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IntroductionPostoperative complications are the most
important factors in determining outcome in the first 72 hours following surgery
It is critical to monitor basic physiological parameters such as renal, cardiovascular and respiratory functions
Postoperative ordersVital signsDiet: NPO until bowel sounds presentIntravenous fluidsCare of drainsInput and output chartPain medication: dose and route of
administrationAntibiotics
Postoperative ordersVenous thrombosis prophylaxisOther medications
Postoperative vascular complicationsVenous thromboembolism (VTE) includes
DVT and PE and are a major but preventable cause of morbidity and mortality
Pulmonary embolismHas few definite symptomsBut onset of respiratory distress with
hypotension, chest pain and cardiac arrhythmias may be harbingers of impending death
Can convert a successful operation into a postoperative fatality
Prevention of VTEUnfractionated heparinLMWHGraduated compression stockingsIntermittent pneumatic compression
stockings
Risk factors of postop thrombosisVirchow’s triad: hypercoagulability, stasis,
trauma to vessels
Diagnosis of VTEVenographyImpedence plethysmographyDoppler ultrasoundMRI/MRI Venography
Treatment of VTEUFHLMWH
Postoperative pulmonary complicationsAtelectasisPneumoniaRespiratory failurePulmonary thromboembolic disease
Risk factors for PPCsAge > 60 yearsCancerCongestive cardiac failureSmoking (within 8 weeks of surgery)Upper abdominal incisionVertical incisionIncision length > 20cm
AtelectasisDefinition not uniform in clinical studiesGenerally accepted criteria include:
impaired oxygenation in a clinical setting where atelectasis is likely
Unexplained fever > 38oCCXR evidence of volume loss or new airspace
opacity
Risk factors for atelectasisAdvanced ageObesityIntraperitoneal atelectasisProlonged anaesthesia timeNG tube placementSmoking
Prevention of atelectasisCessation of smoking (6-8 weeks before
surgery)Laporoscopic procedureDeep breathing exercisesMobilizationAdequate analgesia (epidural or PCA
preferred)Selective gastric decompression
Postoperative pneumoniaHospital-acquired pneumonia (HAP) is
pneumonia that develops 48 hours or more after hospital admission because of an organism that was not incubating at the time of hospitalization
HAP after abdominal surgery increases mortality, hospital stay and hospital charges
Caused by a wide range of bacteria. Also by viruses and fungi in immunosuppressed patients
Pathogens causing early onset (<4 days) HAPStrep pneumoniaMS Staph aureusH influenzaE coliK pneumoniaEnterobacter spp.Proteus spp.Serratia marcescens
Pathogens causing late onset (>5 days) HAPPseudomonas aeruginosaMDR K pneumoniaAcinetobacter spp
HAP: clinical definitionNew opacity on CXR( PA and lat views
preferred) plus 2 of the following:Fever >38oCLeukocytosis or leukopeniaPurulent respiratory secretions
Diagnosis should be supported by sample of lower resp tract secretions-bronchoscopy
HAP treatmentInitial therapy should be given IVCombination therapy for those at risk of MDR
pathogensMonotherapy for those at low risk of MDR
Respiratory failureDef: inability to maintain normal tissue
oxygen transport or the normal excretion of carbon dioxide
Arterial PO2 < 60 mmHg or arterial PCO2 > 45 mmHg generally indicate significant respiratory compromise
Generally managed in ICU including endotracheal intubation
Postoperative care of the urinary bladderMost common postop problem of female bladder
is atony caused by overdistension and reluctance of the patient to initiate the voluntary phase of voiding
Urethral or suprapubic catheter is used7-10 days postop postvoid residuals are
evaluatedIf >100ml catheterization duration is extendedOnce residuals are less than 100ml on 2
successive voidings of >200ml catheters can be removed
Postoperative GIT managenentAdvancing of diets should be individualizedPatients with uncomplicated surgery may be
given a regular diet on the 1st POD if bowel sounds are present, if abdominal exam reveals no distention and patient is not nauseated from anaesthesia
Seriously ill patients may reuire TPN