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SPINAL
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PathophysiologyNormal Spinal CordSpinal cord begins at the foramen magnum in the craniumCord ends at the L1-L2 vertebra levelSpinal nerves continue to the last sacral vertebraThe Human Spine
Spinal CordGray matter- cell bodies of voluntary and autonomic motor neurons
White matter axons of ascending and descending motor fibers
Normal Spinal CordWhite tracts send messages to and from the brainAscending Tracts- carry into higher levels of CNS touch, deep pressure,vibration, position, temperatureDescending Tractsimpulses for voluntary muscle movement
Pyramidal-Voluntary movementsPosterior column (Dorsal)- touch, proprioception, and vibration senseLateral spinothalamic tract- pain and temperature sensation (only tract that crosses within the cord)voluntary movement
Upper Motor NeuronsUMN Originate in cerebral cortexProject downwardResult in skeletal muscle movementInjury = SPASTIC paralysis
Lower Motor NeuronsLMNOriginate at each vertebral levelProject to specific parts of the bodyResult in movement /sensationInjury = FLACCID paralysis
Normal Spinal CordReflex Arc Involuntary response to a stimulus
Where sensory and motor nerves arise from cordSensory fibers enter posteriorSynapse in the grey matterMotor fibers leave anterior Once outside cord join form spinal nerve
reflex movement
Normal Spinal Cord DermatonesSkin innervated by sensory spinal nervesMyotome- muscle group innervated by motor neurons
Nervous System and the Spinal CordANS can be affected by SCISympathetic chains on both sides of the spinal column (T1-L2)Parasympathetic nervous system is the cranial-sacral branch (brainstem, S2-4)
Spinal Cord ProtectionBones- vertebral column7 Cervical12 Thoracic5- Lumbar5- Sacral
Discs- between vertebra
Spinal Cord ProtectionInternal and external ligamentsDuraMeningesCSF in subarachnoid space allow for movement within spinal canal
Etiology of Traumatic SCIMVA- most common causeOther: falls, violence, sport injuriesSCI typically occurs from indirect injury from vertebral bones compressing cord SCI frequently occur with head injuriesCord injury may be caused by direct trauma from knives, bullets, etc
Etiology of Traumatic SCI78% people with SCI are maleTypically young men 16-30Number of older adults rising (>61 yr)Greater complicationsLife Expectancy 5 years less than same age without injury90% go home
Spinal Cord Injury- SCICompressionInterruption of blood supplyTractionPenetrating Trauma
Spinal Cord InjuryPrimaryInitial mechanism of injurySecondaryOngoing progressive damageIschemiaHypoxiaMicrohemorrhageEdema
Spinal Cord InjuryHemorrhage and edema occur in the cord post injury, causing more damage to cord
Extension of the cord injury from cord edema can occur over the first few dayswatch the phrenic nerve!
Initially SCI experience spinal shock depression of all cord & ANS function below injury. Lasts from few min to wks
Spinal and Neurogenic ShockSpinal ShockDecreased reflexes and loss of sensation below the level of injuryMotor loss- flaccid paralysis below level injurySensory loss- loss touch, pressure, temperature pain and proprioception perception below injuryLasts days to months
Spinal and Neurogenic ShockNeurogenic shockDue to loss of vasomotor toneSNS loss results in parasympathetic dominance with vasomotor failureLoss of SNS innervation causes peripheral pooling and decreased cardiac output Hypotension and Bradycardia Orthostatic hypotension and poor temperature control (poikilothermic)
How do you know spinal shock is over?
Clonus is one of the first signs Hyperreflexia of footTest by flexing leg at knee & quickly dorsiflex the footRhythmic oscillations of foot against handclonus
Classifications of SCIMechanism of InjurySkeletal and Neurologic LevelCompleteness (degree) of Injury
Mechanism of InjuryFlexionHyperextension CompressionFlexion /Rotation
Classifications of SCIMechanism of Injury
Flexion (hyperflexion)Most common because of natural protection position. Generally cause neck to be unstable because stretching of ligaments
Classifications of SCIMechanism of Injury
HyperextentionCaused by chin hitting a surface area, such as dashboard or bathtubUsually causes central cord syndrome symptoms
Classifications of SCIMechanism of Injury
CompressionCaused by force from above, as hit on headOr from below as landing on buttUsually affects the lumbar region
Classifications of SCIMechanism of Injury
Flexion/RoatationMost unstableResults in tearing of ligamentous structures that normally stabilize the spineUsually results in serious neurologic deficits
Skeletal levelVertebral level where the most damage to the bonesNeurologic levelThe lowest segment of the spinal cord with normal sensory and motor function on both sides of the body
Levels of Function in Spinal Cord Injury
Classification of SCI- Level of InjurySpinal cord levelWhen referring to spinal cord injury, it is the reflex arc level (neurologic)not the vertebral or bone level.
