or m of dummy

ent i t i es or ment al model s ( cf . , Johnson- Lai r d, 1983) , whi ch ar e

t hen sequent i al l y anal yzed by an associ at i ve pr ocessor . However ,

t hi s i s mer e specul at i on and we know of no di r ect evi dence t hat

may support this hypothesis.

Leavi ng t hese i nt er est i ng quest i ons asi de, we suspect t hat

t her e i s pot ent i al f or f eedf or war d connect i oni st model s t o expl ai n

even a br oader r ange of soci al phenomena i n whi ch t he det ect i on of

covar i at i on pl ays a r ol e. For i nst ance, t he connectionist

appr oach may pr ovi de an al t er nat i ve account f or some i nt r i gui ng

f i ndi ngs i n gr oup st er eot ypi ng, such as i l l usor y cor r el at i ons

( Hami l t on & Gi f f or d, 1976) . I l l usor y cor r el at i on i s t he r obust

phenomenon t hat per cei ver s j udge mi nor i t y gr oups mor e negat i vel y

t han maj or i t y gr oups even when t he pr opor t i on of t hei r posi t i ve

and negat i ve behavi or s i s i dent i cal ( e. g. , t wi ce as much posi t i ve

t o negat i ve behavi or s) . As not ed ear l i er , connect i oni st model s

concei ve of l ear ni ng as a gr adual pr ocess by whi ch t he connect i on

wei ght s i ncr ement al l y i ncor por at e new i ncomi ng i nf or mat i on. Let

us assume t hat t he posi t i ve and negat i ve behavi or s ar e encoded i n

t wo i nput nodes, connect ed vi a modi f i abl e wei ght s wi t h t wo out put

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Causal Attribution 37

nodes r epr esent i ng t he maj or i t y and mi nor i t y gr oup. Because t her e

i s much more behavi or al i nf or mat i on about t he majority gr oup t han

about t he min or i t y gr oup, and because l ear ni ng occur s

i ncr ement al l y, t he connect i ons bet ween t he behavi or al i nput nodes

and t he maj or i t y out put node wi l l gr ow much strong er t han t hose of

t he min or i t y out put node. I n f act , t he maj or i t y ' s connect i ons

wi l l al most r each asympt ot e, t hat i s , t hey wi l l r each wei ght s t hat

r ef l ect t he t r ue pr opor t i on of pos i t i ve and negat i ve behaviors .

I n cont r ast , t he mi nor i t y ' s connect i ons wi l l r each onl y weak pr e-

asymptotic wei ght s, so t hat any di f f er ence bet ween t he per cei ved

strength of posi t i ve and negat i ve behavi or s i s mi ni mal and

insignificant . Thi s r esul t , whi ch can be easi l y s i mul at ed wi t h a

s i mpl e two - la yer f eedf or war d network ( McCl el l and & Rumel hart,

1988) , may be r esponsi bl e f or per cei ver s ' bi ased per cept i on of the

minority group as being l ess positive.

Gi ven t hat connect i oni st model s ar e an i deal i zed r ef l ect i on

of t he neur al wor ki ngs of t he human br ai n, we suspect t hat t hey

wi l l per haps i ncr ease our under st andi ng of ot her causal phenomena,

such as how per cei ver s i nf er di sposi t i onal at t r i but i ons about

ot her per sons usi ng covar i at i on i nf or mat i on ( cf . , Hi l t on, Smi t h &

Ki m, 1995) , and how peopl e f al l pr ey t o ot her causal i l l usi ons,

such as t he cor r espondence bi as ( Gi l ber t , 1989) . We suspect t hat

t hese and many ot her phenomena i n soci al r easoni ng ar e not so much

a t r i cky r esul t of our soci al per cept i ons, societal r ul es, or of

t he demandi ng ci r cumst ances of ever yday soci al l i f e, but s i mpl y

the outcome of a connectionist processing mechanism .

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Causal Attribution 38

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Causal Attribution 43

press .

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Manuscript submitted for publication

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Causal Attribution 44

Table 1.

Mean Attribution ratings per Factor and Condition

Discounting Augmentatio n

Large Set Small Set Large Set Small Set

Sequential Presentation

Person 3.07 < 5.33 8.87 > 7.30

Stimulus 3.13 3.57 7.67 7.23

Summary Presentation

Person 4.03 < 7.67 8.63 > 5.93

Stimulus 2.67 < 5.90 7.90 > 6.03

Note. Significant differences ( p<0.5) are indicated by > or <.

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Causal Attribution 45 Table 2. Fits of the Models to the Discounting and Augmentation Data Model R Model Parameter

Sequential Presentation

Probabilistic Joint .358

Rescorla - Wagner .854 βw = .80

Summarized Presentation

Probabilistic Joint .270

Rescorla - Wagner .751 βw = .80

Note . R = correlation, βw = learning rate parameter.

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Causal Attribution 46

Table 3.

Causal Strength predictions of Rescorla - Wagner's and Pearce's

Network illustrated for Consensus and Distinctiveness.

