Ten Myths About Endodontics: “Fact Versus Pulp Fiction”

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Continuing Education

Ten Myths About Endodontics:“Fact Versus Pulp Fiction”

Authored by John West, DDS, MSD

Upon successful completion of this CE activity 2 CE credit hours will be awarded

Volume 33 No. 9 Page 118

ABOUT THE AUTHORDr. West is founder and director of theCenter for Endodontics in Tacoma,Wash. He graduated from the Universityof Washington Dental School andreceived his MS degree and en do donticcertificate at Boston University, where he

was awarded the Alumni of the Year Award. He is aneducator and clinician, and his focus is interdisciplinaryendodontics. He has authored several textbook chaptersand is an editorial board member for the Journal of Estheticand Restorative Dentistry, Practical Procedures inAesthetic Dentistry, and the Journal of MicroscopeEnhanced Dentistry. He can be reached toll-free at (800)900-7668, via e-mail at the following address:[email protected], or visit the Web sitecenterforendodontics.com.

Disclosure: Dr. West is the co-inventor of Pro Taper, WaveOne,and Calamus technologies, in addition to ProGlider(DENTSPLY Tulsa Dental Specialties).

INTRODUCTIONThe late Drs. Sam Seltzer and I. B. Bender are quoted assaying, a long time ago, that the pulp is a “is a big issueabout a little tissue.”1 Their quote is just as applicable to dayas it was decades ago. There seems to be as much studyand research about a 15-mm piece of tissue as any pieceof tissue in the human body. So what is the big issue?

Fundamentally, endodontic biology and treatment areas basic today as they were in the Seltzer and Bender days.And, the theory of focal infection created pharmacology asthe stepchild to clinical endodontics, which then led to thesimple biology of endodontics: eliminate the source ofendodontic disease, and lesions of endodontic origin(LEOs) will heal or will be prevented where they do not

exist. This simple and richly supported biologic pathway issometimes lost in to day’s endodontic art and science. Ihave identified 10 endodontic myths that have crept into (orback into) endodontic literature and endodontic thoughtpro cesses and treatment considerations in recent years.

The purpose of this inquiry is to evaluate 10 commonmyths that influence daily endodontic decision-makingplans. There are more controversial areas; however, I hopethis article will serve as a starting point to separateendodontic fact from folklore fiction. In all endodontics thereis, of course, only one accurate diagnosis and, while theremay be several treatment plans, the successful endodonticclinicians of the future must do the right thing every day byasking the question, “What would you [the clinician] do ifyou were the patient?” And, if we were to ask the patient thesame question, they will always ask the clinicianconsciously or wonder unconsciously, “What would you doif it were you?” Some myths, though widely believed, maynot be the truth when challenged under the experimentalmodel, involving both the lab bench and the clinical reality.Myths can interfere with the essence of predictability duringthe process of saving an endodontically diseased tooth inthe simplest form of daily endodontic treatment. It has oftenbeen my personal experience that if the scientificobservations are different from the clinical observations,


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Ten Myths About Endodontics:“Fact Versus Pulp Fiction” Effective Date: 9/1/2014 Expiration Date: 9/1/2017

Figures 1a and1b. Myth No. 1:“Large lesionsextending thelength over severalteeth have adiminishedcapacity to heal.”(a) A large lesionof endodonticorigin (LEO)extending frommandibular rightpremolar tomandibular leftpremolar. (b) Post-treatmenthealing at 2 years.

a

b

then something is wrong with the science. My first of the 10endodontic myths that will now be ex plored is just such anexample.

ENDODONTIC MYTH NO. 1“Large Endodontic Lesions Extending the Length ofSeveral Teeth Have a Diminished Capacity To Heal”What are the factors that promote en dodontic healing?2,3

Remove the disease source. This fact is simple, and it isprofound. Get highly skilled at the mechanics of endodonticsand enjoy enormous en dodontic success. Mother Nature haslittle regard for the size of the LEO as a deterrent to healing.The only clinically meaningful determinant is that the LEO iscontained within the attachment apparatus; and, theperiodontal vector is a sequelae of the LEO; and that thegingival crevice sinus tract probes in a precipitous fashion(Figure 1a). You may say, why? It is because, if the resultingendo-perio le sion probes precipitously versus conical, thesinus tract draining “waterfall” is simply wider than narrow.Healing biology treats a narrow or wider sinus tract that re -sults from a necrotic pulp in the very same way: Naturerewards the clinician and pa tient by predictably healing, aftercleaning and disinfecting the entire root canal system ofnecrotic debris, including bacteria, biofilm, and detachedcollagen. Based on re sults, the large endo dontic lesionclinically heals, regardless if the endodontic lesion is linedwith epithelium, is a granuloma, or is an abscess (Figure 1b).Clini cians who have performed endodontics day-in and day-out know this myth size to be false.