the thoracic, lumbar & sacral reflex arcs are higher than where the spinal nerves actually leave through the opening of vertebral bone
Classifications of SCICompleteness (Degree) of Injury
CompleteIncompleteCentral cord syndromeAnterior Cord syndromeBrown-Sequard SyndromePosterior Cord SyndromeCauda Equina and Conus Medullaris
Classification of SCI Completeness (degree) of InjuryComplete (transection)After spinal shock: Motor deficits- spastic paralysis below level of injurySensory- loss of all sensation perceptionAutonomic deficits- vasomotor failure and spastic bladder
Classification of SCI Completeness (degree) of InjuryIncompleteCentral Cord Syndrome
Injury to the center of the cord by edema and hemorrhageMotor weakness and sensory loss in all extremities Upper extremities affected more
Classification of SCI Completeness (degree) of InjuryIncomplete Brown-Squard Syndrome
Hemisection of cordIpsilateral paralysisIpsilateral superficial sensation, vibration and proprioception lossContralateral loss of pain and temperature perception
Classification of SCI Completeness (degree) of InjuryincompleteAnterior Cord Syndrome
Injury to anterior cord Loss of voluntary motor, pain and temperature perception below injuryRetains posterior column function (sensations of touch, position, vibration, motion)
Classification of SCI Completeness (degree) of InjuryincompletePosterior Cord Syndrome
Least frequent syndromeInjury to the posterior (dorsal) columns Loss of proprioceptionPain, temperature, sensation and motor function below the level of the lesion remain intact
Classification of SCI Completeness (degree) of InjuryincompleteConus MedullarisInjury to the sacral cord (conus) and lumbar nerve rootsCauda EquinaInjury to the lumbosacral nerve roots Result- areflexic (flaccid)bladder and bowel, flaccid lower limbs
Clinical Manifestations of SCISkin: pressure ulcers
Neuro: pain sensory lossupper/lower motor deficitsautonomic dysreflexia
Cardio: dysrhythmiasspinal shockloss of SNS control over blood vesselsorthostatic hypotension, poikilothermic
Respiratory-decrease chest expansion, cough reflex & vital capacitydiaphragm function-phrenic nerve GIstress ulcersparalytic ileusbowel- impaction & incontinence
GUupper/lower motor bladderImpotencesexual dysfunctionMusculoskeletaljoint contracturesbone demineralizationosteoporosismuscle spasmsmuscle atrophypathologic fracturespara/tetraplegia
Common Manifestation/ComplicationsUpper and Lower Motor Deficits
Upper motor deficits result in spastic paralysis
Lower motor deficits result in flaccid paralysis and muscle atrophy
Common Manifestations/ComplicationsSpinal cord injuries are described by the level of the injury the cord segment or dermatome levelSuch as C6; L4 spinal cord injury
Terms used to describe motor deficitsPrefix: para- meaning two extremitiestetra- or quadra- all four extremitiesSuffix :-paresis meaning weakness -plegia meaning paralysis
Quadraparesis means what?
Common Manifestations/ComplicationsC1-3 usually fatal- Loss of phrenic innervation ventilator dependent No B/B controlSpastic paralysisElectric w/c with chin/mouth control
Common Manifestations/ComplicationsC6- weak graspHas shoulder/biceps to transfer & push w/cNo bowel/bladder control.
