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NURSING CARE OF THE CLIENT: RESPIRATORY SYSTEM
Nursing Dx: Respiratory Dysfunction
Ineffective Airway Clearance
Impaired Gas Exchange Ineffective Breathing
Pattern Impaired Verbal
Communication
Activity Intolerance
Anxiety Altered Nutrition:
Less than body requirement
Risk for Infection
Respiratory System Its primary
function is delivery of oxygen to the lungs and removal of carbon dioxide from the lungs.
Respiration Process of gas exchange Supply cells with oxygen for carrying on
metabolism Remove carbon dioxide produced as a
waste by-product. Two types of respiration: external and
internal.
Respiratory Assessment
A u scu lta tion(L is te n ing fo r N o rm a l a n d A d ve n tit io u s B rea th S o un d s)
P a lpa tion a nd P ercuss ion
In sp e ction(c lie n t 's co lo r, le ve l o f con sc iou sn ess , em o tion a l sta te )
(R a te , d ep th , q u a lity, rh ythm , e ffo rt re la tin g to resp ira tio n )
H e a lth H is to ry(a lle rg ie s , o ccup a tion , lifes tyle , h e a lth ha b its)
Assessment Review
Vital Signs Respiratory rate & heart rate WNL
Oxygen saturation of 95% or higher
Assessment ReviewPhysical Assessment Speak a sentence of 12 words without
stopping for breath Walk and talk without stopping for breath No cyanosis, pallor, or jaundice Oral mucus membrane & nail beds pink with
rapid capillary refill
Assessment Review Fingertips and nails normal shape, no
clubbing Anterior & posterior diameter of chest 2/3
smaller than lateral diameter Space between each rib larger than breath
of patient’s finger Breathes in through nose & out through
mouth & nose
Assessment Review Breathing quiet Air movement heard in all lobes of both
lungs Sputum production minimal, clear or
white Muscle development even with no
muscle loss on arms & legs Weight proportionate to height; not
underweight
Assessment Review
Psychological Assessment Oriented, not confused
Energy level good, can engage in desired work, recreational & personal activities
Assessment Review
Laboratory Assessment RBC Hemoglobin Hematocrit WBC
WNL for age & gender
Assessment: Inadequate Oxygenation
Resp rapid & shallow Respirations noisy Cannot speak >4 or 5 words without
pausing for breath Change in cognition, acute confusion Decreased oxygen saturation by pulse ox
Assessment: Inadequate Oxygenation
Skin cyanosis or pallor (lighter-skinned pts)
Cyanosis or pallor of lips or oral mucus membranes (pts of any skin color)
Tachycardia Appears to strain to catch breath Fatigue
Physical Assessment: Inadequate O2 Take vital signs Auscultate all lung fields Monitor O2 sat Check recent Hgb, Hct, ABGs Assess cognition Assess use of accessory muscles
Physical Assessment: Inadequate O2 Assess presence of thick or excessive
secretions Assess ability to cough and clear airway
Intervention: Inadequate Oxygenation
Apply O2 & assess response Elevate HOB 30 degrees Suction if needed Notify MD Priortize & pace activities to prevent
fatique
Assessing Lung Sounds
Adventitious Breath Sounds Fine crackles (dry, high-pitched popping…
COPD, CHF, pneumonia)
Coarse crackles (moist, low-pitched gurgling…pneumonia, edema, bronchitis)
Sonorous wheezes (low-pitched snoring…asthma, bronchitis, tumor)
Adventitious Breath Sounds Sibilant wheezes (high-pitched, musical …
asthma, bronchitis, emphysema, tumor)
Pleural friction rub (creaking, grating… pleurisy, tuberculosis, abscess, pneumonia)
Stridor (crowing…croup, foreign body obstruction, large airway tumor)
Diagnosing Respiratory Disorders
Laboratory Tests Hemoglobin Arterial blood
gases Pulmonary
Function Tests Sputum Analysis
Radiologic Studies Chest X-ray Ventilation-
perfusion scan CAT scan Pulmonary
angiography
Respiratory Disorders
Other diagnostic tests Pulse oximetry Bronchoscopy Thoracentesis MRI