Consensus High Low Distinctiveness Low High Low High Configural nodes a E*G E*S*G P*E*G P*E*S*G

Causal Type Positive E*G E*S P*G P*S E E P P G S G S Generalized b P*G P*S P*S P*G E*S E*G E*G E*S Null P*S P*G E*S E*G P P E E S G S G

Note . P = person, E = external, S = stimulus, G = general. a Conf i gur al nodes i n Pear ce' s model that will acquire the

st r ongest wei ght s. b Nul l st r engt h predicted by Rescorla &

Wagner, generalized strength predicted by Pearce.

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Causal Attribution 47

Table 4.

Mean Causal Ratings in function of Causal Strength Type.

Positive Generalized Null

Presentation Sequential 5.46 3.87 2.90 Summary 6.84 4.17 2.82

Note . Means di f f er s i gni f i cant l y bet ween al l t hr ee causal

types ( p<.002)

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Causal Attribution 48

Table 5. Fits of the Models to the Attribution Data Model R Model Parameters

Sequential Presentation

Rescorla - Wagner .706 βw = .62

Configural .742 βw = 1.00; αo = .13

Back - Propagation .152 βw = .81; αm = .52

Summarized Presentation

Rescorla - Wagner .812 βw = .41

Configural .844 βw = .42; αo = .03

Back - Propagation .191 βw = .88; αm = .99

Note . R = maxi mum cor r el at i on; Model par amet er s ar e : βw =

l ear ni ng r at e; αo = at t ent i on f or ot her di mensi ons not pr esent

at i nput ; αm = moment um. Ther e wer e 3 compar i son t r i al s i n t he

sequent i al f or mat ; t hei r number i n t he summar y f or mat was

simulated as 1/5 (see footnote 2).

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Causal Attribution 49

Figure Captions

Figure 1. A cont i ngency t abl e i l l ust r at i ng a cont r ast f act or ( C)

and i t s cont ext f act or ( X) when t he ef f ect i s pr esent and

absent. The letters a - d reflect frequencies.

Figure 2 . A net wor k r epr esent at i on of t he Rescor l a- Wagner model

gi ven a cont r ast f act or ( C) and i t s cont ext ( X) , t oget her

wi t h some i l l ust r at i ve codi ng f or i nf or mat i on pr esent ed

during learning and questions presented during testing.

Figure 3 . A s i mul at i on of causal l ear ni ng wi t h par amet er βw = . 50.

The l ef t panel r ef l ect s a l ear ni ng hi st or y of CX → Effect

and X → No Ef f ect t r i al s; wher eas t he r i ght panel r ef l ect s

X → Effect and CX → No Effect trials.

Figure 4 . A s i mul at i on of di scount i ng and augment at i on wi t h

par amet er βw = . 50, i l l ust r at ed f or a per son ( P) cause and

i t s ext er nal ( E) cont ext . The condi t i ons i nvol ved f i ve

( l ar ge set ) or one ( smal l set ) E → Ef f ect t r i al s, f ol l owed

by one PE → Ef f ect t r i al i n t he di scount i ng condi t i on, or

one PE → No Effect trial in the augmentation condition.

Figure 5 . The conf i gur al net wor k pr oposed by Pear ce ( 1994) ,

illustrated for four factors P, E, S and G.

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Figure 1

C X

X

Effect

No Effect

a b

c d

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Figure 2

Out put Layer

I nput Layer C X

Information coding : C =X =

Question coding : C =X =

10

10

11

01

Associative links

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Figure 3

-1

0

1

0 1 2 3 4 5 6

Trials

Wei

gh

t

X

C

-1

0

1

0 1 2 3 4 5 6

Trials

C

X

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Figure 4

Discounting

Large Set

0

1

0 1 2 3 4 5 6

Trials

Str

eng

ht

P

E

Small Set

0

1

0 1 2

Trials

P

E

Augmentation

Large Set

-1

0

1

0 1 2 3 4 5 6

Trials

Str

eng

ht

P

E

Small Set

-1

0

1

0 1 2

Trials

P

E

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Figure 5

Out put Layer

Conf i gur al Layer

Information coding : PS =ES =

10

11

11

11

Question coding :

10

01

11

P =E =

10

01

00

00

00

PS =ES =

PESG

P E S G

PEG EGESG

I nput Layer

Associative links

Similarity links

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Causal Attribution

Footnotes

1 Thi s pr obabi l i s t i c r ul e i s al so t er med the delta - P rule. To

avoi d any conf usi on wi t h t he delta l ear ni ng al gor i t hm f r om

connectionist learning models, we will not use this term.

2 Gi ven t hat t he amount of compar i son cases was not speci f i ed

i n t he summar y f or mat , t hei r number was est i mat ed by r unni ng

si mul at i ons of t he pr obabi l i s t i c j oi nt model wi t h di f f erent

wei ght s f or t he f r equenci es of t he compar i son cases. The hi ghest

f i t was obt ai ned when t he compar i son cases r ecei ved one f i f t h of

t he wei ght of t he t ar get cases. The si mul at i ons wi t h t he

connect i oni st model s wer e t hen car r i ed out wi t h t he number of

t r i al s of compar i son and t ar get cases adj ust ed t o t hat same

proportion, that is, five target trials for each comparison trial.

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