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8/10/2019 Operative Review of Retinal Detachment
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8/10/2019 Operative Review of Retinal Detachment
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CHAPTER I
TABLE OF CONTENTS
PAGE
I. INTRODUCTION 1
II. ANATOMY 4
III. PATHOPHYSIOLOGY 7
IV. MEDICAL MANAGEMENT 8
V. DIAGNOSIS 12
VI. PROCEDURE PROPER (with Instrumentation) 15
VII. Roles of Circulating and Scrub nurse 24
VIII. Nursing Management 30
a. Nursing Care Plan
i. Pre-Operative Review
ii. Intra-Operative Review
iii. Post-Operative Review
IX. Pharmacology
i. Pre-operative
ii. Intra-operative
iii. Post-operative
X. Bibliography 37
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Types of Retinal Detachment
Rhegmatogenous retinal detachmentA rhegmatogenous retinal
detachment occurs due to a hole, tear, or break in the retina that allows fluid to pass
from the vitreous space into the subretinal space between the sensory retina and the
retinal pigment epithelium.
Exudative, serous, or secondary retinal detachment An exudative
retinal detachment occurs due to inflammation, injury or vascular abnormalities that
results in fluid accumulating underneath the retina without the presence of a hole, tear,
or break.
Tractional retinal detachmentA tractional retinal detachment occurs
when fibrovascular tissue, caused by an injury, inflammation or neovascularization, pulls
the sensory retina from the retinal pigment epithelium.
A substantial number of retinal detachments result from trauma, including blunt
blows to the orbit, penetrating trauma, and concussions to the head. A retrospective
Indian study of more than 500 cases of rhegmatogenous detachments found that 11%
were due to trauma, and that gradual onset was the norm, with over 50% presenting
more than one month after the inciting injury.
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Prevalence Rate
A physician using a "three-mirror glass" to diagnose retinal detachment
The risk of retinal detachment in otherwise normal eyes is around 5 in 100,000 peryear. Detachment is more frequent in the middle-aged or elderly population with rates of
around 20 in 100,000 per year. The lifetime risk in normal eyes is about 1 in 300.
Retinal detachment is more common in those with severe myopia (above 56
diopters), as their eyes are longer and the retina is stretched thin. The lifetime
risk increases to 1 in 20. Myopia is associated with 67% of retinal detachment
cases. Patients suffering from a detachment related to myopia tend to be
younger than non-myopic detachment patients.
Retinal detachment can occur more frequently after surgery for cataracts. The
estimated of risk of retinal detachment after cataract surgery is 5 to 16 per 1000
cataract operations.The risk may be much higher in those who are highly myopic,
with a frequency of 7% reported in one study.Young age at cataract removal
further increased risk in this study. Long term risk of retinal detachment after
extracapsular and phacoemulsification cataract surgery at 2, 5, and 10 years was
estimated in one study to be 0.36%, 0.77%, and 1.29%, respectively.
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http://en.wikipedia.org/wiki/Myopiahttp://en.wikipedia.org/wiki/Dioptershttp://en.wikipedia.org/wiki/Dioptershttp://en.wikipedia.org/wiki/Myopia8/10/2019 Operative Review of Retinal Detachment
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Causes of Retinal Detachment
Retinal detachment can occur as a result of:
Trauma Advanced diabetes
An inflammatory disorder, such as sarcoidosis or cytomegalovirus retinitis
Sagging or shrinkage of the jelly-like vitreous that fills the inside of your eye
It is more likely to develop in people who are nearsighted, or whose relatives had
retinal detachments. A hard, solid blow to the eye may also cause the retina to detach.
Severe trauma to the eye, such as a contusion or a penetrating wound, may be the
cause, but in the great majority of cases, retinal detachment is the result of internal
changes in the vitreous chamber associated with aging, or less frequently, with
inflammation of the interior of the eye.
The Risk Factors of Retinal Detachment
The following factors increase your risk of retinal detachment:
Aging retinal detachment is more common in people older than age 40 Previous retinal detachment in one eye
A family history of retinal detachment
Extreme nearsightedness (myopia)
Previous eye surgery, such as cataract removal
Previous severe eye injury or trauma
Weak areas on the sides (periphery) of your retina
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CHAPTER II
ANATOMY
The inner wall of the back of the eyeball is covered by the retina. It contains light
sensitive cells (photoreceptors) and numerous cells and nerve fibers responsible for
transmitting visual information. Below is the pigment epithelium, rich in vessels
(choroid), ensuring the supply of nutrients and oxygen to the retina.