Assessment: Upper Airway Problems Voice changes
nasal quality if above palate“breathy” or “whispery” if larynx or trachea
Snoring Mouth breathing
Assessment: Upper Airway Problems Change in cognition or LOC or acute
confusion Decreased O2 sat Skin cyanosis or pallor Cyanosis or pallor of lips or oral mucus
membranes Tachycardia & dysrhythmia
Physical Assessment: Upper Airway Problems
Take vital signs Monitor O2 sat Assess for presence of thick or excessive
secretions Assess ability to cough and clear airway Assess nasal drainage & sputum for
color & blood
Physical Assessment: Upper Airway Problems
Check WBC & ABG levels Assess cognition Assess hydration status
Intervention: Upper Airway Problems Suction Apply o2 & assess response Keep HOB elevated 30 degrees Notify MD Ensure venous access
Obstructive Sleep Apnea Intermittent absence of airflow through
mouth & nose during sleep
Occlusion of the oropharyngeal airway
Obstruction causes O2 sat, pO2, and pH to rise & pCO2 to rise
Obstructive Sleep Apnea
Obstructive Sleep Apnea Loud storing
during sleep Excessive daytime
drowsiness Irritability Restless sleep
Obstructive Sleep Apnea Restore airflow Prevent adverse
effects of disorder
Weight reduction Alcohol abstinence Improve nasal
patency Avoid prone
sleeping position
Obstructive Sleep Apnea Treatment of
Choice:Continous positiveairway pressure
(CPAP)
Obstructive Sleep Apnea Tonsillectomy Adenoidectomy
Obstructive Sleep Apnea Uvuloplatopharyngo
plasty
Obstructive Sleep Apnea Disturbed Sleep Pattern Fatigue Ineffective Breathing Pattern Impaired Gas Exchange Risk for Injury Risk for Sexual Dysfunction
Tracheostomy Bypass upper
airway obstruction 1. esophagus 2. trachea 3. tracheostomy tube
Tracheostomy Facilitate removal
of secretions
Tracheostomy
Manage long-term mechanical ventilation
Assessment: Infectious Resp Problems Resp shallow & rapid Decreased O2 sat Skin cyanosis or pallor Cyanosis or pallor of lips & oral mucus
membranes Tachycardia Work hard to inhale & exhale Restless anxious or confused
Physical Assessment: Infections Vital signs Auscultate all lung fields Monitor O2 sat Assess cognition Assess sputum Assess ability to cough & clear airway
Lab Values: Infections Elevated WBC ABG:
pH lower than 7.35HCO3 at or below 24 mmHgPaCO2 at or below 45 mmHgPaO2 below 90 mm Hg
Interventions: Infectious Resp Problems
Administer O2 Upright position with arms resting on
table or armrests Chest physiotherapy/pulmonary hygiene Pace activities to prevent fatigue
Interventions: Infectious Resp Problems
Administer IV, oral, or inhaled drugs Respiratory therapy treatments Reassess resp status after resp therapy Ensure fluid intake 3 liters/day
Sinusitis
Sinusitis
Pain & tenderness Headache, fever,
malaise Nasal congestion Purulent nasal
discharge Bad breath
Sinusitis: Medication Therapy Antibiotics
Oral or topical decongestants
Antihistamines
Saline nose drops or sprays
Systemic mucolytic agents
Sinusitis: Interdisciplinary Care Drain obstructed
sinuses Control infection Relieve pain Prevent
complications
Sinusitis Endoscopic sinus surgery
Sinus Surgery: Caldwell Luc procedure
Sinus Surgery: Antral irrigation
Sinusitis: Health Promotion Promote nasal drainage Encourage liberal fluid intake Judicious use of nasal decongestants Treat any obstructive process
Pneumonia Inflammation of lung parenchyma Infectious: Bacteria, viruses, fungal
protozoa Noninfectious: aspiration of gastric
contents, inhalation of toxic or irritating gases
Can be classified as community acquired, nosocomial, or opportunistic
Pneumonia: Signs & SymptomsPrimary Atypical PNA Fever Headache Myalgias Arthralgias Dry, hacking, non
productive cough
Viral PNA Flu-like symptoms Headache Fever Fatigue Malaise Muscle aches
Pneumonia: Signs & Symptoms
Pneumocystis PNA
Opportunistic infection