Considered level of independence
Common Manifestations/ComplicationsT1-6- full use of upper extremityTransferDrive car with hand controls and do ADLsNo bowel/bladder control
Immediate CareEmergency Care at Scene, ER & ICU
Transport with cervical collarAssess ABCs; O2; tracheotomy/ventIV for life lineNG to suctionFoley
Diagnostic Studies for SCI
X-ray of spinal columnCT/MRIBlood gases
Therapeutic Interventions MedicationsIV methylprednisolone (Solu-Medrol) within 8 hrs to decrease cord edema
Therapeutic InterventionsMedications To control or to prevent complications of SCI and immobility:Vasopressors to maintain perfusionHistamine H2 blockers to prevent stress ulcersAnticoagulantsStool softenersAntispasmodics
Therapeutic InterventionsStabilization/Immobilization
Traction-Gardner-wells tongsHalo
CastsSplintsCollarsBraces
Therapeutic InterventionsSurgery for SCI
Manipulation to correct dislocation or to unlock vertebraeDecompression laminectomySpinal fusionWiring or rods to hold vertebrae together
Nursing Management AssessmentHEALTH HISTOYDescription of how and when injury occurredOther illnesses or disease processesAbility to move, breathe, and associated injury such as a head injury, fractures
Nursing Management AssessmentPHYSICAL EXAM
LOC and pupils- may have indirect SCI from head injuryRespiratory status- phrenic nerve (diaphragm) and intercostals; lung soundsVital signsMotorSensoryBowel and bladder function
Nursing ManagementAssessmentMotor Assessment Upper Extremity
Movement, strength and symmetry
Hand grips
Flex and extend arm at elbow- with and without resistance
Nursing Management Assessment Motor Assessment Lower Extremity
Flex and extend leg at knee with and without resistancePlanter and dorsi flexion of footAssess for Clonus
Nursing Management AssessmentSensory assessment
With the sharp and dull ends of a paperclip have the individual, with their eyes closed identify
Use the dermatome as reference to identify level
C6 thumb; T4 nipple; T10 naval
Nursing Problems/Interventions
1.Impaired mobility2.Impaired gas exchange3. Impaired skin integrity4. Constipation5. Impaired urinary elimination6. Risk for autonomic dysreflexia7. Ineffective coping
1. Impaired Physical MobilityLog roll as a single unit; provide assistance as needed to keep alignment; teach patient Care traction, collars, splints, braces, assistive devices for ADLsFlaccid paralysis- use high top tennis shoes or splints to prevent contractures. Remove at least every 2 hrs for ROM (active ROM best)
1. Impaired Physical MobilitySpastic ParalysisPrevent spasms by avoiding; sudden movements or jarring of the bed; internal stimulus (full bladder/skin breakdown; use of footboard; staying in one position too long; fatigueTreat spasms by decreasing causes; hot or cold packs; passive stretching; antispasmodic medications Assess skin break down thrombophlebitis; remove TED hose at least every shift
1. Impaired Physical MobilityPrevent/treat orthostatic hypotension Abdominal binder, calf compressors, TED hose when individual gets upAssess BP, especially when rising
Teach use of transfer boardAssist Physical Therapy with tilt table as individual gradually gets use to being in an upright position
2. Impaired Gas ExchangePhrenic nerve (C3-5) controls the diaphragm bilaterally. If nerve is nonfunctioning then individual is ventilator dependent.Thoracic nerves control the intercostals muscles for breathing and abdominal muscles aide in breathing and coughing
2. Impaired Gas ExchangeRespiratory rate, rhythm, depth, breath sounds, respiratory effort, ABGs, O2 saturation
Signs of impending extension of SCI up cord to phrenic nerve level (C3-5)Need for ventilatory assistance tracheotomy, ventilator
Quad cough (assistive cough) as needed
3. Impaired Skin IntegrityChange position frequentlyProtection from extremes in temperatureInspect skin at least 2x/day especially over boney prominences Avoid shearing and friction to soft tissue with transfersRemoval of TED hose every 8 hoursNutritional status
4. Constipation
Bowels rely more on bulk than on nervesStimulate bowels at the same time each day. Best after a meal when normal peristalsis occursIndividual may progress from Dulcolax suppository to glycerin then to gloved finger for digital stimulation Assess bowel sounds prior to giving food for the first time paralytic ileus!