In the event of detachment, the light-sensitive retina becomes detached from theouter membrane (RPE). The vision is thereby impaired: the subjects speak for a
blackout before the eyes. When this process affects the macula (yellow spot) the
point where vision is sharpestbecomes totally blurred vision.
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CHAPTER III
PHYSIOLOGY
The development of rhegmatogenous RD is a consequence of both posterior
vitreous detachment and the development of one or more breaks in the retina. Fluid can
then pass from the vitreous cavity through these retinal breaks into a subretinal space,
which extends the detachment once the amount of incoming fluid exceeds the removal
capacity of the retinal pigment epithelium (RPE). Detachment of the posterior vitreous is
considered a major - in fact indispensable - factor in the pathogenesis of
rhegmatogenous RD. However, no preoperative diagnostic technique can accurately
distinguish between a posterior vitreous detachment and a posterior vitreoschisis.
Progression of the detachment depends on many factors, including:
Location of the break: superior faster than inferior
Size of the break: larger faster than smaller
Adhesion of the remaining vitreous gel to the retina: stronger faster
than weaker
Movement of the patient's head and eyes: this is also important
because lack of such movement, as with bilateral patching, can result
in the reattachment of the retina spontaneously, albeit temporarily.
In eyes with tractional RD, the membranes on either surface of the retina are 1)
attached to the retina, and 2) elastic. As the membranes contract, the retina detaches
from the RPE. Accumulation of the subretinal fluid is a secondary event; as part of the
normal fluid transport from the vitreous to the choroid, the fluid simply fills the space
created by the elevated retina.
In serous and haemorrhagic RD, the fluid that accumulates under the neuroretina
separates it from the RPE.
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CHAPTER IV
MEDICAL MANAGEMENT
Medical Management
Currently, no role exists for medical care in the treatment of TRD.
Surgical Care
Depending on the underlying cause and extent of the TRD, surgical intervention
is offered to patients. For instance, a patient with TRD secondary to PDR that does not
threaten the macula probably can be monitored closely. The main surgical goal in all
these cases is to relieve vitreoretinal traction. Traction may be relieved with scleral
buckling techniques and/or with vitrectomy.
In certain cases, combined RRD and TRD may be present. Usually, the retina
becomes detached from the vitreoretinal traction. With further traction, small breaks
may occur causing a combined TRD-RRD. In these cases, the surgical goal is to
identify all the breaks and to close them in addition to the relief of vitreoretinal traction.
In TRD secondary to PVR, usually a broad circumferential element, such as a
287 buckle, is placed. A decision is made whether the crystalline lens needs to
be sacrificed. A complete vitrectomy follows. Inside-out (posterior to anterior)
forceps (not pick) membrane peeling is the preferred dissection method with or
without perfluorocarbon liquid injection. Perfluorocarbon liquid may be injected at
the surgeon's discretion to stabilize the posterior retina. If residual traction
remains, subretinal membranes may need to be excised if causing traction. If
necessary, a relaxing retinectomy is created. A fluid-air exchange is performed.
Endophotocoagulation is followed by either air-silicone oil exchange or air-gas
exchange. If perfluorocarbon liquids are not used, the dissection starts anteriorly
and proceeds posteriorly.
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A randomized controlled clinical trial of a perioperative infusion of 5-fluorouracil
and low molecular weight heparin was not able to demonstrate a better surgical
outcome in eyes with established PVR.
In TRD secondary to PDR, several surgical techniques have been developed. A
scleral buckle usually is not used unless anterior breaks are present.
A central vitrectomy is performed with the vitrector clearing the axial opacities
and the cortical vitreous gel. A large opening is created in the posterior hyaloid
until vitreoretinal adhesions are encountered. Segmentation and/or delamination
of these adhesions (as described by Charles) are used for virtually all diabetic
TRD.
Delamination refers to the separation of the retina from the extraretinal
proliferation. This dissection proceeds from posterior to anterior. Fibrovascular
tissue often bridge separate retinal zones. Segmentation refers to cutting of the
fibrovascular tissue bridge into small separate islands of tissue.
Care must be given to create as few iatrogenic breaks as possible. If breaks are
identified, usually fluid-air exchange with photocoagulation reattaches the retina.
Breaks should be marked with diathermy, so they are identified easily in the air-
filled eye. The incidence of RRD in patients who underwent vitrectomy for PDR
has been reported to be 4.3%. Intraocular bleeding also must be monitoredclosely. Diathermy to active neovascular fronds may be necessary.
