Low Back Pain and Disorders

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    Low Back Pain and

    Disorders o f the Lumbar

    Spine

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    Risk Facto rs

    Occupational Factors (lifting-pulling-pushing-slipping

    Sitting-vibration-dissatisfying)

    Patient -Related Facto rs

    Age(55years old) Sex

    Anthropometric Factors

    Postural Factors

    Muscle Strength

    Smoking

    Psychosocial

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    15>AGE>55

    TRAUMA

    PAIN AT NIGHT

    HISTORY OF CANCER

    WEIGHT LOSS DRUG ABUSE

    FECAL OR URIN INCONTINENCE

    SADDLE ANESTHESIA

    PROGRESSIVE MOTOR WEAKNESS

    MARKED MORNING STIFNESS

    PERIPHERAL JOINT INVOLVEMENT

    SKIN RASH-COLITIS-URETHRAL DISCHARGE

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    Mechanical Low Back Pain

    A descriptiveTERM

    It does not point to a single or particular cause.

    stress or strain to the back muscles, tendons, andligaments

    chronic, dul l , aching pain of vary ing intensi ty that affects

    the lower spine and m ight sp read to the butto cks.

    worsens dur ing the day.

    no associated neurological symptoms or signs,

    correction of static or dynamic postural abnormalitiesishelpful.

    An exercise program consisting of abdominal and backstrengthening exerciseis necessary, and patients often

    improve quickly.

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    Osteoarthritis

    occurs with aging and can begin during the third decade of life.

    If the disease is symptomatic, the associated pain is centered in the lower backand is often increased with movementof the spine.

    Range of motionof the spine may be limited. Pain is often relieved by rest.

    Hypertrophic changes and spurscan compress nerve rootsand causeaddit ion al radicu lar pain.

    Radiographs,particularly after the early stages, are diagnostic.

    When muscle support is poor, the application of an elastic supportto controlpain is advisable. The back support can be used for 6 weekswhile attemptsare made to improve the strength of the supporting muscles.

    Exercises include abdominal and back muscle strengthening exercises(preferably isometric exercise

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    Lumbar Disk Synd rome and

    Lumbosacral radiculopathies Lumbar disk syndrome is a common causeof acute, chronic, or recurrent low

    back pain, particularly in young to middle-aged men,but it also occurs inwomen, older persons, and even adolescents, especially if they are involved inst renuo us phys ical act iv ity.

    Overall, the mean age of the patient with lumbar disk herniation is the early 40s.

    Disk herniation can occur in the midline, but it often occurs to one side.

    Pain may be unilateral, bilateral, or bilateral but more prominent on one side. Irritation or compression of an adjacent nerve root can occur, as is often the case

    with laterally extruded ("squeezed toothpaste") disk herniations (Figs. 40-10 and40-11).

    Different degrees and types of disk herniation can occur.

    Bulging Disk. A bulge and convexity of the disk beyond the adjacent vertebral

    disk margins, but with an intact annulus fibrosus and Sharpey's fibers. Prolapsed Disk.The disk herniates posteriorly through an incomplete defect

    in the annulus fibrosus.

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    Extruded Disk.The disk herniates posteriorly through a complete defectin the annulus fibrosus

    Sequestered Disk. Part of the nucleus pulposus is extruded through acomplete defect in the annulus fibrosus and has lost continuity with thepresent nucleus pulposus.

    The pain often radiates into the buttock.the posterior thigh,and lateral calf

    or to lateral or medial malleoli(in cases of L5 or S1 radiculopathies).

    The pain radiates to the anterior thighin L3or L4 radiculopathies.

    When the disk is extruded, the low back pain is sometimes decreased or even

    relieved, but radicular limb symptomsbecome more prominent.

    The most common levels of lumbar disk protrusion, herniation, or extrusion, indecreasing order of frequency, are L5-Sl, L4-L5, L3-L4, and L2-L3.

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    Midline disk protrusion may cause low back pain but no significantradiculopathy.

    Large midline disk herniations can cause bilateralradiculopathiesor cauda equine syndromesevere enough toproduce sphincter problems.

    Upper lumbar radiculopathiesare less commonly caused by diskdisease. When upper lumbar radiculopathy is evaluated, otheretiologic factors, particularly neoplastic disease,should be ruled out.

    Examination of the back

    paraspinal muscle spasm, loss of lumbar lordosis,

    positive straight-leg-raising test, and, sometimes, crossedstraight-leg-raising signin cases of L5 or S1 radiculop athies.

    The chin-chestmaneuver might cause low back pain because ofupward traction on the cord and lower nerve roots.

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    Dorsiflexionof the foot can also cause stretching of the sciatic nerve andtherefore stretching of the attached tendon nerve root, leading to pain. Thesame findings may be noted when the patient tries to perform heel-walkingortries to bend forward.

    Coughing, sneezing, or straining causes an increasein abdominal

    pressure leading to distention of epidural and intervertebral veins.

    MRI has become a major diagnostic tool in the diagnosis of herniated lumbardisks

    It is also very useful for demonstrating several nondiscogenic entities.However, some herniated disks may be missed by MRI.

    Electromyographyis very helpful for localizing the level of involvement,determining whether root involvement is single or multiple, anddifferentiating a mu lt ip le root f rom a plexus les ion.

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    Most patients with discogenic low back pain respond to conservativemanagement.