My clinical reality is that large LEOs do heal. It is thequality of the endodontic seal that determines en dodontichealing, not the size of the lesion. Large or small, the rationalefor endodontics remains the same biology.

ENDODONTIC MYTH NO. 2“The Root Canal System Is Impossible To Disinfect”Experiments and studies in the 1970s by Gary Grey, thenan endodontic resident at Boston University GraduateEndodontics, demonstrated that an hour per canal ofmanual canal serial filing and reaming, followed byconfirming patency, followed by recapitulation (sequentialre-entry of previous files and reamers) and Gates Gliddendrills with copious sodium hypochlorite irrigation produced

clean histologic root canal radicular preparations (Figure 2). While current irrigation protocols, irrigation solutions,

and activation systems are improving the effectiveness and


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Ten Myths About Endodontics: “Fact Versus Pulp Fiction”

Figures 2a to 2c.Myth No. 2: “Theroot canal systemis impossible todisinfect.” (a)Shaped canals arestill not cleaned asevidenced bydebris still presentin isthmusbetween MB andML canals of amandibular molar.(b and c) Cleanhistological crosssections from a1970s graduatethesis after onehour of canalcleaning andpreparation.

b

a

c

Figure 3. Myth No.3: “Minimallyinvasiveendodontics (MIE)is the newendodonticbenchmark.” Thisis an image of amaxillary molardemonstrating 3separate MB canalconefits. Aconservativerestricted accesswould have likely

prevented the successful discovery and treatment of the 3 MB canals.Thus, MIE to this patient means an access that is as small asappropriate, not as small as possible.

predictability of root canal systemdisinfection, studies suggest that 100%disinfection cannot yet be achieved due tobiofilm structure, evolving resistant bacterialstrains, and anatomic culs-de-sac that areunreachable and protected from currentcleaning and irrigation technique.4,5 Thefuture of disinfection may very well rely onnanotechnology, laser, and/or techniquesand tools not yet thought of.

My clinical reality is that root canalsystems can be effectively and sufficientlycleaned if the clinical time and propertechnique are practiced. Can endodontics dobetter? Always. Of the classic endodontictriangle of (1) disinfection, (2) preparation,and (3) obturation, disinfection is a mainfocus of current endodontic investigation andholds enormous promise for the “little tissue.”

ENDODONTIC MYTH NO. 3“Minimally Invasive Endodontics Is the NewEndodontic Benchmark”Minimally invasive endodontics (MIE) is today’s newbuzzword in endodontics. First of all, there is nothing newabout being conservative; however, what does conservativemean? What is MIE? MIE can be considered a broad termincluding vital pulp treatment, revascularization, coronalaccess, radicular canal shaping, and safe obturationtechniques. However, smaller accesses, for example,generate a potentially dangerous limited view of the pulpchamber, reduced lighting, and magnification. Endodonticmechan ics are hard enough to perform even in unrestrictedaccess let alone through “peep” holes.

Second, the word conservative in endodontics used tomean nonsurgical retreatment versus surgery. How ever, ifnonsurgical retreatment were to risk ferrule loss, or damagethe tooth or restorative structure, and/or aesthetics of anexisting foundation and crown, then a surgical endo donticseal could very well be considered conservative. Sur gerywould especially be conservative if the LEO was onlyapically present. In addition, if the endodontic preparationshape is too slender (such as in the old silver cone days)

and/or not sufficiently funnel shaped, then the capacity for3-D cleaning, shaping, and obturation is lessened and,while the coronal tooth structure is preserved, theprerequisite of filling the root canal system in unmet. This is,of course, a false sense of security and a misuse of thewords conservative and MIE. To save the crown and to losethe LEO is biologic suicide. Some re cent endodonticliterature suggests that most current endodontic shapes aretoo large and weaken the restorability of the tooth, and yetall the data is derived from facial views of overzealousshaping of precious ferrule measured by viewing a finishedresult from the typical buccal view. Re storative literatureverifies the essential part of ferrule preservation is facialand lingual and should be measured in the buccal-lingualaxial cone beam view. None theless, the final endodonticpreparation should take into consideration the finalcircumferential ferrule in order to preserve post-endodonticrestorability (Figure 3).

