Operative Review of Retinal Detachment

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    CHAPTER I

    TABLE OF CONTENTS

    PAGE

    I. INTRODUCTION 1

    II. ANATOMY 4

    III. PATHOPHYSIOLOGY 7

    IV. MEDICAL MANAGEMENT 8

    V. DIAGNOSIS 12

    VI. PROCEDURE PROPER (with Instrumentation) 15

    VII. Roles of Circulating and Scrub nurse 24

    VIII. Nursing Management 30

    a. Nursing Care Plan

    i. Pre-Operative Review

    ii. Intra-Operative Review

    iii. Post-Operative Review

    IX. Pharmacology

    i. Pre-operative

    ii. Intra-operative

    iii. Post-operative

    X. Bibliography 37

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    Types of Retinal Detachment

    Rhegmatogenous retinal detachmentA rhegmatogenous retinal

    detachment occurs due to a hole, tear, or break in the retina that allows fluid to pass

    from the vitreous space into the subretinal space between the sensory retina and the

    retinal pigment epithelium.

    Exudative, serous, or secondary retinal detachment An exudative

    retinal detachment occurs due to inflammation, injury or vascular abnormalities that

    results in fluid accumulating underneath the retina without the presence of a hole, tear,

    or break.

    Tractional retinal detachmentA tractional retinal detachment occurs

    when fibrovascular tissue, caused by an injury, inflammation or neovascularization, pulls

    the sensory retina from the retinal pigment epithelium.

    A substantial number of retinal detachments result from trauma, including blunt

    blows to the orbit, penetrating trauma, and concussions to the head. A retrospective

    Indian study of more than 500 cases of rhegmatogenous detachments found that 11%

    were due to trauma, and that gradual onset was the norm, with over 50% presenting

    more than one month after the inciting injury.

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    Prevalence Rate

    A physician using a "three-mirror glass" to diagnose retinal detachment

    The risk of retinal detachment in otherwise normal eyes is around 5 in 100,000 peryear. Detachment is more frequent in the middle-aged or elderly population with rates of

    around 20 in 100,000 per year. The lifetime risk in normal eyes is about 1 in 300.

    Retinal detachment is more common in those with severe myopia (above 56

    diopters), as their eyes are longer and the retina is stretched thin. The lifetime

    risk increases to 1 in 20. Myopia is associated with 67% of retinal detachment

    cases. Patients suffering from a detachment related to myopia tend to be

    younger than non-myopic detachment patients.

    Retinal detachment can occur more frequently after surgery for cataracts. The

    estimated of risk of retinal detachment after cataract surgery is 5 to 16 per 1000

    cataract operations.The risk may be much higher in those who are highly myopic,

    with a frequency of 7% reported in one study.Young age at cataract removal

    further increased risk in this study. Long term risk of retinal detachment after

    extracapsular and phacoemulsification cataract surgery at 2, 5, and 10 years was

    estimated in one study to be 0.36%, 0.77%, and 1.29%, respectively.

    3

    http://en.wikipedia.org/wiki/Myopiahttp://en.wikipedia.org/wiki/Dioptershttp://en.wikipedia.org/wiki/Dioptershttp://en.wikipedia.org/wiki/Myopia
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    Causes of Retinal Detachment

    Retinal detachment can occur as a result of:

    Trauma Advanced diabetes

    An inflammatory disorder, such as sarcoidosis or cytomegalovirus retinitis

    Sagging or shrinkage of the jelly-like vitreous that fills the inside of your eye

    It is more likely to develop in people who are nearsighted, or whose relatives had

    retinal detachments. A hard, solid blow to the eye may also cause the retina to detach.

    Severe trauma to the eye, such as a contusion or a penetrating wound, may be the

    cause, but in the great majority of cases, retinal detachment is the result of internal

    changes in the vitreous chamber associated with aging, or less frequently, with

    inflammation of the interior of the eye.

    The Risk Factors of Retinal Detachment

    The following factors increase your risk of retinal detachment:

    Aging retinal detachment is more common in people older than age 40 Previous retinal detachment in one eye

    A family history of retinal detachment

    Extreme nearsightedness (myopia)

    Previous eye surgery, such as cataract removal

    Previous severe eye injury or trauma

    Weak areas on the sides (periphery) of your retina

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    CHAPTER II

    ANATOMY

    The inner wall of the back of the eyeball is covered by the retina. It contains light

    sensitive cells (photoreceptors) and numerous cells and nerve fibers responsible for

    transmitting visual information. Below is the pigment epithelium, rich in vessels

    (choroid), ensuring the supply of nutrients and oxygen to the retina.

