DNA DISRUPTION INVIRUSES AND ANIMALS

BY THE USE OF SPECIFICFREQUENCIES OF

ELECTROMAGNETIC RADIATIONWHICH MATCH THE

RESONANCE FREQUENCIES OFTHE DNA SECTION WHEN THEDNA SECTION IS CONSIDERED

AS AN ELECTRICALLYCONDUCTIVE ANTENNA

STRUCTUREBy Gary wade, Physicist, 12 / 29 / 02

OVERVIEW FOR THE LAYMAN - In this article the fact that the mostcommon form of DNA ( B-DNA) is electrically conductive is used to showthat viral, bacterial, and animal (human) DNA can be thought of and usedas tuned "radio" antennas. For example by choosing the proper frequencyof light to match a resonance frequency of the length of DNA in a virus, theoscillating electric field of the light can induce an electric current from thevirus DNA ends (field emission). This field emission can damage the virusDNA end segments and surrounding protein structures and thereby makethe virus non-infective. When considering human DNA gene sets as tunedantenna, specific frequencies in the microwave range can be used atrelatively low power levels for short time intervals to open up specific gene

sets. This allows for resetting of genetic age clocks (restoring telomereson chromosome ends), reversal of some genetic defects, and total tissuerepair (opening up some fetal gene sequences), even from traumaticdamage such as amputations and brain and spinal cord damage. (Note:you can now print out the figures in Landscape mode--Ed 1/30)

THE TECHNICAL DETAILS

DNA when it is in the B–DNA form, the DNA form normally found inabundance in plants and animals, is known to be an electrical conductoralong its core. It is not as good a conductor as a metal, more comparableto that of a semi-conductor or conductive polymer (ref. 1,2,3). Thiselectrical conductivity allows us to consider a B–DNA strand of a fixedlength as a "tuned" antenna. The length of the viral DNA is rather short, thegreat majority ranging from around 2,000 to 200,000 DNA base pairs inlength, depending on the particular virus under consideration. Figure 1Aillustrates a graphically linearized version of a very simple DNA virus,where the DNA length S has been chosen as the half wavelength (S = 1/2Y) resonant antenna for an applied oscillating electromagnetic field offrequency (F). Figure 1B shows a more realistic graphical illustration of thissimple virus where the DNA strand of length S is complexed with a proteincoat in the form of a coil. The coiled DNA has both inductance andcapacitance that will effect the resonant frequency and wave speed of/onthe DNA coil. However, for our purposes the coiled B-DNA to a firstapproximation acts electrically essentially like a straight antenna of lengthS as depicted in Figure 1A. For our purposes, since the (LC) resonancephenomenon does not reduce the field emission, but only shifts thefrequency at which it occurs at most efficiently, we will therefore ignore theinductance and capacitance of the virus coiled DNA. The applied resonantelectric field (specific frequency of light), if of high enough intensity, willdrive the DNA core electrons back and forth to a voltage / electric fieldamplitude at the DNA strand ends, such that electrons are emitted at theends of the DNA by a process similar to the well known phenomenon offield emission from the surface of a metal point.

Field emission from a metal point is a very non-linear process in terms ofthe electric field (goes as the square of the electric field) and the electricfield is approximately proportional to the inverse fourth power of the radiusof curvature of the point of emission. The radius of curvature of the DNAconductive core is only ~ 3 angstroms and a relatively modest appliedelectromagnetic field intensity should, in a resonance situation, inducelarge amplitude electric fields at the DNA strand ends. Since the DNA corehas a much lower density of conduction electrons than a metal core would

have, the field emission probability of the emission (quantum tunneling) ofan electron approaching the DNA strand end should significantly increasebecause there will be a greatly reduced image charge potential barrier.From these conditions we can surmise the strong possibility of substantialelectron field emission from DNA strand ends under the above statedconditions. Furthermore, if these emitted electrons have acquired enoughkinetic energy they can denature and/or destroy the chemical bonds ofDNA coating proteins as well as disrupting any chemical structure in thelocal area. Also, in this protein damaging process, damage to the endDNA base pairs can be expected. In other words, a DNA strand exposedto its resonant electromagnetic frequency at adequate intensity should inshort order become non-functional as a viable virus DNA to infect a host.This proposed phenomenon lends great support to some forms of light orcolor therapy, which have been persecuted by orthodox pharmaceuticaldriven allopathic medicine for eighty plus years now.

