SAFETY DATA SHEET Creation Date: 11/09/2006 Phosphoric acid · 2015-03-27 · SAFETY DATA SHEET Phosphoric acid 25 ≤ c Creation Date: 11/09/2006 Revision date: 10/04/2014 According

SAFETY DATA SHEET

Phosphoric acid 25 ≤ c

Creation Date: 11/09/2006

Revision date: 10/04/2014

According to 1907/2006/EC

SDS: Phosphoric acid 1/18

1. Identification Of The Substance Or Mixture And Of The Supplier:

1.1.Product name:

Chemical name:

Synonyms:

Chemical formula:

INDEX no.:

CAS no.:

EC no.:

REACH registration number

Pre-registration number:

1.2.1. Recommended use:

1.2.2. Uses advised against:

1.3.Company identification:

1.4.Emergency telephone

number:

Phosphoric acid 25%

Phosphoric acid 75%

Phosphoric acid 80%

Phosphoric acid 85%

Phosphoric acid

orthophosphoric acid

H3PO4

015-011-00-6

7664-38-2

231-633-2

01-2119485924-24-XXXX*

05-2114089027-46-0000

Industrial and Professional uses:

1. Production of phosphoric acid

2. Industrial uses: intermediate in chemical synthesis, component

of mixtures /including packaging and distribution/, surface

metalworking agent, cleaning agent, an adjuvant in chemical

industry /including laboratory application/,

3. Professional uses: in fertilizers, surface metalworking, building

industry, a component of detergents and cleaners, facilitates

soldering,

Consumer uses

1. Consumer use: a component of polishes and wax mixtures,

washing and cleaning agents, facilitates soldering, a component of

fertilizers and scale removing agents.

The concentration of phosphoric acid in consumer products does

not exceed 10 % /15 % in scale removing agents/.

2. Phosphoric acid is used in batteries, but no substance emission

is expected in this case and therefore there is no adequate scenario

for consumers /lack of exposure/,

3. Phosphoric acid is used as an additive for foodstuffs, cosmetics

and fodder – these uses are not subject to the REACH Regulation.

Consumer uses of products containing phosphoric acid with a

concentration greater than 15 % are not subject to the safety data

sheet.

not determined

AS INGLE

Ingliste 79004, Rapla maakond, EESTI

372 48 90 190 (8:00 – 17:00)

[email protected] www.ingle.ee

112

Poison Information Centre: http://www.big.be/antigif.htm

International emergency number: Telephone: +49 180 2273-112

* Last 4 digits of the registration numbers are omitted due to the confidentiality issues.

mailto:[email protected]

http://www.big.be/antigif.htm

SAFETY DATA SHEET






2. Hazard identification:

2.1. Classification of the

substance:

Regulation (EC) No

1272/2008/EC:

Directive 67/548/EMÜ

2.2. Label elements:

Regulation (EC) No

1272/2008/EÜ:

2.3. Other hazards:

Met Corr. 1 H290 May be corrosive to metals

Skin Corr. 1B - H314 Causes severe skin burns and eye damage

C Corrosive R34 Causes burns

Phosphoric acid ... %;

Cas: 7664-38-2 EC: 231-633-2

Danger!

H290 May be corrosive to metals

H314 Causes severe skin burns and eye damage

P260 Do not breathe dust/fume/gas/mist/vapours/spray.

P280 Wear protective gloves/protective clothing/eye

protection/face protection

P303 + P361 + P353 If on skin (or hair): Remove/Take off

immediately all contaminated clothing. Rinse skin with

water/shower.

P310 Immediately call a POISON CENTER or doctor/physician.

P305 + P351 + P338 If in eyes: Rinse cautiously with water for

several minutes. Remove contact lenses, if present and easy to do.

Continue rinsing.

P406 Store in a corrosive resistant/… container with a resistant

inner liner.

Adverse human health effects

Phosphoric acid is classified as a corrosive substance which causes

burns.

Phosphoric acid causes tissue damage through direct chemical

action. Tissue proteins are turned into acid albuminates that can

dissolve in concentrated acid. Heamonoglobin is precipitated. As a

strong irritant it causes loss of tension in the nervous system.

Ingestion (swallowing) - phosphoric acid often eats away

oesophagus and stomach walls. The contact area is coloured

brown or black. Precipitated blood looking like coffee grounds can

be often found in stomach. Oesophagus epithelium can be partially

or completely exfoliated.

Eye contact - phosphoric acid can cause cornea exposure and in

more serious cases oedema and necrosis of deeper located tissues.

Skin contact - phosphoric acid causes burns that usually penetrate

SAFETY DATA SHEET






the complete depth of the skin; the wound heal slowly

with scaring and inflammations.

Vapour inhalation - causes pulmonary oedema. Accompanying

physical symptoms are most often wheezing, low blood pressure

and quickened pulse. Haemoptysis and dyspnoea can persist for a

few weeks after a single exposure to acid vapours.

Vapours can cause damage to tooth enamel.