    In the event of detachment, the light-sensitive retina becomes detached from theouter membrane (RPE). The vision is thereby impaired: the subjects speak for a

    blackout before the eyes. When this process affects the macula (yellow spot) the

    point where vision is sharpestbecomes totally blurred vision.

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    CHAPTER III

    PHYSIOLOGY

    The development of rhegmatogenous RD is a consequence of both posterior

    vitreous detachment and the development of one or more breaks in the retina. Fluid can

    then pass from the vitreous cavity through these retinal breaks into a subretinal space,

    which extends the detachment once the amount of incoming fluid exceeds the removal

    capacity of the retinal pigment epithelium (RPE). Detachment of the posterior vitreous is

    considered a major - in fact indispensable - factor in the pathogenesis of

    rhegmatogenous RD. However, no preoperative diagnostic technique can accurately

    distinguish between a posterior vitreous detachment and a posterior vitreoschisis.

    Progression of the detachment depends on many factors, including:

    Location of the break: superior faster than inferior

    Size of the break: larger faster than smaller

    Adhesion of the remaining vitreous gel to the retina: stronger faster

    than weaker

    Movement of the patient's head and eyes: this is also important

    because lack of such movement, as with bilateral patching, can result

    in the reattachment of the retina spontaneously, albeit temporarily.

    In eyes with tractional RD, the membranes on either surface of the retina are 1)

    attached to the retina, and 2) elastic. As the membranes contract, the retina detaches

    from the RPE. Accumulation of the subretinal fluid is a secondary event; as part of the

    normal fluid transport from the vitreous to the choroid, the fluid simply fills the space

    created by the elevated retina.

    In serous and haemorrhagic RD, the fluid that accumulates under the neuroretina

    separates it from the RPE.

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    CHAPTER IV

    MEDICAL MANAGEMENT

    Medical Management

    Currently, no role exists for medical care in the treatment of TRD.

    Surgical Care

    Depending on the underlying cause and extent of the TRD, surgical intervention

    is offered to patients. For instance, a patient with TRD secondary to PDR that does not

    threaten the macula probably can be monitored closely. The main surgical goal in all

    these cases is to relieve vitreoretinal traction. Traction may be relieved with scleral

    buckling techniques and/or with vitrectomy.

    In certain cases, combined RRD and TRD may be present. Usually, the retina

    becomes detached from the vitreoretinal traction. With further traction, small breaks

    may occur causing a combined TRD-RRD. In these cases, the surgical goal is to

    identify all the breaks and to close them in addition to the relief of vitreoretinal traction.

    In TRD secondary to PVR, usually a broad circumferential element, such as a

    287 buckle, is placed. A decision is made whether the crystalline lens needs to

    be sacrificed. A complete vitrectomy follows. Inside-out (posterior to anterior)

    forceps (not pick) membrane peeling is the preferred dissection method with or

    without perfluorocarbon liquid injection. Perfluorocarbon liquid may be injected at

    the surgeon's discretion to stabilize the posterior retina. If residual traction

    remains, subretinal membranes may need to be excised if causing traction. If

    necessary, a relaxing retinectomy is created. A fluid-air exchange is performed.

    Endophotocoagulation is followed by either air-silicone oil exchange or air-gas

    exchange. If perfluorocarbon liquids are not used, the dissection starts anteriorly

    and proceeds posteriorly.

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    A randomized controlled clinical trial of a perioperative infusion of 5-fluorouracil

    and low molecular weight heparin was not able to demonstrate a better surgical

    outcome in eyes with established PVR.

    In TRD secondary to PDR, several surgical techniques have been developed. A

    scleral buckle usually is not used unless anterior breaks are present.

    A central vitrectomy is performed with the vitrector clearing the axial opacities

    and the cortical vitreous gel. A large opening is created in the posterior hyaloid

    until vitreoretinal adhesions are encountered. Segmentation and/or delamination

    of these adhesions (as described by Charles) are used for virtually all diabetic

    TRD.

    Delamination refers to the separation of the retina from the extraretinal

    proliferation. This dissection proceeds from posterior to anterior. Fibrovascular

    tissue often bridge separate retinal zones. Segmentation refers to cutting of the

    fibrovascular tissue bridge into small separate islands of tissue.

    Care must be given to create as few iatrogenic breaks as possible. If breaks are

    identified, usually fluid-air exchange with photocoagulation reattaches the retina.

    Breaks should be marked with diathermy, so they are identified easily in the air-

    filled eye. The incidence of RRD in patients who underwent vitrectomy for PDR

    has been reported to be 4.3%. Intraocular bleeding also must be monitoredclosely. Diathermy to active neovascular fronds may be necessary.

