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Pediatric ECG:A practical Approach Dr ANIL S.R Consultant Pediatric Cardiologist MIMS Calicut

Ecg pediatric

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Pediatric ECG:A practical Approach

Dr ANIL S.R

Consultant Pediatric Cardiologist MIMS Calicut

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• A wriggling neonate• A crying infant• An apprehensive child• A ticklish adolescent

ECG in pediatric Practice

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• Evolution of ECG- Neonate to Adolescent• Identify an abnormal ECG at a given age• ECG and Common congenital heart diseases• ECG abnormalities after surgical interventions• Pediatric arrhythmias

ECG in pediatric Practice

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Normal neonate

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Normal infant

Low voltages of QRS in precordial and limb leads

Low T wave voltages

RV dominance

Right Axis deviation of upto 180 degree

Upright T waves in right precordial leads- Ist week of life

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Normal Child

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Normal adolescent

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Age related changes

• HR decreases• All durations and intervals increases• RV dominance gradually changes to LV

dominance• QRS axis- less rightward shift• R wave in RPLs decreases and in LPLs

it increases. This is reverse for S wave

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LV/RV Mass ratio

30w 1.2:1

33w 1:1

Birth 0.8:1

6m 2:1

Adult 2.5:1

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Preterm infants

Low voltages of QRS

Low T wave voltages

Less RV dominance

Left Axis deviation

Short PR, QRS and QT intervals

More ECG variability

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Leads

Bipolar leads : I , II, IIIUnipolar leads : aVR, aVL, aVF V1 to V6

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LEADS: Bipolar leads : I , II, III

Lead I

Lead II Lead III

LARA

LL

• Selected by Einthoven• Records PD between two

points• Rt leg electrode- ground

wire• II = 1 + 111 (Kirchoff’s Law)

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Laws of ECG

• Depolarization is towards the +ve of a lead= +Ve Deflection

• Depolarization is towards the -ve of a lead= -Ve Deflection

• Depolarization is perpendicular to the lead= Biphasic or No Deflection

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Right atrial Enlargement

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Left atrial Enlargement

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Right Ventricular Hypertrophy

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Left Ventricular Hypertrophy

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Common congenital heart defects

Left to right shuntsStenotic lesions

Cyanotic heart diseases

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Secundum ASD

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Primum ASD.... Left axis deviation and Q in aVL

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Sinus venosus ASD ... Note inverted P waves in III

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Small VSD in a young child ..... No LV forces

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Large VSD with biventricular forces ....... Note Katz Wachtel phenomenon

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VSD Eisenmenger......note loss of q wave in V6

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Large Inlet VSD, ......note left axis

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PDA ... Note prominent LV forces

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PDA in Rubella syndrome patients

Left axis deviation due to injury to conduction tissues

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AS with LVH, note strain pattern

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AS with significant LVH and strain pattern

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Coarctation in infancy ..... Since PAH is common, RV forces are dominant

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Coarctation

• In infancy, due to pulmonary hypertension, Right axis and RVH are common

• In older patients, LVH occurs

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Coarctation in an older child

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Valvar PS

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Sick TOF

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TOF - Transition occurs in V1Importance of right chest leads

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D-TGA in a older child

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Corrected TGA with large VSD.......Note septal Q in right sided leads, no Q in V6

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Common atrium.....mimics a primum ASD, but patient is blue

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Ebstein’s anomaly of tricuspid valve....... Striking RA forces, splintered qrs in V1

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TAPVC

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TAPVC ... ECG shows features of PAH

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Tricuspid atresia..... Left axis and LV forces

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Pulmonary atresia, Intact IVS..... Again LV forces but axis is not leftward

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Single ventricle - RAD with LV forces

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In a cyanotic child:• Right ventricular forces: TOF

TOF with pulmonary atresiaTGATAPVC, Common atrium

• Left ventricular forces:Tricuspid atresiaPulmonary atresia with

IVSHypoplastic right heartSingle ventricleEbsteins

• Bi-ventricular forces: TruncusDORV

• Normal ECG: Pulmonary AV fistula Anomalous systemic venous return

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Provides valuable clues in diagnosis

Invaluable in arrhythmia

Comprehensive assessment before surgery

Read and analyze ECGs

Conclusion