Chemical hazards

Phosphoric acid is a medium acid. At room temperature it is

lasting and chemically low-active; does not oxidise, tends to

condense strongly (polyphosphoric acids are created by

dehydration).

It is resistant to strong reducing agents at room temperature or

even at the temperature of up to about 350 - 400°C, at the

temperature of over 400°C it is easily reduced. At higher

temperatures the phosphoric acid is highly reactive with the

majority of metals and their oxides, and it affects even such

resistant oxides as silica.

Boiling phosphoric acid is highly corrosive for the majority of

practically applied metals and alloys, as well as for ceramics

which acid resistance decreases as the temperature and

concentration grow.

Concentrated and hot H3PO4 reacts with porcelain and platinum.

Adverse environmental effects

After entering the environment, phosphoric acid causes strong soil

and water acidification and is toxic for plants and animals.

Criteria for PBT or vPvB in accordance with Annex XIII of the

REACH Regulation do not apply to inorganic substances.

Phosphoric acid is not identified as a PBT and vPvB substance.

3. Composition / Information of Ingredients:

CAS.: No

EC No.:

Chemical name

Percent:

Classification

7664-38-2 231-633-2 Phosphoric acid

H3PO4

25% ÷ 85% Skin Corr. 1B - H314 C ≥ 25 % Skin Corr. 1B; H314

10 % ≤ C < 25 % Skin Irrit. 2; H315

10 % ≤ C < 25 % Eye Irrit. 2; H319

C R34 C ≥ 25 % C; R34

10 % ≤ C < 25 % Xi; R36/38

4. First aid measures:

4.1.Description of first aid

measures

Inhalation:

Ingestion:

A rescuer should wear personal protective equipment (Section 8).

Vapour inhalation - move the affected away from the area of

contact with acid vapours, keep at rest (immobility) in semi

recumbent or reclining position. Exercise may cause pulmonary

oedema. Protect against heat loss, in case of shortness of breath

administer oxygen, preferably with breathing mask. Medical

attention is required.

Never perform stomach wash-out and never give emetics and

SAFETY DATA SHEET






Skin contact:

Eye contact:

4.2. Most important

symptoms and effects, both

acute and delayed:

4.3. Indication of any

immediate medical attention

and special treatment needed

neutralizing agents (alkalizing). Except for a few glasses of water

or milk to drink do not administer anything orally. Never give

anything by mouth to an unconscious person. Call a physician

immediately.

Skin contact- remove acid rinsing with plenty of water (not hot)

for at least 15 minutes. Never use soap or any neutralization

agents. In case of clothing contamination undress affected person

under water stream, apply a sterile dressing on burns. Medical

attention is required.

In case of contact, immediately flush eyes with plenty of water for

at least 15 minutes (remove contact lenses if easily possible).

(avoid strong water flow for the risk of mechanical corneal

damage). After rinsing apply a sterile medication - free dressing on

the eyes and administer painkillers. Do not apply chemical

antidotes, as the heat produced during the reaction can worsen the

damage. Medical attention is required.

Vapour inhalation - cough, choke, headaches, dizziness, weakness

and after 6-8 hours of latent period pulmonary oedema with

heaviness in the chest, shortness of breath, dizziness, foamy

secretion and cyanosis. Also wheezing, low blood pressure and

quickened pulse may occur.

Ingestion - acute, burning pain in oral cavity, throat and stomach

followed by vomiting and diarrhoea containing dark, coagulated

blood. Blood pressure violently decreases. In oral cavity and oral

cavity environment brown or yellowish colouring can be found.

Glottis oedema may cause breathing difficulty or hypoxia.

Skin contact - acute pain, brown or yellow tissue colouring

Eye contact - pain, burning sensation, lacrimation, photophobia,

conjunctiva congestion and oedema, corneal damage.

No special medical treatment methods are not available in the

literature.

No information on specific product antidote is available.

If poisoning is suspected contact the nearest toxicology dept.

After careful assessment of the victim’s condition the physician

makes a decision on how to proceed.

5. Firefighting measures:

5.1. Extinguishing media

Suitable extinguishing media:

Unsuitable extinguishing

media:

5.2. Special hazards arising

from the substance or

mixture

Use extinguishing media that are safe for the environment; CO2,

foam, fire foam

no extinguishing media which are not allowed are known

In contact with commonly known metals generates flammable

hydrogen, which can form an explosive mixture with air. - There is

a risk of formation of toxic decomposition products (POx) when

exposed to high temperature – continuous monitoring of POx and

SAFETY DATA SHEET






5.3. Advice for firefighters

hydrogen concentration is required.

In case of hot acid reacting with contaminated metals toxic

gaseous phosphine PH3 may be formed.

When extinguishing fire, be sure to wear personal protective

equipment.

Use water spray jet to cool containers and equipment.

Prevent extinguishing media mixed with H3PO4 from entering

surface water or/and groundwater.

Contaminated fire-extinguishing water must be disposed of in

accordance with the regulations issued by the appropriate local

authorities.