My clinical reality: With all due respect and in myhumble opinion, using to day’s ac cess, cleaning, andobturation techniques, the pendulum is swinging in thewrong direction. Modern straight-line un fet tered access andappropriate shapes for the root that house them does notcompromise success.


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Figures 4a and 4b. Myth No. 4: “Short endodontic fillings have a better prognosis than longendodontic fillings.” (a) Diagram shows 9 possible vertical and 3-D combinations ofendodontic obturation. (b) Only D and G are examples of 3-D filling or overfilling. All other 7possibilities are examples of underfilling. (Hash marks represent gutta-percha obturation.)

a

b

9 Obturation possibilities.

How many are 3-D?

ENDODONTIC MYTH NO. 4 “Short Endodontic Fillings Have a Better PrognosisThan Long Endodontic Fillings”While most studies suggest it is better to be short than long,6

the reality may come down to the definitions of long and short(Figure 4a). Is it a vertical measurement or a 3-dimensionalmeasurement? Dr. Herbert Schilder was the first to make alength versus volume distinction as early as the 1970s bysuggesting that “most overfillings are in reality, overextensionsof underfilled canals.” Schilder further defined overfill assealed in 3-dimensions and surplus material. When you thinkabout it, why is “overfilling” a clinical outcome anyway? It isbecause the geometric relationship between canal shape andconefit do not match, due to failure to create deep funnelcanal radicular preparation, or a “false tugback” conefit.Consequently, there is lack of obturation control withsubsequent material beyond the undersealed physiologicterminus. In fact, the only way to fill an endodontic preparationis to either fill it or overfill it (Figure 4b). Any vertical orhorizontal root canal system filling, that is neither filled oroverfilled, is not filled.

My clinical reality is that the only way to create the 3-D endodontic seal is to in fact create the 3-D endodonticseal or overseal. Anything short of the 3-D endodontic sealis undersealed, simply having the capacity to create or notprevent LEOs.

ENDODONTIC MYTH NO. 5 “Multivisit Endodontic Treatment Is More SuccessfulThan Single-Visit Endodontic Treatment” The general thinking behind multi-endodontic visits is that theyprovide an opportunity to place an intracanal medicament, such ascalcium hydroxide placement, for killing remaining bacteria. Wealready know that sodium hypochlorite kills all bacteria within 30seconds, including the AIDS virus. Well-shaped canals are cleancanals, and shaping facilitates cleaning, and 3-D obturationincarcerates any remaining bacteria (Figure 5a). In addition, andperhaps more importantly, the optimal time to know the criticalrelationship between conefit and shape validation is at the conefitvisit. Shaping is finished when the conefit fits (Figure 5b). Theclinician will never know the shape better than right now at the endof endodontic mechanics.

My clinical reality is that the best time to cement a crown

is when the crown fits. The best time to pack the root canalsystem is when the cone fits. The only clinically validcontraindication is if the pa tient’s endodontic tooth were stillsymptomatic.


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Figures 6a and 6b. MythNo. 6: “Previousendodontics has one or 2strikes against it andtherefore the toothshould be removed andtypically replaced with animplant.” (a) Pretreatment of toothtreatment planned forremoval andreplacement. Tooth had 2strikes against it:nonsurgical and surgicalattempt. Just oneproblem—the patient didnot want an implant. (b)Five-month post-treatment ofnonsurgical retreatmentof previousnonsurgical/surgicaltreatment. LEO isradiographically smallerand the patient is now

asymptomatic. What would you have wanted to do (attempt) if it wereyour tooth? For the author, I would have done what this patient hoped for.An implant can always be placed, and implants will be even morepredictable in a few years if the crown/root ratio fails. Remember, though,post-orthodontic short roots usually last the patient’s lifetime.

a

b

Figures 5a and 5b. MythNo. 5: “Multivisitendodontic treatment ismore successful thansingle-visit endodontictreatment.” (a) Conefit. (b) Finishedobturation at same visitas the conefit. Local rootcanal system knowledgeand needed finesse arehighest at the moment of

conefit and is, therefore, the most desirable time to perform the optimalobturation skill.

a b

Figure 7. Myth No. 7:“Maxillary molars have 4canals more than 90% ofthe time.” Example of 4separate canals cleaned,shaped with ProTaperGold (DENTSPLY TulsaDental Specialties), andobturated by verticalcompaction of warmgutta-percha. (Courtesyof Dr. Jordan West.)

