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 Introduction to Movement Dysfunction & Kinetic Chain Assessment Introduction to Movement Dysfunction & Kinetic Chain Assessment Jim Thornton, MA, ATC, NASM-PES Head Athleti c Tra iner Clarion University of PA 2006 EATA Annual Meeting Philadelphia, PA

Kinetic Chain Assessment Kinetic Chain Assessment

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Introduction to Movement Dysfunction&

Kinetic Chain Assessment

Introduction to Movement Dysfunction&

Kinetic Chain Assessment

Jim Thornton, MA, ATC, NASM-PESHead Athletic Trainer – Clarion University of PA

2006 EATA Annual MeetingPhiladelphia, PA

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Objectives

1. Define common human movement imbalances

2. Explain the evidence for common imbalances

3. Define Corrective Exercisea. What is it?

 b. Why is it used?

c. How do you apply it?

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Paradigm Shift• Injury prevention is the primary goal of all

 performance enhancement programs – The primary emphasis of traditional “Sports

Conditioning” programs has been on concentric

force production (how much can you lift) primarilyin the sagittal plane, in a controlled environment

• This may not be the best way to prevent injuries

 – Also, many athletes have pre-existing injuries thatmust be addressed prior to advanced performance

enhancement training

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Foot and Ankle Imbalances• Lateral ankle sprains are the most common

injury suffered in sports during sports participation (Safran, 1999)

 – Denegar, et al 2002 in a retrospective studydemonstrated altered arthrokinematic movement of

the talus (decreased posterior glide) even thoughrange of motion was restored 

 – This is very important because limited posterior

glide of the talus may lead to decreased dorsiflexion• Limited dorsiflexion may lead to other compensations

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Foot and Ankle Imbalances• Bullock-Saxton et al 1994 demonstrated

decreased gluteus maximus muscle activation post ankle sprain

• Beckman et al 1995 demonstrated decreasedgluteus medius muscle activation post anklesprain

 – If an athlete begins an integrated training programand has muscle imbalances in the hip complex

secondary to an ankle sprain, then furthercompensations and possible injury may occur 

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Knee and Hip Imbalances• It has been recognized that the patellofemoral

 joint may be influenced by the segmentalinteractions of the lower extremity

(Fredericson, Powers)

 – Abnormal motions of the tibia and femur in thetransverse and frontal planes are believed to have an

effect on the patellofemoral joint (Ford, Nyland)

 – This abnormal motion may be caused by weaknessin the hip abductors and external rotators (Ireland)

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Knee and Hip Imbalance• Recent kinetic analysis of running reveals that,

although the knee joint primarily moves in the sagittal

 plane, the knee is also subject to significant frontal andtransverse plane moments (McClay)

 – In the absence of sufficient proximal hip strength, the femurmay adduct and internally rotate, further increasing thelateral patellar contact pressure (Lee)

 – (Fredericson) demonstrated that distance runners with ITBSyndrome had weaker hip abduction strength than thecontrol group and their unaffected leg

 – (Ford and Hewett) demonstrated that female athletes landedwith greater total valgus (femur adduction and tibiaabduction) than male athletes and may lead to ACL Tears

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Lumbo-Pelvic-Hip Complex Imbalance• Low back pain is very common in the active

 population (Nadler)

 – In a cross-sectional study of 100 patients (Cibulka)demonstrated unilateral hip rotation ROM asymmetry in patients with SI joint regional pain

 – Hodges and Richardson 1996 reported that slow speed of

contraction of the transverse abdominus during arm and legmovements was well correlated with LBP

 – O’Sullivan et al 1997 found that synergist substitution of therectus abdominus for the agonist transverse abdominusduring the abdominal drawing-in maneuver suggesting lessefficient intersegmental stabilizing mechanisms and greatershear forces at the intervertebral joints

 – Hides et al 1994 demonstrated unilateral atrophy of themultifidus in patients with low back pain

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Current Concepts in Human Movement

Science

• Two distinct yet interdependent muscle systems

 – Stabilization System (Stabilizers)• Primarily involved in joint support

• Broad spectrum of attachments

• Prone to inhibition and weakness

 – Movement System (Mobilizers)

• Superficial muscles associated with extremity movement

• Prone to overactivity and tightness

• Categorized into four common sub-systems

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Understanding Muscle Function

