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Seminar by
DHANYA V THILAKAM
FIRST YEAR MSC NURSING
HYPERTENSION
BY
DHANYA V THILAKAM
FIRST YEAR MSC NURSING
Called the silent killer
About 31% of the adult in the united states
have hypertension
the prevalence increases significantly as
people are older or have other
cardiovascular risk factors
The prevalence also varies by ethnicity with
African Americans having the highest
prevalence
The WHO International society of
hypertension classifies non hypertensive
subjects with a systolic pressure of a130-139
mm Hg or a diastolic pressure of 85-89 mm
Hg of having a “ high ‘ normal blood
pressure
It is defined as a persistent elevation of the
systolic blood pressure at alevel of 140 mm
of Hg or higher and diastolic blood pressure
at a level of 90 mm Hg or higher.
STAGE 1 HYPERTENSION
STAGE 2 HYPERTENSION
SEVERE HYPERTENSION
ESSENTIAL HYPERTENSION
1) GENETIC FACTORS
2) FETAL FACTORS
3) ENVIRONMENTAL FACTORS
4) HUMORAL MECHANISMS
5) INSULIN RESISTANCE
Secondary Hypertension Is Where Blood
Pressure Elevation Is The Result Of A
Specific And Potentially Treatable Cause
. These Patients May Be Amenable To
Curative Treatment , Thereby Sparing
Them From Life-long Medical Therapy
That Is Frequently
Unpleasant, Sometimes Ineffective ,And
Always Expensive
RENAL DISEASES
ENDOCRINE CAUSES
Chron’s syndrome
Adrenal hyperplasia
Phaeochromocytoma
Cushing’s syndrome
Acromegaly
CARDIOVASCULAR CAUSES
The major cause is
coarctation of the aorta
DRUGS
PREGNANCY
hypertension in people who are actually
normotensive except when their blood
pressure is measured by ahealth care
professional .An intermittent vasovagal
response accounts for the transient elevation
in blood pressure
Occurs when the systolic blood pressure is
140mm of Hg or higher but the diastolic
pressure remains less than 90 mm of Hg.It is
thought to emerge because of increased
cardiac output or atherosclerosis induced
changes in blood vessel compliances or both
in older adult
Characterised by a diastolic pressure above
110-120 mm of Hg .It results when
hypertension is left untreated or is
unresponsive to treatment and becomes a
truly severe emergency condition as the
pressure continuous to rise unchecked.
Cardiac •Heart rate
•Inotropic state
•Neural (pons and medulla)
•Humoral (hormones)
Cardiac Output
Renal Fluid Volume Control•Renin–angiotensin
•Aldosterone
•Atrial natriuretic factor
Copyright © 2007, 2004, 2000, Mosby, Inc., an affiliate of Elsevier Inc. All Rights Reserved.
• Chemosensitive cells that respond to changes in pCO2
and pO2 and pH levels (Hydrogen ion).
pO2 and
pH pCO2
Stimulation of
vasomotor
center
CO HRvasoconstrictio
n
BP (speeding return of
blood to the heart and
lungs)
Short-term Regulation of Rising Blood Pressure
Rising blood
pressure
Stretching of arterial
walls
Stimulation of baroreceptors in
carotid sinus, aortic arch, and other
large arteries of the neck and
thorax
Increased impulses to the
brain
Physical examination may reveal no abnormalities other than elevated blood pressure.
Occasionally, retinal changes such as hemorrhages , exudates (fluid accumulation), arteriolar narrowing and cotton-wool spots (small infarctions) occur .
In severe hypertension, papilledema(swelling of the optic disc)may be seen . People with hypertension may be asymptomatic and remains so for many years.
. Coronary artery disease with angina and myocardial infarction are common consequences of hypertension
.Left ventricular hypertrophy occurs in response to the increased workload placed on the ventricle as it contracts against higher systemic pressure
Pathologic changes in the kidneys(indicated by increased blood urea nitrogen{BUN}and serum creatininelevels) may manifest as nocturia.
Cerebrovascular involvement may lead to a stoke or transient ischemia attack (TIA) , manifested by alterations in vision or speech , dizziness , weakness ,a sudden fall , or transient or permanent paralysis on one side (hemiplegia).
Cerebral infarctions accounts for most of the strokes and TIAs in patients with hypertension.
Stroke
Heart attack
Heart failure
Kidney failure
Eye damage
Peripheral artery disease
A thorough health history and physical
examination are necessary .
