Dr Shuja Presentation

Embed Size (px)

Citation preview

  • 8/2/2019 Dr Shuja Presentation

    1/54

  • 8/2/2019 Dr Shuja Presentation

    2/54

    ODONTOGENICINFECTIONS OF THE

    MAXILLO FACIAL &

    NECK REGION

  • 8/2/2019 Dr Shuja Presentation

    3/54

    ETIOLOGY

    1. Pulp disease.

    2. Periodontal disease.

    3. Secondarily infected cysts or odontomes.

    4. Remaining root fragment.

    5. Residual infection.

    6. Pericoronal infection.

  • 8/2/2019 Dr Shuja Presentation

    4/54

    Bacteriology

    Aerobic 7% G +ve Cocci ( Strep, Staph) & G-ve cocci (Neisseria)

    G +ve rods (Corny ) & G-ve rods

    Anaerobic 33% G +ve Cocci ( Strep, Pseudo strep) & G-ve

    cocci(veiollonela)

    G +ve rods (Lacto, Actino ) & G-ve rods (Bacteriodes)

    Mixed 60%

  • 8/2/2019 Dr Shuja Presentation

    5/54

    TYPES

    ACUTE

    In the acute stage infection may remain intrabony or spread into soft tissues in

    following clinical forms:1. Abscess:

    1.Circumscribed collection of pus in a pathologicaltissue space.

    2.Thick walled cavity containing pus.

    3.Aerobes & anaerobes--- large accumulation ofpus--- pointing & drainage.

  • 8/2/2019 Dr Shuja Presentation

    6/54

    AbscessAbscess

  • 8/2/2019 Dr Shuja Presentation

    7/54

  • 8/2/2019 Dr Shuja Presentation

    8/54

    2. Cellulitis:

    1.This is spreading infection of loose CT.

    1.It is a diffuse, erythematous, mucosal or cutaneous

    infection.

    2.It is result of streptococci & does not result in large

    accumulation of pus.

    3.Streptococci produce streptokinase, hyaluronidase.

  • 8/2/2019 Dr Shuja Presentation

    9/54

  • 8/2/2019 Dr Shuja Presentation

    10/54

    3.Fulminating infections:

    1.Spread of infection in various primary spaces in the

    orofacial region.

    2.Here secondary spaces along the pathway of least

    resistance are involved.

    3.Spread of deep cervical spaces and beyond.

  • 8/2/2019 Dr Shuja Presentation

    11/54

    Acute Peri Apical Abscess1

    This is due to vascular dilatation, an exudate of

    neutrophil leucocytes & oedema in the peri apical

    region.

    It is due to persistent irritation from chronic pulpor acute virulent infection, or less host

    resistance.

  • 8/2/2019 Dr Shuja Presentation

    12/54

    Etiology Acute Peri Apical Abscess

    Infective necrosis of pulp

    Caries.

    Traumatic exposure.

    Traumatic necrosis

    Blow on teeth.Mechanical &

    Chemical

  • 8/2/2019 Dr Shuja Presentation

    13/54

    CLINICAL FEATURESAcute Peri Apical Abscess

    1- History of previous pulpitis.

    2- Carious or heavily filled tooth.

    3- Tender and felt extruded in socket.

    4- When pus has formed severethrobbing

    pain5- sensitive to percussion.

    6- Over lying gum may or may not be

    swollen

  • 8/2/2019 Dr Shuja Presentation

    14/54

    TREATMENTAcute Peri Apical Abscess

    Antibiotics ,Analgesics & Drainage through

    pulp chamber.

    Extraction or endodontic treatment.

  • 8/2/2019 Dr Shuja Presentation

    15/54

    Acute Dento Alveolar Abscess

    When pus does not remain confined to the peri

    apical region.

    It perforates the cortex and comes to lie under

    periosteum--- SUB PERIOSTEAL ABSCESS.

    The perforating abscess come into the soft tissues

    then called as ACUTE DENTOALVEOLAR ABSCESS

  • 8/2/2019 Dr Shuja Presentation

    16/54

    CLINICAL FEATURESAcute Dento Alveolar Abscess

    Pain depend on the stage of disease.

    Sub mucosal swelling (Intra Oral).

    Facial swelling (extra Oral).

    Fluctuation may come after few days.

    If untreated may point or burst producing adischarging sinus.

  • 8/2/2019 Dr Shuja Presentation

    17/54

  • 8/2/2019 Dr Shuja Presentation

    18/54

    Radiographic featuresAcute Dento Alveolar Abscess

    Little informative in acute phase except

    little widening of periodontal ligament.

    But previous pathology if present will be

    seen.