Other techniques include the en bloc dissection. En bloc is a name applied to
outside-in delamination where the vitreous is used to pull on the epiretinal
membrane. Outside-in causes more retinal breaks than inside-out, making it a
dangerous maneuver.
Intravitreal bevacizumab has been reported as a preoperative adjunct in
vitrectomy for PDR. Bevacizumab seems to reduce the bleeding associated with
the segmentation and delamination of fibrovascular membranes. However, in
eyes with severe ischemia, the neovascularization regresses rapidly, but the
resulting fibrous scar tissue may lead to the development or progression of TRD.
Therefore, caution should be exercised when injecting these eyes, and patients
should be scheduled for surgery days, and not weeks, after the injection.
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Anti-VEGF agents such as bevacizumab have been used as adjuncts to
vitrectomy. The advantages of using preoperative bevacizumab includes faster
surgery and reduced risk of intraoperative bleeding, which facilitates membrane
dissection.[8, 9, 10, 11] Care must be taken because it has been reported that, in
very ischemic eyes, TRD may occur or progress shortly following intravitreal
bevacizumab.[9, 10] It is speculated that rapid neovascular involution with
accelerated fibrosis and posterior hyaloidal contraction as a response to
decreased levels of VEGF is responsible for this phenomenon. In this
retrospective series, the time from injection to TRD was a mean of 13 days, with
a range of 3-31 days.[9] Therefore, the time between bevacizumab injection and
vitrectomy should not exceed 3 days.
The treatment of TRD secondary to ROP depends on the stage of the disease.
Although many vitreoretinal surgeons advocate an encircling band for stage 4A
ROP, no scientific evidence is available that supports its efficacy. In stage 4B,
vitrectomy is recommended. It is currently unclear if lens-sparing vitrectomy has
any advantages over lensectomy.
For stage 5 ROP, visual and anatomical results have been disappointing, making
some surgeons abandon surgery for these cases. Others have tried vitrectomy
and lensectomy with or without scleral buckling. In these cases, a 2-portvitrectomy technique is recommended since the small size of the eye and orbit
limits ocular manipulation if a 3-port technique is used. The use of intravitreal
triamcinolone as a postoperative adjuvant might improve the rate of retinal
reattachment after vitrectomy.
A recent case series of aggressive posterior ROP suggested that early
vitrectomy with lensectomy in these cases is effective in preventing TRD.
Special attention must be given to avoid iatrogenic retinal breaks because of the
poor prognosis associated with this complication. The goal of surgery is to obtain
macular reattachment.
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CHAPTER V
DIAGNOSIS
History
Patients who present with symptoms of new onset photopsias, floaters or visual
field loss should be suspected of having a retinal tear or detachment until proven
otherwise. Important information in the history includes onset of symptoms, duration of
decreased visual acuity, metamorphopsia, any prior trauma, prior surgery, intraocular
inflammation, hemorrhage, glaucoma and a complete past medical history and review of
systems.
Physical examination
Visual acuity, pupillary examination, visual field testing and intraocular pressure
measurement are important parts of the predilated ophthalmic examination to evaluate
patients with symptoms of retinal detachment. Additional examination to include color
vision and ocular motility should be tailored according to the history provided.
Slit lamp examination of the anterior segment should be completed prior to
dilation. Examination of the vitreous for pigment cells followed by a thorough fundus
examination to include indirect ophthalmoscopy with scleral depression should be
completed. A detailed drawing describing the detachment with location of retinal
pathology should be documented.
If there is no view to the posterior pole such as in hemorrhage or media opacity,ultrasound should be used to evaluate the retinal status.
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Clinical diagnosis
Rhegmatogenous retinal detachment has a characteristic appearance
differentiating it from a tractional or serous detachment. A rhegmatogenous retinal
detachment has a corrugated appearance and undulates with eye movements.
Tractional detachments have smooth concave surfaces with minimal shifting with eye
movements. Serous detachments show a smooth retinal surface and shifting fluid
depending on patient positioning.
Laboratory/Ancillary testing
Laboratory testing is only indicated in traction or exudative detachments. If a
cause for the traction retinal detachment cannot be determined by history, furtherlaboratory analysis may be required to determine if diabetes, sickle cell, carotid disease
or another systemic or ocular process is the source for proliferative retinopathy.
