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NeuroPathicOsteoArthropathy
5-Year Mortality Rates
Armstrong et al. Int Wound J. 2007;Dec;4(4):286. CA = Carcinoma.PAD = Peripheral artery disease.
Hysteria and Neuropathic Feet - Christopher Hendrix
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Learning Objectives
Note differences between sensory, motor, and autonomic neuropathy
Impact each has in towards the development of Charcot Foot
Identify 3 components of an effective management plan
William Musgrave
British Physician and Antiquary 1655 – 1721 Practiced in Exeter Writings included topics of arthritis Credited with first reference to
Neuroarthrosis
Jean-Martin CharcotBorn 1825 – Paris, France29 November 1825 – 16 August 1893
Founder of modern neurology
Salpetriere Hospital
Recognized neurological changes
Described “Pied Tabe’tique”
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Paul Wilson Brand, MD•July 17, 1914 - July 8, 2003
•Missionary Physician in India
•Recognized the effects of loss of sensation on hands•and feet of patients with Hansens Disease
•Advanced surgical techniques
•Total Contact Casting
•“The Gift of Pain”
CHARCOT Who’s Who in this room ?
FreudBabinskiToothTouretteMarie
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“Eichenholz Classification” of
Disease Progression
STAGE 0 - ZERO
What’s the Diagnosis ???
CHARCOT
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Charcot Foot
Charcot FOOT
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“Eichenholz Classification” of
Disease Progression
•Stage 1 – destruction •Stage 2 – coalescence •Stage 3 – consolidation
Brodsky Classification of Disease LOCATION
Type ITarsal-Metatarsal Jt.Lesser Tarsus
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Type IIPeri-Talar
Type IIIaAnkle – TibioTalarType IIIbPosterior OsCalcis
Type IIIaAnkle – TibioTalarType IIIbPosterior OsCalcis
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Trepman-Brodsky Type IV and Type V
Multiple sites and forefoot
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CHARCOT
EVERY 30 SecondsA lower limb is lost due to the effects of Diabetessomewhere in the world
-International Diabetes Foundation
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Assessment
ADA Recommendations
“All individuals with diabetes should receive an annual foot examination to identify high-risk foot conditions.”
American Diabetes Association: Preventive foot care in people with diabetes. Diabetes Care: 26(1) S78-S79, January, 2003.
Neurological ExamSensory Neuropathy
Semmes-Weinstein monofilament 5.07-applies 10 grams of pressure.
Detects protective sensation. Patients C/O burning, tingling, numbness
in feet.
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Neurological ExamMotor Neuropathy Causes weakness of the intrinsic
musculature of the foot. Over-powering of extrinsic musculature. Leads to dorsal contraction of digits
(hammer toes). Causes “slapping” of the foot during gait. Creates areas of increased plantar pressure,
especially along the metatarsal heads.
Neurological ExamSensory Neuropathy Loss of Protective Threshold Loss of Vibratory Sense Loss of Proprioception Profound Anesthesia Places Patient at Risk for Injury from
Chronic Micro Repetitive Traumas w/o Rocognition
Neurological ExamAutonomic Neuropathy Disturbances in the sympathetic nervous
system. Absence of sweat production, causing dry,
scaly skin. Alters vascular perfusion, increasing risk
for Charcot Arthropathy.
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Semmes-Weinstein Monofilament
Simple Sensory Testing
Alcohol pad – cool sensationCotton wisp – light touchSW monofilament - protective
Semmes-Weinstein Monofilament
5.07 monofilament10g force
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Mechanical
“High Pressure Areas”
Neuropathic
Musculoskeletal ExamOsseous deformities
Bunions Hammer Toes Bony Prominences Pes Cavus/Planus
Hallux Abducto Valgus(Bunion Deformity)
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Osseous Deformities
NEUROARTHROPATHY
SPONTANEOUS PATTERNS OF FRACTURE AND DISLOCATION WITH A PREDILECTION FOR THE MIDFOOT
SEEN ONLY WITH SENSORY NEUROPATHY
DIABETES, ETOH, HEAVY METALS, LUETIC, IDIOPATHIC
Charcot NeuroarthropathyIncidence
An acute event will only develop in the presence of neuropathy
Neuroarthropathy will not occur in ischemic feet
13% of diabetics with sensory neuropathy will develop an event
80% of events affect Lisfranc’s joint or the midtarsal joint
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Charcot NeuropathyPathogenesis
Tarsal prominence Reversal of Arch Intrinsic Atrophy Pull of the triceps Inadequate ability to
resolve midfoot shear
CHARCOT
CHARCOT
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The Diagnosis of Acute Charcot Neuroarthropathy
High index of suspicion!!!!
Unilateral edema Unilateral warmth Elevated ESR, alkaline
phosphatase XRays may not be
helpful if fragmentation has not yet occurred
“Eichenholz Classification” of
Disease Progression
STAGE 0 - ZERO
What’s the Diagnosis ???
Treatment of Acute Charcot Neuroarthropathy
The best treatment is early recognition and immobilization in a non WB cast
The incidence of acute Charcot in the other extremity during the period of treatment is significant (25-30%)
Protect contralateral limb Patients should be
confined to a wheelchair until consolidated
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Treatment of acute Charcot events
Elevation Rest Compression Immobilization Accomodation with
molded shoes, bracing, etc
Surgical Intervention?
WHY TREAT? The bones are soft,
fractured, and displaced
The patient is neuropathic
The pt. may be anemic, may have poor vision, poor circulation, etc.
Medical management of acute Charcot events
Jude et al.Bisphosphonates in the treatment of Charcot neuroarthropathy: a double-blind randomised controlled trial. (N=39)
Level I evidence
Diabetologia. 2001 Nov;44(11):2032-7
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CharcotDeformity
Off-load with a CROW Prosthesis
Surgical Salvage/Reconstruction
Surgical Intervention
Osseous Resection Tendon Transfer Charcot Reconstruction
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Charcot Arthropathy:“Diabetic Patient’s Worst Nightmare!”
“There is nothing we can do for you”
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Very Unstable!
Goals of Surgery
Before & After
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CHARCOT-Long Term Bracing AFO Brace on Shoe CROW PTB Amputation
Total Contact Casting
Have to Control Swelling
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Recommendations…..
Final ThoughtsThorough evaluation of patientThorough evaluation of neuropathyEtiology of NeuropathySelection of Treatment ModalitiesDynamic CareProactiveProtectivePreventitive
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Patient Cases
Patient Case
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