6 · Web viewDr. MINU. P. Post Graduate Student, Post Graduate Dept. of Repertory and Case taking, DBHP Sabah’s. Dr. B. D. Jatti Homoeopathic Medical College,

From: Dr. MINU. P. Post Graduate Student, Post Graduate Dept. of Repertory and Case taking, DBHP Sabah’s. Dr. B. D. Jatti Homoeopathic Medical College,Hospital & P.G. Research Center, D.C. Compound, Dharwad-580001.

To: The RegistrarRajiv Gandhi University of Health Sciences, Karnataka, Bangalore.

Through:

The Principal DBHP Sabha’s. Dr. B. D. Jatti Homoeopathic Medical College, Hospital & P.G. Research Center, Dharwad-580 001.

Respected Sir,

Subject: Submission of Completed Proforma of Synopsis for Registration of Subject for Dissertation.

I request you to kindly register the below mentioned subject against my name for the submission of dissertation to the Rajiv Gandhi University of Health Sciences, Bangalore in partial fulfillment for the award of the degree of M.D. (Homoeopathy) in Repertory.

Title of Dissertation:

“A COMPARATIVE STUDY ON LOGICO-UTILITARIAN REPERTORIES AND CLINICAL REPERTORIES IN THE

MANAGEMENT OF TYPE 2 DIABETES MELLITUS”

I am herewith enclosing completed proforma of synopsis for registration of subject for dissertation.

Thanking you,

Place: Dharwad Yours faithfully Date:

(Dr. MINU. P)

“A COMPARATIVE STUDY ON LOGICO-UTILITARIAN REPERTORIES AND CLINICAL REPERTORIES IN THE


SYNOPSIS

Submitted to

RAJIV GANDHI UNIVERSITY OF HEALTH SCIENCES KARNATAKA, BANGALORE,

BY

Dr. MINU. P

ThroughDPHP’s

DR. B. D. JATTI HOMOEOPATHIC MEDICAL COLLEGE, HOSPITAL & P.G.

RESEARCH CENTER, D.C. COMPOUND, DHARWAD-580001.

(KARNATAKA)

In partial fulfillment of requirement for the

DOCTOR OF MEDICINE (HOMOEOPATHY)

HOMOEOPATHIC REPERTORY

Under the valuable guidance of

DR. SUDHANSU SEKHAR MOHARANA

PROFESSOR

Dept. of Hom. Repertory. DR. B. D. JATTI HOMOEOPATHIC MEDICAL COLLEGE, HOSPITAL & P.G.

RESEARCH CENTER, D.C. COMPOUND, DHARWAD-580001.

(KARNATAKA)

RAJIV GANDHI UNIVERSITY OF HEALTH SCIENCES, KARNATAKA, BANGALORE.

Annexure-II

REGISTRATION OF SUBJECT FOR DISSERTATION

1. NAME OF CANDIDATE & ADDRESS

Dr. MINU. P. Post Graduate Student,Post Graduate Dept of Repertory and Case taking,Dr. B. D. Jatti Homoeopathic Medical College, Hospital & P.G. Research Center,Dharwad-580001

PERMANENT ADDRESS Dr. MINU. P. D/o. P. Gangadharan58/1, “Sree Ganga”J.R. Colony, New ThippasandraBANGALORE-560075

2.NAME OF THE INSTITUTION

D.B.H.P. Sabha’sDr. B. D. Jatti Homoeopathic Medical College, Hospital & P.G. Research Center, D. C. Compound, Dharwad-580001

3.COURSE OF STUDY AND SUBJECT

M. D. (HOMOEOPATHY) REPERTORY

4.DATE OF ADMISSION TO COURSE 07-07-2010

5.TITLE OF THE TOPIC “A COMPARATIVE STUDY ON LOGICO-

UTILITARIAN REPERTORIES AND CLINICAL REPERTORIES IN THE


6 BRIEF RESUME OF THE INTENDED WORK 6.1 NEED FOR THE STUDY:

In the vast majority of type 2 diabetics the principal abnormality is one

of the reduced insulin sensitivity. However, if insulin secretion is

insufficient to compensate, impaired glucose tolerance or frank

diabetes results. This typically occurs as a result of β cell exhaustion.