• Stabilizers

 – Gluteus Medius

 – Transverse Abdominus

 – Internal Oblique

 – Multifidus – Lower Trapezius

 – Serratus Anterior 

 – Rotator Cuff  – Deep Neck Flexors

• Mobilizers

 – Gastrocnemius – Quadriceps

 – Hamstrings

 – Adductors – Hip Flexors

 – Rectus Abdominus

 – Erector Spinae – Latissimus Dorsi

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Understanding Muscle Function

• Stabilizers

 – Delayed recruitment

 – Reacts to pain and

 pathology with inhibition

 – Loss of jointstabilizations

 – Leads to synergistic

dominance

• Mobilizers

 – Become overactive

 – Reacts to pain and

 pathology with spasm

 – Develops myofascialadhesions which alter

(LTR, ATK)

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Lateral Sub-System• Muscles Involved 

 – Gluteus Medius

 – Tensor Fascia Latae

 – Adductor Complex

 – Quadratus Lumborum

• Function

 – Frontal plane stability

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Posterior-Oblique Sub-System• Muscles Involved 

 – Latissimus Dorsi

 – Thoracolumbar Fascia

 – Gluteus Maximus

• Function

 – Transverse plane

stabilization

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Anterior-Oblique Sub-System• Muscles Involved 

 – Internal Oblique and Hip

Adductor Complex

 – External Oblique and Hip

External Rotators

• Function – Transverse plane

stabilization

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Deep Longitudinal Sub-System• Muscles Involved 

 – Erector Spinae

 – Thoracolumbar Fascia

 – Gluteus Maximus

 – Biceps Femoris

 – Peroneals

• Function

 – Force transmission from

the ground to the trunk 

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Kinesiopathological Model

• If one component of the human movement

system is not functioning optimally, than

PREDICTABLE PATTERNS of dysfunctionwill develop and initiate the cumulative injury

cycle

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Myofascial

Neuromuscular

Control

SensorimotorIntegration

 Articular Neural

Kinetic Chain

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Length-tension Relationships• There is a direct

relationship between

tension development in amuscle and the length of

the muscle

 – There is an optimumlength at which a muscle

can generate maximum

tension

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Force-couple Relationships

• Muscles work synergistically to reduce force,

dynamically stabilize and concentrically

 produce force in all three planes of motion

• The CNS is designed to optimize the selection

of muscle synergies

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Arthrokinematics• Articular partners have

 predictable movement

 patterns

 – Roll

 – Slide

 – Glide

 – Translation

• These patterns are

controlled by the CNS and

the surrounding muscles of

the kinetic chain

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 Altered Length-

tension Relationships

Initiation of theCumulative

Injury Cycle

Tissue Fatigue

 Altered Sensorimotor Integration

 Altered Force-

Couple Relationships

 Altered

 Arthrokinematics

DYSFUNCTION

 Altered Neuromuscular 

Efficiency

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Results from Human Movement

System Impairment

• Reciprocal Inhibition

• Synergistic Dominance

• Arthrokinetic Inhibition

• Relative Flexibility

• Pattern Overload 

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Reciprocal Inhibition

• Increased neural drive or

decreased extensibility

of an agonist will

decrease the neural drive

to the antagonist

 – Leads to synergistic

dominance

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Synergistic Dominance

• The NMS phenomenon

that occurs whensynergists and stabilizers

compensate for prime

movers duringfunctional movement

 patterns

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Relative Flexibility

• The Human Movement

System will take the

 path of least resistance

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Pattern Overload 

• Repetitive recruitment of

the same muscle fibers,in the same ROM/Plane

of motion and at the

same speed createstissue overload and

eventually injury

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Kinesiopatholocigal Model

Low Back Extension

Lengthened ShortenedJoint

Dysfunction

Gluteus Maximus Abdominal Complex

Erector SpinaeHip Flexors

Lumbar SI-Joint

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Integrated Training

General Guidelines:

• Identify all kinetic-chain imbalances.

• Correct all kinetic chain imbalances

• Develop proper structural integrity of the kinetic chain before activity-

specific training.

• Integrate functional movements in the plane of motion, range of

motion and speed of motion that replicates the training activity

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What is the Solution?

