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IncidanceLess than 1 percent in hospital delivaries Commonest is rheumatic type f/b congenital hrt
disRatio is 3;1 in developing countries to o 1;1 in
developed countriesRh lesios are –ms(most common ),mr, as,ar Congenital types are –septal defects ,PDA,PULMO
STENOSIS,FELLOTS TETROLOGY
OTHER COARCTATION OF AORTA ,HYPERTENSIVE,SYPHILITIC AND CORONARY CARDIAC DS
Cardiac output; 4.5 to 6.251 30% increaseStroke volume 65 to 75 ml increasesPulse70 to 85Bp mild diastolic reduction in bpVenous pressure 10 to 20 cm of h2o 100 %
riseperipheral resistance markedly diminishedBlood viscosity 4.7 t0 3.8 -decres
Normal hrt –enough resrved power that can cope up with extra load
Damaged hrt has less reserved power to withstand against extra load
So ,chance of failure ,which usually occure at 30 wks ,during labour or at soon after labour
additional factors responsible for deterioration are advancing age ,arrythmia,left vent hypertrophy,h/o hrt failure,and apperance of risk factors like infection,anaemia,pre-eclampsia,excessive wt gain,multiple pregnancyand inadeqate supervision
Maternal depend on -nature of lesion , -functional capacity of hrt -quality of medical supervision during pregnancy- apperance of risk factor a mentioned aboveMortality on maternal side is least in rheumatic type
about less than one percent but increses upto 50% in cynotic hrt dis and max death occures after birth of baby
Other causes of death than cardiac failure are pulmo oedema,embolism ,active rhumatic carditis ,SABE,and rapture of cerebral aneurism in coarctation of aorta
FETAL PROGNOSIS;good but incresed chances of prematurity ,iugr ,and chance of abortion ,and chance of congenital malformation increses if either of parents suffering from congenital hrt dis
Symptoms Progressive nocturnal dyspnoea and
orthopnoea,nocturnal cough ,hemoptysis,syncope and chest pain
Criteria to find out organic hrt lesion Diastolic murmur,systojic murmur with
thrill,cardiac enlargement,presence of arrythmia
Othrs are cluubing ,cynosis, NVE and split s2Studies done for diagnosisECG,X-RAY AND ECHO
Grd-1 no limitation of physical activityGrd 2 mild compromised ordinary
physical activity causes discomfortGrd 3 mark compromised less than ordinary
physical activity cause discomfortGrd 4 severerly compromised discomfort
at rest
Grp 1 minimal risk –0 to 1 % mortality ASD ,VSD,PDA ,PS and TS Corrected Fallot’s Tetralogy Prosthetic Valves MS—related with NYHA class 1 and 2.
Group 2: moderate risk.5-15% mortality. MS, related with NYHA 3 and 4. AS Coarcrtation of Aorta without Valvae involvment. Uncorrected Fallot’s tetralogy Previous MI. Marfan’s with normal Aorta
Group 3: major risk—25-50% mortality. PHT, Aortic coarctation with valvular involvement Marfans’ with aortic involvment.
In cases of congenital heart ds. With PHT.Artificial valvesAF.Warfarin in nonpregnant------ as soon as
prgnacy is diagnoised, 5000 IU heparin SC BD up to 12 wks---shift on Warfarin up to 36 wks ---after that shift on heparin till 7 days postpartum .
DECISION OF ADMISSION:Grade 1 NYHA –2 wks prior to EDD.Grade 2 –at 28 wks in specially unfavourable
surrondings.Grade 3 and 4—as soon as prgnancy is
diagnosed.
Emergency Admission:Deterioration of functional grading .Appearance of pregnancy complications like
pre-eclampsia, anemia and abnormal wt. gain.
INTRAPARTUM MANAGEMENT:No place for induction.Induction only for obst. Indications.
FIRST STAGE:FIRST STAGE:Bed rest.Lt lateral position—to reduce aortocaval pressure by
gravid uterus.In majority –epidural labour analgesia.O2 inhalation.Restricted IV fluid ---less than 75ml/hr to prevent
pulmonary edema.Prophylactic antibiotics to prevent purperial
endocarditis.Monitor PR, RR, ---if PR> 110/min ---rapid
digitalization by 0.5 mg digoxin IM /Slowly IV.
SECOND STAGE:SECOND STAGE:If tendency to delay –cut short second stage –by
vaccum /forceps.NO ERGOMETRINConcentrated oxytocin infusion.THIRD STAGE:THIRD STAGE:CONVENTIONAL MANAGEMENT:Slight to moderate blood loss is benificial IV frusemide may be accompanied.INDICATIONS FOR CS:Only for obst. Indications.Elective CS:--Elective CS:--coarctation of aorta , mycotic
cerebral aneurysmEpidural analgesia.
