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Ms. Sophie is a 74 year old female who will be admitted to your floor for complaint of shortness of breath with activity and wound on left ankle. She reports a regular diet of fast food, potato chips, and chocolate. Loves salty food. She also states that she often feels stressed and depressed because of her weight however, she eats ice cream when she gets stressed. She does not exercise and often feels very tired. She has had two toes amputated from her left foot and she complains that her feet “fall asleep” easily. Patient states “feet look puffy.” She also has “moments” of chest pain. She walks with a cane.
Smoker High cholesterolDiabetesMI x 3 yearsCardiac stentDNRCHF AnginaPVDCADHTNPTCAMeds: Furosemide, Nitroglycerin,Captopril, Metoprolol
Smoker - ↑BP, dysrhythmia, PVD
High Cholesterol – occlusion of arteries
Diabetes - ↑risk of MI & CVA (TIA)
MI – ischemia, damage
Cardiac stent – open occluded artery
CHF – Left sided Lungs
R ight sided Rest of body
Angina – chest pain associated with ↓coronary artery blood flow
PVD - ↑thrombus
HTN – sex, aging, HX, overweight, inactivity
Obesity - ↑demand, ↑adipose tissue
Stress - ↑ cortisol
Nutrition - ↑ salt
Age
Fall Risk
Metabolic syndrome risk
Activity level
Blood pressure: 160/90 RR 16 Pulse 74 Fingerstick: 450 Height: 5’2” Weight: 180 lbs. Pedal pulses not palpable Lower extremity edema Left lower extremity edematous, red, warm BLE shiny, lack of hair, cool to touch Stage III wound on LLE ankle
PVD – mostly affects lower extremities - Risk factors: age > 50, HX of heart disease, Type I diabetes, HTN, obesity, physical inactivity, smoking - Symptoms: fragile skin on legs/feet, hair loss on legs, non-healing wounds, gangrene, complaint of “numbness” - Diagnosis: angiogram - Complications: Stroke, MI, amputation
Most common type of heart disease Arteries to heart become hardened and narrow Plaque →↑cholesterol→atherosclerosis→ ↓blood supply→angina or MI Over time = heart failure
MEDS: Furosemide,Heparin,Captopril,MetoprololEKGTelemetryEchocardiagramStress testUltrasoundAngiogramTEDS/SCD’s right legDoppler LLE
Furosemide Nitroglycerin Heparin
Insert drug table here
MEDICATION EXPECTED OUTCOME
Cardiac glycosides Increase cardiac contractility; decrease heartrate
Antihypertensives Decrease blood pressure
Vasopressors Increase blood pressure
Antiarrhythmics Regulate heart rhythm
Nitrates Relieve angina
Antilipids Decrease cholesterol levels
Diuretics Reduce fluid volume
Anticoagulants Decrease potential for clot formation
4th ICS &
5th ICS
Normal Sinus RhythmP wave – atrial depolarization
Q,R,S complex- ventricular depolarization
ST segment-complete depolarization of ventricles
T wave- ventricular repolarization
PR interval = 0.12 – 0.20 seconds (or 3 small – 1 large box)QRS complex = 0.06 – 0.12 seconds (or 1.5 – 3 small boxes)QT interval = 0.34-0.43 seconds (or 8.5 – 11 small boxes)Heart rate = # of PQRST complexes that occur in 6 seconds multiplied by 10
Small square = 0.04 secondsLarge square = 0.20 seconds Five small squares = a large squareFive large squares = 1 second
Leads I, II, III, aVR, aVL & aVF are measurements of the frontal plane
V1, V2, V3, V4, V5 & V6 are measurements of the horizontal plane
RA LA
HEART
RL LL
-continuous monitoring
Clouds over grass Smoke over fire Chocolate is good
for your heart
EKG
Telemetry
Assist with leg exercises, positioning, SCDs/TEDS
Medication :Anticoagulation prophylaxis, diuretics, I & O
Assessment
Education
TEDS(Thromboembo
lic Deterrent Stockings)
SCD’s(Sequential Compression Device)
o Test to see how blood moves through arteries and veinso Combines traditional ultrasound with dopplero No preparation needed
http://www.youtube.com/watch?v=yzxSrLa1d0g
o Use of radiopaque dye to outline vessels looking for blockage or narrowing
http://www.youtube.com/watch?v=N7nghr9TpSU
o Percutaneous Transluminal Coronary Angioplastyo Catheter placed through IV in neck, arm or groino Balloon tip inflated to compress blockageo Possible stent placement
Placed with balloon catheter
Locks in open position
Monitor for dysrythmias
Check peripheral pulses & VS Assess pain Assess bleeding or hematoma formation at
insertion site Maintain pressure dressing & immobilize limb for
approximately 6 hours Monitor for urination within 6 hours Administer fluids Document
http://www.youtube.com/watch?v=x_Z0GF6AuTw
Records cardiovascular response to increased activity
http://www.youtube.com/watch?v=2XR6etAY_-w&feature=related
-Ultrasonic waves- Detect structural defects-Can be done at bedside
DNR – do not rescusitate
DNI – do not intibate
MOLST – Maryland Order for Life Sustaining Treatment
1. Smoking – most modifiable risk factor for cardiovascular disease
2. Medication3. Edema4. Pain management5. Activity intolerance6. Diet
Peripheral intravascular device
Inserted by nurses/techs
Short term
Triple Lumen Central Line Can be placed at bedside Placed into internal jugular Risk of pneumothorax or air
embolism Short term
Hickman Placed on OR Terminates in superior vena cava
near entrance to right atrium Tunnelling Long term
Peripherally Inserted Central Catheter
Terminates in subclavian or superior vena cava
Small diameter Long term
Mediport Placed in OR Terminates in superior
vena cava near entrance to right atrium
Self sealing port Long term
Huber needle Non-coring needle used
to access mediport
oFirst responderoBedside nurseoCharge nurseoTeam/Code LeaderoRecorderoRunneroRespiratory/AnesthesiaoCode cart nurseoNursing supervisoroSecurity
Emergency meds
Defibrillator
Blood drawing supplies
Central line kit
Backboard
Laryngoscope
Oropharyngeal airway
60-100 pbm
Bradycardia - <60 bpm
Tachycardia - >100 bpm
Ectopic foci in walls of ventricles
↓ cardiac output
Intrinsic ventricular rate is only 20-40 bpm
Premature ventricular contraction Irritable focus in ventricle More likely to occur during bradycardia A beat, not a rhythm CauseBigeminy – PVC’s with every other beatTrigeminy – PVC’s every third beatQuadreminy – PVC’s with every fourth beat
• Foci in ventricles takes over
• When 3 or more PVC’s occur in a row at a rate greater than 100 bpm lasting longer than 30 seconds
• Treatment: cardioversion & CPR. Long term-antiarrythmic drugs
Rapid, disorganized depolarization of ventricles
No coordination of ventricular and atrial contractions
No palpable pulse
Treatment: Defibrillation & CPR. Long term: ICD
Pulseless electrical activityElectrical activity is present but no
contractionNo cardiac output, no tissue
perfusion, death
No electrical activity
Immediate loss of oxygen supply to brain, heart and tissue
Holter monitoro Worn for 1-3 dayso Records activity of heart
Pacemakero Used to augment or replace
the natural pacemaker of the heart
o Can be used for bradycardia/tahcycardia, damage to heart from MI, CHF
ICD – Implantable Cardiac Defibrillator
o To treat ventricular tachycardia and ventricular fibrillation
o Patients say a shock is like being kicked in the chest
o Another person touching the person will feel the shock