Equation 1 is the universal relationship between wavelength (Y),frequency (F), and wave propagation velocity (V) for all wavephenomenon.

V = Y F ; Equation 1.

For some wave phenomena V is frequency dependent. However, inour interested area it should be a near constant value. Considering theDNA strand of Figure 1A as a half wave antenna (S = Y/2) and puttingY in terms of S into equation 1 we obtain:

F = ( V ) / 2S ; Equation 2.

The DNA strand of Figure 1A could just as well have been consideredas a full wave antenna (Y = S), a three half wave antenna ( Y = 2S / 3), a two wave antenna ( Y = S / 2 ), etc.. Equation 2 is a special caseof the general equation describing the possible resonance frequenciesthat the DNA antenna can support, namely Equation 3.

F = NV / 2S ; where N = 1,2,3, … Equation 3.

Figures 2 A, B, C, and D illustrates N = 1, 2, 3, and 4 of equation 3, wherethe curves represent the boundary envelope for the maximum value that theoscillating voltage reaches at each point along the DNA strand. Forexample, consider the point C on the DNA strand of Figure 2A. In Figure 3,the voltage point C is plotted verses time for the situation depicted inFigure 2A. The plot of the voltage verses time for any point along the DNAstrand looks like Figure 3, except the maximum amplitude varies frompoint to point. The voltage at a point on the DNA in one envelope oscillates180 degrees out of phase with the voltage at a point in the adjacentenvelope. If the DNA strand of Figures 2A, B, C, and D are exposed to"light" frequencies, which are somewhat off the resonance frequenciesgiven by Equation 3, standing waves will still be formed on the DNA strandsimilar to those illustrated in Figures 2A, B, C, and D. However, themaximum voltage amplitude on the DNA ends will decrease significantlywith increasing frequency shift off the resonance frequencies.

The virus illustrated in Figures 1A and Figure B are very simple and do nothave the commonly observed lipid covered protein capsid coat of manycommon viruses that infect animals and plants. Just such a common virusis illustrated in Figure 4. The virus of Figure 4 looks a great deal differentthan the virus of Figure 1. However, to a first approximation it is only thelength of the DNA segment that is dominate in determining the resonancefrequency. The shape of the DNA strand is not too critical in most cases.An example of a good exception to this would be a lambda bacteriophage.In the lambda bacteriophage the DNA is very tightly packed under highcompression into a many turned and layered coil which has significantinductance and capacitance. Also, the electrical conductivity could besignificantly increased do to the high compression of and highly restrictedmovement of the lambda DNA in its capsid.

Let us use Equation 3 and some possibly reasonable values for V, S, andN to obtain approximate electromagnetic frequency ranges that coulddisrupt or destroy some virus. Let our virus of interest have a DNA strandof 20,000 DNA base pairs. The average length per base pair in B-DNA is3.4 angstroms (an angstrom equals ten to the minus ten meters). Thisimplies that the DNA strand length (S) is 6.8 microns. Let us guesstimatethat V is .9 the speed of light.

Let N = 1, 2, 3, and 4. Then:

F = N ( 1.98 x 10 exp.13 sec.-1) , Equation 4.

F1 = 1.98 x 10 exp.13 sec.-1 ----------------------- Y1 = 15.113 microns

F2 = 3.97 x 10 exp.13 sec.-1 ----------------------- Y2 = 7.5565 microns

F3 = 5.95 x 10 exp.13 sec.-1 ------------------------ Y3 = 5.0377microns

F4 = 7.94 x 10 exp.13 sec.-1 ----------------------- Y4 = 3.7783 microns

All of these resonance frequencies / wavelengths are in the infraredband range. If we had chosen a virus of 2,000 DNA base pairs, thenthe wavelengths would have been ten times smaller.