5. Impaired Urinary Elimination
Flaccid bladder (lower motor neuron lesion) No reflex from S2,3,4Automatic empting of bladder Urine fills the bladder and dribbles outNeed Foley or freq intermittent self catheterization
Spastic bladder (upper motor neuron lesion) Reflex arc but no connection to or from brain Reflex fires at will Bladder training- trigger points to stimulate empting; self catheterization
6. Risk for Autonomic DysreflexiaSCI above T6 Results in loss of normal compensatory mechanisms when sympathetic nervous system is stimulatedLife threatening- if goes unchecked BP can result in cerebral hemorrhage
Vasodilatation symptoms above SCIVasoconstriction symptoms below SCIThe cause of SNS stimulation
6. Risk for Autonomic Dysreflexia
Elevate head of bed- causes orthostatic hypotension Identify cause/alleviate- if full bladder- cath; if skin- remove pressure, if full bowel- empty, etcRemove support hose/abdominal binderMonitor blood pressure- can get > 300 SGive PRN medication to lower BPIf above not effective call physician
7. Ineffective Coping/Grief and Depression
Assess thoughts on quality of life; body image; role changesPhysical and psychological supportMost common SCI is 15-30 yeas old and generally a risk taker this greatly affects their perception of life and rehabilitation
7. Ineffective Coping/sexualityMaleFemaleUMN lesionreflexogenic (S2,3,4) erectionsLMN lesion psychogenic erections (psychological stimulation)
Ejaculation/fertility may be affectedhormones more than nerves regarding fertility. C-section because of chance for autonomic dysreflexia during labor. Lack of sensation/movement affects sexual performance
7. Ineffective Coping/sexualityAssess readiness/knowledge/your abilityUse proper terminologySuggestions: empty bladder before sexwithhold fluids and antispasmodicscertain positions may increase spasmsexplore new erogenous zonespenile implantsRefer to specially trained counselor
Home CareAssess psychological, physiological resourcesneed for rehabilitation (in-house or out patient)need for community resources
Home assessment
Whats new in SCI treatment?Superman breatherYouTube - Superman breather USA
Kevin Everetthypothermia treatment for SCIStanding TallTravis Roy- 11 Seconds
Stem Cell treatment for SCILipitor for SCI
Case study- Jim Valdez1. Why does Jim have flaccid paralysis on admission to ICU?2. What symptoms indicate that he is in spinal shock? What was done about these symptoms?3. How will we know when he is out of spinal shock?4. How does progressive mobilization assist with orthostatic hypotension? What else can be done?5. What are realistic functional goals for Jim?
Spinal Cord AnatomyFunction of disc is to allow for mobility of the spine and act as shock absorberspinal cord anatomy
Pathophysiology/Etiology Located between vertebral bodiesComposed of nucleus pulposus a gelatinous material surrounded by annulus fibrosis- a fibrous coilSpinal nerves come out between vertebra
Herniated DiscHerniated nucleus pulposus, (HNP) slipped disc, ruptured discHNP- annulus becomes weakened/torn and the nucleus pulposus herniates through it.Risk Factors-Standing erectAging changesPoor body mechanicsOverweightTrauma
Common Manifestations/ComplicationsHNP compressesSpinal nerve (sensory or motor component) as it leaves the spinal cordOr the cord itself- the white tracts within the cord- rare
Common Manifestations/ComplicationsSensory root or nerve usually affected pain, parenthesis, or loss of sensationMotor root or nerve may be affected paresis or paralysis
Manifestations depend on what nerve root, spinal nerve is being compressed which dermatomesRadiculopathy- pathology of the nerve root
Common Manifestations/Complications Lumbar HNPMost common site for HNPL4-5 disc- the 5th lumbar nerve rootposterior sensory nerve or root compressed
Classic symptoms- low back sciatica painpain increases with increase in intrathoracic pressure
herniated disc L4-L5
Other Symptoms Lumbar HNP:Postural changesUrinary/male sexual function changesParesis or paralysisFoot dropParesthesiasNumbnessMuscle spasmsAbsent cord reflexes
Common Manifestations/Complications Cervical HNPC5-C6 disc- affects the 6th cervical nerve root
Pain- neck, shoulder, anterior upper arm to thumbAbsent/diminished reflexes to the armMotor changes- paresis or paralysisSensory- paresthesias or painMuscle spasms
Therapeutic Interventions- Diagnostic TestsX-ray identify deformities and narrowing of disk spaceCT/MRIMylogram p1336Nerve conduction studies (EMG) detect electrical activity of skeletal muscles
Treatment- ConservativeBed rest with firm mattress log roll side lying position with knees bent and pillow between legs to support legsAvoid flexion of the spine brace/corset, cervical collar to provide supportMedicationsnon-narcotic analgesics, anti-inflammatory, muscle relaxants, antispasmodics and tranquilizers
Treatment- ConservativeHeat/cold therapy to decrease muscle spasmsBreak the pain-spasm-pain cycleUltrasound, massage, relaxation techniquesProgressive mobilization with approved exercise program includes abdominal/thigh strengtheningTeaching good body mechanicsWeight lossTENS unit