ENDODONTIC MYTH NO. 6 “Previous Endodontics Has One or TwoStrikes Against It and, Therefore, theTooth Should Be Removed and TypicallyReplaced With an Implant”In reality, the same predic tability and properconcomitant mechanics have the sametreatment rationale as original endodontictreatment, al though a higher skill set and ahigher degree of intention are required. How -ever, when a nonsurgical retreatment re sults inthe removal of too much tooth structure,rendering the tooth unrestorable, an endodonticthird strike does warrant removal andreplacement. But, this error is not inevitablesince careful disassembly management, pri orto new shaping, is im perative and can bepredictably achieved. In addition, endo dontictreatment, in clu ding re treatment, has successlevels comparable to im plants, and also withbetter patient acceptance7-9 (Fig ure 6).

My clinical reality is that the capacity forsuccessful endo dontic retreatment is thesame as the capacity for endodonticnonsurgical treatment: 100% capacity. Theonly difference is the technical skillsfrequently need to be taken to another level.

ENDODONTIC MYTH NO. 7 “Maxillary Molars Have Four Canals MoreThan 90% of the Time”If you ask any endodontic audience whatpercentage of maxillary molars have 4 canals,most feel obliged to say more than 90%. Theclinical reality and the literature suggest asignificantly different expectation: that, while most teethdemonstrate a fourth chamber orifice, less than half of theseorifices lead to separate canals (Figure 7).10-13 So keeplooking diligently, but do not beat yourself up if you cannot“follow” the canal to length as it may very well unite or crosswith MB1. Nonetheless, a significant lateral portal of exit (POE)could emanate from the coronal portion of MB2, so don’t giveup carefully following into MB1; and, if this were the case, then

shape it and pack it with the same precision as MB1. My clinical reality is that maxillary molars must be treated

as if they possess 2 or more separate mesiobuccal canals.However, the fact is that as many as half do converge one wayor another. This is no reason not to successfully find, follow, andfinish these canals as they may converge and then emergeseparately again. In addition, a significant POE may branch fromthe converging canal itself.


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a b

Figures 8a to 8d. Myth No. 8: “Endodontically treated teeth discolor in the aesthetic zone.”(a) Clinical pre-op photo of necrotic and discolored maxillary left central incisor. (b) Post-opphoto at 12 years showing no color change. In fact, the tooth is still lighter than adjacentcentral incisor. (c) Careful restorative layering and polishing prevented microleakage andtherefore prevented re-discoloration. (d) Lingual view showing no microleakage aroundlingual access repair margin.

c d

Figures 9a to 9c. Myth No. 9: “Endodontically treated teeth are weaker.” (a) Pretreatmentimage of maxillary right central incisor. Note damage to crown has already occurred as aresult of caries, not endodontics. (b) Beginning of backpack following shaping with ProTaperGold and vertical compaction of warm gutta-percha. (c) Endodontic Finish where theendodontic access and radicular shape have not weakened the tooth in any way.

a b c

ENDODODONTIC MYTH NO. 8 “Endodontically Treated Teeth Discolor in theAesthetic Zone” Why do some endodontically treated teeth discolor?Discoloration is due to insufficient coronal seal, failure toproperly remove necrotic tissue, failure to finish obturation oneto 2 mm apical to the cemento-enamel junction, or the failureto clean sealer and/or obturation material from the pulpchamber access (Figure 8a).

My clinical reality is that, if properly restored,endodontically treated teeth do not readily re-discolor, sincemicroleakage will be prevented or significantly delayed. Infact, if the same focus, technique, and attention to detail isplaced on access finishing as the original access cavity,such as layering the access restorative materials andemploying a proper polishing protocol, then endodonticallytreated teeth will retain their restored color (Figure 8).

ENDODONTIC MYTH NO. 9 “Endodontically Treated Teeth Are Weaker”The teeth that require endodontics are weakened before

the endodontics due to caries and/or previous caries andlarge and deep restorations (Figure 9a). The endodonticaccess, like removing the top of the Duomo from the SantaMaria del Fiore Cathedral (in Florence, Italy) has littleinfluence on the strength of the structure. The endodonticaccess is almost insignificant compared to the damage ofrestorative procedures14 (Figures 9b and 9c).