6. Accidental release measures:

6.1.Personal precautions,

protective equipment and

emergency procedures

6.2. Environmental

precaution

6.3. Methods and material for

containment and cleaning up

6.4.Reference to other

sections

In case of a serious accident:

- remove all the persons that are not directly involved in the rescue

operation from the hazard area (downwind)

- notify the local authorities, Police, Chemical Rescue Unit and

Road Administration

Start removing the damage yourself, and when it poses too

excessive hazard, wait for specialist group Persons involved in

rescue operation should wear protective cloths and protective

masks of respiratory tract.

Avoid contact with skin and eyes. Do not breathe vapours.

Do not allow acid contact with metals.

Protect the area from acid entering the sewage system,

watercourses and water reservoirs.

In case of small spillage, contain the spill by binding (as far as

possible).

Cover up spilled acid with non-combustible absorbent material

(preferable milled limestone, sand, absorbents eligible for acid

spillage removal), collect in container made of acid-resistant

material and transfer to neutralization. Clean up the affected area

To reduce the harmfulness, neutralize with diluted NaOH solution

or Na2CO3 solution.

Refer to SECTION 13 for disposal considerations.

Refer to SECTION 8 for individual protection measures.

7. Handling and Storage:

7.1. Precautions for safe

handling

Use as intended, by trained and suitably equipped with protective

equipment staff only, handle with special care due to the hazards –

see section 2.

Prevent substance release to environment (entering sewage

system) by using protective trays.

Do not:

- eat and drink in workplace.

SAFETY DATA SHEET






7.2. Conditions for safe

storage, including any

incompatibilities

7.3.Specific end use(s)

- smoke outside the designated areas.

Avoid contact with eyes and skin. Thoroughly wash hands before

break and after work.

Technical measures: It is required to use adequate classical

exhaust ventilation in the way described in the annex to the safety

data sheet. The boundary value of professional exposure

applicable in Europe has to be observed. Use only acid-resistant

materials.

Do not inhale vapour. Never pour water or any water solutions to

tanks or containers with acids. Dissolution and neutralization

reactions are highly egzotermic in such cases. Avoid splashes.

Observe caution. Always add acid to water. Do not mix it with

inadequate materials (see subsection 10.5).

Keep in steel rubberised containers or in an acid-resistant steel

container placed in open air on concrete trays tiled with acid-

resistant tiles. Smaller quantities of acid can be kept in transport

packages in well-ventilated rooms or in open air or on concrete

trays tiled with acid-resistant tiles (accidental spillage protection).

Do not stack packages with the substance one on another. Observe

general rules concerning the storage of chemicals; close the

packaging for the time of not using the substances. Do not expose

the substance to direct influence of sunshine.

Do not store in temperatures close to the freezing temperature.

Avoid freezing of the substance (85% at +21,1 oC, 80% at +4,6

oC

and 75% at -17,5 oC; the actual point of freezing is again

unpredictable). After melting the chemical’s physical and

chemical properties recover.

Adequate materials: stainless steel, high-density polyethylene,

glass.

Collective storage is permissible only with materials of the same

hazard class.

Detailed information on specific end uses is contained in exposure

scenarios.

The exposure scenarios contained in annexes depict the most

disadvantageous conditions of human and environmental

exposure.

Lack of information about uses other than mentioned in subsection

1.2.

Lack of information about the required special approach to the

product.

8. Exposure controls/ Personal protection:

8.1. Control parameters

DNELS

TWA (8 hr exposure limit): 1 mg/m3

STEL (15 min exposure limit): 2 mg/m3

DNELs (worker) Exposure Exposure pattern DNEL (workers)

SAFETY DATA SHEET






route

Inhalation

and Dermal

Acute and Long-

term systemic

effects

As no acute toxicity hazard and no

systemic toxicity has been identified,

there is no requirement to derive acute

DNELs.

Phosphoric acid as such is not expected

to become systemically available in the

body. Once absorbed, phosphoric acid

is distributed widely in the body as

phosphate, which is an essential

element to organisms and its

metabolism in the human body is

regulated. Phosphoric acid can be

locally absorbed by ingestion,

inhalation and dermal contact but is not

expected to elicit a systemically toxic

effect.

Long term systemic

Inhalation

Acute local effects

A DNEL for acute toxicity should be

derived if an acute toxicity hazard

(leading to C&L) has been identified

and there is a potential for high peak

exposures (REACH Guidance

document R8, appendix R8 -8: Acute

toxicity). The substance is not

classified for acute exposure via

inhalation. In addition, no peak

exposure is warranted for this

substance. A European Short term

Occupational exposure limit

recommended from Scientific Expert

Group is available for phosphoric acid

and is STEL 15 min: 2 mg/m3. This

value is based on the Rushing

observation on phosphorus pentoxide to

the ACGIH TLV committee

Long term local

effects

A DNEL for long term inhalation,

workers has been calculated to be 2.92

mg/m³ on the basis of an OECD 422

study.

However, A European Long term

Occupational exposure limit (OEL)

recommended by the Scientific Expert

Group (SCOEL and HSE) is available

for phosphoric acid: 8 hour TWA: 1

mg/m3. This value is based on the

Rushing observation on phosphorus

pentoxide to the ACGIH TLV

committee. Nevertheless as this value is

more conservative it will be used in the

exposure assessment and determination

of the risk characterization ratio for

phosphoric acid.

