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Cardiac Cycle
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Dr. QURBA KIRAN
DPT
Physiology
PRESENTATION
Cardaic cycle
Cardaic cycleCARDAIC CYCLE
Cardiac Cycle
• The cardiac events that occur from beginning of one heart beat to the beginning of the next are called the cardiac cycle.
• Initiated by spontaneous generation of AP in SA node.
• Atria act as PRIMER PUMPS for ventricles & ventricles provide major source of power for moving the blood through the vascular system.
Cardiac Cycle
• Cardiac cycle refers to all events associated with blood flow through the heart– Systole – contraction of heart muscle– Diastole – relaxation of heart muscle
ATRIAL SYSTOLE
the end of diastole
Atrial Systole Blood normally flows continually
from great veins into arteria 75% flows directly the atria into ventricle before the arteria contracts. 25% of filling of ventricles – arterial
contraction Arterial contraction is complete
before the ventricle begins to contract.
ISOVOLUMETRIC CONTRACTIONPressures & Volumes
• The AV valves close when the pressure in the ventricles (red) exceeds the pressure in the atria (yellow).
• As the ventricles contract isovolumetrically -- their volume does not change (white) -- the pressure inside increases, approaching the pressure in the aorta and pulmonary arteries (green).
Atrial SystoleECG
• p wave – atrial depolarization• impulse from SA node results in depolarization &
contraction of atria ( Rt before Lt )• PR segment – isoelectric line as depolarization
proceeds to AV node.• This brief pause before contraction allows the
ventricles to fill completely with blood.
Atrial SystoleHeart Sounds
• S4 - end of arterial emptying after arterial contraction. • rapid emptying of atrium into non compliant ventricle• physiological : children , young adults • pathological : angina, MI,
Beginning of Ven.SystoleIsovolumetric Contraction
• The atrioventricular (AV) valves close at the beginning of this phase
• Mechanically, ventricular systole is defined as the interval between the closing of the AV valves and the opening of the semilunar valves (aortic and pulmonary valves).
• Electrically, ventricular systole is defined as the interval between the QRS complex and the end of the T wave (the Q-T interval).
Isovolumetric ContractionPressure & Volume Changes
• The AV valves close when the pressure in the ventricles (red) exceeds the pressure in the atria (yellow).
• As the ventricles contract isovolumetrically -- their volume does not change (white) -- the pressure inside increases, approaching the pressure in the aorta and pulmonary arteries (green).
Isovolumetric ContractionECG
• The QRS complex is due to ventricular depolarization, and it marks the beginning of ventricular systole.
Isovolumetric ContractionHeart Sounds
• S1 is d/t closure of AV Valves .
Ejection
• The Semilunar valves ( aortic , pulmonary ) open at the beginning of this phase.
• This at first 1/3 rapid ejection (70%) & remaining 2/3 slow ejection.(30%)
ECG & Heart Sounds• In rapid ejection part of the
ejection phase there no specific ecg changes / heartsounds heard.
Slow Ejection
• At the end of ejection, the semilunar valves close.
ECG & Heart Sounds
• T wave – slightly before the end of ventricular contraction
• it is d/t ventricular repolarization
• heart sounds : none
Rapid Inflow ( Rapid Ven. Filling)
• Once AV valves are open the blood that has accumulated in atria flows into the ventricle
Volume changes
• Ventricular volume increases rapidly as blood flows from the atria into the ventricles.
Rapid Inflow ( Rapid Ven. Filling)ECG & Heart Sounds
• ECG : no deflections• Heart sounds : S3 is heard –
Initial passive filling of ventricles
• physiological : children, atheletes, pregnancy
• pathological : High output states, ASD,VSD
REDUCED VENTRICULAR FILLING (Diastasis)
Diastasis
• remaining blood which has accumulated in atria slowly flows into the ventricle.
DiastasisVolume changes
• Ventricular volume increases more slowly now. The ventricles continue to fill with blood until they are nearly full.
ECG & Heart Sounds
• ECG : no deflections• Heart Sounds : none
COMPLETE PICTURE OF CARDAIC CYCLE
Phases of the Cardiac Cycle
Figure 18.20