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The Cardiovascular
System
Ms. Louradel Ulbata-Alfonso,MAN, RN
Heart • A hollow muscular organ• Located in thorax between
2 lungs• 4 chambers• 2 atria (atrium) & 2
ventricles• 4 valves• 2 separate pumps (R & L
sides)• Right side receives blood
from the body and sends it to the lungs (pulmonary)
• Left side receives blood from lungs and sends it to the body (systemic)
• PMI –midclavicular line @ 5th IC space
Position of Heart
3 LAYERS:
1. ENDOCARDIUM2. MYOCARDIUM3. EPICARDIUM
Layers of the HeartLayers of the Heart
Pericardium
Myocardium
Fibrous pericardiumSerous pericardium (parietal layer)
Pericardial spaceSerous pericardium (visceral layer - Epicardium)
Endocardium
• The heart lies between the lungs in a region called the mediastinum.
• heart is wrapped by some membranes that also hold the heart in its position relative to the diaphragm and lungs.
The PericardiumThe pericardium is the set of membranes
around the heart. It is actually composed of three layers of membranes.
• visceral pericardium - the innermost• parietal pericardium - the middle, • fibrous pericardium - the outer one is the
extra one, and is tough.
•Pericardial Cavity- tiny space between the visceral pericardium and the parietal pericardium •15-50 ml•Pericardial Effusion VS Cardiac Tamponade
Layers of the HeartLayers of the Heart
Pericardium
Myocardium
Fibrous pericardiumSerous pericardium (parietal layer)
Pericardial spaceSerous pericardium (visceral layer - Epicardium)
Endocardium
CORONARY ARTERIES• L & R coronary arteries –supply arterial
blood to the heart• Originate from the aorta above the aortic
valve leaflets• Perfused during diastole• ↑ HR → ↓ Blood flow → myocardial
ischemia• Heart has high metabolic requirements
(extract 70-80% of the oxygen delivered)
LEFT CORONARY ARTERIES• 3 BRANCHES
• 1. Left main coronary artery• 2. Left anterior descending artery –
anterior wall of the heart• 3. Circumflex artery – circles around to the
lateral left wall of the heart
CORONARY ARTERIES
RIGHT CORONARY ARTERY• Right coronary artery
- inferior wall of the heart
• Posterior descending artery- posterior wall of the heart
- Drains into the coronary sinus
CORONARY ARTERIES
Coronary Arteries
Atherosclerosis is an accumulationof fat on the inner walls of arteries.
When coronary arteries become partially blocked….angina
When coronary arteries becomesignificantly blocked….myocardialinfarction
Chambers of the Heart
Valves of the Heart• 4 valves• One way flow• Leaky valve = heart murmur• 2 atrioventricular valves
– Left AV valve- bicuspid or mitral– Right AV valve- tricuspid
• 2 semilunar valves– Pulmonic semilunar valve– Aortic semilunar valve
Valves of the Heart
Tricuspid Valve
Mitral Valve
Pulmonic Valve
Aortic Valve
Atrioventricular Valves1. Right AV valve• Between right atrium and right ventricle• Also called the tricuspid valve because it has three
cusps.• Cusps close when right ventricle
contracts….preventing blood from going back up into the right atrium
2. Left AV valve• Between the left atrium and the left ventricle• Also called the bicuspid valve because it only has
two cusps• Also called the mitral valve• Cusps close when left ventricle contracts….preventing
blood from back up into the left atrium
Semilunar Valves1. Pulmonary semilunar valve• When right ventricle contracts, blood is forced through
this valve to enter pulmonary trunk2. Aortic semilunar valve• When left ventricle contracts, blood is forced through
this valve to enter the aorta
• Papillary muscles – located on the sides of the ventricular walls and and connected to the valve leaflets by thin fibrous bands called CHORDAE TENDINAE
CHORDAE TENDINAE
2 Pumps
Blood flow in the Heart
CARDIAC ELECTROPHYSIOLOGY
Cardiac Conduction System
The cardiac conduction system generates and transmits impulses that stimulate contraction of the myocardium.
Under normal circumstances, the conduction system first stimulate the contraction of the atria and then the ventricles.
CONDUCTION SYSTEM
SA NODE- main regulator of HR - transmit impulse to the surrounding atrial
muscle.AV NODE- transmit impulses to the
surrounding ventricular muscle.BUNDLE OF HIS- continuation of AV Node
and has a left and right bundles and fuse with purkinje fibers.