Occasionally, insulin sensitivity is normal but insulin production is

reduced (e.g. maturity-onset diabetes of the young [MODY]); diabetes

again results.

The prevalence of type 2 in Britain is around 2.3% type 2 diabetes

occurs in migrant populations to industrialized countries as in Asia and

Afro-Caribbean immigrants to the UK.

Type 2 DM is a multi-factorial disease with genetical and

environmental factors including possible infections. Precipitating

causes like physical or emotional stress. At present there is an

outerageous evidence to say Indians are prone to type 2 diabetes due to

diseased life style rather than genes. Which ultimately harms our

healthy genes and turning them against us.

Insulin, which was discovered by Fedrick Bunting and Charles Best in

1921, Since then it had saved millions of lives all over the world.

Modern medicines contribute immensely and saved millions of people.

Majority of the patients are geriatric, most drugs are metabolized in the

liver. With age the ability of the liver to degrade and is diminished

which leads to more toxicity. Thus the side effect of oral hypoglycemic

agents and the development of Insulin hypersensitivity and resistance

must be taken into consideration. Though insulin therapy is considered

to be live saving, but there are some controversial aspect about the side

effects of insulin therapy. The most common side effects of insulin

therapy is low blood sugar. Other side effects of insulin therapy is

redness, swelling or itching at the site of injection, worsening of

diabetic retinopathy, changes in distribution of body fat

(lipodystrophy), allergic reactions, sodium retention and general body

swelling, a type of chest discomfort, cough and dyspnea. It is also seen

that there is weight gain, lipoatrophy and lipohypertrophy. The side

effects of oral hypoghycemic agents, Hypoglycaemia, allergic reaction,

dilutional hyponatraemia, cholestasis, lactic acidosis, Anorexia,

Nausea, vomiting, diarrhoea, Abdominal fullness, Bloating,

Hypoglycemia (mild), transient visual disturbance, occasionally transits

elevation of liver enzymes, sinusitis, upper respiratory tract infection,

hepatic injury, headache, Anaemia and oedema, malabsorption of vit

B12, and folic acid, muscle pain, weakness, weight again.

Homeopathy has been said to be having much efficacy in treating type

2 Diabetic mellitus because of its systematic analysis and holistic

approach of considering body, mind and disease and with the concept

of individualization and dynamization. One endeavours to treat all the

symptoms at one time and with one drug. The selection of the potency,

the repetition and the time of releasing the remedial force depend upon

an accurate assessment of the pathogenesis as well as the Pathology

and the qualitative assessment of susceptibility and sensitivity. Thus,

all this criteria is considered for an efficient out come in the

management of the case.

Logical utilitarian Repertories have distinctive principles of their own.

Therefore cases have to be selected to fit them with the principles Eg:

Kent’s Repertory. Synthesis repertory, Boenninghausen’s

characteristics materia medica and Repertory by CM Boger etc.

Clinical Repertory have many clinical rubrics under different systems.

Eg. clinical Repertory by Oscar. E. Boericke, clinical Repertory by J.

H. Clarke etc. making treatment of type 2 DM more inclusive.

Kent’s Repertory is rich in rubric of generalities and mind. Synthesis

repertory is rich in ailment from sub-rubric of the mind and rubric of

generals and characteristics particulars. Boger Boenninghausen’s

characteristics and repertory by CM Boger is rich in pathological

generals and clinical rubrics with rich in modalities sensations and

concomitants. Clinical repertory by Oscar. E. Boerick is rich in clinical

rubrics. All the headings are presented under definite captions in the

following order, course, type, location, character of the pain,

concomitants and modalities. Technical names of diseases are

bracketed, thereby assuming a subsidiary place, which is in strict

accord with Homoeopathic requirement. Clinical Repertory by J. H.

Clarke is rich in clinical condition, clinical relationships,

temperaments, dispositions, constitutions and states. Thus clinical,

pathological generals, physical and mental generals are of immense

value in treatment of type 2 DM.

Thus a comparative study on logico-utilitarian repertories and clinical

repertories is undertaken to ascertain the Homoeopathic management of

type 2 Diabetic Mellitus.

Hence, this study is undertaken to determine the comparative efficacy

of logico-utilitarian repertories and clinical repertories.

