Dental Anomalies II new.pdf

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    Anomalies of Structure Enamel

    Dentin

    Cementum

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    Enamel Tooth structure abnormalities result from disruption

    during the histodifferentiation, apposition, andmineralization stages of tooth development.

    Enamel defects manifested as:

    Hypoplasia

    Hypocalcification

    Can be:Heritable defects

    Environmental defects

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    Hypoplasia: disturbance of matrix deposition,characterized by irregular enamel in thickness ordeficient in structure.

    Hypocalcification: disturbance in mineralization,characterized by normal enamel in thickness but partof it is poorly mineralized

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    Amelogenesis Imperfecta Distinct patterns of inheritance.

    Defective enamel, so on radiograph the pulpal outline

    appears normal and root morphology. Hypoplastic vs hypomaturation vs hypocalcified

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    Hypoplastic AmelogenesisImperfecta

    Occurs in histodifferentiation

    stage of tooth developmentThin Enamel resulting in highsensitivity to thermal stimuli

    Lack of contact points between

    teethRough smooth or randomlypitted enamel

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    Hypomaturation AmelogenesisImperfecta

    Defect in enamel matrix

    apposition normal enamel thickness

    a low value of radiodensity andmineral content

    porous surface that becomesstained

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    HypocalcificationAmelogenesis Imperfecta

    Defect in the calcification

    stage of enamel formation Qualitatively the matrix is

    poorly calcified with aresultant fracturing of the

    enamel surface exposingdentin surface especially atincisal edge

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    Environmental Enamel HypoplasiaSystemic causes

    Nutritional deficiencies (Vit. A ,C, D, Ca+ PO4)

    Severe Infections, Fever ( Rubella , Syphilis ..)Asthma

    Neurologic defects as cerebral palsy

    Radiation

    Flouride

    Syndromes ??

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    Localized Enamel Hypoplasia Local infection,

    Local trauma,

    Iatrogenic surgery as occurs in cleft palate closure Primary tooth overretention.

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    Turner hypoplasia Turner hypoplasia is a

    classic example ofhypoplastic defects inpermanent teethresulting from localinfection or trauma tothe primary precursor

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    Pre-operativePost-operative

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    Full mouth rehabilitation of

    Amelogenesis Imperfecta

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    DentinDentinogenesis Imperfecta

    defect during the histodifferentiation stage of toothdevelopment

    defect of predentin matrix results in amorphic,disorganized, and atubular circumpulpal dentin(which is high in organic content and containsinterglobular calcification)

    The mantle dentin is normal.

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    Dentinogenesis Imperfecta Shields type 1 occurs with osteogenesis imperfecta.

    Defect in collagen formation

    Blue sclera, brittle bones, bowing of limbs Periapical radiolucencies

    Bulbous crowns

    Obliteration of pulp chambers

    Root fractures

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    Dentinogenesis Imperfecta Shields type 2, (hereditary opalescent dentin)

    Periapical radiolucencies

    Bulbous crowns Obliteration of pulp chambers

    Root fractures

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    Dentinogenesis imperfecta (Hereditary opalescent dentine)

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    Dentinogenesis Imperfecta Shields type 3

    a predominance of bell-shaped crowns

    involves teeth with a shell-like appearance andmultiple pulp exposures.

    It has occurred exclusively in a triracial isolated groupin Maryland known as the Brandywine population

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    Dentin Dysplasia Inherited dentin disorders resulting in characteristic

    features involving the circumpulpal dentin and rootmorphology.

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    Dentin DysplasiaShields type 1 (Radicular Dentin

    Dysplasia, rootless teeth)

    Normal crown morphology with

    an amber translucency). The roots tend to be short and

    sharply constricted.

    Multiple periapical

    radiolucencies and absent pulpchambers.

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    Dentin DysplasiaShields type 2

    Involves amber-colored primary teeth closelyresembling dentinogenesis imperfecta

    Permanent teeth appear normal, but radiographicallythey demonstrate thistle-tube-shaped pulp chamberswith multiple pulp stones

    no periapical radiolucencies are visible

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    Regional Odontodysplasia Localized arrest in tooth development thought to

    result from a regional vascular developmentalanomaly.

    Affected teeth have thin layers of poorly calcifiedenamel and dentin with large, diffusely calcified pulpchambers and shortened, poorly defined roots

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    Regional Odontodysplasia Ghost teeth radiographic appearance with shortened

    roots and shell-like crowns

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    Cementum Epidermolysis bullosa dystrophica, an inherited

    vesicular and bullous disease of the skin and mucousmembranes,

    involves formation of fibrous, poorly calcified acellularcementum and overproduction of cellular cementum.

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    Hypophosphatasia

    Failure of bone to mineralize properly, which isassociated with low serum alkaline phosphatase levels.

    Osteoporosis

    bone fragility

    premature loss of primary incisors

    Failure of cementum formation

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    REFERENCES:

    Pediatric Dentistry : Infancy through AdolescencePinkhamCh. 4 (Anomalies of Developing Dentition)