Y1 = 1.51 microns ( top of infrared band )

Y2 = .7556 microns ( Very top of infrared band )

Y3 = .5037 microns ( green light )

Y4 = .3778 microns ( bottom of ultraviolet band )

These results are potentially rather significant because there are many.many viruses that have DNA bases pair counts in the 2,000 to 20,000 andbeyond range which cause serious health problems / diseases in peoplefor which we currently have no viable treatments. We now have availabletunable lasers, which can cover the top of the infrared band through thevisible band into the ultraviolet band. These tunable lasers can be used totreat the blood directly or can be used in intense pulse scanning mode totreat the surface tissue and at depth in some cases. It is necessary to usea relatively narrow band of frequencies (specific color) to efficiently andeffectively destroy the virus. If broad spectra light (i.e. white light) is used,the conduction electrons in the B-DNA core see the composite random

oscillating electric field from all the different colors (frequencies) and willnot form a strong resonant or near resonant electrical oscillation that isrequired to destroy the virus by the method discussed above. There arealso very strong light sources commercially available, which producefrequency bands from the infrared through the ultraviolet. With appropriatefiltering of these light sources, a viable whole body treatment modality canbe envisioned. It is also conceivable to fire or cook into certain ceramics,certain molecules and mono atomic elements to obtain narrow bandelectromagnetic frequency emissions from the ceramics upon raising themto the appropriate temperature and then filtering out what is not wanted. Itis even conceivable to "filter" infrared light from a very intense blackbodysource to be used in treatment.

What we have been considering here for treatment on humans can ofcourse be used on animals and plants in general. We can even imagineprotecting one cell plants and animals as well as bacteria from viral attackby the use of specific frequencies of electromagnetic fields. Also, we maywish to wonder about finding the resonant frequencies for the chromosomeDNA of certain bacteria. Can we disrupt or denature the chromosomal andor plasmid DNA of bacteria using resonant standing wave electromagneticradiation? For example, imagine a bacterial plasmid that codes for anenzyme that destroys some anti-biotic. If this plasmid has a shortelectrically non-conductive segment such as Z-DNA, then the above usedantenna formula can be used to destroy the plasmid. Imagine a small room(chamber) where a patient would stand in their nature suit bathed in alldirections by a light as brilliant as the sun, but of only a very narrowfrequency range (a specific color). Thirty seconds in the room and you gohome just fine.

Now that you are familiar with the concept of various virus DNA lengthsbeing treated as a tuned antenna, let us extend the concept to thechromosome size scale. Research has shown that the relaxedchromosome in the cell nucleus can be considered as a series of genesand gene sets sequences each gene set to be read all together or not atall. These gene sets are often separated/partitioned from each other bycombinations of promoter and blocker proteins and or Z-DNA segments.Z-DNA is generally formed and maintained by the interaction betweenspecific short DNA base pair sequences and certain ion complexes. Z-DNA is not electrically conductive and acts like an insulator separating twoB-DNA conductors. Z-DNA effectively partitions chromosome B-DNA intoa set of tuned antennas. Furthermore, the proteins that complex to thesurface of the chromosome undoubtedly affect the electrical conductivity ofthat local B-DNA base pair sequence or region. In some cases the

conductivity may increase and in others it may decrease. In some casesthis B-DNA region may gain significant resistance even to the point ofbecoming effectively non-conductive. Also, at any one moment there areoften many B-DNA transcription enzymes at work on a single chromosometranscribing gene sequences.