6.2 REVIEW OF LITERATURE HISTORICAL REVIEW

1. The name diabetes is derived from the Greek word for ‘Syphen’. It is

disease of great antiquity, and some of its salient feature have been

known since the secondary century AD. When Aretaeus described it as

a melting of the flesh and limbs into the urine. Thomas Willis noted

that the urine was ‘wonderfully sweet as if it were inbued with honey

or sugar. Hence the term mellitus, derived from the Latin word for

honey.1 Diabetes is first recorded in English in the form diabetic in a

medical text written around1425. In 1776, Matthew Dobson confirmed

that the sweet taste was because of an excess of a kind of sugar in the

urine and blood of people with diabetes. Diabetes Mellitus appears to

have been a death sentence in the ancient era. Hippocrates makes no

mention of it, which may indicate that he felt the disease was

incurable. Aretaeus did attempt to treat it but could not give a good

prognosis, he commented that “life (with diabetes) is short, disgusting

and painful. Sushruta (6th century BCE) identified diabetes and

classified it as Madhumeha. The further identified it with obesity and

sendentary life style, advising exercises to help “cure it. The ancient

Indians tested for diabetes by observing whether ants were attracted to

persons urine, and called the ailment “Sweet urine, disease”

(Madhumeha). In 1910, sir Edward Albert Sharpey Schofer suggested

that people with diabetes were deficient in a single chemical that was

normally produced by the pancreas–he proposed calling this substance

insulin, from the Latin insula, meaning island, in reference to the

insulin producing islets of langerhans in the pancreas.2

2. Diabetes Mellitus – Comprises a heterogeneous group of metabolic

diseases that are characterized by chronic hyperglycemia and

disturbances in carbohydrate, lipid and protein metabolism. These

diseases result from defects in insulin secretion, insulin action or both.3

3. Classification of Diabetic Mellitus:

Type-1: Insulin dependent diabetic mellitus.

Type-2: Non insulin dependent diabetic mellitus.

Other specific types are due to Genetic defect, Genetic defect of

insulin action, drug induced, viral infection, associated with genetic

syndromes, uncommon forms of immune-mediated diabetes. Excess

endogenous production of hormonal antagonists to insulin.

Gestational diabetes4.

4. Type 2 diabetes mellitus it was previously called maturity onset

diabetes or non insulin dependent diabetes mellitus of obese and non-

obese type.5 There is both reduced sensitivity of tissues to insulin and

impaired regulation of insulin secretion.6

5. The prevalence diabetes mellitus in India is currently reported around

13-15%.7 The overall prevalence of type 2 diabetes in Karnataka was

found to be 16%, among males 18.8% and among females 14.4%.8

Type 2 diabetes is the commonest form of diabetes affecting 5-7%

adults in western society.9 In 2000, according to World Health

Organization, at least 171 million people world wide suffer from

diabetes, or 2.8% of the population.10

6. Type 2 diabetic mellitus is principally a disease of the middle aged and

elderly usually above the aged of 40 years.11

7. Genetic factors are important in the etiology of type 2 diabetes as

shown by marked differences in susceptibility in different ethnic group

and by studies in monozygotic twins where concordance roles for type

2 diabetes approach 100%. Environmental factors, epidemiological

studies provide evidence that type 2 diabetes is associated with

overeating, especially when combined with obesity and under activity.

Obesity probably acts as a diabetogenic factor only in those who are

genetically predisposed to insulin resistance and to β-cell failure. The

risk of developing type 2 diabetes increases tenfold in people with a

body mass index > 30kg/m2.12

8. The basic metabolic defect in type 2 DM is either a delayed insulin

secretion relative to glucose load (impaired insulin secretion) or the

peripheral tissues are unable to respond to insulin (insulin resistance).

Insulin resistance is the most prominent feature of type 2 DM is the

lack of responsiveness of peripheral tissues to insulin, especially of

skeletal muscle and liver. There is increased hepatic synthesis of

glucose. Hyperglycaemia in obesity is related to high levels of free

fatty acids and cytokines. (eg. TNF-l and adiponectin) affect peripheral

tissue sensitivity to respond to insulin. In impaired insulin secretion,

there is failure of β-Cell function to secrete adequate insulin due to the

1) Amylin which forms fibrillar protein deposits in pancreatic islets in

longstanding cases of type 2 DM and also due to

2) Metabolic environment of chronic hyperglycemia surrounding the

islets (glucose toxicity) may paradoxically impair islet cell function.