    Other techniques include the en bloc dissection. En bloc is a name applied to

    outside-in delamination where the vitreous is used to pull on the epiretinal

    membrane. Outside-in causes more retinal breaks than inside-out, making it a

    dangerous maneuver.

    Intravitreal bevacizumab has been reported as a preoperative adjunct in

    vitrectomy for PDR. Bevacizumab seems to reduce the bleeding associated with

    the segmentation and delamination of fibrovascular membranes. However, in

    eyes with severe ischemia, the neovascularization regresses rapidly, but the

    resulting fibrous scar tissue may lead to the development or progression of TRD.

    Therefore, caution should be exercised when injecting these eyes, and patients

    should be scheduled for surgery days, and not weeks, after the injection.

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    Anti-VEGF agents such as bevacizumab have been used as adjuncts to

    vitrectomy. The advantages of using preoperative bevacizumab includes faster

    surgery and reduced risk of intraoperative bleeding, which facilitates membrane

    dissection.[8, 9, 10, 11] Care must be taken because it has been reported that, in

    very ischemic eyes, TRD may occur or progress shortly following intravitreal

    bevacizumab.[9, 10] It is speculated that rapid neovascular involution with

    accelerated fibrosis and posterior hyaloidal contraction as a response to

    decreased levels of VEGF is responsible for this phenomenon. In this

    retrospective series, the time from injection to TRD was a mean of 13 days, with

    a range of 3-31 days.[9] Therefore, the time between bevacizumab injection and

    vitrectomy should not exceed 3 days.

    The treatment of TRD secondary to ROP depends on the stage of the disease.

    Although many vitreoretinal surgeons advocate an encircling band for stage 4A

    ROP, no scientific evidence is available that supports its efficacy. In stage 4B,

    vitrectomy is recommended. It is currently unclear if lens-sparing vitrectomy has

    any advantages over lensectomy.

    For stage 5 ROP, visual and anatomical results have been disappointing, making

    some surgeons abandon surgery for these cases. Others have tried vitrectomy

    and lensectomy with or without scleral buckling. In these cases, a 2-portvitrectomy technique is recommended since the small size of the eye and orbit

    limits ocular manipulation if a 3-port technique is used. The use of intravitreal

    triamcinolone as a postoperative adjuvant might improve the rate of retinal

    reattachment after vitrectomy.

    A recent case series of aggressive posterior ROP suggested that early

    vitrectomy with lensectomy in these cases is effective in preventing TRD.

    Special attention must be given to avoid iatrogenic retinal breaks because of the

    poor prognosis associated with this complication. The goal of surgery is to obtain

    macular reattachment.

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    CHAPTER V

    DIAGNOSIS

    History

    Patients who present with symptoms of new onset photopsias, floaters or visual

    field loss should be suspected of having a retinal tear or detachment until proven

    otherwise. Important information in the history includes onset of symptoms, duration of

    decreased visual acuity, metamorphopsia, any prior trauma, prior surgery, intraocular

    inflammation, hemorrhage, glaucoma and a complete past medical history and review of

    systems.

    Physical examination

    Visual acuity, pupillary examination, visual field testing and intraocular pressure

    measurement are important parts of the predilated ophthalmic examination to evaluate

    patients with symptoms of retinal detachment. Additional examination to include color

    vision and ocular motility should be tailored according to the history provided.

    Slit lamp examination of the anterior segment should be completed prior to

    dilation. Examination of the vitreous for pigment cells followed by a thorough fundus

    examination to include indirect ophthalmoscopy with scleral depression should be

    completed. A detailed drawing describing the detachment with location of retinal

    pathology should be documented.

    If there is no view to the posterior pole such as in hemorrhage or media opacity,ultrasound should be used to evaluate the retinal status.

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    Clinical diagnosis

    Rhegmatogenous retinal detachment has a characteristic appearance

    differentiating it from a tractional or serous detachment. A rhegmatogenous retinal

    detachment has a corrugated appearance and undulates with eye movements.

    Tractional detachments have smooth concave surfaces with minimal shifting with eye

    movements. Serous detachments show a smooth retinal surface and shifting fluid

    depending on patient positioning.

    Laboratory/Ancillary testing

    Laboratory testing is only indicated in traction or exudative detachments. If a

    cause for the traction retinal detachment cannot be determined by history, furtherlaboratory analysis may be required to determine if diabetes, sickle cell, carotid disease

    or another systemic or ocular process is the source for proliferative retinopathy.

    Since exudative detachments may be due to a systemic or ocular inflammatory

    process, laboratory investigation may be indicated.