Identify Causative Factors

Correct Weak Links

ReconditionBigger Engines or Better

Brakes

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ICE: Integrated Corrective Exercise

• Identify the kinetic chain Imbalance responsible for the

movement inefficiency and the biomechanical overload 

• Correct the Imbalance – Inhibit the overactive

• Self-Myofascial Release

 – Lengthen the overactive

• Static Stretching – Activate the under-active

• Active-Isolated Muscle Activation

 – Integrate functional movement patterns

• Integrated Isolation

• Empower your client

 – Give your client an individualized corrective exercise plan

 – Give your client an individualized Fitness and/or Performance

Enhancement Program

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Identifying the Weak Link:

• Posture

• Gait

• Flexibility assessment• Neuromuscular assessment

• Overhead-squat test

• Single-leg balance excursion

• Single-leg squat test

• Multiplanar lunge test

• Multiplanar step-up test

• Push-up test• Overhead medicine-ball

throw

• Multiplanar vertical jump/hop

• Multiplanar horizontal jump/hop

• Shark skill test

• Multiplanar cone jump/hop

test• Speed tests

 – Straight-ahead speed

 – Lateral speed and agility

 – Sport-specific – Speed endurance

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Transitional Movement Assessment

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Single-leg Squat Assessment

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The Overhead Squat

1. Feet

2. Knees

3. Hips (Lumbar Spine)

4. Shoulders

5. Head  

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 Normal Movement Assessment

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Can You Guess the Chief Complaint?

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What are Contributing Factors?

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Foot & Ankle: Foot Turns Out

 Normal Abnormal

Foot Turns Out: Note the 1st MTP Joint in relation to the medial

malleolus. In a normal foot the 1st MTP joint will appear along the same

 plane as the medial malleolus. However in a foot that is turned out the 1st

MTP joint will appear lateral to the medial malleolus.

StraightExternal

Rotation

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Foot & Ankle: Foot Flattens

 Normal Abnormal

Foot Flattens: Note the height of the longitudinal arch of the foot. It should be in a neutral

 position with a slight curve distinguishable and if the foot flattens it will appear to be flat

along the floor. Another indicator of the foot flattening is the Achilles tendon. Note in the

neutral picture how the tendon is straight, however when the foot flattens note the lateral

angle that is produced by the Achilles tendon.

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Foot & Ankle: Heel of Foot Rises

 Normal Abnormal

Heel of Foot Rises: Note the heel of the foot rising off of the floor. If the

heel stays firmly planted on the floor then there is no abnormality.However any rise of the foot from the floor indicates an abnormal

movement pattern.

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Foot & Ankle: Single-Leg Foot Flattens

 Normal Abnormal

Foot Flattens Single-leg: Note the height of the longitudinal arch of the foot. It should be in

a neutral position with a slight curve distinguishable and if the foot flattens it will appear to be flat along the floor. Another indicator of the foot flattening is the medial malleolus will

appear larger than the lateral malleolus. In the neutral picture both medial and lateral

malleolus are even.

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Knee: Moves Inward 

 Normal Abnormal

Knee Moves Inward: Note a line drawn from the patellar tendon which bisects theankle. This line should be perpendicular to the ground. If the line is leaning

toward the midline of the body then the knee is moving inward.

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Knee: Moves Outward 

 Normal Abnormal

Knee Moves Outward: Note a line drawn from the patellar tendon which bisectsthe ankle. This line should be perpendicular to the ground. If the line is leaning

away from the midline of the body then the knee is moving outward.

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Knee: Single-leg Moves Inward 

 Normal Abnormal

Single-leg Knee Moves Inward: Note a line drawn from the patellar tendon which bisects the ankle. This line should be perpendicular to the ground. If the line is

leaning toward the midline of the body then the knee is moving inward.

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K Si l l M O t d

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Knee: Single-leg Moves Outward 

 Normal Abnormal

Single-leg Knee Moves Outward: Note a line drawn from the patellar tendonwhich bisects the ankle. This line should be perpendicular to the ground. If the line

is leaning away from the midline of the body then the knee is moving outward.

LPHC L B k R d

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LPHC: Low Back Rounds

 Normal Abnormal

Low Back Rounds: Take notice of the area from approximately the mid back through the Sacral Complex. If the area is rounding then this area

will appear as a thoracic or convex curve.

LPHC L B k A h

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LPHC: Low Back Arches

 Normal Abnormal

Low Back Arches: Take notice of the area from approximately the mid back through the Sacral Complex. If the area is arched then this area will

appear with an excessive lumbar or convex curve.

LPHC E i F d L

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LPHC: Excessive Forward Lean

 Normal Abnormal

Excessive Forward Lean: Imaginary lines that are created by the shins and torso

of the client if extended out should remain parallel. If these lines would cross

immediately or shortly after extending them then the person does have excessive

forward lean.