No DNELs can be derived for dermal local effects as the

concentration limit for corrosivity of H3PO4 is 25%. The

concentration range of 10 -25% is established for irritation,

therefore calculation of a DNEL is not required due to protective

measures already in place.

SAFETY DATA SHEET






PNEC

DNELs (general population):. Exposure

route

Exposure pattern DNELs (general population):.

Inhalation

and Dermal

Acute systemic

effects

As no acute toxicity hazard and no

systemic toxicity has been identified,

there is no requirement to derive acute

DNELs.

Phosphoric acid as such is not expected

to become systemically available in the

body. Once absorbed, phosphoric acid

is distributed widely in the body as

phosphate, which is an essential

element to organisms and its

metabolism in the human body is

regulated. Phosphoric acid can be

locally absorbed by ingestion,

inhalation and dermal contact but is not

expected to elicit a systemically toxic

effect.

Long term systemic

effects

Inhalation

Short-term, local

effects

No assessment of peak-exposures is

warranted on the basis of an acute

inhalation study.

Long term local

effects

The DNEL for long-term exposure -

local effects - inhalation, general

population is (437.5 mg/m³)/600 = 0.73

mg/m³

No DNELs can be derived for dermal local effects as the

concentration limit for corrosivity of H3PO4 is 25%. The

concentration range of 10 -25% is established for irritation,

therefore calculation of a DNEL is not required due to protective

measures already in place. In addition, no data are available to

warrant the calculation of oral DNELS.

PNECs

Compartment PENEC Remarks

PNEC aqua

(freshwater)

Not

calculated

The predominant adverse effects of

phosphoric acid in aquatic systems are pH-

related effects, as a result of the acidic

nature of phosphoric acid. As phosphoric

acid is a triprotic acid it will dissociate up

to 3 times; releasing a phosphate anion

(H2PO4-, HPO4

2- or PO4

3-) and a H

+ ion at

each dissociation. The fate of the H+ ions

(and subsequently the resultant pH) will

depend on the chemical composition of the

receiving water body. The pH of the

receiving body can vary significantly

between different ecosystems and

geographical locations and also the pH

change as a consequence of anthropogenic

phosphoric acid is influenced by the

buffering capacity of the receiving water.

Changes in the pH of a water body can

have potentially toxic effects on the

endogenous aquatic organisms present.

However, the sensitivity of the organisms

to pH changes vary significantly according

PNEC aqua

(marine water)

Not

calculated

PNEC aqua

(intermittent

releases)

Not

calculated

SAFETY DATA SHEET






to location or species (pH ranges of

tolerance for the major taxonomic groups,

fish, crustaceans and algae, are commonly

report to vary between pH 6.0 and pH 9.0).

The phosphate anions are not considered to

be toxic to the aquatic environment as

phosphate anions are naturally widespread

both in the environment and within

organisms and cells, and they are readily

assimilated as an essential nutrient by the

aquatic organisms. It is therefore not

considered appropriate to derive a PNEC

aqua on the basis of single-species

ecotoxicology data as the main risk factor

has been determined to be a phosphoric

acid related change in the pH of the

receiving water body rather than direct

toxicity of phosphoric acid to the target

organisms (in a laboratory study the test

media would be buffered to pH-neutral

thus sequestering the H+ ions and as such

no adverse effects would be expected).

In addition, pH-related effects are

considered to be of lower risk in marine

water due to its buffering capacity being

greater than that of freshwater.

PNEC STP Not

calculated

No data are available for STP

microorganisms. In water phosphoric acid

will progressively dissociate to phosphate

anions (PO43-) and hydrogen cations (H+).

The release of H+ ions will lead to a net

decrease in the pH of the receiving water

body. However, in the case of waste water

treatment plants, the pH levels are typically

adjusted to ensure a neutral discharge to

the receiving water and in order to prevent

pH-related inhibitory effects on the growth

of STP microorganisms. The neutralisation

of phosphoric acid would result in the STP

microoganisms being exposed to

phosphate. Phosphate is an essential

nutrient for activated sludge systems and

does not result in adverse effects. It is

therefore not considered appropriate or

meaningful to derive a PNEC STP for

phosphoric acid.

PNEC

sediment

(freshwater)

Not

calculated

No data are available for sediments.

Phosphoric acid will progressively

dissociate in water to give H+ and PO4

3-

ions. Due to its high water solubility and

low vapour pressure it is predicted that any

un-dissociated phosphoric acid will remain

in the water phase and will not absorb onto

particulate. The PO43-

ion is likely to

absorb onto sediment, however, PO43-

ions

are ubiquitous in the environment and are

found naturally in soil, water and sediment.

They are assimilated by species residing in

PNEC

sediment

(marine water)

Not

calculated

SAFETY DATA SHEET






the sediment, water or soil and are essential

for maintaining a good chemical balance.