    Operation is considered when definite radiculopathyand neurologicaldeficitsare present, especially when they are persistentor progressive.

    However, in the spectrum of discogenic low back pain, patients in this groupare a definite minority.

    Large midline disk protrusionswith cauda equinasyndromerequire urgenttreatmentand decompression. .

    However, in many patients with lumbar disk syndrome, the major difficulty islow back pain with only mild, slight, or no evidence of radiculopathy.

    The standard surgical procedure is open laminectomy and discectomy.

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    Posttraumatic Compression

    Fracture Posttraumatic compression fracture usually results from compressive

    flexion trauma.

    It can also occur spontaneouslyin patients with osteoporosis,osteomalacia, multiple myeloma, hyperparathyroidism, andmetastatic cancer.

    The upper lumbar spine or the middle to lower thoracicspine ismost commonly affected.

    The painusually is present immediately after the fracture and isoften localized.

    There may be accompanying paraspinal muscle spasm, and the

    range of motion of the related level of the spine is limited.

    Plain radiography, CT, MRI, or bone scanning may be needed-toestablish the diagnosis.

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    Posttraumatic Compression

    Fracture Sedative rehabilitative measures, especially in the acute phase,

    including application of cold for the first 24 to 48 hours, analgesics,and muscle relaxants, are often necessary.

    The pain can be managed with use of a back support, such as athoracolumbar supportthat functions on the basis of three-point

    contact. For provision of extension in cases of thoracic compression

    fractures, the three points of contact are the base of the sternum, thesymphysis pubis, and the lumbar spine, as in the Jewett brace

    When therapeutic exercisesare to be prescribed. extensionrather

    than flexion exercises should be utilized. Flexion exercises can increase the incidence of vertebral body

    wedging and compression fractures. Extension exercises are effectivefor strengthening back muscles at any age.

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    Spondylo lys is and

    Spondylo l is thesis

    Spondylolysis refers to a bony defect in the parsinterarticularis.

    Bilateral spondylolysisof the lumbar spine can lead to

    anterior slipping of the vertebral body on its adjacentvertebraand cause spondylolisthesis(in Greek, spondylomeans "vertebra" and listhesis means "sliding on a slipperysurface").

    Five types of spondy lol isthesis: (1) dysp last ic, (2)

    isthm ic (3) degenerative, (4) traumatic, and (5)patho log ical . To these catego ries, a sixth catego ry issom et imes added: postsurg ical or iatrogenicspondylol is thesis .

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    Spondylolysis or spondylolisthesis maycause back pain.

    However, the presence of a pars defect (spondylolysis) oreven spondylolisthesis in a patient with back pain does not

    necessari ly indicate a cause-and-effect relat ionsh ip.

    Spondylolisthesis is two to four times more common inmales.

    The pars defect is at L5 in 67% of persons, at L4 in 15%to 30%, and at L3 in 2%.

    It is rare in the cervical region.

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    Spondy lol is thesis can also cause

    compression o f nerve roots and lead to

    radicu lar pain or neuro log ical defic i ts in

    the lower extrem it ies.

    The lumbar lordos isis often exaggeratedin patients with spondylolisthesis, and range

    of motion of the lumbar spine may be

    limited.

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    For grades 1 and 2 spondylolisthesis and in olderpatients, nonsurgical treatment isrecommended.

    The physical therapeutic procedures consist ofapplication of heat and massagefor reduction ofpain and stiffness.

    Special attention can be given to reduc ing thet ightness o f the hip f lexo rs, hams tr ings, and

    Achi l les tendons.

    A program of stretching exercisesisrecommended. Dur ing stretch ing o f the backand lower extrem it ies, f lexion o f one hip

    (related knee) at a time helps reduce the strain

    on the lumbar sp ine.

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    LUMBAR CANAL STENOSIS

    DJD is the most common cause

    Pseudo claudication is the most common

    manifestation and often is bilateral Sensory symptoms(66%)

    LBP(70%)

    DTR AB.(50%) Weakness(40%)

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    Level of stenosis:

    L4-5 L3-4 L2-3 L5-S1 T12-L1

    Treatment:strengthening the abdominal and lumbarflexors

    Abdominal binder

    NSAIDS Surgical

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    AS.

    It mainly affects spine

    Sacroiliitis is usually the first manifestation

    Age 20-35

    Males>females

    HLA-B27+(80-90%)

    Morning stiffness & pain in the lower backimprove with activity

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    BACK EXT.EXC.

    Deep breathing Exc.

    Posture training

    ROM of the proximal joints Stretching EXC.

    Flexed Posture to be avoided

    HEAT & MASSAGE before EXC. Evaluation of chest expantion(

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    NEOPLASTIC DISEASE

    HALLMARK:pain at rest particularly noctural

    painBony metastasis is the most common

    cause

    Sometimes spInal metastasis is the first

    manifestation of canser

    Lung-prostate-breast

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    LBP in pregnancy

    Prevalence at 49-76%

    Risk factors:

    History of prior LBP

    Previous pregnancy related LBP

    LBP during menses

    Pregnant woman s age

    The risk of IBP during pregnancydecreases withage

    Pain has a peak at 36 weeks then decreases

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    Sacroiliac pain

    Nacturnal back pain

    Mechanical pains

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    LBP of pregnancy may not disappear with

    delivery

    Other cause Remission of RA

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    PROPHYLAXIS

    TREATMENT