Treatment- SurgeryLaminectomy- removal of a portion of the lamina to relieve pressure and to get to the herniated nucleus pulposus that is protruding outherniated disc repairForaminotomy Enlargement of the bony overgrowth at the opening which is compressing the nerve
Treatment- Surgery
Microdiskectomy Use of electron microscope through a small incision to remove a portion of the HNP that is displaced
If cervical HNP, usually use the anterior approach in the neckanterior cervical fusion
Treatment- SurgerySpinal fusion removes most of the disc and replaces it with bone usually from the patient iliac crestFusion also with rods, pins, synthetic proteinFlexibility is lost at the site- requires longer hospital stayspinal fusionArtificial DiscCombination of metal and plasticAttached to vertebrae above and below
Prevention of HNPBack school approach-Causes of HNPLearn how to prevent Good body mechanicsExercises to strengthen leg and abdominal muscles
Change in life-style or occupation
Nursing Assessment Specific to HNP Health HistoryAssess for risk factors- The cumulative effect of standing erect and daily stress Aging changes in disc/ligaments Poor body mechanics OverweightTraumaEmployment History of pain and other neuro changes
Nursing Assessment Specific to HNP Physical Exam Use similar methods to assess as utilized SCI
Muscle strength and coordinationSensationsharp/dull of paperclip using dermatome as referencePain evaluation- pain scalePre/Post-op assessment
Post-Op Assessment for HNP
Sensory/motor assessment- care not to injure op siteAssess for CSF drainage or bleeding from op siteEncourage turn (log roll, cough, deep breath) Assess for postural hypotensionespecially if client was on bed rest for several days/weeks prior to surgery
Post-op Assessment for HNPIf Anterior Cervical- Assess injury to the carotid, esophagus, trachea, laryngeal nerve (speech- hoarseness)Assess respiration, neck size, swallowing and speech
If Post-Op Lumbar- Assess bowels sounds, voiding. Minimize stress of post-op site- flat with pillow between knees, log roll, etc
Nursing Problems/Interventions 1. Acute PainPost surgery the individual may have similar pain as pre-op due to lack of resiliency of the spinal nerves to bounce back quickly
Donor site (illiac crest) may cause more pain than laminectomy
Individual may be in a pain-spasm-pain cycle, therefore may need both antispasmodic as well as analgesic
2. Chronic PainSurgery may not relieve pain
Nonpharmalogical methods to control pain
Pain clinic
3. ConstipationAs a result of bed rest and decreased mobility and fear of pain with straining of stool
Constipation prevention methods fluids, diet, etc
4. Home CareWhen riding in a car, take frequent stops to move and stretchPrevention Back school approachMay have to deal with pain as a chronic conditionMay need to make life/job changes
Spinal Cord TumorsCNS is made up of neural tissue and support tissue
These tissues undergo changes and result in spinal cord tumors
Blood vessels and bone also can be part of the tumor
Intramedullary- arise from neural tissues of the spinal cord
Extramedullary- arise from tissues outside the spinal cord may be benign or malignantIntradural-from the nerve roots or meninges in subarachnoid spaceExtradural- from the epidural tissue or vertebra
Classification by originPrimary- originating in the spinal cord or meninges
Secondary- metastases from other parts of the bodyMost spinal cord tumors are found in the thoracic region
Spinal cord tumors can compress (benign), invade the neural tissue, or cause ischemia to the area because of vascular obstruction
Common Manifestations/ComplicationsSymptoms depend on the anatomical level of the spinal column, the anatomical location, the type of tumor and the spinal nerves affected
Pain that is not relieved by bed rest is the most common presenting symptom
Other symptoms are similar to those found with HNP or spinal cord injury- sensory or motor
Common Manifestations/ComplicationsManifestations of thoracic cord tumorParesis & spasticity of one leg then the otherPain back & chest, not relieved by bedrest Sensory changes Babinski reflexBowel (ileus); bladder dysfunction (UMN in type)
Therapeutic Interventions
Diagnostic tests include:X-ray of the spinal columnMyelogramLumbar puncture with CSF analysis
Therapeutic InterventionsMedications spinal tumorsControl pain- narcotic analgesics, epidural catheter, PCA, NSAIDs
Reduce cord edema and tumor size- Steroids- high dose Dexamethasone
Therapeutic InterventionsSurgery for spinal cord tumorsLaminectomy to remove or to decrease the size (decompression laminectomy) of the spinal cord tumorSpinal fusion or the insertion of rods if several vertebra involved and the column is unstable
Radiation to reduce size and control pain
Nursing AssessmentHealth historyPain, motor and sensory changes, bowel and bladder changes, Babinski reflex.