3) Elevated free fatty acid level (lipotoxicity in these cases may worsen

islet cell function).13

9. CLINICAL FEATURES:

1) Polydipsia, Polyuria, nocturia, weight loss, pruritis vulvae/ balanitis,

impotence.14

2) Weakness, easy fatigability, giddiness, blurring of vision, muscle pain,

cramps and paraesthesias.15

10. DIAGNOSIS OF DIABETES

1. Based on sign and symptoms:

Polyuria, polydipsia, Polyphagia, weight loss, non healing wounds,

recurrent styes, pruritis Vulvae, recurrent urinary tract infections.16

2. Based on Investigation:

Symptoms of diabetes plus causal plasma glucose concentraction

≥ 200 mg/dl(11.1mm ol/c).17

Fasting plasma glucose ≥ 126mgldl (7.0 mmol/c/).17

1 hr post load glucose < 200mg/ dl during an oral glucose tolerance

Test (OGTT).18

11. COMPLICATIONS

- The main metabolic complication in type 2 diabetes mellitus is

Hyperosmolar non-ketonic coma.19 The late complications are Diabetic

retinopathy, Diabetic nephropathy, Diabetic neuropathy, Cardiovascular

diseases,20 Diabetic foot, Diabetic skin – diabetic non healing wounds,

Candidiasis, Diabetic infections21

12. INVESTIGATION

1. Urinalysis: Glycosuria may suggest the presence of diabetes but requires

confirmation with a blood test. Conversely, absence of glycosuria does not

exclude diabetes. The presence of ketones, blood, protein, nitrites and

leucocytes should be noted.

2. Blood glucose: The American Diabetes Association has recently

recommended that the diagnosis of diabetes mellitus should be made on the

basis of a fasting venous plasma glucose level of ≥ 7.0 mmol/L which should

be confirmed on a second occasion. It can also be made however in a

symptomatic patient whose random venous plasma glucose is ≥ 11.1 mmol/L.

3. Others

- Routine blood test: Urea and electrolytes, HbA1c, lipid profile, thyroid

function.

- ECG and chest radiograph (especially in the older patient)

Retinal picture / eye screening.22

13. DIET MANAGEMENT

The importance of diet in the management of diabetes varies with the type of

disease. In non insulin dependent patients not treated with exogenous insulin,

more rigorous adherence to diet is required, since the endogenous insulin

reserve is limited. Such patients cannot respond to the increased demand

produced by excess calories or increased intake of rapidly absorbed

carbohydrate. Medical nutrition therapy for people with diabetes should be

individualized, with consideration given to eating habits and other lifestyle

factors. Nutrition recommendations are then developed to meet treatment

goals and desired outcomes. Flexibility in use of ordinary foods important

patients and families. The first decision is the caloric content of the diet, based

on the need to gain, lose or maintain current weight.23

14) Homoeopathic treatment aims to cure the sickness of the patient by

stimulating the vital dynamic or the power of resistance of the sick person. But

if the power of resistance is weakened by morbific influences of toxic and

parasitic agents also called chronic miasms. Dr. Hahnemann has advised to

remove these obstructions to cure chronic miasms so that no hindrance may

come in the way of cure. Deficiency of insulin is the chief obstruction to cure

diabetes mellitus. Therefore, where insulin deficiency is present and cannot be

stimulated by homoeopathic and specific remedies it should be supplied in

order to remove the obstruction to cure.24

15) In the literature of the new system we found on the one hand report of

decided benefit in the diabetic cases from general and symptomatic treatment,

and on the other certain complete or proximate cures with medicines

presumbly homoeopathic to the essential lesions. Of these hughes gave

attention on phosphoric acid and the salts of uranium. Dr. Stiegele had an

interesting experience with syzgium and arsenicum, where arsenicum was

considered to be antidiabetic.25

16) Urine-Sugar: Acet-ac., all-s., amyl-n., arg-m., ars., benz-ac., Bov., calc.,

calc-p., carb-ac., carb-v., chel., chin., coff., colch., conv., cupr., cur., elaps,

ferr-m., Helon., hep., iris, kali-chl., kali-n., kali-p., kreos., lac-d., lach., lac-

ac., lec., lith., Lyc., lycps., lyss., mag-s., med., mosch., morph., nat-s., nit-ac.,

op., petr., Ph-ac., Phos., pic-ac., Plb., podo., rat., sil., sulph., sul-ac., Tarent.,