Abrupt onset Fever Tachypnea SOB
Dry, nonproductive cough
Respiratory distress
Intercostal retractions
Cyanosis
Pneumonia
Interdisciplinary care
Prevention Pneumococcal
vaccine Influenza vaccine
Medications Antibiotics Bronchodilators Agents to liquefy
mucus
Pneumonia
Treatment Oxygen therapy Chest
physiotherapy
Nursing Diagnosis Ineffective airway
clearance Ineffective
breathing pattern Activity
intolerance
Theresa A 20 year old college student Lives in a small dormitory with 30 other
students. Four weeks into the Spring semester, she
was diagnosed with bacterial pneumonia Admitted to the hospital
Teresa: High Priority Intervention Specimens for culture are taken prior to
beginning the antibiotic
Administering prior to cultures may make it impossible to determine the actual agent causing the pneumonia.
Theresa: Bacterial Pneumonia
Sputume culture results most frequent strain of found in
community-acquired pneumonia Streptococcus pneumoniae
Teresa: Clinical Manifestations Fever
stabbing or pleuritic chest pain
tachypnea
Elderly Weakness Fatigue lethargy Confusion poor appetite
without classic s & s
Treatment: Bacterial Pneumonia Started on Penicillin G
Response between 1 & 2 days
Complications of Pneumonia Atelectasis
Hypotension & shock
Pleural effusion
Impaired gas exchange
Pneumonia: Impaired Gas Exchange Results in hypoxia
Earliest sign and symptom of which is a change in the level of consciousness.
Interventions Oxygen by nasal cannula Plan for periods of rest during activities
of daily living. Monitor pulse oximetry readings every 4
hours. What oxygen delivery system would be
most effective for Theresa?
Nasal Cannula
Low flow delivery device 2 l/min = ~28% Higher flow rates (>5 l/min) dry nasal
membranes
Simple Face Mask
Flow rates 6-12 l/min Delivers 35-50% O2 Pt comfort issues (Maybe used for
Mr. Howe if SOB)
Non-Rebreathing Mask
Delivers accurate, high concentrations of oxygen
Achieves 60-90% O2 delivery
Oxygen Conserving Cannula
Built in oxygen reservoir
30-50% O2 delivery Increased comfort
Nebulizers/Humidifiers 02 is drying to mucous membranes Nebulizers
Bubble-through humidifier >4 l/min
Humidifiers Heated water
Tuberculosis Infection of the
lung tissue
Mycobacterium tuberculosis
TuberculosisSpread through
dropletnuclei: Coughing Sneezing Speaking Singing
Tuberculosis: Risk Factors Overcrowded, poor
living conditions Poor nutritional status Previous infection Inadequate treatment
of primary infection leads to multi-drug resistant organisms
Close contact to infected person
Immune dysfunction; HIV infection
LTC facilities, Prisons
Elderly Substance abuse
Tuberculosis
Caseation necrosis Inhaled bacteria multiply Tubercle is formed Infected tissue dies Cheeselike center forms
TuberculosisIf patient has adequateimmune response: Scar tissue develops
around tubercle Walls off bacilli Infected, does not
develop TB
Inadequate immuneresponse TB can develop
rapidly
Reactivation TB
Suppressed immune system due to Age Disease Use of immunosuppressive drugs
Tuberculosis: Signs & Symptoms Fatigue Weight loss Anorexia pm fever
Dry cough Later productive,
purelent/blood tingled
Night sweats
Tuberculosis: Interdisciplinary Care Early detection Accurate diagnosis Effective disease
treatment Preventing spread
to others
Tuberculin test Intradermal PPD
(Mantoux) test Multiple-puncture
(tine) testing
TB: Goals of Medication Treatment Make the disease noncommunicable to
others
Reduce symptoms of the disease
Affect a cure in the shortest possible time
Tuberculosis: Nursing Diagnosis Deficient Knowledge
Ineffective Therapeutic Regimem Management
Risk for Infection
Mr. Howe c/o dyspnea progressive wt
loss for several months
Productive cough Night sweats
“wringing wet”
Dx: R/O TB What additional
questions should you ask about Mr. Howe’s cough?