PURKINJE FIBERS- terminal branches of the conduction system and are responsible for carrying the wave of depolarization to both ventricular walls.04/28/23
04/28/23
2 TYPES OF SPECIALIZED ELECTRICAL CELLS IN THE HEART• 1. NODAL CELLS• 2. PURKINJE CELLS
• These provide synchronization
ELECTRICAL CELLS
• Nodal Cells – SA and AV nodes• Purkinje Cells – Purkinje Fibers
3 Physiologic Properties of the Electrical Cells
Excitability. The ability of the heart to depolarize in response to a stimulus. Once stimulated, the whole heart muscle contracts. It is influenced by hormones, electrolytes, nutrition, oxygen supply, medications, infection, and nerve characteristics.- “ The ability to RESPOND”
Automaticity/Rhythmicity. The ability of cardiac cells to initiate an impulse spontaneously and repetitively, without external neurohormonal control.
Conductivity. The ability of the heart muscle fibers to propagate/ transmit electrical impulses along and across cell membranes.
CONDUCTION SYSTEM OF THE HEART
• SA NODE (Primary pacemaker)↓
AV NODE ↓
Bundle of his ┴
R bundle L bundle branch branch
┬
PURKINJE FIBERS↓
VENTRICULAR CONTRACTIONS
Conduction System of the Heart
CARDIAC ELECTROPHYSIOLOGY
CARDIAC ELECTROPHYSIOLOGY
CARDIAC ACTION POTENTIAL• IONS1. Sodium2. Potassium3. Calcium
3 PHASES1. Resting membrane state2. Depolarization3. Repolarization
REFRACTORY PERIODS• Myocardial cells must COMPLETELY
repolarize (rest) before they can depolarize/ stimulated again
• 2 PHASES:1. Effective/ Absolute refractory period2. Relative refractory period
ABSOLUTE REFRACTORY• Cell is COMPLETELY UNRESPONSIVE
to any electrical impulse• It is INCAPABLE of initiating early
depolarization
RELATIVE REFRACTORY• If an electrical stimulus is stronger than
normal, the cell may depolarize early or prematurely
• Early depolarization of atrium or ventricle → premature contractions → DYSRHYTHMIAS
CARDIAC HEMODYNAMICS• Principle that fluids flow from a region of
higher pressure to a region of lower pressure
Cardiac Cycle- refers to the events of one complete heart beat. The length of the cardiac cycle is usually about 0.8 sec.
• Systole (contraction of the muscle)- there is ventricular pumping, the chambers of the heart become smaller as the blood is ejected. Occurs secondary to depolarization of cells
• Diastole (relaxation of the muscle)- there
is ventricular filling, the heart chambers fill with blood in preparation for subsequent ejection.
Cardiac Output
• Volume of blood ejected per minute– Each ventricle ejects approximately 70mL of
blood/ beat
• Averages between 4-8L/min• CO = Stroke volume X heart rate =70 ml X 60 beats/min =4,200 ml/min
Changes in SV and HR affects cardiac output
Controls of the heart
1. AUTONOMIC NS- sympathetic and parasym
2. CNS and baroreceptors and chemoreceptors
Baroreceptors- aortic arch and R & L internal carotid arteries- sensitive to changes in BP
Chemoreceptors- sensitive to CO2 level
Subdivisions of ANS
• Parasympathetic – acetylcholine produces inhibitory response
• Sympathetic – catecholamines stimulate– Increase heart rate – Beta 1 receptors– Dilate smooth muscles – Beta 2 receptors– Vasoconstrict vessels – Alpha receptors
Stroke Volume Is Determined By Three Factors
• Preload• Afterload• Contractility
Preload• Degree of stretch of myocardial fibers• Determined by the volume of blood in left
ventricle (LV) at end of diastole• Increased blood volume – increased
preload- increased cardiac output (CO)• Decreased blood volume – decreased
preload – decreased cardiac output (CO)
Frank- Starling Law-the critical factor controlling stroke volume is how much the cardiac muscle cells are stretched just before the contract. The more they are stretched, the stronger the contraction will be. The important factor stretching the heart muscles is the venous return.
Factors Which Increase Preload
• IV fluids• Blood• Vasoconstriction
Factors Which Decrease Preload
• Diuretics• Dehydration• Hemorrhage• Vasodilation
Afterload
• Resistance or pressure the ventricles must overcome to pump blood out
• Left ventricle (LV) affected by systemic vascular resistance (SVR)
• Right ventricle (RV) affected by pulmonary vascular resistance (PVR)
• Related to arterial pressure or diameter of arteries
• As pressure increases, resistance increases, afterload increases
• As pressure decreases, resistance decreases, afterload decreases
Contractility
• Force generated by the myocardium when it contracts – inotropic property
END