Ter., thuj., Uran., zinc.26

17) Saccharine: Amy-n., arg., Ars., aur., bar-c., carb-v., chin., colo., con.,

curar., helo., kali-bi., kali-c., kali-p., kre., led., lyc., mag-c., meph., merc.,

mur-ac., Nat-m., nat-s., nit-ac., pho., PHO-AC., pic-ac., plb., ran-b., sec-c.,

sep., Sul., tarx., Thu., zin.27

18) Diabetes mellitus: (URINE-Sugar) allox,ars-br, brid-fr, cortico, cur, galeg,

helon, kali-act, lac-ac, lyc, mag-act, merc-d, morind-l, mosch, nat-m, Nat-s,

Op, pancr, phos, plb, rhus-t, sulph, syzyg, tarent, Uran-n, vinc-r.28

19) DIABETES–Sugar-Acet. Ac.; Adren.; Arg. m.; Arg. n.; Arist.; Arn.; Ars.;

Ars. i.; Aur.; Aur. m.; Bell.; bor. ac.; Bov.; Bry.; Caps.; Carb. ac.; Cham.;

Chel; Chim.; Eup. pur.; Fel.; Ferr. i.; Ferr. M.; Fl. Ac.; glon.; Glyc.; Hell.;

Helon.; Iod.; Iris; Kali act.; Kali br.; Kreos.; Lac. ac.; Lach.; Lec.; Lyc.;

Lycps. V.; Lyss.; Morph.; Mosch.; Murx.; Nat.m.; Nat. s.; Nit. Ac.; Nux v.;

Op.; Pancr.; Phase.; Ph.ac.; Phos.; Pic. Ac.; Plb.; Plb. i.; Podo.; Rhus. ar.;

sil.; Squil.; Stry. ar.; Sulph.; Syzyg.; Tarent.; Tarax.; Ter.; Uran. n.; Urea.;

Vanad.29

20) Diabetes: Ac.x., Aln., Am. Ac., Ank., Arg., (Ag.n.), Ari., Arn., As. Br.,

Asp., Bov., Calc., Ca. p., Cbl. x., Carl., cod., clch., Col., Cur., e. pu., Fe. i., Fe.

m., Fe. p., Hlon., Iod., k. ac., k.br., Kis., Lc. v., Lc. v. d., lc. x., Lrs., lcs., Mag.

s., med., Mos., Mur., na. m., Na. p., Na.s., Phas., Phlo., Ph. x., Pi.x., Plnt.,

Rat., Sac. l., Sac. o., Snc., Scil., Sec., Sil., Sti., Sul., Su. x., Syz., trx., Trl., ure.,

vic.30

6.3 AIMS AND OBJECTIVES OF THE STUDY

1. The comparative evaluation of the efficacy of both the Logico-

Utilitarian general repertories and clinical repertories.

2. Study of the clinical and general rubric from both in Logico-utilitarian

general repertories and clinical repertories in the management of type

2 diabetes mellitus.

3. To study the clinical presentation of type 2 diabetes Mellitus.

7. MATERIALS AND METHODS

7.1 PRIMARY SOURCE:

The subject for this study will be collected from OPD/IPD/ Rural camp of

Dr. B. D. Jatti Homoeopathic Medical College, Hospital and Post Graduate

Research Centre, Dharwad.

7.2 METHOD OF COLLECTION OF DATA (including sampling

procedure, if any,) Simple random sampling method.

Definition of study subject:

Subjects are considered on the basis of clinical presentations i.e., polydipsia,

polyuria, nocturia, weight loss, weakness, giddiness, blurring of vision,

muscle pain, cramps, routine investigation and urine analysis, duration of 6

weeks or more.

Following are inclusion criteria:

1) Subjects of age above 40 years and both sexes, and all ethnic groups.