Toxicity to sediment organisms via this

route is not predicted and as such further

toxicity studies to determine a PNEC

sediment are not considered to be

scientifically justified.

PNEC soil Not

calculated

No data are available for soil organisms.

Phosphoric acid occurs naturally and

widely in low concentrations in soils, In

addition, phosphoric acid is widely used as

a plant fertiliser, either as such or as a

processing residue in superphosphates, and

as a soil stabiliser (through reactions with

aluminium molecules in clays). When

spilled on to soil phosphoric acid will

initially infiltrate downwards, but in most

cases it will dissociate to PO43-

and H+ ions

in the soil pore water, and/or react with

minerals present in the soil, in particular

calcium, iron and aluminium . Except in

very specific circumstances (acidic soils,

certain mineral soil types, very high dosage

of phosphoric acid) phosphoric acid will

not therefore penetrate beyond the surface

layer of soil and will not reach the

groundwater table. Phosphate loss to

surface water is usually related to run-off

or

erosion of soil particles, which carry the

attached phosphates. Accidental release of

significant quantities of concentrated

phosphoric acid into soil may cause soil to

expand as the phosphoric acid is

neutralised by the lime already present in

the soils resulting in calcium phosphate

minerals replacing calcium carbonate.

Phosphate ions released into soil are not

anticipated to result in toxicity to soil-

dwelling organisms as phosphate is an

essential nutrient. Toxicity to terrestrial

organisms via this route is only considered

to be as a result of localised pH changes; as

such it is not justified to further investigate

the effects of phosphoric acid on terrestrial

organisms as the potential effects are

highly variable according to location and

soil composition and a reliable PNEC soil

cannot be calculated. If phosphoric acid

does reach the groundwater table any

remaining phosphoric acid will dissociate

to PO43-

and H+ ions; the resultant ions may

be dispersed and diluted in the ground

water.

PNEC oral Not

calculated

Phosphoric acid is not considered to pose a

risk of secondary poisoning as it is not

anticipated to bioaccumulate. Phosphoric

acid will progressively dissociate into

SAFETY DATA SHEET






8.2. Exposure controls

Personal protecion:

Respirators:

Eyes:

Skin:

Environmental exposure

controls

PO43-

and H+ ions in aqueous

environments; these ions are not lipophillic

and will therefore not accumulate in fatty

tissues. It not considered necessary to

derive a PNEC to take into account the risk

of secondary poisoning and no further

testing is considered necessary.

Additional information in the Exposure scenarios for identified

uses attached to this safety data sheet.

Protective measures and hygiene: Handle in accordance with good

industrial hygiene and safety practice. Thoroughly wash face and

hands after work, take off contaminated clothing. Do not eat or

drink anything during working with phosphoric acid.

Applied technical measures of control

It is necessary to:

- ensure proper ventilation in the workplace (and exhaust

ventilation in requires situations and according to the exposure

scenarios),

- in the direct vicinity of the direct exposure areas provide the

possibility of rinsing the eyes and taking a shower,

- use phosphoric acid in closed systems or open containers

equipped with locks,

- transport phosphoric acid by pipelines,

- fill/empty containers using automatic systems (suction pumps,

etc.),

- use a handling device /e.g. pincers, holders/ to avoid direct

contact with the acid and exposure by splashing oneself,

- observe principles of good practice.

usually not required. When vapours/aerosols are generated, use

acid vapour respirator. If the product concentration in the air is not

known, use automatic respirator.

required - close-fitting safety goggles appropriate for chemicals or

face shield /according with EN 166/ or a face shield.

It is recommended to place an eye-rinsing device i.e. eye-wash

station next to the system where phosphoric acid is used.

required – impermeable gloves resistant to chemicals

/recommended gloves made of neoprene, chloroprene or PVC /,

protective boots and acid-proof clothing.

thermal hazards – the substance does not pose a thermal hazard, no

specific requirements are applicable.

Avoid uncontrolled discharge of phosphoric acid solutions to

sewers and surface waters. In case of discharge, regular pH control

is necessary. Minimize pH change in surface waters during

discharge.

9. Physical and Chemical Properties:

Appearance:

Odour:

Clear colourless transparent solid or viscous liquid, Handbook data

no data

SAFETY DATA SHEET






Odour threshold

pH

Freezing/ melting point

Boiling point

Flash Point

Evaporation rate

Flammability

Explosion Limits

Vapor Pressure

Vapor density

Water solubility:

Relative density

n-octanol/water

Autoignition Temp

Decomposition temperature

Viscosity

not applicable

< 1 pH <1

100g/l = pH 3.0 (as measured in algal growth inhibition study).