The retinas are examined and laboratory
studies are performed to assess possible
target organ damage
.Routine laboratory tests include urinalysis
, blood chemistry (i.e analysis of sodium
, potassium ,creatinine ,fasting glucose ,and
total and high-density lipoprotein{HDL}
Cholesterol levels) and a 12 lead
electrocardiogram (ECG),
Left ventricular hypertrophy can be assessed
by echocardigraphy .
Renal damage may be suggested by elevation
in BUN and creatinine levels or by
microalbuminuria or macroalbuminuria .
Additional studies, such as creatinine
clearance , reninlevel , urine tests , and 24-
hour urine protein may be performed .
The goal of hypertension treatment is to prevent complications and death by achieving and maintaining the arterial blood pressure at 140/90 mm Hg or lower.
JNC 7 specifies a lower goal pressure of 130 /80 mm Hg for people with diabetes mellitus or chronic kidney disease, which is defined as either a reduced glomerular filtration rate (GFR)resulting in a serum creatinine of greater than 1.3 mg/dL in a women or greater than 1.5 mg/dL in men , or albuminuria of greater than 300mg/dL.
1. diuretics
2. β-adrenoceptor antagonists (β-blockers)
3. Ca-channel blockers
4. inhibitors of angiotensin (ACE-
inhibitors/AT1-blockers)
5. α-adrenergic blockers
FOOD GROUPS No of servings
/day
Grains and grain products 7 or 8
Vegetables 4 or 5
Fruits 4 or 5
Low fat or fat free dairy food 2 or 3
Meat ,fish , and poultry 2 or fewer
Nuts ,seeds and dry beans 4 or 5 weekly
ASSESSMENT
A complete history is obtained to assess for
signs and symptoms that indicate target
organ damage (i.e , whether specific tissues
are damaged by the elevated blood pressure
). Such manifestation may include angina
pain ; shortness of breath; alternation in
speech ,vision or balance ;nose bleeds
;headaches; dizziness ‘ or nocturia
During the physical examination , the nurse must also pay specific attention to the rate , rhythm ,and character of the apical peripheral pulses to detect effects of hypertension on the heart and blood vessels. A thorough assessment can yield valuable information about the extent to which the hypertension has affected the body and about any other personal ,social , or financial factors related to the condition. For example , some patient’s ability to adhere to an antihypertensive medication regimen may be influenced by the financial resources to buy the medication and health insurance
Deficient knowledge regarding the relation between the treatment regimen and control of the disease process
Non-compliance with therapeutic regimen related to side effects of prescribed therapy
altered tissue perfusion ( renal,cerebral, cardio pulmonary,and peripheral ) relatedred to pheripheral blood flow
Altered nutrition : High Risk for more than Body requirements related to learned eating behaviours ,ethic and cultural values, lack of social support for weight loss , and or imbalance between activity level and caloric intake
Fatigue related to altered body chemistry
(medications)
altered sexuality pattern related to effects
of medical treatment ( drugs)
ineffective individual coping related to
effects of chronic illness and major changes
in lifestyle
The major goals of the patient include
understanding of the disease process and its
treatment , participation in a selfcare
program , and absence of complications
INCREASING KNOWLEDGE
PROMOTING HOME AND COMMUNITY - BASED
CARE
TEACHING PATIENTS SELF – CARE
MONITORING AND MANAGERING POTENTIAL
COMPLICATIONS
Research finding demonstrate that weight loss
, reduced alcohol and sodium intake , and regular
physical activity are effective lifestyle adaptations to
reduce blood pressure
Studies also show that diets high in fruits , vegetables
,and low –fat dairy products can prevent the
development of hypertension and can lower elevated
blood pressure
Consumption of garlic reduces hypertension
The prognosis from hypertension depends on a
number of features ;
The level of blood pressure
The presence of target-organ changes
Coexisting risk factors for cardiovascular
disease , such as hyperlipidaemia , diabetes
,smoking ,obesity ,male sex
Age at presentation
Fauci AS, Braunwald E, Isselbacher KJ, Wilson JD, Martin JB, Kasper DL,et al, editors. Harrison's principles of internal medicine. 14th ed. New York:McGraw Hill; 1998
Suzanne C. Smelter, Brenda G Bare,Brunner& Suddarth ‘s Textbook of Medical Surgical Nursing,12 th edn,Lippincott;2010
Praveen Kumar ,Michel Clark ,Kumar & clarkclinical Medicine ;5th edn ;Saunders 2002
en.wikipedia.org/wiki/Essential_hypertension
circ.ahajournals.org/content/101/3/329.full
www.ncbi.nlm.nih.gov/pubmed/11785064