    Fluorescein angiography may be indicated to further clarify exudative processes

    such as macular degeneration, central serous chorioretinopathy, and Vogt-Koyanagi-

    Harada syndrome or other uveitic processes. Ultrasound is a useful imaging modality to

    evaluate choroidal masses or posterior scleritis.

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    CHAPTER VI

    PROPER PROCEDURE

    Pars Plana Vitrectomy

    Patients are brought to the operating room in an eye bed that has an appropriate

    head rest and the capability to have a wrist rest secured to it. Once the bed is

    positioned next to the operating microscope and locked, the bed is made completely

    flat, and the patient is positioned so that the head lies comfortably on the head rest.

    The wrist rest is then appropriately secured so that its height is at the level of the

    patients zygoma and the apex of the patients head is about 1 cm from the rest. The

    patients arms should be appropriately secured so that they do not hang off the side of

    the bed. A bed sheet can be wrapped around the patients torso and secured with

    hemostats to prevent inadvertent movement during the procedure.

    Either the older 20-gauge system or the newer 23- and 25-gauge systems may be used

    for vitrectomy. Certain technical details are specific to the vitrectomy system used.

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    The conjunctiva and tenon layer are incised to expose the sclera. This is done with

    Westcott scissors superonasally, superotemporally, and inferotemporally. Once bare

    sclera is exposed, light cauterization is applied over the planned sclerotomy sites to

    obtain hemostasis.

    A caliper is then used to measure 4 mm from the limbus in phakic eyes and 3.5

    mm in pseudophakic or aphakic eyes in the inferotemporal quadrant. This distance is

    marked on the sclera with the caliper, and 7-0 or 8-0 double-armed polyglactin suture is

    used to place 2 radial bites on either side of the mark. These bites should be about 1.5

    mm long and 1.5 mm from each other. The suture is cut so as to leave tails

    approximately 2 cm long on each side

    Instruments used in Pars plana vitrectomy

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    Equipment

    Pars plana vitrectomy requires highly specialized equipment that is found only in an

    operating room (OR) that is specially equipped for vitreoretinal surgery. Generally, the

    following are needed:

    An eye bed on which a wrist rest for the surgeon can be secured

    An operating microscope

    A mechanical vitrector

    A wide-angle viewing system

    Calipers

    Westcott scissors, forceps, and needle holders

    An argon indirect laser or endolaser device

    An endoillumination system

    A bipolar cautery

    Intraocular instruments (eg, forceps, scissors, and flute needle)

    Scleral depressor

    Sulfur hexafluoride (SF6) and octafluoropropane (C3F8) gases

    Silicone oil

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    CHAPTER VII

    ROLES OF CIRCULATING AND SCRUB NURSE

    Roles of a Circulating Nurse

    The Circulating nurse is responsible for managing the nursing care of the patient within

    the OR and coordinating the needs of the surgical team with other care provider

    necessary for completion of surgery,

    Observes the surgery and surgical team from broad perspective and assists the team tocreate and maintain a safe and comfortable environment for the patient

    Asses the patients condition before, during and after the operation to ensure an optimal

    outcome for the patient and;

    Must be able to anticipate the scrub nursesneeds and be able to open sterile packs,

    operate machinery and keep accurate records

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    Duties of a circulating nurse

    Before an operation

    Checks all equipment for proper functioning such as cautery machine, suction

    machine, OR light and OR table

    Make sure theater is clean

    Arrange furniture according to use

    Place a clean sheet, arm board (arm strap) and a pillow on the OR table

    Provide a clean kick bucket and pail

    Collect necessary stock and equipment

    Turn on aircon unit

    Help scrub nurse with setting up the theater

    Assist with counts and records

    During the Induction of Anesthesia

    Turn on OR light

    Assist the anesthesiologist in positioning the patient

    Assist the patient in assuming the position for anesthesia

    Anticipate the anesthesiologists needs

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    After the patient is anesthetized

    Reposition the patient per anesthesiologists instruction

    Attached anesthesia screen and place the patients arm on the arm boards

    Apply restraints on the patient

    Expose the area for skin preparation

    Catheterize the patient as indicated by the anesthesiologist

    Perform skin preparation

    During Operation

    Remain in theater throughout operation

    Focus the OR light every now and then

    Connect diatherapy, suction, etc.