Assessing Cough How it feels How bad it is What makes it better or worse When it started Amount, color, odor, and consistency of
sputum
Mr. Howe Diagnostic test
expected for patient
Mantoux test Sputum for acid-
fast bacillus Chest X-ray History and
Physical Examination
Mantoux Test Positive result only indicate exposure or
has received BCG immunization
BCG immunization: Eastern Europe and countries where TB is endemic
Is not diagnostic for active TB
Mantoux Test Give upper 1/3 surface of the forearm Needle is inserted with bevel up 0.1 ml of purified derivative (PPD) inserted
intradermally) Read 48-78 hrs Induration 1.5 mm or greater is + (HIV or
immunosuppressed pts 5 mm or greater +
Sputum Studies Sputum Samples
Expectoration tracheal suction
Bronchoscopy Used to
identify infecting organisms
Confirm presence of malignant cells
early morning 15 ml required Obtain prior to
antibiotics Ask pt to rinse
mouth before collecting specimen
Mr. Howe: Bronchoscopy ordered
Preparation Informed consent NPO after midnight Explain procedure, obtain baseline vs &
ABG Atropine may be ordered to dry secretions
Bronchoscopy
Mr. Howe: Post Bronchoscopy
Complications Aspiration
Infection
Pneumothorax
Mr. Howe: Post Bronchoscopy Care NPO until gag reflex Monitor vital signs Assess for dyspnea, hemoptysis, & tachycardia Notify MD if fever, difficulty breathing Semi-Fowler’s position Give H2O as first fluid Inform pt of possible expectoration of blood
tingled mucus
Tuberculosis: Drug Therapy
Mr. Howe’s Medication Regime Chemotherapy are
all Hepatotoxic
Ethambutol optic neuritis skin rash
Rifampicin n/v Thrombocytopenia turns all bodily
secretions a red-orange color (tears, sweat, etc)
Mr. Howe’s Medication RegimeINH peripheral neuritis
(take Vitamin B 6 in conjunction to prevent)
hepatotoxicity GI upset
Streptomycin 8th cranial nerve
damage routine hearing
test caution in renal
disease
Mr. Howe’s Medication RegimePyrazinamid Heptoxicity hyperuricemia monitor uric acid & hepatic function
Mr. Howe’s Hospital Care Teach handwashing, cover nose and
mouth when coughing, sneezing Droplet Isolation-negative pressure room Special particulate respirator mask Psychosocial support-reinforce need to
take medication
Mr. Howe’s Teaching Plan Preventive measures to avoid catching
viral infections Taken drugs in combination to avoid
bacterial resistance Take meds at the same time of day on an
empty stomach Follow med regimen 6-12 months as
prescribed
Mr. Howe’s Teaching Plan Adequate nutritional status Annual check-up Annual Check-up: liver function tests Notify MD if signs of hepatitis,
hepatoxicity, neurotoxicity, & visual changes occur
Thoracentesis Used to obtain pleural fluid
for analysis Needle inserted between
ribs second and third intercostal spaces
Fluid withdrawn with syringe or tubing connected to sterile vacuum bottle
ThoracentesisPre-Procedure Informed consent-
explained & signed
Inform about pressure sensations that will be experienced during the procedure
Baseline vital signs
Make sure that a CXR has been completed
Thoracentesis: Positioning Lying on the unaffected side with the
bed elevated 30 – 40 degrees Sitting on the edge of the bed with her
feet supported and her arms and head on a padded overbed table.
Straddling a chair with her arms and head resting on the back of the chair.
