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8/13/2019 Anaphylactic shock.ppt
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Macam danpathofisiologi shock
Drg Yayun Siti Rochmah SpBM
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C (Circulation)
Assessment of organ perfusion
- Level of conciousness
- Skin color and temperature
- Pulse rate and character
- Urinary output
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SHOCK
An abnormality of the circulatory system
that result in inadequate organ perfusion
and tissue oxygenation
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DEFINITION SHOCK:- It is generalized
inadequency of blood flow throughout
the body to the extent that the body
tissue are damaged because of too
little flow, especially too little delivery ofoxygen and other nutrients to tissue cell.
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Batasan shock
GANGGUAN SIRKULASI
DISERTAI MENURUNNYAPERFUSI JARINGAN
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Shock indeks
NADI
TENSI(SISTOLE) = 0,5
SHOCK INDEKS = 1,0 BAHAYA
SHOCK INDEKS = 1,5 MANIFEST
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TYPES:-
SHOCK
HYPOVOLMIC CARDIOGENIC
ANAPHYLACTIC SEPTICEMIC NEUROGENIC
DISTRIBUTIVE
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CLASSIFICATION OF SHOCK
Trauma : - Haemorrhagic
- Non haemorrhagicCardiogenicTension pneumothoraxNeurogenic
Septic
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….. Classification of shock
Hypovolemic : - Haemorrhage- Diarrhoea
- Burn
Distributive
- Septic- Anaphylaxsis- Spinal cord injury
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….. Classification of shock
Cardiogenik :
- Arrytmias- Heart failure- Myocardial contusion / infarction
Obstructive- Tension pneumothorax
- Cardiac tamponade- Haemopneumothorax
Disscociative
- Profound anemia
- Co poisoning
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ETIOLOGY: 1)HYPOVOLMIC:>
>HAEMORRHAGE
>TRAUMA
>BURNS
>DEHYDRATION 2)SEPTIC:>
>Gm +ve SEPTICEMIA
>Gm -ve SEPTICEMIA
3)CARDIOGENIC:>
>DEFICIENCY OF EMPTYING
>DEFICIENCY OF FILLING
>OUTFLOW OBSTRUCTION
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Jenis shock
Shock dengan tahanan vaskuler
sistemik tinggi : syok kardiogenik,
syok hipovolemik
Shock dengan tahanan vaskular
sistemik rendah : syok septik, syok
anafilaktik
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CO = SV X F
preload C after loadEDV SVRVR
BP = CO X SVR
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FISIOLOGI SHOCK
CARDIAC
OUTPUTMENINGKAT
Mengurangi stimulasi baroreseptorA. Carotis dan arcus aorta :
kenaikan output sistem sarafsimpatis
1. Vena besar berkontraksi, vol.Darah sirkulasi meningkat,
cardiac output naik (Hk.Starling)
2. Takhikardia3. Vasokonstriksi arteriol, tahanan
perifer meningkat Tahanan sistemikvaskular meningkat
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PATOFISIOLOGI SHOCK
VOLUME DARAHBERKURANG
Kegiatan simpatis
meningkat
Iskemik splanknikIskemik renal
Depresi RES
ENZIM2 LISOSOMDILEPAS (PROTEOLITIK)
Toksin vasoaktifdilepas
Asidosis metabolik
Kontraksi arteri >>
Iskemia jaringan
Reaksi kardiogenikFaal renal, serebralberkurang, respirasi
menurun, faal kontraksi jantung<<
Kerusakan endotel,cairan hilang,
kelainan metabolik
Shock Syndrome
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PATHOGENESIS
HYPOVOLUMIC & SEPTIC
DECREASED EFFECIENCY CIRCULATING VOLUME
DECREASED VENUS RETURN TO HEART
DECREASED CARDIAC OUTPUT
DECREASED BLOOD FLOW
DECREASED SUPPLY OF OXYGEN
ANOROXIA
SHOCK
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CARDIOGENIC
CARDIAC OUTPUT
DECREASED BLOOD FLOW
DECREASED SUPPLY OF OXYGEN
ANOROXIA
SHOCK
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SHOCK RECOGNITION AND MANAGEMENT
• Recognize signs of inadequate perfusion
and oxygenation
• Identify probable cause• Restore perfusion
• Re-evaluate patient response
• Immediate involvement by specialists
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CLINICAL FEATURES:-
>RAPID PULSE (TAKHIKARDIA)
>PULSE MAY BE WEAK (INDEKS !)
>RAPID BREATHING(TAKHIPNEU)
>ANXIETY>COOL,CALMMY SKIN (CLAMMY)
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>WEAKNESS
>PALE SKIN COLOR(PALLOR)
>SWEATING,MOIST SKIN
>DECREASED OR NO URINE
OUTPUT
>LOW BLOOD PRESSURE
>CONFUSION>UNCONCIOUSNESS
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CLINICAL SIGNS
1231. Tachycardia
2. Vasoconstriction
3. cardiac output
4. Narrow pulse pressure
5. MAP
6. blood flow
Remember :
Compensatory mechanisms
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Akibat shock pada tubuh
Asidosis laktat dan gangguan pernafasan
karena kompensasinya
Adanya PH rendah menekan kontraktilitasmiokard, tensi turun
Vasodilatasi sebagai akibat akhir : hipotensi
Gagal organ spesifik otak : kesadaran turun
Gangguan perfusi ginjal : GGA Perfusi usus menurun, basorbsi toksin bakteri
usus : hipotensi, sepsis, “shock splanknik”
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Shock anaphylactic
Terjadi karena pelepasan histamin
Berakibat edema paru akut,
bronkospasme dan edeme mukosasehingga menyebabkan obstruksi jalannafas akut
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terapi
Oksigen
Infus
Adrenalin
Kortikosteroid
Intubasi dan ventilasi
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