Pain relief:Continuous Epidural analgesia is recomended
in most cases.Contraindicated –intracardiac shunts, PHT,
and aortic stenosis because it may cause reversal of shunt by causing hypotension and reduction of output in AS which is dependant on preload—in such condition Narcotic analgesia or GA preferred.,
Commonest RHD.Commonest RHD.Normal area of valve—3 to 6 cm2, symptoms Normal area of valve—3 to 6 cm2, symptoms
appears below 2.5 cm2 area.appears below 2.5 cm2 area.With NYHA grade 1 and 2 –mortality less than With NYHA grade 1 and 2 –mortality less than
1%.1%.Grade 3 and 4—5 to 15%Grade 3 and 4—5 to 15%Epidural analgesia preferred and restrict IV fluid.Epidural analgesia preferred and restrict IV fluid.Valvotomy: in case of unresponsive cardiac Valvotomy: in case of unresponsive cardiac
failure with pregnancy beyound 12 wks.failure with pregnancy beyound 12 wks.Best time for surgery—14 to 18 wks.Best time for surgery—14 to 18 wks.Anticoagulant therpy and antibiotics to prevent Anticoagulant therpy and antibiotics to prevent
SABE and thromboembolism.SABE and thromboembolism.
Commonly congenital , less commonly rheumatic.
Maternal mortality---significant 15-20% and perinatal loss 30%.
Long term bedrest .Epidural analgesia—contraindicate d and no
fluid restricted and to be given more than 125 ml/hr.
ASD: most common , tolerate pregnancy and labour well CCf unresponsive to medical treatment—required surgery.
Shunt reversal is major risk which develops in hypovolemia and hemorrhagic conditions and hypotension.
Peripartum prophylactic antibiotics
PDA: Tolerate pregnancy well. PHT—cause of maternal death. Surgical correction can be performed provided there is no PHT. Epidural to be avoided. Fetal loss up to 7%. And 4% chance of same lesion in child.
VSD: Defect < 1.25 cm2—generally. Moderate Lt to RT shunt and moderate PHT –women tolerates well Risk of reversal leads to circulatory collapse and cynosis. Epidural analgesia and hypotension to be avoided. Fetal loss up to 20% and 8% chance to get same malformation in fetus.
Fallot’s tetralogy;4 lesions are –VSD, PS, RVH and overriding of
aorta.Uncorrected pts are at high risk but usually
corrected stages pts are found.Complications like –SABE, brain abscess and
cerebral embolism are common.Maternal mortality –5 to 10% Perinatal –30 to 40%.IUGR common.Systemic hypotension is dangerous –even leads
to death.
EISENMENGERATION’S SYNDROME:It is presence of ASD VSD and PDA with PHT
with Rt to LT flow from shunts.Maternal mortality –50% and perinatal loss is
very high .Usually termination –should be considered
and Suction and Evacuation is preferred method.
OTHER CONGENITAL HEART DS: Coarctatiton aorta: Major risk of dissection, BE, and rupture of Intracranial aneurysm. Maternal mortality—3 to 8% and fetal loss up to 25% and elective CS
preferred to minimize dissection associated with labour. BP is to be maintained throughout pregnancy and prophylactic antibiotics. Primary PHT: Characterized by thickening of muscular layer of arteriols Maternal mortality—40% and fetal outcome bad. Vasodilators like hydralazines is to be given even in absence systemic HT. Marfan’s Syndrome: Autosomal dominant. 50% chance of transmission to offspring Dilatation of aorta >40 mm ---contraindicated for pregnancy. Beta blocker ---to reduce hemodynamic load on ascending aorta even in
absence of systemic HT. If there is any dilatation of aorta during prgnancy---elective CS.
CARDIOMYOPATHY:Manifestations appear usually in last month of
pregnancy to 5 mths after delivery.Etiology –not known.Usually multiprous, young 25 to 35 yrs of age and in
second and third postpartum month.Sx;; dyspnoea, nocturnal cough, weakness and
palpitaions.Examinations;; Tachycardia, arrythmia, peripheral
edema, and creopts .X-ray---enlaged heart,ECHo-=--dilated chambers particularly lt. ventricalTreatment: bed rest ,
digitalis ,diuretics ,anticoagulants Vaginal delivery favorable Unfavourable cases ---cesaren section.