Post Thoracentesis Apply pressure to
puncture site Assess bleeding &
crepitus Semi-fowlers or
puncture site up
Monitor for blood-tingled mucus
Assess for hypoxemia,
Assess for tachycardia
Assess breath sounds
Why is a chest x-ray ordered post procedure?
Assessment: Lower Resp Problems Resp shallow and rapid Decreased oxygen saturation Skin cyanosis or pallor Cyanosis or pallor of lips & mucus
membranes Tachycardia Work hard to inhale & exhale
Assessment: Lower Resp Problems Restless & anxious Thin compared to height Muscles of neck appear thick Arm & leg muscles appear thin Clubbed fingers Chest is barrel shaped Rib space more than a finger breath apart
Physical Assessment: Lower Resp Problems
Take vital signs Monitor O2 sat Assess cognition Assess sputum Assess ability to cough & clear airway
Lab Values: Lower Resp Problems Elevated RBC, HCT, HGB Elevated WBC ABGs
ph <7.35HCO3 > 24mm HgPCO2 > 45 mm HGPaO2 < 80 mm Hg
Interventions: Lower Resp Problems Upright position Chest Physiotherapy O2 low to maintain resp of 16 breaths minute Pace activities Administer inhaled drugs Respiratory therapy Fluid intake at least 3L daily
Bronchitis Common in adults
Risk factors Impaired immune
defenses Cigarette smoking
Acute bronchitis follows a viral URI
Chronic bronchitis is a component of COPD
Bronchitis Viral, bacterial
or inflammatory Irritants cause
increased mucus production and mucosal irritation
Acute Bronchitis
Bronchitis: Signs & Symptoms Non-productive
cough
Later becomes productive
Paroxysmal cough
Chest pain
Moderate fever
General malaise
Bronchitis
Treatment Symptomatic Rest Increased fluid
intakeNursing Intervention teaching
Medications ASA or tylenol Broad spectrum
antibiotic Cough
expectorant
Asthma Chronic inflammatory disorder of the
airways
Brief (acute asthma fatal)
Persistent irritation of the airways
Asthma: Risk Factors Allergies Family history occupational exposure Respiratory viruses Exercise in cold air Emotional stress
Asthma: Triggers Allergens Resp tract infection Exercise Inhaled irritants Secondhand smoke Medications
Asthma: Acute/early response Vasoconstriction
Edema
Mucus production
Asthma: Patho Inflammatory
mediators released
Activation of inflammatory cells
Bronchoconstriction
Airway edema
Impaired mucus clearing
SOB trapping of air
impairs gas exchange
Asthma: Signs & Symptoms Chest tightness Cough, dyspnea,
sheezing Tachycardia,
tachypnea, prolonged expiration
Fatigue, anxiety apprenhension
Respiratory failure Breath sounds
may improve right before failure
Asthma: Treatment Control symptoms Prevent acute
attacks Restore airway
patency Restore alveolar
ventilation
Long term control Anti-infammatory
agents Long acting
bronchodialators Leukotriene
modifiers
Asthma: Treatment
Quick relief Short acting
adrenergic stimulants
Anticholinergic drugs
Methylxanthines
Administration methods
Metered-dose inhaler (MDI)
Dry powder inhaler (DPI)
Nebulizer
Chronic Obstructive Pulmonary Disease
A collective term used to refer to chronic lung disorders
Air flow into or out of the lungs is limited
John Emphysema for 25 years
H/O smoking Diagnosis: Bronchitis
John: Cigarette Smoking Major causative factor in the
development of respiratory disorders lung cancer cancer of the larynx Emphysema chronic bronchitis
During assessment you note the presence of a “barrel chest”.
“air trapping” in the lungs
Barrel Chest
Slow progressive obstruction of airways Airways narrow Resistance to airflow increase Expiration slow and difficult Result: mismatch between alveolar
ventilation and perfusion, leading to impaired gas exchange
Major symptoms to assess John for
You should be alert for the followingpresenting symptom of COPD?