Handbook data

41.1 oC at 1013 hPa /purity 98,8% ¤– according to EC guidelines

A.1- differential scanning calorimetry

25 % H3PO4: -7 °C

30 % H3PO4: +11,8 oC

70 % H3PO4: -43 °C

75 % H3PO4: -17 °C

85 % H3PO4: +21 °C

296.5 oC at 1013 hPa/purity 98,8% – according to EC guidelines

A.2- Siwolobow method

25 % H3PO4: +113 °C

30 % H3PO4: +115 oC

70 % H3PO4: +127 °C

75 % H3PO4: +135°C

85 % H3PO4: +151 °C

not applicable; According to Regulation No. 1907/2006, the flash

point does not need to be assessed for inorganic chemicals.

not applicable

not applicable; Data waiver based on observations in use and

chemical structure of the material.

not applicable

4 Pa at 20 0C ; Handbook data – weight of evidence approach.

25 % H3PO4: 29,8 mm Hg at 30 °C

30 % H3PO4: 28,9 mm Hg at 30 °C

70 % H3PO4: 13,1 mm Hg at 30 °C

75 % H3PO4: 10,0 mm Hg at 30 °C

85 % H3PO4: 3,95 mm Hg at 30 °C

not applicable

> 1000 g/l of water at 20 0C ; Handbook data – weight of evidence

approach

1.840 g/cm3 at 38

0C/ purity 98,8% ; EU Method A.3. (Klimisch

Reliability 1)

25 % H3PO4: 1,1493 g/cm3 (+15 °C)

30 % H3PO4: 1,1837 g/cm3 (+15 °C)

70 % H3PO4: 1,5271 g/cm3 (+15 °C)

75 % H3PO4: 1,5788 g/cm3 (+15 °C)

85 % H3PO4: 1,6928 g/cm3 (+15 °C)

Not applicable; According to Regulation No. 1907/2006, the

partition coefficient n-octanol/water does not need to be assessed

for inorganic chemicals.

No auto-ignition anticipated; Based on prediction

not applicable

600 mP at 20 0C for the concentration of 105 % H3PO4; Handbook

data – weight of evidence approach

25 % H3PO4: 1,8 cSt at 20 °C

30 % H3PO4: 2,2 cSt at 20 °C

SAFETY DATA SHEET






Explosive properties

Oxidizing Properties

Conductivity

70 % H3PO4: 11,0 cSt at 20 °C

75 % H3PO4: 15,2 cSt at 20 °C

non-explosive; Based on prediction

non-oxidizing; Based on prediction

25 % H3PO4: 0,175 Ω-1

*cm-1

at 25°C

30 % H3PO4: 0,138 Ω-1

*cm-1

at 25°C

70 % H3PO4: 0,06 Ω-1

*cm-1

at 25°C

75 % H3PO4: 0,055 Ω-1

*cm-1

at 25°C

85 % H3PO4: 0,05 Ω-1

*cm-1

at 25°C

10. Stability and reactivity:

Reactivity:

Chemical Stability:

Possibility of hazardous

reactions:

Conditions to Avoid:

Incompatible materials:

Hazardous Decomposition

Products:

At room temperature, phosphoric acid is lasting and chemically low-

active; it does not oxidise, tends to condense strongly

(polyphosphoric acids are created by dehydration).

Phosphoric acid is resistant to strong reducing agents at room

temperature or even at the temperature of up to about 350 - 400°C, it

is easily reduced at the temperature of over 400°C.

In contact with commonly known metals, it generates flammable

hydrogen, which can form an explosive mixture with air.

- high temperature,

- light

– contact of hot acid with metals

– nitromethane,

– alkalis,

– metals,

– metal oxides.

Materials ineligible to be used jointly – iron and its compounds,

steel, aluminium and its compounds

Emission of toxic fumes POx, when heated up to high temperatures.

In case of hot acid reaction with contaminated metals, toxic gaseous

phosphine PH3 may be formed.

11. Toxicological Information:

11.1. Acute toxicity:

Oral:

Dermal:

Inhalation:

11.2. Irritation and

corrosion:

LD50 = 1.7 mL / 100g bw/day, for a 10% solution of 75.4%

phosphoric acid (equivalent to 2600 mg/kg bw). Not classified.

Method: Female rats. Method similar to OECD 423, Klimisch

reliability 2.

No reliable data – substance is corrosive to skin.

No reliable data - Substance is corrosive to skin and is not

considered to be acutely toxic via the inhalation route.

Skin corrosion/irritation:

Corrosive Cat 1B (concentrations ≥25% w/w)

Method: SCL according to Regulation EC No.1272/2008 (EU CLP),

Annex VI, Table 3.1.

Serious eye/damage/irritation:

Corrosive (concentrations ≥25% w/w).

Eye irritant (concentrations ≥10 % w/w < 25% w/w)

SAFETY DATA SHEET






11.3. Respiratory or skin

sensitization

11.4. CMR effects:

11.5. STOT effects:

STOT - single exposure :

STOT - repeated exposure :

Aspiration toxicity :

11.6. Further information:

Method: SCL according to Regulation EC No.1272/2008 (EU CLP),

Annex VI, Table 3.1.

Not investigated – substance is corrosive.

No data to suggest substance is a sensitiser.

Germ cell mutagenicity

Not considered to be mutagenic.

Method: Orthophosphoric acid is not expected to produce germ cell

genotoxic damage and was found to be negative in the following

tests:

- OECD 471 (Klimisch reliability 1)



Carcinogenicity

Not considered to be a carcinogen.