The transcription enzyme splits the DNA at its location into two separatesingle DNA base sequence strands during transcription and thereforestops electrical conductivity at this location. Figures 5 shows a smallsection of chromosome which illustrates the situation just described. So, itshould be clear that the chromosomes could be considered as a set ofboth isolated and coupled tuned antennas as illustrated in Figure 6, whereonly the electrical properties of the chromosome are dwelt upon. The tunedantennas of the chromosome illustrated in Figure 6 are generally muchlonger in length than those of viruses and therefore have fundamentalresonance frequencies considerably lower. Namely in the low gigahertzrange. For example, when the cell phone companies say / claim that thereis no scientific proof that cell phone use is harmful, they lie. It has beenknown since the 1960’s that R.F. in the 1 gigahertz range can causechromosome damage and breakage. It was originally proposed to use thisfact to study the phenomena of chromosome damage and breakage. Thenext time you use a cell phone ask yourself: Do I feel lucky ? Well, do youfeel lucky? Well, do you?

There are potentially great benefits available from exposing animal cells tospecific sets of microwave frequencies for brief time intervals at theappropriate intensities. Various gene sets can be opened up withphenomenal results. For example, resetting the telomere " time " clocks incells giving people vastly extended youthfulness, reversing geneticdiseases, and completely repairing massive body tissue damage such asspinal cord injuries, brain injuries, organ damage, and amputations. Asbold as the above statements may seem, their truth or validity is easy tosee when you consider a few facts and observations. First, consider fetaldevelopment, the fetus develops out of a fertilized egg in a totallygenetically orchestrated/programmed fashion just like clockwork. Largenumbers of gene sequences are expressed and then shut down usually notto be expressed again during the individual’s normal lifetime. Note thatfetuses have been operated on in the womb, and when born have noscaring evident, i.e. complete tissue repair. This ability is shut downsometime before birth. Just as your development to birth is geneticallyprogrammed, your death is also. As you age with the need for continuouscell division for tissue repair and maintenance, you loose telomeres on theends of each of the chromosomes of the dividing cells. As this process

proceeds the length of the telomeres get shorter and shorter and the celldivision rate continually decreases to the point that health can not bemaintained and you die. Experimentally from tissue regenerationexperiments done with salamanders and rats( Dr. Robert Becker’s work) itis known that amputated limbs of mammals can be re-grown (ref. 4). Thatis total tissue regeneration. Cells were made to become embryonic-like(de-differentiation) and then to multiply and then differentiate into all theneeded body cell types to reform the missing amputated limb. In thisprocess gene sequences that had been shut down to be never accessedagain were opened up and expressed and apparently the chromosomesdid not initially suffer telomere shorting.

Analyses of Becker’s work shows that it is the drastic change in theconcentration of positively charged metal ion complexes in the cellcytoplasm surrounding the chromosomal DNA that causes thededifferentiation of cell structure (ref. 5). In the re-growing of rat arms, itwas the exposure of the cells at the amputation site to feeble negativeelectric currents that formed high hydroxyl ion concentrations around theamputation site, which attracted the positively charged metal ions into theregion. This high hydroxyl ion concentration brought in high positive metalion concentrations to mask the excess negative hydroxyl ion charge. Also,hydrogen ions were neutralized and chlorine ions were "forced" out of theregion (significantly depleted). The cells in this region being bathed in ahigh PH and high positive metal ion concentration, are moved into anotherinternal (cytoplasm) dynamic equilibrium positive metal ion concentrationstate in which the various positive metal ion concentrations are greatlyincreased and their concentration ratios significantly changed. Thesemetal ion complexes interact with both DNA binding proteins and withspecific ion complexes on specific DNA base pair sequences to eitherform and or undo Z-DNA short segments at the beginning of some genesets. For the DNA reader enzymes to express the information in a genesequence set it has to mount onto a specific promoter protein which iswrapped onto a specific sequence of B-DNA at the beginning of a genesequence set. The promoter protein in turn needs the blocker protein whichcommonly share their binding DNA sequence site removed from the site,so as to allow the promoter protein to move to the proper exact DNA basepair sequence for the DNA reader enzyme mounting of the promoterprotein and DNA transcription to begin. The blocker proteins are usuallyremoved by other proteins sent from other gene sequences being read inthe cell nucleus. If the promoter protein is being blocked by its neededspecific base pair sequence being in the Z-DNA configuration, then this Z-DNA must be converted to the B-DNA configuration for the gene settranscription to begin.

transcription to begin.