Increased dyspnea Sputum production
EmphysemaJohn is medicated with a bronchodilator to reduceairway obstruction. Assess for Dysrhythmias Central nervous system excitement Tachycardia
Purse Lip BreathingRecommended for John to: Decrease respiratory
rate
Increase alveolar ventilation
Reduce functional residual capacity
Venturi Mask is prescribed for John because: Moderate Oxygen Flow Delivers precise, high-
flow rates 24%-50%
Humidification available Requires face mask
BronchiectasisA chronic dilation of thebronchi caused by: pulmonary TB
infection chronic upper
respiratory tract infections
complications of other respiratory disorders
Obstruction of a pulmonary artery by a bloodborne substance
Pulmonary Embolism:
Common Cause: Deep vein
thrombosis
Pulmonary Embolism
Other sources of Pulmonary Emboli Fat Emboli
From fractured long bones Air Emboli
From IVs Amniotic fluid Tumors
Mrs. Perkins Mrs Perkins is suspected of having a
pulmonary embolus.
What diagnostic test confirms this diagnosis?
Pulmonary Embolism The plasma D-dimer test is highly specific for
the presence of a thrombus. An elevated d-dimer indicates a thrombus
formation and lysis.
What assessment data would support that Mrs. Perkins has experienced a pulmonary embolus?
Clinical Manifestations of Pulmonary Embolus
Sudden, unexplained dyspnea, tachypnea or tachycardia
Cough Chest pain Hemoptysis Sudden changes in mental status
(hypoxia)
Diagnosing Pulmonary Embolism Ventilation-Perfusion Scan
Nuclear imaging test Determines percentage of each lung that is
functioning normally
Pulmonary Angiography
Pulmonary Embolism
Mrs. Perkins pulse oximetry has decreasedto 90%. What does this indicate?
The normal pulse oximeter reading is 93% - 100%.
A reading of 90% indicates Mrs Perkins has an
arterial oxygen level of about 60
Pulmonary Embolism
With a diagnosis of PE, what intervention is crucial for
Mrs. Perkins?
Institute and maintain bedrest Bedrest reduces metabolic demands and
tissue needs for oxygen.
Management: Pulmonary Emboli Anticoagulation therapy
Heparin Coumadin for ~6 months
Thrombolytic therapy Use very cautiously only for acute, massive
PE Urokinase, Streptokinase & tPA
Inferior Vena Cava filter
Mrs. Perkins
Mrs. Perkins is receiving a heparin drip.The bag hanging is 20,000 units/500 ml of D5W infusing at 22 ml/hr. How many units
ofheparin is Mrs Perkins receiving each
hour?
Heparin Infusion 880 units20,000 divided by 500 = 40 units
If 22 ml are infused per hour, then 880 units
of heparin are infused each hour40 x 22 = 880
Heparin TherapyWhat nursing interventions should you implement forMrs Perkins receiving Heparin? Keep protamine sulfate readily available Assess for overt & covert signs of bleeding Avoid invasive procedures and injections Administer stool softeners as ordered
Pulmonary EmbolismMrs Perkins PT is 12.9 and PTT is 98. What are your implications for administering heparin to Mrs
Perkins?
A normal PTT is 39 seconds 58-78 is 1.5 to 2 times the normal value and is
within the normal therapeutic range A PTT of 98 means Mrs Perkins is not clotting;
medication should be held.
Pulmonary EmbolismThe doctor has ordered Coumadin for Mrs.Perkins. PT = 22 PTT = 39 INR = 2.8
What action should you implement Give the Coumadin because the
theurapeutic INR level is 2-3. What is the antidote for Coumadin?
Pulmonary Embolism: Teaching Use a soft bristle toothbrush to reduce the
risk of bleeding
Avoid aspirin Aspirin is an antiplatlet which may
increase bleeding tendencies.
Pulmonary Embolism: Teaching Wear a medic alert band
Increase fluid intake to 2-3L day (increases fluid volume which prevents DVT the common cause of PE)
IVC Filters
Greenfield Filter
Bird’s Nest Filter