  • 8/2/2019 Dr Shuja Presentation

    19/54

  • 8/2/2019 Dr Shuja Presentation

    20/54

    Treatment

    Acute Dento Alveolar Abscess

    Same i.e. endo- or ext-.

    Intra or extra oral drainage

  • 8/2/2019 Dr Shuja Presentation

    21/54

    CHRONIC PERI APICAL

    PERIODONTITIS1

    When the irritation in the peri apicaltissues persists either due to, incomplete resolution

    In complete treatment of acute periodontitis orpulpitis leading to necrotic pulp

    a forgotten blow or massive fillings orunsuccessful R.C.T lead to chronic

    periodontitis. This goes on painlessly and become

    chronic

  • 8/2/2019 Dr Shuja Presentation

    22/54

  • 8/2/2019 Dr Shuja Presentation

    23/54

    Skin Sinus Due Chronic infectionSkin Sinus Due Chronic infection

    from deciduous molarfrom deciduous molar

  • 8/2/2019 Dr Shuja Presentation

    24/54

    FACIAL SPACE INFECTIONFACIAL SPACE INFECTION

    Fascialined areas-- potential spaces thatdo not exist in healthy persons.

    Filled by pus or exudation during infection.

    Neurovascular structure - compartments.

    Loose areolar CT------ Clefts

  • 8/2/2019 Dr Shuja Presentation

    25/54

    Primary facial spacesPrimary facial spaces

    Primary spaces are adjacent to tooth bearing

    area & are directly involved by infection.

    Primary maxillary spaces.

    Canine Buccal

    Infratemporal.

    Primary mandibular spaces. Submental.

    Buccal.

    Submandibular.

    Sublingual.

  • 8/2/2019 Dr Shuja Presentation

    26/54

  • 8/2/2019 Dr Shuja Presentation

    27/54

  • 8/2/2019 Dr Shuja Presentation

    28/54

  • 8/2/2019 Dr Shuja Presentation

    29/54

  • 8/2/2019 Dr Shuja Presentation

    30/54

  • 8/2/2019 Dr Shuja Presentation

    31/54

  • 8/2/2019 Dr Shuja Presentation

    32/54

  • 8/2/2019 Dr Shuja Presentation

    33/54

    Secondary spacesSecondary spaces

    MASTICATORY SPACES

    Masseteric.

    Pterygomandibular.

    Superficial & deep temporal.

  • 8/2/2019 Dr Shuja Presentation

    34/54

  • 8/2/2019 Dr Shuja Presentation

    35/54

    CERVICAL SPACES

    Lateral pharyngeal

    Retropharyngeal

    Prevertebral

  • 8/2/2019 Dr Shuja Presentation

    36/54

  • 8/2/2019 Dr Shuja Presentation

    37/54

  • 8/2/2019 Dr Shuja Presentation

    38/54

    High Risk Infections or LethalHigh Risk Infections or Lethal

    complicationscomplications

    Orbital & peri orbital cellulitis.

    Cavernous sinus thrombosis

    Ludwigs angina

    Cervical cellulitis ( Lung Abscess &

    Mediastinitis)

  • 8/2/2019 Dr Shuja Presentation

    39/54

    Orbital & Periorbital cellulitisOrbital & Periorbital cellulitis

  • 8/2/2019 Dr Shuja Presentation

    40/54

  • 8/2/2019 Dr Shuja Presentation

    41/54

    Cavernous Sinus ThrombosisCavernous Sinus Thrombosis

  • 8/2/2019 Dr Shuja Presentation

    42/54

  • 8/2/2019 Dr Shuja Presentation

    43/54

  • 8/2/2019 Dr Shuja Presentation

    44/54

    LUDWIG S ANGINALUDWIG S ANGINA

  • 8/2/2019 Dr Shuja Presentation

    45/54

  • 8/2/2019 Dr Shuja Presentation

    46/54

    Cervical CellulitisCervical Cellulitis

  • 8/2/2019 Dr Shuja Presentation

    47/54

  • 8/2/2019 Dr Shuja Presentation

    48/54

    Principles of managementPrinciples of management

    Determine the severity of infection

    Evaluate the state of patients host defensemechanism

    Determine , whether treated by GDP or refer tospecialist

    Appropriate antibiotic & their properadministration

    Treat infection surgically

    Diet & i-v fluids

    Evaluate pts frequently

  • 8/2/2019 Dr Shuja Presentation

    49/54

    Surgical ManagementSurgical Management

  • 8/2/2019 Dr Shuja Presentation

    50/54

  • 8/2/2019 Dr Shuja Presentation

    51/54

  • 8/2/2019 Dr Shuja Presentation

    52/54

  • 8/2/2019 Dr Shuja Presentation

    53/54

  • 8/2/2019 Dr Shuja Presentation

    54/54