2) Subjects clinically diagnosed to be having uncomplicated type 2

diabetes mellitus.

Following are Exclusion criteria:

1) Subjects with any complications of type 2 diabetes mellitus. Eg:

diabetic nephropathy, diabetic retinopathy, diabetic neuropathy etc.

2) Subjects with type 2 Diabetes Mellitus associated with any other

systemic disease.

Study sampling design:

Sampling size: Prevalence rate of diabetes mellitus in our OPD/ IPD/

peripheral OPD is 5%. Considering the 95% confidence interval at 5%

permissible error, sampling size worked out to be 76 cases. Since it is time

bound study, all admitted IPD, OPD and peripheral OPDs, cases are included

in my study period.

Study design:

The study will be a non-controlled longitudinal prospective case study.

Subjects will be selected on the basis of non-probability judgmental sampling

technique from the population of type 2 DM patients attending the OPD or

admitted to the IPD or attending peripheral OPD.

Follow up:

Patients will be seen every 15 days for 3 months and then monthly once till

the end of study period. Examination is done periodically.

Parameters used are:

Change in Clinical Findings like the Presenting Symptoms, Signs and

Investigations.

A) Controlled: Patient feels relief in the presenting symptoms either

in intensity and/ or in frequency and both fasting and post prandial

blood sugars controlled till end of the study.

B) Improved. Patient feels better of symptoms and blood sugars are

controlled but complaints reappears again and again i.e. both

fasting and post prandial blood sugar increases again and again.

C) No improvement:

i) Status quo (neither relief of symptoms nor both fasting and

post prandial blood sugars are not controlled).

ii) Drop out cases.

Study period: Nov-2010 to Nov-2012.

Statistical tests:

Appropriate test will be used depending upon the data available at the end of

the study.

7.3 DOES THE STUDY REQUIRE ANY INVESTIGATIONS OR INTERVENTIONS TO BE CONDUCTED ON PATIENTS OR OTHER HUMANS OR ANIMALS? IF SO DESCRIBE BRIEFLY.

The study requires following investigations to be conducted on patients. The study requires any following investigations to be conducted on patients.

1) FBG2) PPBG3) GLYCOSYLATED HAEMOGLOBIN4) RBS (When needed)5) URINE SUGAR IN FBG & PPBG6) URINE SUGAR IN RBS (When required)7) Routine urine and microscopic test to eliminate diabetic nephropathy 8) Eye examination (when necessary) to eliminate the diabetic

retinopathy7.4 HAS ETHICAL CLEARANCE BEEN OBTAINED FROM YOUR

INSTITUTION IN CASE OF 7.3? Yes, Ethical clearance has been obtained from the institution.

8 List of reference: 1. Walter J.B., I.C. Talbot. Walter and Israel General pathology. 7th Edition.

Edinburgh: Churchill livingstone; 1996. Page no: 593.

2. http://en.wikipedia.org

3. Carpenter, Griggs, losscolzo. Andreoli cecil Essential of medicine. 5th

Edition. Hart court India Private Ltd. New Delhi; 2001. Page No: 583.

4. Boon Nicholas A., Nicki R. Colledge. Brain. R. Walkder. Davidson’s

Principals and Practice of medicine. 20th Edition. Edinburgh: Churchill

livingstone; 2006 Page no:813.

5. Mohan Harsh. Text book of Pathology. 5th Edition. New Delhi: Jaypee

Brothers medical publishers (p) Ltd; 2005. Page no: 843.

6. Kaushik Sadhana. Insulin Strategies in type 1 and type 2 diabetes mellitus.

Current Medical Journal of India. Vol XIII/No.4; July 2007. Page no: 32.

7. Shah Siddharth N. API Text Book of Medicine. 7th Edition. Mumbai: The

Association of Physicians of India; 2003. Page No: 1097.

8. http://www.ijddc.com

9. Macroforlance Peter. S., Robin Reid, Robin Callender. Pathology

illustrated. 5th Edition. Edinburgh: Churchill livingstone; 2000. Page No:

659.

10. http://en.wikipedia.org



12. Boon Nicholas A., Nicki R. Colledge. Brain. R. Walkder. Davidson’s

Principals and Practice of medicine. 20th Edition. Edinburgh: Churchill

livingstone; 2006 Page no: 814.



