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THE CARDIOVASCULAR  S  YSTEM CORONARY  ARTERY DISEASE By : Group 3

Presentasi 30 April Fixxxxxx

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THE CARDIOVASCULAR S YSTEM 

CORONARY ARTERY DISEASE By : Group 3

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 ANATOMY  AND PHYSIOLOGI THE 

CARDIOVASCULAR SYSTEM 

Heart

The heart is greatest in the cardiovascular system.

The heart has 2 sides:Right side & left side

The heart have 4 roows:

right atrium, left atrium,

right ventricle, left ventricle

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BLOOD VESSEL 

The blood vessel:

 Arteries

arteriolesCapilllaries

Venules

Veins

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CORONARY ARTERY DISEASE 

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P ATOPHYSIOLOGI 

Pathophysiology disorder as the discussion of

abnormalities in other organs, the discussion of

abnormalities in the arteries is also differentiated by

the underlying process, namely: congenital

abnormalities, inflammatory disorders, degenerativedisorders, immunologic disorders, neoplasms and

other disorders is unclear pathophysiology.

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ETIOLOGYCauses  Why 

Atherosclerosis  Fatty plaques deposit and narrow the arteries over time Congenital defects  Irregular vessel shapes can cause plaques and other debris

to become trapped narrowing the vessels Coronary artery spasm  Creates a temporary vessel blockage 

Dissecting aneurysm 

An aneurysm creates a bulging out of the vessel wall due to

 pressure. This can cause atherosclerotic plaque formation at

the site of the aneurysm, which causes further weakening of  the artery wall. A blood clot may form at the site and

dislodge, increasing the chance of stroke Infectious vasculitis  Inflammation of the vessels contributes to growth of plaque

in the arteries Syphilis  If left untreated, syphilis can cause inflammation of the

vessels, which leads to growth of plaque in the arteries High blood levels of C-reactive

 protein (CRP) CRP levels rise when there is protein (CRP) inflammation.

The inflammation process contributes to the growth of plaque in arteries 

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Unhealthy blood cholesterol levels. High blood pressure.

Smoking.

Insulin resistance 

Diabetes 

Family history of early heart disease.

Lack of physical activity 

Overweight or obesity.

Age 

Unhealthy diet 

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CLINICAL APPEARANCES

Signs and symptoms  Why 

MI  The arteries become narrowed due to fatty plaque buildup (atherosclerosis) andnot enough oxygen reaches the heart, causing ischemia 

Angina The arteries become narrowed due to fatty plaque buildup (atherosclerosis) and

not enough oxygen reaches the heart, causing ischemia. The ischemia causes

chest pain High blood pressure  Atheromas grow, causing narrowing of the arteries and calcium accumulation in

the arteries Decreased peripheral pulses  Decreased elasticity of the arteries and the narrowed lumen contribute to

decreased peripheral circulation  Nausea and vomiting  Angina 

Fainting  Decreased blood flow prevents oxygenation of the brain Sweating  Angina 

Cool extremities  Decreased peripheral circulation Shortness of breath  Decreased cardiac output leads to decreased lung perfusion 

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DIAGNOSTIC ASSESMENT

EKG (Electrocardiogram) 

Blood Tests  Chest X Ray 

Ankle/Brachial Index 

Echocardiography 

Computed Tomography Scan  Stress Testing 

Angiogram 

Electron beam computerized tomography

(EBCT): Magnetic resonance angiography (MRA):

Myocardial perfusion imaging with thallium 201during treadmill exercise 

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Risk factor for Coronary Artery Disease includedyslipidemia, smoking,hypertension, male gender

(women are protected untilmenopause), aging, non-white race, family history,obesity, sedimentary

lifestyle, diabetes mellitus,metabolic syndrome,elevated homocysteine, andstress.

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THE THERAPY 

blockers, and angiotensin converting enzymeinhibitors) to promote a favorable balance of

oxygen supply and demand.

 Antilipid medications to decrease blood

cholesterol and tricglyceride levels in patientswith elevated levels.

 Antiplatelet agents to inhibit thrombus

formation.Folic acid and B complex vitamins to reduce

homocysteine levels

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......

Coronary angioplasty , or balloon

angioplasty  

Coronary artery bypass graftoperation:  Atherectomy , Laser

angioplasty  

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NURSING INTERVENTION  Monitor blood pressure, apical heart rate, and

respirations every 5 minutes during an anginal

attack. Maintain continuous ECG monitoring or obtain a

12-lead ECG, as directed, monitor for arrhythmiasand ST elevation.

Place patient in comfortable position and administer

oxygen, if prescribed, to enhance myocardialoxygen supply.

Identify specific activities patient may engage inthat are below the level at which anginal painoccurs.

Reinforce the importance of notifying nursing staffwhenever angina pain is experienced.

Encourage supine position for dizziness caused byantianginals.

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Be alert to adverse reaction related to abrupt

discontinuation of beta-adrenergic blocker and

calcium channel blocker therapy. These drug must

be tapered to prevent a “rebound phenomenon”;tachycardia, increase in chest pain, and

hypertension.

Explain to the patient the importance of anxiety

reduction to assist to control angina. Teach the patient relaxation techniques.

Review specific factors that affect CAD

development and progression; highlight those risk

factors that can be modified and controlled toreduce the risk.

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TERIM K SIH T PERH TI NY