Method: Not required for REACH. No data to suggest likelihood of

carcinogenicity.

Reproductive toxicity

Not considered to be a reproductive or developmental toxicant

Method: Two studies: Rat, OECD 422 Klimisch 1 and klimisch

reliability 2 (for read-across from study on monosodium phosphate)

NOAEL 500 mg/kg bw/day

Developmental toxicity: NOAEL (monosodium phosphate): 410

mg/kg bw/day

STOT-single exposure – all routes

No STOT SE classification – however substance is corrosive and

likely to cause respiratory irritation.

STOT-Repeated exposure - all routes

Oral: NOAEL: 250 mg/kg bw/day

Inhalation: no data

Dermal: no data

Method: Rat, OECD 422, Klimisch reliability 1.

No aspiration hazard identified

Method: Not applicable.

Possible routes of exposure

Inhalation is the most likely route of exposure. Inhalation of

phosphoric acid vapours/mists may cause immediate irritation and

pain.

Causes burns on contact with skin.

SAFETY DATA SHEET






Causes burns on contact with eyes.

Causes burns of gastrointestinal tract in case of ingestion.

Health effects of acute exposure

Vapours and dusts above 5 mg/m3 cause conjunctiva congestion,

eyes pain and lacrimation; irritation of upper respiratory tract with

cough, burning sensations of throat, shortness of breath, larynx

oedema, haemoptysis.

Toxic pulmonary oedema may occur. Skin contamination with

solution causes burns with coagulation necrosis. Extensive burn may

cause shock. Eye contamination causes eyelid and conjunctivitis

burns. Poisoning through the digestive tract causes burns of oral

cavity mucosa, throat and gullet with risk of bleeding from the

digestive tract and shock.

Health effects of chronic exposure:

Inflammatory states of eyes and skin, chronic inflammatory states of

upper respiratory tract. Long-term exposition to acid vapours may

cause teeth erosion and later jaw necrosis. Irritation of bronchial

tubes and chronic cough as well as frequent episode of

bronchopneumonia may occur.

12. Ecological Information:

12.1. Ecotoxical effects:

Fish:

Invertebrate toxicity:

Algal toxicity:

12.2. Biodegration

12.3. Bioaccumulation

12.4. Mobility in soil

12.5. Results of PBT and

vPvB assessment

Median Lethal pH (96 hr): 3-3.25 for Lepomis macrochirus.

Method: No guideline followed

EC50(48 h) ≥ 100 mg/l (nominal) – Method: Daphnia magna

(OECD TG 202) EU MEthod C.2

EC 50/LC50 72 h ≥ 100 mg/l (nominal) – Method: Desmodesmus

subspicatus (algae), OECD 201, EU Method C.3

72h NOEC = 100 mg/l (nominal)

Basis for effect: growth rate.

Orthophosphoric acid is an inorganic substance, therefore there is no

need to test it for biodegradation capacity.

Not applicable to inorganic substances.

Phosphoric acid is not absorbed by soil. In most cases, it dissociates

into the ions of PO43-

and H+ in the water present in pores of the soil

and/or reacts with minerals present in the soil, particularly calcium,

iron and aluminium. Except for strictly specified circumstances

(acid soil, certain types of mineral soil, very large doses of

phosphoric acid), phosphoric acid will not penetrate into the

groundwater through the layer of soil.

As per the requirements concerning information and chemical safety

assessment, Chapter R.11: PBT Assessment, the PBT and vPvB

assessment criteria listed in Annex XIII to the Regulation are not

SAFETY DATA SHEET






12.6. Other adverse effects

applicable to inorganic substances. Therefore, there is no need to

further test the PBT properties for phosphoric acid.

There are no available data on sediment and land toxicity. The

substance does not pose hazard to sediment or land organisms. An

increased concentration of orthophosphoric acid may case a

decrease of pH, which can have a local harmful influence on

organisms.

According to the criteria of European classification and labelling

system, there is no need to classify the substance as hazardous to the

environment.

13.Disposal Considerations:

13.1. Disposal:

13.2. Contaminated

packaging:

13.3. EC Waste Code:

Non-used, expired or contaminated phosphoric acid should not be

disposed of into sewage system. Dispose of waste product in

accordance with all government regulations.

Acid neutralization Phosphoric acid is neutralized with excess of

10% lime milk. It is completely clarified within 10 to 20 hours, but

the majority of solids are precipitated in 1 hour.

Phosphor disposal from water and drains: with precipitating

methods. Precipitating process is carried out in parallel to biological

treatment process in an aeration tank, where iron (III) salts and

optionally aluminium salts are added. Precipitated iron (III)

orthophosphate (FePO4) is a water- insoluble salt. Suspension of this

salt is absorbed in activated sludge and removed from treated

sewage.

Packaging, that can not be reused after cleaning must be disposed or

recycled in accordance with all federal, national and local

regulations.

Accepted waste codes/according to executory provisions of Act on

waste classified as hazardous.