Since exudative detachments may be due to a systemic or ocular inflammatory
process, laboratory investigation may be indicated.
Fluorescein angiography may be indicated to further clarify exudative processes
such as macular degeneration, central serous chorioretinopathy, and Vogt-Koyanagi-
Harada syndrome or other uveitic processes. Ultrasound is a useful imaging modality to
evaluate choroidal masses or posterior scleritis.
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CHAPTER VI
PROPER PROCEDURE
Pars Plana Vitrectomy
Patients are brought to the operating room in an eye bed that has an appropriate
head rest and the capability to have a wrist rest secured to it. Once the bed is
positioned next to the operating microscope and locked, the bed is made completely
flat, and the patient is positioned so that the head lies comfortably on the head rest.
The wrist rest is then appropriately secured so that its height is at the level of the
patients zygoma and the apex of the patients head is about 1 cm from the rest. The
patients arms should be appropriately secured so that they do not hang off the side of
the bed. A bed sheet can be wrapped around the patients torso and secured with
hemostats to prevent inadvertent movement during the procedure.
Either the older 20-gauge system or the newer 23- and 25-gauge systems may be used
for vitrectomy. Certain technical details are specific to the vitrectomy system used.
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The conjunctiva and tenon layer are incised to expose the sclera. This is done with
Westcott scissors superonasally, superotemporally, and inferotemporally. Once bare
sclera is exposed, light cauterization is applied over the planned sclerotomy sites to
obtain hemostasis.
A caliper is then used to measure 4 mm from the limbus in phakic eyes and 3.5
mm in pseudophakic or aphakic eyes in the inferotemporal quadrant. This distance is
marked on the sclera with the caliper, and 7-0 or 8-0 double-armed polyglactin suture is
used to place 2 radial bites on either side of the mark. These bites should be about 1.5
mm long and 1.5 mm from each other. The suture is cut so as to leave tails
approximately 2 cm long on each side
Instruments used in Pars plana vitrectomy
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Equipment
Pars plana vitrectomy requires highly specialized equipment that is found only in an
operating room (OR) that is specially equipped for vitreoretinal surgery. Generally, the
following are needed:
An eye bed on which a wrist rest for the surgeon can be secured
An operating microscope
A mechanical vitrector
A wide-angle viewing system
Calipers
Westcott scissors, forceps, and needle holders
An argon indirect laser or endolaser device
An endoillumination system
A bipolar cautery
Intraocular instruments (eg, forceps, scissors, and flute needle)
Scleral depressor
Sulfur hexafluoride (SF6) and octafluoropropane (C3F8) gases
Silicone oil
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CHAPTER VII
ROLES OF CIRCULATING AND SCRUB NURSE
Roles of a Circulating Nurse
The Circulating nurse is responsible for managing the nursing care of the patient within
the OR and coordinating the needs of the surgical team with other care provider
necessary for completion of surgery,
Observes the surgery and surgical team from broad perspective and assists the team tocreate and maintain a safe and comfortable environment for the patient
Asses the patients condition before, during and after the operation to ensure an optimal
outcome for the patient and;
Must be able to anticipate the scrub nursesneeds and be able to open sterile packs,
operate machinery and keep accurate records
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Duties of a circulating nurse
Before an operation
Checks all equipment for proper functioning such as cautery machine, suction
machine, OR light and OR table
Make sure theater is clean
Arrange furniture according to use
Place a clean sheet, arm board (arm strap) and a pillow on the OR table
Provide a clean kick bucket and pail
Collect necessary stock and equipment
Turn on aircon unit
Help scrub nurse with setting up the theater
Assist with counts and records
During the Induction of Anesthesia
Turn on OR light
Assist the anesthesiologist in positioning the patient
Assist the patient in assuming the position for anesthesia
Anticipate the anesthesiologists needs
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After the patient is anesthetized
Reposition the patient per anesthesiologists instruction
Attached anesthesia screen and place the patients arm on the arm boards
Apply restraints on the patient
Expose the area for skin preparation
Catheterize the patient as indicated by the anesthesiologist
Perform skin preparation
During Operation
Remain in theater throughout operation
Focus the OR light every now and then
Connect diatherapy, suction, etc.