My clinical reality is that the microscope-designedendodontic access cavity and root canal radicular preparationsdo not cause a tooth to be weaker post-endodontic treatment.“Weak ness” is caused, instead, by what happens to the toothbefore endodontics is needed: caries, subsequent restorativecavity preps, followed by the restoration itself.

ENDODONTIC MYTH NO. 10 “The Ni-Ti System That I Use Makes the BiggestDifference” Every instrument company in the world reports that they makethe best Ni-Ti shaping system. At last count, there are morethan 40 systems, but they seem to come and go frequently.They can’t all be right, can they? So what and where is the


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Figures 10a to 10f. Myth No. 10: “The Ni-Ti system I use makes the biggest difference.” (a) Pretreatment of mandibular left second molar.(b) Endodontic Finish using ProTaper Gold. Note: MIE access! (Courtesy of Dr. Jason West.) (c) Endodontic Finish using ProTaper Gold.Note elegant shapes follow the original anatomy. (Courtesy of Dr. Jordan West.) (d) Pretreatment image of maxillary right first premolar. (e)Endodontic Finish, again shaped with ProTaper Gold. (f) Second molar ProTaper Gold shaping and obturation with vertical compaction ofwarm gutta-percha. If this were your tooth, would you prefer the first molar result or the second molar? Of course, the second molar has nothad the time to heal yet, but I will bet my professional reputation that it will!

a b c

d e f

truth? It is my thinking that the best Ni-Ti system cannot makea “bad” dentist a “good” dentist. However, the best Ni-Ti systemcan make a “good” dentist a “great” endodontic clinician.

I have been an endodontic clinician and educator formore than 35 years. What I love about my work is that I amstill learning and still improving my performance. I am aspassionate about the processes and results as I was whenI started. Now, if you have read this article this far, it is atthis time that I am obligated to share something: Ni-Ti hasmade the biggest difference. It is amazing how far improvedNi-Ti design has come in the last 5 years, but without adoubt, the foremost and most recognizable name inendodontic shaping has and continues to be ProTaper[DENTSPLY Tulsa Dental Specialties]. Why? ProTaper has3 critical distinctions: (1) it produces predictable andreproducible deep shape for easy 3-D obturation; (2) it isthe only system that understands the tageting role ofShapers for shaping coronal restrictive dentin and the roleof Finishers to finish “connecting the dots of the radicularcontinuously tapering funnel” from apical constriction tocanal orifice while maintaining MIE preparations (Figures10a to 10c); and (3) the shaping sequence is safe, super-efficient, and simple since it is always the same. And yetsome clinicians experienced the Fin ishers as too efficientand, given new metallurgies, not flexible enough. Problemsolved! The new ProTaper Gold (DENTSPLY Tulsa DentalSpecial ties) allows Sha pers and Finishers to easily andsafely crawl down canals and carve deep shapes that areconsistent and appropriate for MIE endodontics, while atthe same time providing perfect funnel preparations foreasy 3-D cleaning and obturation15 (Figures 10d to 10f).

My clinical reality is that there is no doubt that theclinician is the greatest variable in endodontic success.However, when the best Ni-Ti concepts and geometries areembraced by clinicians who are dedicated to being theirbest, the best happens.

LESSONS LEARNEDPreparing this article reinforceded 4 lessons that I havelearned along the way.

1. Diagnosis. Proper diagnosis is key in deciding theexact mode of endodontic treatment or if endodontictreatment is in the patient’s best interest.

2. Treatment Planning. The value of developing anexperienced team of specialists cannot be overestimated.Inter disciplinary collaboration gives the patient the besttreatment plan choices and decisions.

3. Endodontic Mechanics. An understanding of thephysiology and biology of endodontic healing and success isimperative. In this way, misleading myths will play a reducedrole and endodontics will retain its rightful place in providingoptimal oral health.

4. Appropriate Endodontics. The author of this article hastreated endodontic patients for nearly 40 years, and it is clearthat endodontics has improved during this time period.Endodontics is now less invasive. Endodontics is moreprecise. When possible, nonsurgical endodontic retreatmentshould be chosen over surgical retreatment if restorativestructure or anterior aesthetics is not compromised. Thereare some extremely difficult cases that are best treated withremoval and replacement. How ever, well-planned and well-executed endodontics will reduce trauma to the patient,facilitate endodontic success, and improve post endodonticpredictability and stability. When the right tooth is chosen,when the right endodontics is done right, endodontic mythsvaporize and improved biologic, structure, functional, andaesthetic outcomes become an attainable norm for thedentist and his or her interdisciplinary team.