    Position kick buckets on the operating side

    Replenishes and records sponge/ sutures

    Ensure the theater door remain closed and patient s dignity is upheld

    Watch out for any break in aseptic technique

    End of Operation

    Assist with final sponge and instruments count

    Signs the theater register

    Ensures specimen are properly labeled and signed

    After an Operation

    Hands dressing to the scrub nurse

    Helps remove and dispose of drapes

    Helps to prepare the patient for the recovery room

    Assist the scrub nurse, taking the instrumentations to the service (washroom)

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    Roles of a Scrub nurse

    Works directly with surgeon within the sterile field, passing instruments, sponges

    and other items needed during the procedure

    Members of the surgical team who prepares and preserves a sterile field in which

    the operation can take place

    Responsible for the sponge counts, the blades and needles and instruments

    check throughout the operation

    Has a job requiring anticipation, quick reaction and conscientious observation as

    well as knowledge of anatomy and of operative procedures

    Duties of a Scrub Nurse

    Before an operation

    Ensures that the circulating nurse has checked the equipment

    Ensures that the theater has been cleaned before the trolley is set

    Prepares the instruments and equipment needed in the operation

    Uses sterile technique for scrubbing, gowning and gloving

    Receives sterile equipment via circulating nurse using sterile technique

    Performs initial sponges, instruments and needle count, checks with circulating nurse

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    When surgeon arrives after scrubbing

    Perform assisted gowning and gloving to the surgeon and assistant surgeon as

    soon as they enter the operation suite

    Assemble the drapes according to use. Start with towel, towel clips, draw sheet

    and then lap sheet. Then, assist in draping the patient aseptically according to

    routine procedure

    Place blade on the knife handle using needle holder, assemble suction tip and

    suction tube

    Bring mayo stand and back table near the draped patient after draping is

    completed

    Secure suction tube and cautery cord with towel clips or allis

    Prepares sutures and needles according to use

    During an operation

    Maintain sterility throughout the procedure

    Awareness of the patients safety

    Adhere to the policy regarding sponge/ instruments count/ surgical needles

    Arrange the instrument on the mayo table and on the back table

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    Before the Incision Begins

    Provide 2 sponges on the operative site prior to incision

    Passes the 1st knife for the skin to the surgeon with blade facing downward and

    a hemostat to the assistant surgeon

    Hand the retractor to the assistant surgeon

    Watch the field/ procedure and anticipate the surgeons needs

    Pass the instrument in a decisive and positive manner

    Watch out for hand signals to ask for instruments and keep instrument as clean

    as possible by wiping instrument with moist sponge

    Always remove charred tissue from the cautery tip

    Notify circulating nurse if you need additional instruments as clear as possible

    Keep 2 sponges on the field

    Save and care for tissue specimen according to the hospital policy

    Remove excess instrument from the sterile field

    Adhere and maintain sterile technique and watch for any breaks

    End of Operation

    Undertake count of sponges and instruments with circulating nurse

    Informs the surgeon of count result

    Clears away instrument and equipment

    After operation: helps to apply dressing

    Removes and siposes of drapes

    De-gown

    Prepares the patient for recovery room

    Completes documentation

    Hand patient over to recover room

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    CHAPYTER X

    BIBLIOGRAPHY

    Books:

    Medical Surgical Nursing by Brunner and Suddarths 12 Edition Volume 1 and 2

    MIMS 2011

    Nurses Pocket Guide 11thEdition by Alice Murr

    Sources:

    http://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-

    20022595

    http://en.wikipedia.org/wiki/Retinal_detachment

    http://emedicine.medscape.com/article/798501-overview

    http://emedicine.medscape.com/article/798501-overview#a0101

    http://www.medicinenet.com/retinal_detachment/article.htm

    http://www.nhs.uk/conditions/retinal-detachment/Pages/Introduction.aspx

    http://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-20022595http://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-20022595http://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-20022595http://en.wikipedia.org/wiki/Retinal_detachmenthttp://en.wikipedia.org/wiki/Retinal_detachmenthttp://emedicine.medscape.com/article/798501-overviewhttp://emedicine.medscape.com/article/798501-overviewhttp://emedicine.medscape.com/article/798501-overview#a0101http://emedicine.medscape.com/article/798501-overview#a0101http://www.medicinenet.com/retinal_detachment/article.htmhttp://www.medicinenet.com/retinal_detachment/article.htmhttp://www.nhs.uk/conditions/retinal-detachment/Pages/Introduction.aspxhttp://www.nhs.uk/conditions/retinal-detachment/Pages/Introduction.aspxhttp://www.nhs.uk/conditions/retinal-detachment/Pages/Introduction.aspxhttp://www.medicinenet.com/retinal_detachment/article.htmhttp://emedicine.medscape.com/article/798501-overview#a0101http://emedicine.medscape.com/article/798501-overviewhttp://en.wikipedia.org/wiki/Retinal_detachmenthttp://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-20022595http://www.mayoclinic.org/diseases-conditions/retinal-detachment/in-depth/CON-20022595