Position kick buckets on the operating side
Replenishes and records sponge/ sutures
Ensure the theater door remain closed and patient s dignity is upheld
Watch out for any break in aseptic technique
End of Operation
Assist with final sponge and instruments count
Signs the theater register
Ensures specimen are properly labeled and signed
After an Operation
Hands dressing to the scrub nurse
Helps remove and dispose of drapes
Helps to prepare the patient for the recovery room
Assist the scrub nurse, taking the instrumentations to the service (washroom)
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Roles of a Scrub nurse
Works directly with surgeon within the sterile field, passing instruments, sponges
and other items needed during the procedure
Members of the surgical team who prepares and preserves a sterile field in which
the operation can take place
Responsible for the sponge counts, the blades and needles and instruments
check throughout the operation
Has a job requiring anticipation, quick reaction and conscientious observation as
well as knowledge of anatomy and of operative procedures
Duties of a Scrub Nurse
Before an operation
Ensures that the circulating nurse has checked the equipment
Ensures that the theater has been cleaned before the trolley is set
Prepares the instruments and equipment needed in the operation
Uses sterile technique for scrubbing, gowning and gloving
Receives sterile equipment via circulating nurse using sterile technique
Performs initial sponges, instruments and needle count, checks with circulating nurse
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When surgeon arrives after scrubbing
Perform assisted gowning and gloving to the surgeon and assistant surgeon as
soon as they enter the operation suite
Assemble the drapes according to use. Start with towel, towel clips, draw sheet
and then lap sheet. Then, assist in draping the patient aseptically according to
routine procedure
Place blade on the knife handle using needle holder, assemble suction tip and
suction tube
Bring mayo stand and back table near the draped patient after draping is
completed
Secure suction tube and cautery cord with towel clips or allis
Prepares sutures and needles according to use
During an operation
Maintain sterility throughout the procedure
Awareness of the patients safety
Adhere to the policy regarding sponge/ instruments count/ surgical needles
Arrange the instrument on the mayo table and on the back table
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Before the Incision Begins
Provide 2 sponges on the operative site prior to incision
Passes the 1st knife for the skin to the surgeon with blade facing downward and
a hemostat to the assistant surgeon
Hand the retractor to the assistant surgeon
Watch the field/ procedure and anticipate the surgeons needs
Pass the instrument in a decisive and positive manner
Watch out for hand signals to ask for instruments and keep instrument as clean
as possible by wiping instrument with moist sponge
Always remove charred tissue from the cautery tip
Notify circulating nurse if you need additional instruments as clear as possible
Keep 2 sponges on the field
Save and care for tissue specimen according to the hospital policy
Remove excess instrument from the sterile field
Adhere and maintain sterile technique and watch for any breaks
End of Operation
Undertake count of sponges and instruments with circulating nurse
Informs the surgeon of count result
Clears away instrument and equipment
After operation: helps to apply dressing
Removes and siposes of drapes
De-gown
Prepares the patient for recovery room
Completes documentation
Hand patient over to recover room
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CHAPYTER X
BIBLIOGRAPHY
Books:
Medical Surgical Nursing by Brunner and Suddarths 12 Edition Volume 1 and 2
MIMS 2011
Nurses Pocket Guide 11thEdition by Alice Murr
Sources:
http://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-
20022595
http://en.wikipedia.org/wiki/Retinal_detachment
http://emedicine.medscape.com/article/798501-overview
http://emedicine.medscape.com/article/798501-overview#a0101
http://www.medicinenet.com/retinal_detachment/article.htm
http://www.nhs.uk/conditions/retinal-detachment/Pages/Introduction.aspx
http://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-20022595http://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-20022595http://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-20022595http://en.wikipedia.org/wiki/Retinal_detachmenthttp://en.wikipedia.org/wiki/Retinal_detachmenthttp://emedicine.medscape.com/article/798501-overviewhttp://emedicine.medscape.com/article/798501-overviewhttp://emedicine.medscape.com/article/798501-overview#a0101http://emedicine.medscape.com/article/798501-overview#a0101http://www.medicinenet.com/retinal_detachment/article.htmhttp://www.medicinenet.com/retinal_detachment/article.htmhttp://www.nhs.uk/conditions/retinal-detachment/Pages/Introduction.aspxhttp://www.nhs.uk/conditions/retinal-detachment/Pages/Introduction.aspxhttp://www.nhs.uk/conditions/retinal-detachment/Pages/Introduction.aspxhttp://www.medicinenet.com/retinal_detachment/article.htmhttp://emedicine.medscape.com/article/798501-overview#a0101http://emedicine.medscape.com/article/798501-overviewhttp://en.wikipedia.org/wiki/Retinal_detachmenthttp://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-20022595http://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-20022595