LPHC: Weight Shift

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LPHC: Weight Shift

 Normal Abnormal

Weight Shift: Taking a line extending from the cervical spine through the thoracic and

lumbar spine that is parallel to the ground should bisect the LPHC resulting in equal parts

falling on either side of the line. If the LPHC is split unevenly resulting in a larger

 percentage on one side of the line then there is a weight shift on the side of the line that has

the larger percentage of the LPHC.

LPHC: Single leg Lateral Hip Shift

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LPHC: Single-leg Lateral Hip Shift

 Normal Abnormal

Single-leg Lateral Hip Shift: Taking a line originating from the patellar tendon

and bisecting the quadriceps should be parallel to the ground. If the line moves

away from the midline then there is a lateral hip shift.

Upper Body: Arms Fall Forward

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Upper Body: Arms Fall Forward 

 Normal Abnormal

Arms Fall Forward: A line bisecting the torso and head should be noted. If this

line travels parallel along the arms and the arms cover the ears of the subject then

there are no abnormalities present. If the line does not parallel the arms and you

can see the ears then the arms have fallen forward.

Dynamic Assessments

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Dynamic Assessments

Valid and Reliable Tests

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Valid and Reliable Tests

• Orthopedic Assessment

(Magee)

• Muscle lengthassessment with

Goniometer (Brosseau,

 Norkin)• Single-leg Balance

Excursion Test

(Olmsted)

• Overhead Medicine Ball

Throw (Stockburger)

• Vertical Jump (Manske)

• Single-leg Vertical Hop

(Manske)

• Horizontal Jump

(Manske)

• Single-leg horizontal

hop (Manske)

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So, once you find it…how will you

address it?

ICE: Integrated Corrective Exercise

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ICE: Integrated Corrective Exercise

• Identify the kinetic chain Imbalance responsible for the

movement inefficiency and the biomechanical overload 

• Correct the Imbalance

 – Inhibit the overactive

• Self-Myofascial Release

 – Lengthen the overactive

• Static Stretching

 – Activate the under-active

• Active-Isolated Muscle Activation

 – Integrate functional movement patterns

• Integrated Isolation

• Empower your client

 – Give your client an individualized corrective exercise plan

 – Give your client an individualized Fitness and/or PerformanceEnhancement Program

A Deeper Look at Dysfunction

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A Deeper Look at Dysfunction

ICE: Integrated Corrective Exercise

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ICE: Integrated Corrective Exercise

• Identify the kinetic chain Imbalance responsible for the

movement inefficiency and the biomechanical overload 

• Correct the Imbalance

 – Inhibit the overactive

• Self-Myofascial Release

 – Lengthen the overactive

• Static Stretching

 – Activate the under-active

• Active-Isolated Muscle Activation

 – Integrate functional movement patterns

• Integrated Isolation

• Empower your client

 – Give your client an individualized corrective exercise plan

 – Give your client an individualized Fitness and/or PerformanceEnhancement Program

Where are they going now?

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Where are they going now?

• A comprehensive training approach that strivesto improve all components necessary to allow

each individual to achieve optimum performance (Clark 2000, Kraemer 2004)

 – Flexibility

 – Core Strength – Neuromuscular Efficiency

 – Power 

 – Strength – Cardiorespiratory Efficiency

Summary

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Summary

• Must appreciate movement as a whole when looking at postural& functional assessments

• Identify the weak link 

• Provide isolated exercise to increase motor unit recruitment – Can only recruit motor units to the degree of dynamic joint stabilization

• Integrate to CNS by:

 – Multiplanar exercise selection – Variable speeds

 – ROM

 – Resistance (mode, frequency)

 – Acute variables (reps, sets, intensity, tempo, rest interval)

• Progress in training systematically to safely achieve goals

Further Reading Suggestions

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Further Reading Suggestions

• Professional Development – Kinesiology of the Musculoskeletal System (Neumann 2002)

 – Diagnosis and Treatment of Movement Impairment Syndromes(Sahrmann 2001)

 – Foundations of Clinical Research, 2nd ed (Portney & Watkins 1999)

 – Evidence-Based Medicine, 2nd ed (Sacket 2000)

 – Performance Enhancement Specialist (NASM 2001)

 – California University of Pennsylvania’s online, 12-month MS degree

• Personal Development – How Full is Your Bucket (Rath 2004)

 – The Leadership Challenge (Kouzes & Posner 2002)

 – Patch Adams (1998: 1 hr 56 min)