06 01 04 – phosphoric and phosphorous acid

16 03 03 – inorganic wastes containing dangerous substances,

including phosphoric acid,

16 05 06 – laboratory chemicals consisting of or containing

dangerous substances, including mixtures of laboratory chemicals ,

16 05 07 – discarded inorganic chemicals consisting of or

containing dangerous substances, including phosphoric acid,

15 01 10 – packaging containing residues of or contaminated by

dangerous substances

14. Transport Information

ADR / RID

UN-No

Class

Pacing group

Shipping Name:

1805

8

III

PHOSPHORIC ACID, solution

SAFETY DATA SHEET






Environmental hazards

Special precautions for

user:

Other information

Not classified for the environment.

In consideration of the hazards /refer to SECTION 2/, wear personal

protective equipment as per SECTION 8

Transport in bulk according to Annex II of MARPOL 73/78 ad the

IB Code: not applicable – no in bulk transport is predicted

- passenger plane: transport forbidden.

- labelling for air transport: corrosive substance; EmS No.: F-A, S-B

15. Regulatory Information:

15.1. Regulatory

Information:

15.2 Chemical safety

assessment

List of EC laws:

Regulation (EC) No 1907/2006 of the European Parliament and of

the Council of 18 December 2006 concerning the Registration,

Evaluation, Authorisation and Restriction of Chemicals (REACH),

establishing European Chemicals Agency, amending Directive

1999/45/EC and repealing Council Regulation (EEC) No 739/93

and Commission Regulation (EC) No 1488/94 as well as Council

Directive 76/769/EEC and Commission Directives 91/155/EEC,

93/67/EEC, 93/105/EC and 2000/21/EC

Council Directive 91/689/EEC of 12 December 1991 on hazardous

waste, amended with Council Directive 94/31/EC

Regulation (EC) No 94/62 of the European Parliament and of the

Council of 20th December 1994 on packages and waste packages

Directive 2006/12/EC of the European Parliament and of the

Council of 5th April 2006 on waste

Regulation (EC) No 1272/2008 of the European Parliament and of

the Council of 16 December 2008 on classification, labelling and

packaging of substances and mixtures, amending and repealing

Directives 67/548/EEC and 1999/45/EC, and amending Regulation

(EC) No 1907/2006.

Commission Regulation (EU) No 453/2010 of 20th May 2010

amending Regulation (EC) No 1907/2006 of the European

Parliament and of the Council on the Registration, Evaluation,

Authorisation and Restriction of Chemicals (REACH).

Chemical Safety Assessment was carried out for phosphoric acid.

Exposure scenarios are enclosed in Annex 1-4 hereto.

16. Other Information:

14.1. Full text of R-, H- and

Abbreviations and acronyms:

H314 Causes severe skin burns and eye damage.

H315 Causes skin irritation.

H319 Causes serious eye irritation.

R34 Causes burns.

R36/38 Irritating to eyes and skin

P234 – Keep only in original container

P264 – Wash hands thoroughly after handling.

PRECAUTIONARY STATEMENT - REACTION:

P301 + P330 + P331 – If swallowed: rinse mouth, do not induce

vomiting

P363 – Wash contaminated clothing before reuse.

P304 + P340 – If inhaled: remove victim to fresh air and keep at

SAFETY DATA SHEET






14.2. Recommended

restrictions on use

14.3. Further information

14.4. Sources of key data

used to compile the Safety

Data Sheet.:

14.5. Modifications in this

version:

rest in a position comfortable for breathing.

P321 – Specific treatment (see on the label)

P390 – Absorb spillage to prevent materials damage

P405 – Store locked up

PRECAUTIONARY STATEMENT - DISPOSAL:

P501 – Dispose of contents/container as per current national and

international regulations.

ACGIH = American Conference of Industrial Hygienists

AF = Assessment factor

DNEL = Derived no effect level

EC50 = Median effect concentration

LC50 = Median lethal concentration

LD50 = Median lethal dose

LEV = Local Exhaust Ventilation

NOAEL = No observed adverse effect level

NOEC = No observed effect concentration

PBT Persistent bioaccumulative toxic

PEC = Predicted effect level

PNEC = Predicted no effect level

PRE = Personal Respiratory Equipment

OEL = Occupational Exposure Limit

SCOEL = Scientific Community on Occupational Exposure Limits

SDS = Safety data sheet

STOT–SE = Specific target organ toxicity – single exposure

STOT–RE = Specific target organ toxicity – repeated exposure

STP = Sewage treatment plant

TWA = Time-weighted average

vPvB = Very persistent very bioaccumulative

Before using working and safety instructions should be read

through work and safety instructions

This information is based on our present state of knowledge. It

shall describe our products regarding safety requirements and shall

not be construed as a guarantee or statement of condition and /or

quality.

Used delivery MSDS

Added: exposure scenario

Documents

SAFETY DATA SHEET Creation Date: 11/09/2006 Phosphoric acid · 2015-03-27 · SAFETY DATA SHEET Phosphoric acid 25 ≤ c Creation Date: 11/09/2006 Revision date: 10/04/2014 According