So, we have a situation in which it is empirically known that positive metalion concentrations in the cell nucleus can drastically alter the access andexpression of gene sequence sets. Now how do these positive metal ioncomplexes, which interact / complex with: 1) DNA directly, 2) with negativeion complexes complexed with DNA, and 3) DNA’s complexed proteins,interact to open up specific gene sequence sets? Consider a Z-DNAsequence blocking the binding site of a promoter protein and thereforeblocking DNA reader enzyme transcription activity. By significantlychanging the ionic environment at the Z-DNA site, the Z-DNA can beconverted to the B-DNA form and the promoter protein can move into thesite and bind with it and then the reader enzyme can start transcription.One simple way to modify the ionic environment is to apply a significantoscillating electric field at the Z-DNA location. This is easily done byexposing the electrically conductive B-DNA segment, in which the Z-DNAis at one or both of the antenna end points of, to one of the antennas’resonant frequencies. The oscillating voltage induced in the antenna will beat a maximum at the antenna ends, the Z-DNA location. Once theoscillating voltage/electric field strength at the antenna end goes oversome minimum value it will be the dominate field determining ionconcentrations in the region and whether or not ion complexes will complexwith the Z-DNA. Specifically, an oscillating electric field suppressescomplexing of ion complexes with DNA and therefore suppresses Z-DNAformation and opens up Z-DNA blocked gene sequence sets fortranscription. Care must be taken not to build to strong of a resonantvoltage/electric field on the antenna end region to avoid DNA damage.Similar oscillating voltage/electric field interactions between ioncomplexes, DNA sequences, and blocker proteins can be invoked toremove some blocker proteins and therefore start gene sequencetranscription.

CONCLUSION: By the judicious use of various frequencies ofelectromagnetic energy we can destroy unwanted microbes, overridegenetic defects, control genetic expression in such a way as to effectivelyhalt and reverse aging, and repair and regenerate the body totally, evenform amputations and serve brain and spinal injuries. We are potentially atthe start of a brave new world for medicine, biophysics, and biology(energy medicine, very few drugs required). Let us not let this technologybe developed only by the military and the medical elite. Always remember:1) The AMA is a monopolistic trade association interested incontrolling/owning your illness care rights and your illness care money, 2)The pharmaceutical companies are not our friends and DO NOT HAVEOUR BEST INTERESTS AT HEART. THEIR GOD IS MONEY. AS LONG

AS THEY CAN GET YOU TO TAKE THE SYMPTOM SUPPRESSINGDRUG AND NOT TAKE CARE OF THE FUNDAMENTAL CAUSE OFYOUR PROBLEM, YOU WILL NEED TO KEEP GOING TO THEIRLICENSED DRUG DEALER (YOUR ALOPATHIC DOCTOR) ANDSPEND YOUR MONEY. YOU ARE THEIR CASH COW. CAN YOU SAYMOO?

References:

1) Variable Range Hopping and Electrical Conductivity along the DNADouble Helix by Z. G. Yu and Xueyu Song, Physical Review Letters, 25, June2001 Volume 86, Number 26.

2) Charge Transport along the Lambda DNA Double Helix, P. Tran,B. Alavi, and G. Grunen, Physical Review Letters, Volume 85,Number 7, Page 1564-1567.

3) DNA and Conducing Electrons, H. W. Fink, Visions andReflections, Cell, Mol., Life Sci (CMLS), Volume 58, 2001, Page 1-3.

4) The Body Electric (Electromagnetism and the Foundation of Life),by Robert O. Becker, M.D., and Gary Selden, ISBN 0-688-06971-1.

5) A Physicist’s View of the Use of Feeble Electric Direct Currents ToRepair Tissue and Replace Body Parts (Part One), by Gary Wade,Health Freedom News (The magazine of The National HealthFederation, Monrovia, CA), February 1996, Page 22 – 33.