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PEMERIKSAAN PENUNJANG & DD ANGINA PEKTORIS

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PEMERIKSAAN PENUNJANG & DD ANGINA PEKTORIS

Text of PEMERIKSAAN PENUNJANG & DD ANGINA PEKTORIS

PEMERIKSAAN PENUNJANG

PEMERIKSAAN PENUNJANGUNTUK MENYINGKIRKAN DIAGNOSIS BANDINGEKGThe ECG is pivotal for identifying patients with ongoing ischemia as the principal reason for their presentation as well as secondary cardiac complications of other disorders.Professional society guidelines recommend that an ECG be obtained within 10 min of presentation.ST-segment elevation MIST-segment depression and symmetric T-wave inversions at least 0.2 mV in depth MI in the absence of STEMI , indicative of higher risk of death or recurrent ischemia

Abnormalities of the ST segment and T wave may occur in a variety of conditions, including pulmonary embolism, ventricular hypertrophy, acute and chronic pericarditis, myocarditis, electrolyte imbalance, and metabolic disorders. Notably, hyperventilation associated with panic disorder can also lead to nonspecific ST and T-wave abnormalities. Pulmonary embolism is most often associated with sinus tachycardia but can also lead to rightward shift of the ECG axis, manifesting as an S-wave in lead I, with a Q-wave and T-wave in lead III In patients with ST-segment elevation, the presence of diffuse lead involvement not corresponding to a specific coronary anatomic distribution and PR-segment depression can aid in distinguishing pericarditis from acute MI.RADIOGRAFI DADAThe chest radiograph is most useful for identifying pulmonary processes, such as pneumonia or pneumothorax. Findings are often unremarkable in patients with ACS, but pulmonary edema may be evident. Other specific findings include widening of the mediastinum in some patients with aortic dissection, Hamptons hump or Westermarks sign in patients with pulmonary embolism, or pericardial calcification in chronic pericarditis.BIOMARKER JANTUNGLaboratory testing in patients with acute chest pain is focused on the detection of myocardial injury.Because of superior cardiac tissue-specificity compared with creatine kinase MB, cardiac troponin is the preferred biomarker for the diagnosis of MI and should be measured in all patients with suspected ACS at presentation and repeated in 36 h.Testing after 6 h is required only when there is uncertainty regarding the onset of pain or when stuttering symptoms have occurred.PX LAB LAINOther laboratory assessments may include the D-dimer test to aid in exclusion of pulmonary embolism.Measurement of a B-type natriuretic peptide is useful when considered in conjunction with the clinical history and exam for the diagnosis of heart failure.B-type natriuretic peptides also provide prognostic information regarding patients with ACS and those with pulmonary embolism.EKOKARDIOGRAFIin patients with an uncertain diagnosis, particularly those with nondiagnostic ST elevation, ongoing symptoms, or hemodynamic instability, detection of abnormal regional wall motion provides evidence of possible ischemic dysfunction. Echocardiography is diagnostic in patients with mechanical complications of MI or in patients with pericardial tamponade. Transthoracic echocardiography is poorly sensitive for aortic dissection, although an intimal flap may sometimes be detected in the ascending aorta.

CT-ANGIOGRAPHYCoronary CT angiography is a sensitive technique for detection of obstructive coronary disease, particularly in the proximal third of the major epicardial coronary arteries.contrast-enhanced CT can detect focal areas of myocardial injury in the acute setting as decreased areas of enhancement. At the same time, CT angiography can exclude aortic dissection, pericardial effusion, and pulmonary embolism.

MRICardiac magnetic resonance (CMR) imaging is an evolving, versatile technique for structural and functional evaluation of the heart and the vasculature of the chest.Gadolinium-enhanced CMR can provide early detection of MI, defining areas of myocardial necrosis accurately, and can delineate patterns of myocardial disease that are often useful in discriminating ischemic from non-ischemic myocardial injury.CMR can be a useful modality for cardiac structural evaluation of patients with elevated cardiac troponin levels in the absence of definite coronary artery disease.MRI also permits highly accurate assessment for aortic dissection but is infrequently used as the first test because CT and transesophageal echocardiography are usually more practical.