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DENTAL ANOMALIES
UNDER THE GUIDANCE OF:
DR.M.K.JINDAL
PRESENTED BY:BUSHRA FARHANBDS 2010 BATCH
DENTAL ANOMALIES
NUMBER SIZE
SHAPE STRUCTURE
ALTERATIONS IN NUMBER OF
TEETHAlteration in tooth number occur usually during initiation or dental
lamina stage of dental development
The alteration may produce extra or missing teeth
ANODONTIA
HYPODONTIA
OLIGODONTIA
SUPERNUMERARY TEETH
ANODONTIA•Congenital absence of teeth
because of failure of development of tooth germs.•Total anodontia is rare.
•Partial anodontia (hypodontia) is more common.
SUPERNUMERARY TEETH Supernumerary teeth are
additional number of teeth, over and above the usual number for
the dentition Mostly seen in
a. Gardner's syndrome,b.Cleidocranial dysostosis syndrome
c. Cleft palate (or cleft lip)
CLASSIFICATION
CONICAL
SUPPLEMENTAL
TUBERCULATE
ODONTOME
CONICAL
• Most commonly found in permanent dentition.
• Usually present as mesiodens between the incisors.
TUBERCULATE
• Commonly located on palatal side of central incisors.
• Possess more than 1 tubercle or cusp.• Associated with delayed eruption of
incisors.
SUPPLEMENTAL• Duplication of teeth in normal series
& found at the end of a tooth series.• Most commonly found in permanent
maxillary lateral incisors.
ALTERATIONS IN SIZE OF TEETH
Alteration in tooth size originate during the Bell stage or
proliferation stage of tooth development.
MICRODONTIA MACRODONTIA
MICRODONTIA• Teeth that are smaller than normal.• Most commonly affects maxillary
lateral incisors or maxillary third molars.
• Occur in a condition known as pituitary dwarfism.
• Can cause spacing in primary and permanent dentition.
MACRODONTIA• Teeth that are larger than
normal• Hemifacial hypertrophy
• Can cause crowding in primary n permanent dentition
ALTERATIONS IN SHAPE OF TEETH•Abnormalities in shape can
originate during the morphodifferentiation stage of
tooth development and are manifested as alterations in
crown and root form.
FUSION
GEMINATION
CONCRESCENCE
DENS INVAGINATUS(DENS IN DENTE)
DENS EVAGINATUS
TALON CUSP
TAURODONTISM
DILACERATION
HYPERCEMENTOSIS
FUSIONUnion of 2 normally separated tooth
germs. Must involve the dentin.
Cause :some physical force or pressure produces contact of tooth germs.Early contact: 2 teeth may be
completely united.Late contact: union of roots only.
GEMINATIONIncomplete attempt of a tooth
germ to divide into two.Tooth has two crowns or a large
crown partially separated.Single (common) root and root
canal.Etiology of this condition is
unknown
CONCRESCENCEFusion occurring after root
formation has been completed.Teeth united by their cementum.
Mostly association with the maxillary second and third molars.Difficulty in tooth extraction.
DENS INVAGINATUSInvagination in the surface of tooth
crown before calcification has occurred.
Also called tooth within a tooth.Can cause development and
spread of dental caries.Maxillary lateral incisor is the most
frequently affected tooth.
DENS EVAGINATUSTubercle or cusp located in the
center of the occlusal surface.Affect predominantly premolar and
molar teeth.Tubercle wears off relatively
quickly causing early exposure of the accessory pulp horn that
extends into the tubercle.
TALON CUSPAccessory cusp located on the
lingual surface of maxillary or mandibular teeth
Pattern resembling an eagle's talon.
Maxillary central or lateral incisor are often involved
TAURODONTISM Crowns of normal size and shape but have
large rectangular bodies. Pulp chamber is dramatically increased in its
apico-occlusal heights. Apically displaced furcations. Short roots and pulp canals.
Involves molar tooth. Seen in association with amelogenesis
imperfecta. Not recognizable clinically but on a radiograph.
DILACERATIONAbnormal bend in the root of a
tooth.Result of trauma to a developing
tooth.Difficulties during extraction or
root canal therapy.
HYPERCEMENTOSISExcessive build-up of cementum
around toothEvident on a radiograph
Affects vital teethExact cause not known
Mostly seen in periapical inflammation, tooth repair and teeth that are not in occlusion
ALTERATIONS IN STRUCUTRE OF TEETH
AMELOGENESIS IMPERFECTA
DENTINOGENESIS IMPERFECTA
DENTIN DYSPLASIA
ODONTODYSPLASIA
AMELOGENESIS IMPERFECTA
• Disturbance in enamel development
•Normal dentin & root•Etiology: alteration of genes
involved in the process of formation & maturation of the enamel.
•Three general types
• Defects in matrix formation
HYPOPLASTIC
• Defects of matrix structure and of mineral deposition.
HYPOCALCIFIED
• Alterations in enamel rod and rod sheath structures.
HYPOMATURE
DENTINOGENESIS IMPERFECTA
A hereditary abnormality in the formation of dentin.
Teeth varies from gray to brownish violet to yellowish brown color.
Crown fractures easily because of abnormal DEJ.
Pulp chambers and root canals may be partially or completely obliterated.
Radiographically, the teeth exhibit thin, short roots.
DENTIN DYSPLASIARare disturbance of dentin
formation.Normal enamel but atypical dentin
with abnormal pulp morphology.It is subdivided into two types:
TYPE 1 RADICULA
RTYPE 2
CORONAL
TYPE 1 (RADICULAR) Normal color & shape in both
dentition. Exfoliate with little or no trauma.
Short or abnormal roots.Pulp chambers & canals are usually
obliterated.20 % of teeth with type I disease
have apical radiolucencies.
TYPE 2 (CORONAL)Primary dentition appears as in D.I.,
but permanent dentition is normal. Obliteration of the pulp chamber in
deciduous dentition.Abnormally large pulp chamber in
permanent dentition. Roots are normal in shape &
proportion
ODONTODYSPLASIA
• Hypoplastic & hypocalcified dentin & enamel
•central incisors > lateral incisors >canines (maxillary)•Delayed eruption.
•Ghostlike appearance in image.•Marked reduction in amount of dentin.•Thin enamel , less dense as usual.
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