Physical examSimilar to physical assessment for HNP
Nursing Problems/Interventions1. AnxietyMetatastic tumor vs benign spinal cord tumorEducation and support system
2. Risk for constipationFrom spinal cord compression, narcotics, bed restAdjust fluid and diet
Nursing Problems/Interventions3. Impaired physical mobilityFrom bed rest and motor involvementBasic nursing- ROM, etc 4. Acute painFrom compression or invasion of tumorAssess and treat
5. Sexual dysfunctionMale sacral reflex arc (S 2,3,4) interferenceSimilar care as discussed with SCI
Nursing Problems/Interventions6. Urinary retentionReflex arc (S2,3,4) interference can cause neurogenic bladder as discussed with SCI
7. Home careRehabilitationHome evaluationSupport groupscase study
A 30-year-old was admitted to the progressive care unit with a C5 fracture from a motorcycle accident. Which of the following assessments would take priority?
Bladder distensionNeurological deficitPulse ox readingsThe clients feelings about the injury
While in the ER, a client with C8 tetraplegia develops a blood pressure of 80/40, pulse 48, and RR of 18. The nurse suspects which of the following conditions?
Autonomic dysreflexiaHemorrhagic shockNeurogenic shockPulmonary embolism
A 22-year-old client with quadriplegia is apprehensive and flushed, with a blood pressure of 210/100 and a heart rate of 50 bpm. Which of the following nursing interventions should be done first?
Place the client flat in bedAssess patency of the indwelling urinary catheterGive one SL nitroglycerin tabletRaise the head of the bed immediately to 90 degrees
The nurse is caring for an elderly client diagnosed with a herniated nucleus pulposus of L4-L5. Which scientific rationale explains the incidence of a ruptured disc in the elderly?The client did not use good body mechanics when lifting an object.There is an increased blood supply to the back as the body ages.Older clients develop atherosclerotic joint disease as a result of fat deposits.Clients develop intervertebral disc degeneration as they age.
A client is admitted with a spinal cord injury at the level of T12. He has limited movement of his upper extremities. Which of the following medications would be used to control edema of the spinal cord?Acetazolamide (Diamox)Furosemide (Lasix)Methylprednisolone (Solu-Medrol)Sodium bicarbonate
A client with a cervical spine injury has Gardner-Wells tongs inserted for which of the following reasons?
To hasten wound healingTo immobilize the surgical spineTo prevent autonomic dysreflexiaTo hold bony fragments of the skull together
Which of the following interventions describes an appropriate bladder program for a client in rehabilitation for spinal cord injury?Insert an indwelling urinary catheter to straight drainageSchedule intermittent catherization every 2 to 4 hoursPerform a straight catherization every 8 hours while awakePerform Credes maneuver to the lower abdomen before the client voids.
A client has a cervical spine injury at the level of C5. Which of the following conditions would the nurse anticipate during the acute phase?
Absent corneal reflexDecerebate posturingMovement of only the right or left half of the bodyThe need for mechanical ventilation
The nurse is evaluating neurological signs of the male client in spinal shock following spinal cord injury. Which of the following observations by the nurse indicates that spinal shock persists?
Positive reflexesHyperreflexiaInability to elicit a Babinskis reflexReflex emptying of the bladder
Your T1 spinal cord injured patient complains of a headache. You should
Give him prn TylenolDisimpact his bowelsCall the doctorTake his blood pressure
What can the nurse do to best speed the patients recovery from a laminectomy of L5?
Keep patient flat in bedTeach the back school approachMedicate for pain q2 hoursAmbulate as soon as orders permit
Your patient has a malignant metastatic lesion at T8 and is in for palliative radiation. What is your main goal with this patient?
Teach patient self catheterizationEnsure patient receives pain medication as neededEncourage patient to discuss fearsAmbulate twice a shift
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