REFERENCES1. Hargreaves KM, Goodis HE, eds. Seltzer and

Bender’s Dental Pulp. Chicago, IL: QuintessencePublishing; 2002.

2. Ng YL, Mann V, Gulabivala K. Outcome of secondaryroot canal treatment: a systematic review of theliterature. Int Endod J. 2008;41:1026-1046.

3. Ng YL, Mann V, Gulabivala K. A prospective study ofthe factors affecting outcomes of nonsurgical rootcanal treatment: part 1: periapical health. Int Endod J.2011;44:583-609.

4. Ricucci D, Siqueira JF Jr, Bate AL, et al. Histologicinvestigation of root canal-treated teeth with apicalperiodontitis: a retrospective study from twenty-fourpatients. J Endod. 2009;35:493-502.

5. Ricucci D, Siqueira JF Jr. Fate of the tissue in lateralcanals and apical ramifications in response topathologic conditions and treatment procedures. J Endod. 2010;36:1-15.

6. Ng YL, Mann V, Rahbaran S, et al. Outcome of


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primary root canal treatment: systematic review of theliterature—Part 2. Influence of clinical factors. IntEndod J. 2008;41:6-31.

7. Doyle SL, Hodges JS, Pesun IJ, et al. Retrospectivecross sectional comparison of initial nonsurgicalendodontic treatment and single-tooth implants. JEndod. 2006;32:822-827.

8. Iqbal MK, Kim S. For teeth requiring endodontictreatment, what are the differences in outcomes ofrestored endodontically treated teeth compared toimplant-supported restorations? Int J Oral MaxillofacImplants. 2007;22(suppl):96-116.

9. Gatten DL, Riedy CA, Hong SK, et al. Quality of life ofendodontically treated versus implant treated patients:a University-based qualitative research study. JEndod. 2011;37:903-909.

10. Kim Y, Lee SJ, Woo J. Morphology of maxillary firstand second molars analyzed by cone-beam computedtomography in a Korean population: variations in thenumber of roots and canals and the incidence offusion. J Endod. 2012;38:1063-1068.

11. Silva EJ, Nejaim Y, Silva AI, et al. Evaluation of rootcanal configuration of maxillary molars in a Brazilianpopulation using cone-beam computed tomographicimaging: an in vivo study. J Endod. 2014;40:173-176.

12. Weller RN, Niemczyk SP, Kim S. Incidence andposition of the canal isthmus. Part 1. Mesiobuccal rootof the maxillary first molar. J Endod. 1995;21:380-383.

13. Stropko JJ. Canal morphology of maxillary molars:clinical observations of canal configurations. J Endod.1999;25:446-450.

14. Reeh ES, Messer HH, Douglas WH. Reduction intooth stiffness as a result of endodontic andrestorative procedures. J Endod. 1989;15:512-516.

15. Ruddle CJ, Machtou P, West JD. Endodontic canalpreparation: new innovations in glide pathmanagement and shaping canals. Dent Today.2014;33:104-107.


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POST EXAMINATION QUESTIONS

1. Based on results, the large endodontic lesionclinically heals, regardless if the endodontic lesion islined with epithelium, is a granuloma, or is anabscess.

a. True b. False

2. Of the classic endodontic triangle of (1) disinfection,(2) preparation, and (3) obturation, disinfection is amain focus of current endodontic investigation andholds enormous promise for the “little tissue.”

a. True b. False

3. With today’s composites and resin cementsimproving in strength, the final endodonticpreparation does not need to take into considerationthe final circumferential ferrule in order to preservepost-endodontic restorability.

a. True b. False

4. Any vertical or horizontal root canal system fillingwhich is neither filled nor overfilled, is not filled.

a. True b. False

5. We already know that sodium hypochlorite kills allbacteria within 30 seconds, but not the AIDS virus.

a. True b. False

6. If the same focus, technique, and attention to detailis placed on access finishing on the original accesscavity, then endodontically treated teeth will retaintheir restored color.

a. True b. False

7. The teeth that require endodontics are weakenedbefore the endodontics due to caries and/or previouscaries and large and deep restorations.

a. True b. False

8. In the author’s opinion, the clinician is the greatestvariable in endodontic success.

a. True b. False


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Ten Myths About Endodontics: “Fact Versus Pulp Fiction”