14. Kausal Kamal, Rakesh Kaushal. Text book of practice of medicine with

Homoeopathic Therapeutics. Second revised and enlarged edition. New

Delhi: B. Jain Publishers (P) Ltd. reprint edition 2007: page no: 485.

15. Shah Siddharth N. API Text Book of Medicine. 7th Edition. Mumbai: The

http://en.wikipedia.org/

http://www.ijddc.com/

http://en.wikipedia.org/

Association of Physicians of India; 2003. Page No: 1111.

16. Paul Swapan. Dare to be non-diabetic. National Journal of Homoeopathy.

Mumbai: Vishpala parthasarathy. Vol.10/ No.10; Oct 2008. Page No: 15

17. Golwalla ASP. F., Sharukh A. Golwalla. Medicine for students. 22nd

Edition. Mumbai: Distributed by national book depot; 2008. Page No:440.

18. Kasper, Braunwald, Fauci, Hauci, Hauser, Longo, Jameson. Harrisons

Principles of Internal Medicines. 14th Edition. New York: Mc Graw-Hill

medical Publishing division; 1998: Page no: 2069.




20. Carpenter, Griggs, losscolzo. Andreoli cecil Essential of medicine. 5th

Edition. Hart court India Private Ltd. New Delhi; 2001. Page No:594-598.

21. Paul Swapan. Dare to be non-diabetic. National Journal of Homoeopathy.

Mumbai: Vishpala parthasarathy. Vol.10/ No.10; Oct 2008. Page No: 15

22. Firth John D., M.Gurnell. Medical master class-Endocrinology. London:

Royal college of physicians. Page No: 101.




24. Mathur K. N.. Diabetes Mellitus its diagnosis and Homoeopathic

treatment. New Delhi: B.Jain Publishers (P) Ltd; Reprint Edition 2008;

page no: 55.

25. Hughes R. The Principles and practice of homoeopathy. Reprint Edition.

New Delhi: B. Jain Publishers Pvt. Ltd; 1999. Page no:629-630.

26. Kent J. T. Repertory of the homoeopathic meteria medica with word

Index. 6th Edition. New Delhi: Indian Books and periodicals publishers;

August 2006. Page No: 691.

27. Boger. C. M. Boger Boenninghausen’s characteristics and Repertory. New

Delhi: B. Jain Publishers (p) Ltd; Reprint Edition 2008. Page No: 622.

28. Frederick Schroyens. Synthesis repertorium Homoeopathicum

syntheticum. Edition 8.1. New Delhi: B. Jain Publishers (p) Ltd;

December 2002. Page no.: 1767.

29. Boerick Oscar. E.. The clinical Repertory. 9th Edition. New Delhi: B. Jain

Publishers (P) Ltd; 2002. Page No: 828.

30. Clarke. J. H. A Clinical Repertory to the dictionary of Materia Medica.

New Delhi: B. Jain Publishers Pvt. Ltd.; Reprint edition 2001. Page

No:54.

9Signature of the candidate

10

Remarks of the guide

11 Name and Designation of (IN BLOCK LETTER)

11.1

Guide

Dr. SUDHANSU. S. MOHARANA, M.D. (Homoeo) PROFESSORPost Graduate Dept. of Repertory and Case taking, DBHP Sabah’s. Dr. B. D. Jatti Homoeopathic Medical College,Hospital & P.G. Research Center, D.C. Compound, Dharwad-580001.

11.2Signature

11.3Co-Guide (If any)

11.4Signature

11.5

Head of the Department

Dr. SUDHANSU. S. MOHARANA, M.D. (Homoeo) PROFESSOR Post Graduate Dept. of Repertory and Case taking, DBHP Sabah’s. Dr. B. D. Jatti Homoeopathic Medical College,Hospital & P.G. Research Center, D.C. Compound, Dharwad-580001.

11.6Signature

12Remarks of the Principal

12.1Signature

Documents

6 · Web viewDr. MINU. P. Post Graduate Student, Post Graduate Dept. of Repertory and Case taking, DBHP Sabah’s. Dr. B. D. Jatti Homoeopathic Medical College,